The reason UConn Health Center is financially floundering is because they and their Friendly-Local-University-Partners-in-Crime, Yale, lost out on over a billion dollars in grant money ($1.009 billion, to be exact) from Sept 2007 to June 2008.  

The NIH is sick of funding no treatments and no outcomes and no benefits to the humans for whom the US Department of Health and Human Services allegedly work. 

The NIH gave out $400 million to everyone-but-Yale-and-UConn, and the Howard Hughes Institute gave out $600 million to everyone-but-Yale-and-UConn.  And the Department of Agriculture took Plum Stupid Island away from Yale because of all their bumbling accidental releases, Lyme Disease almost certainly being  one of them, according to outbreak-range studies performed by SUNY, Stony Brook [PubMed ID: 3577493].

UConn Health Center staff participated in a crime over which the CT Attorney General, Richard Blumenthal, sued civilly, and subjects of the suit included employees of New York Medical College (NYMC), Valhalla, NY, a former Catholic Medical College.

In other words, if UConn Health Center were to be given a new hospital, it would be a reward for committing a huge international crime, and a crime in which UConn and Yale were partners with BigInsurance, namely Kaiser-Permanente (who is still at NYMC literally training new MDs) and American International Group.

Mr. Blumenthal is a former US Attorney and his staff lawyers first told me in 2003 to file it as a scientific fraud and racketeering crime with the USDOJ in New Haven.  Since the New Haven USDOJ refused to prosecute the crime, Mr. Blumenthal’s office sued civilly for “exclusionary practices” and “monopolization” of the “Lyme Disease Guidelines” three years later.

The last failed HIV vaccine trial, in which the NIH was a participant, had to be stopped in the Spring of 2008 because Yale and UConn never told anyone that their nearly identical vaccine (to the HIV vaccine, Pam3Cys), LYMErix (Pam3Cys), “approved” by the FDA in December 1998, was never a vaccine and created the same immune suppression outcomes as the failed HIV vaccine and the failed tuberculosis vaccines:
http://www.actionlyme.org/PAM3CYS_IMMUNE_SUPPRESSION.htm
http://www.actionlyme.org/FUNGAL_VACCINES.htm
 

Yale and UConn knew LYMErix Pam3Cys was never a vaccine and they know “Lyme Disease” is not an autoimmune arthritis in a knee, which they now claim.  Yale and UConn used to call it The New Great Imitator,
http://www.actionlyme.org/CHP_9_IDSA_REVIEWS.htm
because it produced outcomes like ALS, MS, Lupus, and Cancer.   Later we found out that OspA or Yale’s LYMErix “vaccine” itself, Pam3Cys, caused most of these New Great Imitators because it suppressed the immune system or gummed up the natural course of events in developing immunity.

LYMErix turned off the immune system resulting in the activation of latent viruses of all kinds  -resulting in Cancer and MS - and it also tolerized people to fungal antigens or fungal infections, which are thought to be responsible for ALS in some cases.

It was that big of a lie and a global disaster costing the entire world over 10 years in wasted lives,  time and misdirected scientific discovery.  Yale and UConn lied to the FDA about the outcomes of LYMErix.
http://www.actionlyme.org/DICKSON_FDA_SUBMISSION_FULL.htm

If they had not decided to lie about the outcomes of these vaccines ahead of time, they would have stopped in 1998 or earlier to discover why OspA caused a disease like chronic Lyme.  Instead, Yale and UConn further tortured the victims of their crimes, slandering and persecuting LYMErix and Lyme victims and activists, if not worse.

Here is the overall scheme of the Lyme RICO and how it related to BigInsurance:

The Lyme crymes and the cabal are about an intended monopoly on vector borne diseases "test kits" and "vaccines" and could not have happened without the Bayh-Dole Act. 

