Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine."

19 Dec 2016


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Crymes on Video

CDC whistleblowers explain CDC is 200% corrupt [SPIDER]

KDickson's Blog:  Exported Fungal Antigens Inhibit Apoptosis

Crazy Nutcase Lyme non-non-profits (fun reading, anyway)


Occam's Razor (it's really about EBV/CMV)


IDSA: "Vaccines are too many, not vetted, & babies too young"

Fb DOJ protest group

CDC knew OspA caused disease


BlumenthalAntiTrust Lawsuit

Exosomes, Blebs


CDC Admits Fraud, 2016
Dattwyler, 1988
Golightly, 1988
Dressler, 1994
BarbourFish, 1993
Dearborn, 1994

Pathogenic Fungi

Predicting all of GW Bush's warcrimes, Oct 2000





161219 - Lyme Crooks Wormser and Steere - and even the "CDC officer" criminal Paul Mead - finally admit Late Lyme and LYMErix diseases are immunosuppression outcomes.  
'Say the "TLR2/1 agonism" (immunosuppression) is probably the "more important" driver of the disease outcome.

Abstract (published Dec 15, 2016):

Full Text:

Mario Philipp has long been the guy who says Borrelia and OspA causes immunosuppression, so look at all of his reports:

Ref 79, above, (Mario Philipp): Interleukin-10 anti-inflammatory response to Borrelia burgdorferi, the agent of Lyme disease: a possible role for suppressors of cytokine signaling 1 and 3.


You will recall that when I blew this whistle at the FDA on Lyme (Dearborn) and LYMErix on Jan 31, 2001, I mentioned Dattwyler's "suppression of Natural Killer cell activity" report in response to Lyme and OspA, and I also mentioned that Mario Philipp had published that OspA caused immunosuppression via IL-10:

The "Govt" has never thanked us for getting rid of that TLR2/1-agonist fungal toxin of a non-vaccine.


The crooks report above, saying the more serious disease outcome may be due to its fungal-ish agonism of TLR2/1, is a little bit of a screwed up way of saying it, but they, the CDC and the Lyme criminals, finally admitted their whole Lyme story was a lie.   Notice that ILADS has NEVER said what Late Chronic Lyme was.  They lost their opportunity to be the ones to bring the truth forward as I had predicted, also.


The next reports show your "Post-Lyme disease" is actually "Post-Septic Shock Syndrome," and depends on how much OspA-ish antigens (triacyl/fungal lipoproteins) have ruined your immune system. Whoever says "inflammation" or "autoimmunity" about Lyme in the same sentence is a fraud (unless they're talking about autoimmunity to the secondary infections, like MS or Lupus).

If people don't believe me, they might as well believe Gary Wormser and take these 2 reports to their "doctors" for a cure for post-sepsis syndrome (there isn't). Or at least to help you get Social Security Disability.

The immunosuppression from a tick bite is a well known thing - from the experiments trying to make a vaccine out of fungal lipoproteins for Tuberculosis. They all failed the same way LYMErix failed (see the Occam's Razor) - they made people sicker and "more susceptible to other infections."






Other people who say the CDC,, NYMC, Harvard, Tufts, etc are criminal frauds on Lyme and LYMErix disease, and that Lyme and LYMErix are diseases of immunosuppression and not the opposite, inflammation or HLA-linked bad-knees:

Luft & Dattwyler (SUNY-SB - 1988 !!)
NIH (Fauci, Martin, Marques)
Persing and Schoen (Mayo, Yale)
Duray (US Army, & Yale)
Baumgarth and Barthold (UC Davis)
(Medvedev, Harding, Redmond, Philipp, etc)


2) Norman Latov

Norman Latov on how OspA vaccination caused the same disease as chronic Lyme:

Neuropathy and cognitive impairment following vaccination with the OspA protein of Borrelia burgdorferi.

“Neurological syndromes that follow vaccination or infection are often attributed to autoimmune mechanisms. We report six patients who developed neuropathy or cognitive impairment, within several days to 2 months, following vaccination with the OspA antigen of Borrelia burgdorferi. Two of the patients developed cognitive impairment, one chronic inflammatory demyelinating polyneuropathy (CIDP), one multifocal motor neuropathy, one both cognitive impairment and CIDP, and one cognitive impairment and sensory axonal neuropathy. The patients with cognitive impairment had T2 hyperintense white matter lesions on magnetic resonance imaging. The similarity between the neurological sequelae observed in the OspA-vaccinated patients and those with chronic Lyme disease suggests a possible role for immune mechanisms in some of the manifestations of chronic Lyme disease that are resistant to antibiotic treatment.”


3) Donald Marks

Donald H. Marks - an OspA vaccine trial administrator - on how LYMErix caused the same disease as chronic Lyme:

Neurological complications of vaccination with outer surface protein A (OspA).

”A wide range of neurological complications have been reported via the medical literature and the VAERS system after vaccination with recombinant outer surface protein A (OspA) of Borrelia. To explore this issue, 24 patients reporting neurological adverse events (AE) after vaccination with Lymerix, out of a group of 94 patients reporting adverse events after Lymerix vaccination, were examined for causation. Five reports of cerebral ischemia, two transient Ischemic attacks, five demyelinating events, two optic neuritis, two reports of transverse myelitis, and one non-specific demyelinating condition are evaluated in this paper. Caution is raised on not actively looking for neurologic AE, and for not considering causation when the incidence rate is too low to raise a calculable difference to natural occurrence.”