The monopoly involved Kaiser-Permanente (still) at New York Medical College and the deal was: No one is allowed to have any illness signs nor is treatment to be paid for, until the alleged "vaccine" is ready, and then everyone will be notified about how serious that particular vector borne disease is, and that they better get the "vaccine.”

THE DATA UCONN, IDSA, YALE, and NYMC REFUSED TO TURN OVER TO CT ATTORNEY GENERAL RICHARD BLUMENTHAL:

1) “DEARBORN:”

There was a meeting of all the national labs who did Lyme testing in 1994 in Dearborn, Michigan, in which the labs were invited to “participate in the proceedings.”   The labs said at that meeting that the new diagnostic standard (blood test for Lyme) proposed by Allen Steere (Yale) was between 8% and 22% accurate, or missed 92 to 78% of all cases.  In other words, there was no consensus, but we got this standard anyway:
http://www.actionlyme.org/CRYMEDISEASE_CHP3_B.htm

There's none of that strain B31 OspA in Europe - he says, based on this study, and
OspA is a varying antigen, says CDC officer Alan Barbour:
http://www.actionlyme.org/BARBOUR_MUTANTS_1992.htm

That was how Allen Steere In Europe set up the bogus Dearborn diagnostic standard for the monopoly.

One wonders how one study alone, by Allen Steere in Europe - ALONE - abusing a young student, Frank Dressler, could possibly be the world's diagnostic standard for "Lyme Disease," when the CDC says we are to ignore sci-med reports from Europe (the reverse is clearly true- we should ignore everything published in America).
http://www.actionlyme.org/CRYMEDISEASE_CHP3_B.htm
 

Mr. Blumenthal has this Dearborn booklet that shows the above is true, since I gave his office a copy in 2003, at the time they referred me to the USDOJ.  You can be sure Gary Wormser of New York Medical College never turned over to Mr. Blumenthal his own report which shows that the Dearborn standard only detected 9/59 cases (in IgG), or missed 85% of all cases, since the basis of the “guidelines” is this 1994 Dearborn “case definition.” 

In other words, the “guidelines” are about a bogus entity, “Lyme Disease” renamed and designed at Dearborn to suit BigInsurance’s bottom line and the falsification of the outcome of Yale’s OspA-patent, the LYMErix vaccine.

So, the testing for Lyme is fraudulent and was meant to set up a bogus vaccine trial:  If no one has “Lyme Disease” – and this new Dearborn standard misses ~85% of all cases - then no one will have a “case” of “Lyme Disease” after they were vaccinated with Yale’s vaccine, LYMErix, and the vaccine will be shown to be “safe and effective.”

They committed this crime and we Lyme victims undid it.  The vaccine was removed from the market, but the diagnostic standard remains.  This Dearborn standard only detects the hypersensitivity (allergy) form of Lyme that results in ONLY a bad knee, and no neurologic or chronic fatigue signs.

Insurance companies did not want to pay for long term intravenous treatment for Lyme, since the cost could be $100,000 per year.  So, Yale and UConn worked in cahoots with BigInsurance to narrow disease definitions.  It was a RICO that revolved around vector-borne diseases, which this clique literally called “a gold mine of virulence determinants” (vaccine candidates).

2) IDSA's TREATMENT FAILURE REPORTS:

LINK TO MEDLINE, ALL, "Reviews of Infectious Diseases" (the former name of the IDSA journal), 1989 Supplement 6; this is the status summary that led to the creation of the ALDF.com cabal at New York Medical College with Kaiser-Permanente's takeover of "Lyme Disease"

It was John J. Connolly of CastleConnolly.com and the former Chairman of the ALDF.com cabal, who sold out New York Medical College to Kaiser.  (Connolly is yet another disgrace-to-the-race, in addition to McSweegan and "US Attorney" Kevin J. O'Connor of the DCF-Party-And-Screw for the DCF-Rowlandgate "national string of pediatric jails" enterprise, TREA.)

 

IDSA, the cabal, the ALDF.com; Yale, UConn and NYMC refused to turn over to Mr. Blumenthal 20 or so of their own reports that shows that the treatment of Lyme Disease (OspA-Relapsing Fever or Plum Island Mycoplasmal Borreliosis) failed.   Here are those publication numbers by PubMed, the National Library of Medicine database
http://www.ncbi.nlm.nih.gov/sites/entrez?db=pubmed :
 

At autopsy, lymphoid mononuclear cells were observed surrounding the intracerebral vessels in one section. Using Dieterle silver stain, a spirochete was present in the cortex and another was exterior to a leptomeningeal vessel."
http://www.annals.org/cgi/content/full/121/8/560  (--Allen Steere)
 

d) CDC Officer Mark Klempner on intracellular spirochetes persisting past treatment with ceftriaxone:
1634816;
full text- Fibroblasts protect the Lyme disease spirochete, Borrelia burgdorferi, from ceftriaxone in vitro.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=1634816[uid]
 

e) CDC Officer and owner of the other, ImmuLyme, OspA patent, Alan Barbour (vancomycin and mouse brains):
8913478;  In vivo activities of ceftriaxone and vancomycin against Borrelia spp. in the mouse brain and other sites.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=8913478[uid]
 

[Now, remember, the spirochetes' OspA-shedding or auto-vaccination or spirochetal blebbing of these fungal antigens (Pam3Cys-OspA) causes immune-suppression.  Chronic Lyme is like being continually auto-vaccinated with OspA or Pam3Cys or LYMErix:

Says CDC Officer Alan Barbour:

Many researchers believe that the secret to B. burgdorferi's infectivity and inflammatory capacity lies in the interaction of its surface proteins with the host's immunological system. Yale researcher Stephen Barthold, a veterinarian and professor of comparative medicine who developed the first mouse model of Lyme disease, studies the expression of B. burgdorferi surface proteins throughout various stages of the spirochete's life cycle. He finds that during the early stages of infection, B. burgdorferi avoids immune detection by decreasing its expression of surface proteins or cloaking its expressed surface proteins under a layer of slime. "It's using some sort of stealth-bomber-type mechanism," he says. Or, using another diversionary tactic called blebbing, the spirochete can pinch off bits of its membrane in order to release its surface proteins. Explains Barbour: "It's like a bacterial Star Wars defense program," in which released surface proteins might intercept incoming host antibodies, keeping the spirochete safe from immunological attack. ]
 

f) Russell Johnson, advisor to the ALDF.com (the original “enterprise”) in a patent on chronic Lyme being due to chronic infection:
US Patent:  4,721,617:
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=4,721,617.PN.&OS=PN/4,721,617&RS=PN/4,721,617

"The chronic forms of the disease such as arthritis (joint involvement), acrodermatitis chronica atrophicans (skin involvement), and Bannwarth's syndrome (neurological involvement) may last for months to years and are associated with the persistence of the spirochete…

"The infection may be treated at any time with antibiotics such as penicillin, erythromycin, tetracycline, and ceftriaxone. Once infection has occurred, however, the drugs may not purge the host of the spirochete but may only act to control the chronic forms of the disease.
 

Russell Johnson is also the editor of this 1976 textbook, The Biology of Parasitic Spirochetes:

"The ability of the borrelia, especially tick-borne strains to persist in the brain and in the eye after treatment with arsenic or with penicillin or even after apparent cure is well known (1).  The persistence of treponemes after treatment of syphilis is a major area which currently requires additional study  (3,5,10,11)." 
-Jay Sanford, US Military Hospital, Bethesda, MD   
 

g, h, i) Yale’s and Allen Steere’s Congenital Lyme treatment failure reports:
4003991; Maternal-fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=4003991[uid]


3130607; Borrelia burgdorferi in a newborn despite oral penicillin for Lyme borreliosis during pregnancy:
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=3130607[uid]

"The death of the newborn was probably due to respiratory failure as a consequence of perinatal
brain damage."-- Yale Department of Pathology. 
 