[It's not “Autoimmune.”  It’s Subimmune.  This Subimmunity represents the entire class of the DSM's VooDoo Somatoformia – as well as cancer.  Cancer is in the Subimmune class, at the other end of the immunity spectrum from Autoimmunity.   This fact or condition completely flips the entire medical paradigm where you have to have a biomarker that is above-, or more-than- the normal range.  Lyme is not an inflammatory disease.  There are always negative correlations to biomarkers of autoimmunity or illness or infection except when using sophisticated DNA techniques using spinal fluid, in particular in these chronic, waste-basket diseases.  Henceforth, Autoimmunity will be an obsolete word that connotes the previous Medical Establishment where BigPharma is going to “block” something with their drugs.  They are dinosaurs.  You can’t block a mechanism that is already permanently blocked and you can’t unblock it.
It could be that a person has an HLA-linked outcome to one of the secondary infections like Epstein-Barr or HHV-6, reactivated by the AIDS-like Lyme and LYMErix.  Those people would for instance have the official, hypersensitivity outcomes of MS or Lupus or whatever.  But they are not also called Incompetent Incantation-ators and they are not mistreated by the entire universe (family, friends, Social Security, “doctors,” everyone, including ILADS and the non profits).]



Who else says OspA alone is responsible for the immunosuppression with brain inflammation that is characteristic of "Chronic Lyme?"


4) Ben Luft   SUNY-SB

Ben Luft at the 1998 FDA Vaccine Meeting on LYMErix:

 "The point that I wanted to make in regard to the study is that there is very heavy dependence on serologic confirmation. And when we start thinking about the adverse events, *** it was stated originally when we got the overview of the disease that the disease is really quite protean. And actually the adverse events are very similar to what the disease manifestations are.**** And if you start to, as I think Dr. Hall was eluding to -- if you start to kind of say well how often do you actually become seropositive, you can start to have a different take on when someone has an adverse event or whether it is disease specific or infection specific versus vaccine specific. And I think that that is an important issue that we have to deal with. ..."




5.)  Dattwyler and Luft in 1989, in IDSA's own "journal," saying "treatment fails in half the cases." And the OspA vaccine caused the same systemic disease, say the crooks, themselves.  See the Occam's Razor report for who says so. 

Steere was at this 1994 FDA meeting: 

DATTWYLER:  "-- the ones that (sic) failed to mount a vigrous immune response tended to do worse.  So, there is an inverse correlation between the degree of serologic response and the outcome. 
"So, individuals with a poor immune response tend to have worse disease."

1994 FDA Meeting Transcripts where Ray Dattwyler tells the FDA Vaccine Committee that the
patients with low or no antibodies are the sickest:


Ray Dattwyler (SUNY-SB) says in 1988 that exposure to Lyme or even just its supernatants (OspA-like molecules) turn off the NK cell activity (or is immunosuppressive):

Effect of B burgdorferi Culture on Normal PBL


"...when lymphocytes are cultured in the presence of growing Bb there is a marked inhibition ( p < .0005 ) of NK activity on days 3, 5, and 7 when compared to lymphocytes cultured in BSKII media in the absence of spirochetes.  This effect is not due to a selective depletion or or toxicity to endogenous NK since viability studies and monoclonal antibodies demonstrate no significant changes after culture with the organism.




"The inhibition is directly attributable to the organism or its supernatants from the organism also inhibit NK without prior exposure (data not shown)."


Full Text:  Golightly, 1988.pdf

From: Modulation of natural killer cell activity by Borrelia burgdorferi.

Golightly M, Thomas J, Volkman D, Dattwyler R.
Department of Pathology, State University of New York, Stony Brook 11794.
PMID: 3056196 [PubMed - indexed for MEDLINE]
Ann N Y Acad Sci. 1988;539:103-11.


Dattwyler on Seronegative Lyme and how that is like fungal diseases:

DOWNLOAD THE FULL TEXT of Dattwyler talking about seronegative, neuroLyme, HERE:
dattwyler1988 (1).pdf

Lyme is like Jeopardy!  Ask the right question:  "Something that causes a 'multi-system disease' and is IDSA's own 'New Great Imitator.'" 

The answer is, "What is OspA?"






6) Dave Persing, Mayo Clinic, Robert Schoen, Yale

Dave Persing  who together with Yale’s Robert Schoen developed this test in 1994 or 1995 says this about the similarities between Lyme and LYMErix disease (you cant tell them apart):

"Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure..."






7.) THE NIH (Martin and Marques):

Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression (2006).