3480464;  Stillbirth following maternal Lyme Disease  (Willy Burgdorfer, SUNY)
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=3480464[uid]
 

Of course, Yale and UConn now say that “Lyme Disease” is “not a problem in pregnancy.”  Well, sure, if they have redefined Lyme to only an autoimmune arthritis in a knee.  No one would say a hangnail, carpal tunnel, or a stubbed toe affected pregnancies either.
 

j) CDC, Mayo Clinic, SUNY, Tulane Autopsy Reports: (Dr. Kenneth B. Liegner, Armonk, NY):

4 cases studies wherein the victims were treated multiple times with antibiotics; the tissues sent to the above 4 labs; all came back positive by staining or DNA for Borrelia spirochetes. (Journal of Spirochetal and Tick Borne Diseases, Lyme Disease Foundation, www.Lyme.org )

k) 1989 IDSA Reviews, SUNY’s Raymond Dattwyler and Benjamin Luft quoting Lenny Sigal and Allen Steere:
2682965  A perspective on the treatment of Lyme borreliosis.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=2682965[uid]
 

“Clinical studies have documented the efficacy of antibiotics, but therapy has failed in as many as 50% of cases of chronic infection. Although new antibiotic regimens appear promising, the optimal treatment of this infectious disease remains to be determined.”

What happened was that once BigInsurance saw this series of reports by IDSA in 1989, they freaked out and decided they were “just taking over medicine and the MDs had better just get used to the idea.”—John J. Connolly, former president of the Catholic Medical School, New York Medical College, who sold out the college to Kaiser-Permanente…  Kaiser-Permanente, you can verify independently, is still at NYMC literally training MDs.

This statement on record by John J. Connolly is on record at the New Haven USDOJ… collecting dust.

l) CDC Officer Alan Barbour in 1986 stating that neurotropism (spirochetes going after nerve tissue for a meal and a home) was not trivial, that spirochetes were formally stored in rodent brains and that a treatment for neurosyphilis that he recommends would be to treat the patient with Relapsing Fever spirochetes: 3540570  The Biology of Borrelia Species
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=3540570
 

“When using borreliae for pyrotherapy (induce a fever) of neurosyphilis, the authors of this report recommend that no more than 30-40 passages in mice be made before inoculation of the strain into humans.”

"The propensity for borrelia to go to the brain of infected mammals suggests that the relationship between these spirochetes and neural tissues is not trivial.  Further study of this attraction and the interaction that follows may reveal the basis for the significant nerve and brain involvement in Lyme borreliosis"—

 
m) Dattwyler, Benach, Pachner and “early brain invasion”:
i) Dattwyler at the 1994 June, FDA Meeting Minutes/Transcript regarding OspA Lyme vaccines:

“Ben Luft and myself, and a number of our colleagues, have looked at early CNS invasion using PCR, and found that, in individuals with multiple erythema migrans rashes, or a single lesion or erythema migrans and major constitutional symptoms, about 2/3rds of those individuals will have Borrelia burgdorferi DNA in their cerebrospinal fluid, which we take as evidence of early central nervous system invasion of this organism.”

Now, UConn and Yale – and the BigInsurance companies they prostitute for - maintain that “Lyme only happens in your knee,” because knee diseases do not require the expensive intravenous ceftriaxone.  Brain diseases require intravenous medications because that is the modality for bacterial meningitis, the efficacy of which depends on maintaining a high concentration of the drug in the blood.  High concentrations of antibiotics into the area of the brain and meninges is just not achieved with oral antibiotics.

ii) 2215944; Borrelia burgdorferi infection of the brain: characterization of the organism and response to antibiotics and immune sera in the mouse model.-  Andrew Pachner
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=2215944[uid]

Organism was cultured from the brain early in the course of infection, and this isolate, named N40Br, was further studied in vitro. The plasmid content of N40Br was different from that of the infecting strain, implying either a highly selective process during infection or DNA rearrangement in the organism in vivo.
 