"The spirochete Borrelia burgdorferi is the agent of Lyme disease, which causes central nervous system manifestations in up to 20% of patients. We investigated the response of human brain microglial cells, glial progenitors, neurons, astrocytes, as well as peripheral blood monocytes to stimulation with B. burgdorferi. We used oligoarrays to detect changes in the expression of genes important for shaping adaptive and innate immune responses. We found that stimulation with B. burgdorferi lysate increased the expression of Toll-like receptors (TLRs) 1 and 2 in all cell types except neurons. However, despite similarities in global gene profiles of monocytes and microglia, only microglial cells responded to the stimulation with a robust increase in HLA-DR, HLA-DQ, and also coexpressed CD11-c, a dendritic cell marker. In contrast, a large number of HLA-related molecules were repressed at both the RNA and the protein levels in stimulated monocytes, whereas secretion of IL-10 and TNF-alpha was strongly induced. These results show that signaling through TLR1/2 in response to B. burgdorferi can elicit opposite immunoregulatory effects in blood and in brain immune cells, which could play a role in the different susceptibility of these compartments to infection." 


The NIH-RML patent explaining how Lyme causes LYMErix-disease (“stealth bomber”):

"The invention relates to novel antigens associated with Borrelia burgdorferi which are exported (or shed) in vivo and whose detection is a means of diagnosing Lyme disease. The antigens are extracellular membrane vesicles and other bioproducts including the major extracellular protein antigen. Another object of the invention is to provide antibodies, monoclonal and/or polyclonal, labeled and/or unlabeled, that are raised against the antigens. A further object of the invention is to provide a method of diagnosing Lyme disease by detecting the antigens in a biological sample taken from a host using the antibodies in conventional immunoassay formats. Another object of the invention is to provide kits, for the diagnosis of Lyme disease, comprising the antibodies and ancillary reagents. The advantage of the antibodies used in the invention is that they react with the antigens from geographically diverse strains of Borrelia burgdorferi, but do not react with antigens from related Borrelia spirochetes.",217,872.PN.&OS=PN/5,217,872&RS=PN/5,217,872

The shed blebs (or exosomes or vesicles) have LYMErix on them (delayed fuse or “time bomb”):

Characterization of multiprotein complexes of the Borrelia burgdorferi outer membrane vesicles.

"Although we uncovered the existence of at least 10 distinct OM complexes harboring several unique subunits, the complexome is dominated by the frequent occurrence of a limited diversity of membrane proteins, most notably P13, outer surface protein (Osp) A, -B, -C, and -D and Lp6.6."



UCSF says Lyme and LYMErix-Disease are a diseases of immunosuppression, and they say that half the Lyme/tick bite victims remain ill and are not cured with antibiotics
Feb 12, 2016:

“Early Lyme disease prior to antibiotic therapy was characterized by marked upregulation of Toll-like receptor signaling but lack of activation of the inflammatory T-cell apoptotic and B-cell developmental pathways seen in other acute infectious syndromes,” wrote the study’s authors. “Six months after completion of therapy, Lyme disease patients were found to have 31 to 60% of their pathways in common with three different immune-mediated chronic diseases. No differential gene expression signature was observed between Lyme disease patients with resolved illness to those with persistent symptoms at six months post-treatment.”

"Six months after treatment, 15 of the 29 patients in the study had fully recovered, while 13 had persistent symptoms, and one had dropped out.



See more below from UC Davis, and in the Occam's Razor report. The Great Imitator is EBV, the Great Initiator is the fungal antigen OspA, shed by spirochetes - their own phylum, and not regular "bacteria" - on blebs, says the NIH.  America is a really stupid country, when you consider that nearly every disease is an outcome of the first immune disordered step - inhibition of apoptosis of infected B cells.

Look at the experience of Lewis Bull, East Lyme, CT, back in the OspA "vaccines" trials days (late 1990s) to see exactly what these evil bastards of the CDC (Barbour, Johnson, Steere) had planned from the beginning to do with LYMErix - "multisystem (read: "sepsis")," Chronic Fatigue Syndrome-like - outcomes.   'Says it all.

University of California, Davis, says the CDC, Allen Steere, Gary Wormser, Edward McSweegan, et al must be lying their faces off:  Lyme and LYMErix cause permanent immunosuppression or is like a B cell AIDS. 


Lyme is not an "inflammatory," "autoimmune disease," it is the Great Initiator of latent viruses, as a fungal antigen shedder, SPIROCHETE (its own phylum).  Fungal antigens like triacyl lipopeptides (OspA) have always been known to cause immunosuppression. The complete opposite of an "autoimmune" or "inflammatory" disease.  The only biomarkers will be signs of nerve and brain degradation in the cerebral spinal fluid; no antibodies in the blood.

 So what exactly is OspA? 

“I did not get the vaccine so this does not concern me.”   Oh, yes, you got the vaccine.  Everyone with Lyme got LYMErix.  Here is what LYMErix is, and how this "vaccine-was-the-disease" works:

This is image is of Pam3Cys or OspA.  What is variable is the protein end "("peptide"), but what is immunogenic (seen as an invader to the immune system) are the fatty acids or acyl groups and the very electronegative cysteine core:


Pam3Cys is a triacylated lipoprotein, the degree of acylation is equated with its toxicity.   So what is acylation?  It’s the zig-zaggy lines that mean Carbon-Carbon-Carbon, yes, hydrocarbons, like margarine or octane.  Exactly, the name just refers to the number of carbons in each carboxyl or acyl group.  Palmitic (the Pam in Pam3Cys) has X number of carbons, gasoline, 8, linoleic acids, like 14. Look up what are alkanes then add a COOH group and you have one of these fatty acids. 

Something highly acylated like this (3 or more fatty acids hanging off) are managed by Toll-like Receptor (TLR) 2 and TLR1, together.  Therefore a “TLR2/1-agonist” is another term that generally refers to lipoproteins like those from Borrelia, mycoplasma, mycobacteria, and others like Brucella.  (But they can manage other compounds.)

This thing, Pam3Cys and fungal lipid molecules like it, is shed with the blebs.  In other words, like this:

The likes of OspA is on these blebs.  They go to the brain, inflame it, get eaten up by immune cells - which renders them incompetent-, they go to the kidneys (LUAT), etc. You will find this to be so in an NIH-owned patent (5,217,872) and elsewhere.

So, the fungal antigens are on the shed blebs and they go everywhere and they render the immune cells incompetent, resulting in an AIDS-like disease.  Everyone who has Lyme disease also has LYMErix disease. 


9.) Cadavid and the NIH say these shed vesicles or blebs go to the brain and inflame it:








In other words, the NIH is saying that thanks to exposure to shed blebs with TLR2/1 agonists like OspA on them, the HLA molecules will be downregulated and that you will make no antibodies.  This mechanism is mimicked elsewhere, in other fungal diseases models.  It is also CALLED endotoxin tolerance.


Endotoxin Tolerance Inhibits Lyn and c-Src Phosphorylation and Association with Toll-Like Receptor 4 but Increases Expression and Activity of Protein Phosphatases.

“Endotoxin tolerance protects the host by limiting excessive 'cytokine storm' during sepsis, but compromises the ability to counteract infections in septic shock survivors. It reprograms Toll-like receptor (TLR) 4 responses by attenuating the expression of proinflammatory cytokines without suppressing anti-inflammatory and antimicrobial mediators, but the mechanisms of reprogramming remain unclear. In this study, we demonstrate that the induction of endotoxin tolerance in human monocytes, THP-1 and MonoMac-6 cells inhibited lipopolysaccharide (LPS)-mediated phosphorylation of Lyn, c-Src and their recruitment to TLR4, but increased total protein phosphatase (PP) activity and the expression of protein tyrosine phosphatase (PTP) 1B, PP2A, PTP nonreceptor type (PTPN) 22 and mitogen-activated protein kinase phosphatase (MKP)-1. Chemical PP inhibitors, okadaic acid, dephostatin and cantharidic acid markedly decreased or completely abolished LPS tolerance, indicating the importance of phosphatases in endotoxin tolerization. Overexpression of PTPN22 decreased LPS-mediated nuclear factor (NF)-x03BA;B activation, p38 phosphorylation and CXCL8 gene expression, while PTPN22 ablation upregulated LPS-induced p65 NF-x03BA;B and p38 phosphorylation and the expression of TNF-α and pro-IL-1β mRNA, indicating PTPN22 as an inhibitor of TLR4 signaling. Thus, LPS tolerance interferes with TLR4 signaling by inhibiting Lyn and c-Src phosphorylation and their recruitment to TLR4, while increasing the phosphatase activity and expression of PP2A, PTPN22, PTP1B and MKP1.


10)  Anthony Fauci: Director Anthony Fauci actually owns a patent for the treatment of immunosuppression diseases like Lyme, other fungal diseases, like inhalation crypto-mold and Tuberculosis

"Doctors" don't know what are the diagnostic codes for post-sepsis syndrome, so they cant really help you get Disability, and that is why none can help you in any way.  See the link, below, to the "Big Picture"/Fauci page for that data.

We can tell you what the disease *is,* but we cant tell you how to train your "doctors" to help you.  Sorry.  We're at a total loss if people with "MD" after their names cant see the obvious.  That is the thing about cults, like cop cults and jail guard cults and government employee cults.  The cult protects itself, first, that is one of the primary characteristics of a cult. The second it that the cult leaders tell the cult members that there is something SPECIAL !! about them, or that they are UNIQUE !! or owners of SPECIAL, SECRET KNOWLEDGE !! (appealing to vanity).

The best example I can think of it psychiatry: The head of NIMH said it was all bogus, invalid, and crazy bullshit (Thomas Insel) but few to none of the rest of them say it.  In fact, there is a push on now to get more psychiatrists to literally drug more children.  Yes, that was their claim.  They need more non-valid, non-scientific "MDs" to sell the brain-damaging psych drugs to children.  And this is while the head of the NIMH said none of this DSM bullshit was real.

There is no National Institute of Immunosuppression outcomes to Infectious Diseases (NIIID), there is only the opposite, *ALLERGY,* or "too many antibodies" or "autoimmune diseases" - of which ANTHONY FAUCI is the head (NIAID).  Yet, you can see here that Fauci knows all about the immunosuppression/opportunistics (EBV) outcomes of fungal diseases like Lyme and other infections that cause chronic fatiguing/dementia-causing post-sepsis syndrome, here (he has a patent for the treatment of it)

Why, you may ask, does the abuse us?  We suggest reasons in the article linked below, and they're even more sinister than the abuse of tick bite sepsis victims that you have seen so far.


See more at:



OspA or the LYMErix vaccine was Pam3Cys; here we find it loosens the blood brain barrier permanently, as part of the damage:

OspA or the LYMErix vaccine or the fungal triacyl lipopeptide causes immunosuppression and permanent brain damage, but the US Govt only tortured us instead of thanked us for blowing the whistle on it and forcing it off the market.

Where are our heroism or public service medals?   Not gonna happen, why? 



Cryme Disease.  Here is how it works: 

B cells eat OspA-ish, fungal-ish antigens (highly lipidated) and then go limp (no longer do their jobs; this is called tolerance, there are no antibodies being produced).

And so do you, go limp
(post-sepsis, or endotoxin tolerance and cross-tolerance is the same thing as Chronic Fatigue Syndrome or Fibromyalgia). 

The psychopaths who "work" for the CDC and Yale said this OspA fungal endotoxin was a "vaccine," the opposite.

So, the cryme, saying OspA was a vaccine, is actually the disease, post-sepsis immunosuppression, with reactivated latent herpesviruses, etc.  It's simple.

They used to put Thimerosal in vaccines to prevent fungal growth - or OspA-ish antigens -, because such fungal antigens cause immunosuppression.  (You can't make this up: Thimerosal was invented to prevent the OspA Lyme "vaccines.")

See more at Cryme Disease, the movie:  

Join us at:; ours is the only group that explains this disease to you, and shows you the current status of the disease and testing for it.  However, it is an activism group whose goal is to have the CDC and Yale crooks busted by the USDOJ.  We offer no treatment notions, because there really aren't any.  (Please do NOT even ask, that's not what we do.)


UCSF says Lyme and LYMErix-Disease are a diseases of immunosuppression, and they say that half the Lyme/tick bite victims remain ill and are not cured with antibiotics
Feb 12, 2016:

“Early Lyme disease prior to antibiotic therapy was characterized by marked upregulation of Toll-like receptor signaling but lack of activation of the inflammatory T-cell apoptotic and B-cell developmental pathways seen in other acute infectious syndromes,” wrote the study’s authors. “Six months after completion of therapy, Lyme disease patients were found to have 31 to 60% of their pathways in common with three different immune-mediated chronic diseases. No differential gene expression signature was observed between Lyme disease patients with resolved illness to those with persistent symptoms at six months post-treatment.”

"Six months after treatment, 15 of the 29 patients in the study had fully recovered, while 13 had persistent symptoms, and one had dropped out.


Baumgarth, UCDavis:

"For months after infection, those germinal centers fil to produce the specific cells -- memory B cells and antibody-producing plasma cells -- that are crucial for production of lasting immunity.  In effect, the bacteria prevent the animal's immune system from forming a "memory" of the invading bacteria and launching a protective immune response against future infections. 

"The researchers found that following Borrelia burgdorferi infections, this process even prevented the induction of strong immune responses to an influenza infection."

Ray Dattwyler, 1989, in IDSA's journal:

They're obviously all saying the same thing, from 1989 until 2016.  You have a 50% chance of being effed for life if you are bitten by a tick, because the disease is really about immunosuppression, like AIDS.


the United States of STUPID could you see the likes of the's founders, Edward McSweegan and Durland Fish
, not knowing spirochetes were their own phylum (are not regular "bacteria"), are relapsing fever organisms, that the nature of the relapse was antigenic variation (vaccines would do no good), and that the McSweegan-Fish gang never even asked what OspA was (fungal, TLR2/1 agonist, causes permanent immunosuppression, never could have been a vaccine).  The CDC deployed the stupid and cowardly Allen Steere who went into Rheumatology to avoid VietNam (there are no old ladies who need aspirin among deployed US combat forces), probably because he was known to be too stupid to ask any questions about ticks or spirochetes.

What is the simplest way to explain that the ALDF dot com / Yale / CDC Lyme cabal threw out of the Dearborn, "2-tiered" "case definition" the very same disabling, chronic neurologic, "New Great Imitator" disease that was caused by the fungal toxin, Pam3Cys or OspA?  They knew OspA vaccination causes neurologic, immunosuppression disease before the Oct 1994 Dearborn, Michigan fake consensus conference ever took place (chronology here).
In order for it to be prosecuted as a criminal case you have to show they knew ahead of time that there was this problem with their "vaccine" choice and that they CHANGED the testing definition after that, such as to leave out the chronic neurologic Lyme definition - the very disease caused by the fungal OspA vaccines

This is from June, 1993, by Alan Barbour and Durland Fish:

They said this ˄ BEFORE the CDC's Dearborn-"Let's-Leave-Out-the-Chronic-Neurologic-Form-of-the-Disease-Which-Is-Also-Caused-by-OspA-Injection-Alone" fake consensus conference took place (Oct, 1994).  So, these criminals would have known there was a problem with OspA injections making people sick.  After all, OspA is a fungal endotoxin, TLR2/1-agonist.

The current, 1994, "Dearborn" case definition only detects the 15% of cases, only the late-, HLA-linked- arthritis cases, no early cases, and then these CDC (vaccine patent holding) criminals say this late manifestation needs no treatment.  The truth is that chronic neurologic disease is mainly caused by immunosuppression, reactivated Epstein-Barr and cross tolerance to other infections, or, technically, is post-sepsis syndrome or a kind of a B cell AIDS (see the Occam's Razor).

In this criminal case, you have to show to the DOJ that they CHANGED the diagnostic criteria from an older one that allowed more people to be diagnosed (the 1990 CDC version).  If the DOJ does not respond, clearly this was a bioweapon accident of some sort.  The circumstantial scientific, epidemiological evidence from Plum Island as the original outbreak area says so.    (Do the Russians know by now?  They knew in 2006.  The Chinese, a year later.  Site stats.)

The US press cant handle it, and no one has ever written about it in any book.

Whoever does not know what Lymerix-disease is, does not know what Lyme disease is.  This includes ILADS dot org and all the Lyme non-profits.  One has to know what the antigen is, in order to know what it does.  Ad it turns out, that Lyme and Syphilis as "Great Imitators" are really Great Detonators of latent viruses and general immunosuppression, also known as post-sepsis syndrome.  It turns out that Pam3Cys (OspA) or triacyl lipopeptides are functional mimics of E. coli's Lipid A, a serious fungal (TLR2/1-agonist ENDOTOXIN.  Think about that.  The CDC wanted to inject everone with several blobs of bathroom scum.  And that's what happens with a tick bite, too.

Baumgarth, UCDavis:

"For months after infection, those germinal centers fil to produce the specific cells -- memory B cells and antibody-producing plasma cells -- that are crucial for production of lasting immunity.  In effect, the bacteria prevent the animal's immune system from forming a "memory" of the invading bacteria and launching a protective immune response against future infections.

"The researchers found that following Borrelia burgdorferi infections, this process even prevented the induction of strong immune responses to an influenza infection."

Cadavid on Septic Spirochetes:

"Finally, spirochetal lipoproteins have more prominant pro-inflammatory effects compared to other bacterial lipoproteins and synthetic lipopeptides (



I heard through the grapevine that GSK and others involved are intending to "throw Allen Steere under the bus" (verbatim) for this crime of falsifying the testing.
I replied to that insider that that would not be throwing him under the bus because he really was the one who falsified the testing in Europe (Germany; why Germany?).

This video, inside this blog post, has done what the non-non-profits have never been able to do in ~30 years:  Explain the crime, and give you some actionable intelligence - what you can do.


Two important bits of news came out mid-November, 2016. 

One was regarding how the Epstein-Barr virus - and fungal antigens like those shed by spirochetes - inhibit apoptosis (search for that term here) of infected cells, rendering them "immortalized" (you could call them B cell zombies, really) or "immature" or "Epstein-Barr transformed" B cells, causing cancer or chronic fatigue syndrome or "chronic Lyme." 

The truth is in the BCL2 class genes, manipulation of apoptosis.  Dysfunction, here, is the first step in nearly all disease.

And the other was about
the FDA telling Senators Blumenthal, Durbin, et al, to "Screw Off," that "No," the FDA "would not be doing their jobs, assuring Lyme or any other testing is valid."

You heard it all here first, folks.  And all along.  The FDA did not even look at the falsified case definition for "Lyme disease" (1994, Dearborn) when they "approved" LYMErix, obviously.  I did that for them.  LYMErix was the opposite of a vaccine, the case definition was falsified by Allen Steere in Europe and the CDC at Dearborn, and the FDA finally ordered it off the market with an ultimatum to SmithKline after the November 2001 hearing on adverse events that were not "bad knee" but the systemic post-septic shock disease we call "Chronic Lyme, - the very outcome that was left out of the case definition in 1994 when the CDC falsified it.

Government Welfare.  Everyone should have such a Govt job to not only do nothing, but put out proclamations about how they're not going to do their jobs even when specifically asked to do so by the legislators.  Good one.

And and the "non-profits" of Lyme?  They can't and haven't told you anything about these 2 phenomena:  How OspA alone causes chronic illness as a fungal toxin, reactivating Epstein-Barr via immunosuppression, or how if the FDA was forced to do their jobs, Yale and the CDC criminals would be prosecuted for falsifying the case definition to leave out this very immunosuppression and "reactivation of EBV, et al" result.

There it is.  No one who alleges to "help" you, can or will.   There it is, from the fake non-profits to the black market "LLMDs," to blowhard "lawyers" who don't know "data" from "hearsay," to the FDA either in the past or presently, and who now issue their own demands about what they're not gonna do from now on... to the CDC, NIH, to the to the ends of the earth.  Exceptional America, can't or won't do what they claim to be.  No how no where not ever.  But they want you to worship them.

You should be shocked.  There it all is, right there.  ILADS and the non-profits have made no announcement regarding these 2 news items that came out this week.  We checked.  They can't.  

You live in one big giant asylum.  Put the walls up, Mr. Trump.  Close the ports....

161118;  In case you just fell off the cabbage truck yesterday, "Lyme disease" is not even a real thing
Spirochetes ruin your immune system by shedding fungal antigens, by going directly to and damaging the B cell germinal centers in the lymph nodes, and by reverting to a spheroplast form in hostile environments (antibiotics or spinal fluid).  But the CDC staff members formed a fake non-profit called the ALDF dot com and held a bogus conference in Dearborn MI, in 1994 to falsify the diagnostics of this primarily immunosuppression disease.  It was the only way they could sell their own patented DNA for test kits and vaccines. 

So, "Lyme disease" became only the HLA-linked hypersensitivity outcome or arthritis (this is FRAUD and Racketeering).  Fungal-ish antigens shed by spirochetes are more serious endotoxins than the typical lipopolysaccarides of common bacterial infections.  TLR2/1 agonists (fungal-ish, or triacylated lipoproteins) such the shed Outer Surface Proteins (Osps) and the Variable Major Proteins (Vmps) of Borreliae cause disease by turning off the immune system to more than just other fungal antigens.  This is called cross-tolerance.   And Yale University said this dangerous, moldish, bathroom scum-type toxin, a triacyl lipoprotein, OspA, was a "vaccine" rather than an immune-suppressing, sepsis-causing toxin.


New, 161118; Exported Fungal OspA-ish Antigens Also Inhibit Apoptosis and that is how they cause disease.   
You've read about this mechanism in the Occam's Razor and elsewhere on ActionLyme since 2004.



New:  How to Un-Break Americans' Brains:  Keep Hitting them Over the Head With the Obvious. There is no National Institute of Post-Sepsis or Immunosuppression Diseases (NIIID), because to do so would betray the source of the Autism pandemic.  (Yet you hear these things every day on the TV commercials.)


"Translational Medicine" - Hilarious:

'Something Rockefeller University studied and knew about in 1922: that spirochetes are permanent infections and live in the lymph nodes, destroying immunity,... and we hear about it again in 2015 by a lady scientist, Nicole Baumgarth!!
I would say that was OUTSTANDING "translational" medicine, where all the words and years in between were lost,... and we had to have the Craazy Eddie McSweegan gang and a fake FUNGAL lipoprotein Lyme vaccine.  And no one in the ENTIRE ever asked what OspA was, nor to this day will admit what OspA was (Pam3Cys).

Good translating.  Good word-saying, all around.  Needs 27 million more dollars....

Go ahead, check it:


University of California, Davis, says the CDC, Allen Steere, Gary Wormser, Edward McSweegan, et al must be lying their faces off:  Lyme and LYMErix cause permanent immunosuppression or is like a B cell AIDS. 



Great translating from 1922.  'Needs more money because the NIH has not donated enough to that cesspool called the Ivy League, for them to still not understand what spirochetes are/do, despite a million new cases of DISABILITY per year in the USA, alone.

Why doesn't the CDC want you to know that Post-Lyme and Chronic Fatigue/ME is really Post-SEPSIS-Syndrome?

Because the mechanisms of illness - acquired immune deficiency particularly from the likes of TLR2-agonists or fungal-antigen-bearing/shedding infections or even contamination of childhood vaccines with the likes of a fungal antigen like LYMErix -, are the same in Post-Sepsis and Vaccine-Damage Autism.  

The reason the babies are actually GETTING those live, attenuated, brain-tropic viruses from the vaccine vials is due to immunosuppression and/or contamination causing the immunosuppression. 

The reason someone came up with the idea for a Rubella vaccine in the first place was because Rubella causes the
neurodevelopmental brain damage we call "Autism."  Think about it. The kids are GETTING the VIRUSES and they're all brain-tropic.

The Infectious Diseases Society of America says these vaccines are given too many, too young, and are not properly vetted -
please see for yourself, their exact language; the adverse events are a lot like sepsis events.  <<< See all 3 videos, they are all related, and you'll see - finally - that this is simple, and regarding models of disease you know about and have seen before (dual infections and synergy).

The Autism pandemic is the result of babies getting the actual brain-tropic viruses that are in the vaccine vials,
either because those children are immunosuppressed or because the vaccines are contaminated with the likes of fungal antigens like LYMErix. 

Yes, the *ENTIRE* is that stupid.  Add to that, everyone with "MD" after their names.  YES, ALL OF THEM are THAT STUPID.

You have seen the MRIs of the heads of the ZIKA brain damaged children in the news.  The scientific literature says Borna virus is the model for the "neurodevelopmental brain damage" we call Autism.  The IDSA says vaccines are given too many, and too young, and none of them are properly vetted, and this was true since the 1970s.  The scientific literature on those MMR qualifications shows those children were never followed longer than a few days.  The MMR vaccine qualifications were even more criminal than the LYMErix stunt.  Go look for yourself on Pubmed.  You'll do it if you give a sh*t about other people.  But if you don't, you won't bother. 

It is the same with activism, in general.

If you are looking for the very simplest explainer on the Lyme crymes, see the RICO complaint already filed with the USDOJ.  I was talking to lawyers after all, so I had to really dumb it down.


Cryme Disease, the movie:    

The fungal-ish OspA vaccine caused the same post-sepsis, immunosuppression disease as the one thrown out of the case definition at Dearborn.

Steere used a seronegative Lyme assay to assess chronic neuro Lyme, a disease he later falsified and now claims never existed.  Steere recently claimed in a radio interview that (June 14, 2016) he never knew Lyme caused a chronic neuro outcome.  The CDC would like to throw Allen Steere under the bus,... and Steere is trying to avoid being thrown under the bus.  Obviously not happening.




IF you are the kind of person who wants a simple one-liner, single model, single, simple general idea to explain something, this would be it (Dattwyler and Fauci, below).  OspA is a known immune-suppressor, not a "vaccine."

We are not about to prove that OspA is a triacyl lipoprotein also called a basic Pam3Cys molecule type, is fungal, is a more serious endotoxin than LPS, a TLR2/TLR1 agonist, and that other spirochetal lipoproteins are triacylated, if not Pam3Cys exactly.  We can assume anyone knows how to use the National Library of Medicine or else the United States and European patent databases or even just pull the one arm of one-armed information bandit named Google and discover that, for instance, State University of New York, Stony Brook’s, Raymond Dattwyler has a patent for an inhalation form of OspA. 

It, the OspA in Dattwyler’s OspA patent, is intended to attenuate (lessen, or dampen) any suffocating inflammatory response people might suffer from the likes of the dual infections of, for example, influenza and pneumonia, such as what happened in the 1918 Spanish Flu epidemic [FAUCI].  During that pandemic, the healthy people succumbed to the secondary pneumonia.  That is, the strong, mucus-y inflammatory response is what killed 20+ million people back then. They choked to death.  (One infection invites another, this is well-known).

Let’s just go ahead and have a look at what Raymond Dattwyler says about OspA as Pam3Cys and how inhaling a fungal antigen like this, OspA or Pam3Cys or a triacyl lipoprotein might tolerize you against the deadly pneumonia part of Pandemic Flu-moniaâ:


"A lipidation/processing reaction has been described for the intact OspA gene of B. burgdorferi. The primary translation product of the full-length B. burgdorferi OspA gene contains a hydrophobic N-terminal sequence, of 16 amino acids, which is a substrate for the attachment of a diacyl glyceryl to the sulflhydryl side chain of the adjacent cysteine (Cys) residue (at position 17). Following this attachment, cleavage by signal peptidase II and the attachment of a third fatty acid to the N-terminus occurs. The completed lipid moiety, a tripalmitoyl-S-glycerylcysteine modification, is termed Pam3Cys (or is sometimes referred to herein as Pam(3)Cys or Pam3Cys). It has been suggested that the lipid modification allows membrane localization of proteins, with polypeptide portions exposed as immune targets. In addition to serving as targets for the immune response, Pam3Cys-modified proteins, such as OspA, have been reported to act as potent inflammatory stimulants though the toll-like 2 receptor mechanism (TLR2).


The patent is actually against inhalation Inhalation Bubonic Plague.
You MIGHT want to listen to us.

NIAID's Chief Anthony Fauci on the 'monia part of 1918 Spanish Flumonia
® being what causes people to choke to death -- it was inflammatory response in the lungs to the fungal-ish pneumonia:

 2008 Oct 1;198(7):962-70. doi: 10.1086/591708.

Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemicinfluenza preparedness.



Despite the availability of published data on 4 pandemics that have occurred over the past 120 years, there is little modern information on the causes of death associated with influenza pandemics.


We examined relevant information from the most recent influenza pandemic that occurred during the era prior to the use of antibiotics, the 1918-1919 "Spanish flu" pandemic. We examined lung tissue sections obtained during 58 autopsies and reviewed pathologic and bacteriologic data from 109 published autopsy series that described 8398 individual autopsy investigations.


The postmortem samples we examined from people who died of influenza during 1918-1919 uniformly exhibited severe changes indicative of bacterial pneumonia. Bacteriologic and histopathologic results from published autopsy series clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory-tract bacteria in most influenza fatalities.


The majority of deaths in the 1918-1919 influenza pandemic likely resulted directly from secondary bacterial pneumonia caused by common upper respiratory-tract bacteria. Less substantial data from the subsequent 1957 and 1968 pandemics are consistent with these findings. If severe pandemic influenza is largely a problem of viral-bacterial copathogenesis, pandemic planning needs to go beyond addressing the viral cause alone (e.g., influenza vaccines and antiviral drugs). Prevention, diagnosis, prophylaxis, and treatment of secondary bacterial pneumonia, as well as stockpiling of antibiotics and bacterial vaccines, should also be high priorities for pandemic planning.


So, if OspA was obviously something that causes too-much inflammation-and-then-immunosuppression (endotoxin tolerance), it could not have been a vaccine.  And if OspA was not a vaccine, then certain people not only lied it onto the market, they falsified the Dearborn case definition to leave out the 85% of us who do not have a genetic background for a tendency to have Rheumatoid Arthritis (HLAs).

So, IF you are the kind of person who wants a simple one-liner, single model, single, general idea to explain something, this would be it.  OspA is a known immune-suppressor, not a "vaccine."  It does the OPPOSITE of what vaccines are intended to do, which is produce antibodies.  It is a more severe endotoxin than LPS.

"TLR2 mediates inflammatory responses to a wide variety of lipidated microbial components, including bacterial lipoproteins, atypical lipopolysaccharides, and lipomannans (). Among these microbial agonists, bacterial lipoproteins are by far the most potent (, )."


See the rest of the videos here:  Crymes on Vimeo