2345299; Borrelia burgdorferi in the central nervous system: experimental and clinical evidence for early invasion.  Jorge Benach
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=2345299[uid]

This experimental evidence for early central nervous system invasion was pursued in studies of the human disease. Specific B. burgdorferi antigens could be detected in the cerebrospinal fluid of patients with early Lyme disease by use of murine monoclonal antibodies as probes.

 

It is therefore indisputable that Lyme Borreliosis is a disease of the brain and not of the knee.

n) IDSA staff referring to a report by University of Rhode Island where within one minute after the addition of whole rabbit blood, the spirochetal cyst form reverted back to the intact spirochete form (IDSA/Yale/UConn falsely claim that the cyst or the spheroplast form is an end-stage or an “almost dead” stage, when in fact, it has been well-known through history to be a dormant phase):

10658658; Serum-starvation-induced changes in protein synthesis and morphology of Borrelia burgdorferi.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=10658658[uid]
 

UConn, Yale and NYMC IDSA crooks – the cabal sued by Mr. Blumenthal - referring to the above report:
http://scholar.google.com/scholar?q=link:http://mic.sgmjournals.org/cgi/content/abstract/146/1/119
meaning they read it;

and in fact the US Army advises soldiers to be careful not to inhale spirochetal cysts when around dried animal urine; US Army Manual (LEPTOSPIROSIS):
http://www.afpmb.org/pubs/dveps/haiti.pdf

 

o) Russell Johnson discusses in his article in the 1989 Infectious Disease Reviews (the name of IDSA’s former journal) how it can take a few months to reculture the spheroplast form of the spirochete back into the intact spirochete form:
2682963  Isolation techniques for spirochetes and their sensitivity to antibiotics in vitro and in vivo.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=2682963[uid]
 

3)  THE PRIMERS SHELL GAME and RELAPSING FEVER:

The vaccines and test kit candidates this cabal intended to corner the market on (in grant money or free, taxpayer-funded venture capital for their biotech products in addition to manipulating “disease definitions” on behalf of BigInsurance) required patenting sections of DNA to be spliced into E coli and the like (“Biologics”).  They call these sections of DNA – that they OWN- recombinants.

Recombinant OspA or recombinant Pam3Cys comes from plasmid DNA and not the single linear chromosome that is the main DNA code for these spirochetes.  The plasmid DNA is VARIABLE DNA or the DNA that changes as a result of the mechanism by which Relapsing Fever Borrelia – the genus to which Lyme Disease belongs – are, in fact Relapsing Fever organisms.

That is, through variation in this plasmid DNA, the spirochetes change their outer surface proteins (Osps, like OspA, like LYMErix, or the HIV glycoproteins gp 120 and gp 41) rendering antibodies no good, or the patient relapses.

Antibodies do no good and neither do vaccines, obviously.
 

Taxonomically, the differences in the Relapsing Fever spirochetes – since the differences can’t be in the variable plasmid DNA – is in the non-variable backbone, if you will, of spirochetes, the flagellin.  So, all spirochetes are categorized by differences in their flagellin.

The UConn/Yale crooks are aware of this, but most MDs are not.  Whenever Yale/UConn and the other members of this cabal want to find spirochetes to patent, they search ticks using flagellin DNA primers (or amplimers, or a short DNA template), or they use other BORRELIA-SPECIFIC RNA, like the BORRELIA SPECIFIC intragenic spacer RNA.

When these crooks pretend to be looking for Borrelia DNA in humans, they use the wrong primers.  They use the bogus variable DNA primers like OspA.  They also insist that there is no “Lyme Disease” in areas where they know there are other Relapsing Fever spirochetes. 

In short, according to this criminal gang, if people don’t have the OspA gene, they don’t have “Lyme Disease” and don’t need to be treated.

I hope you can follow this shell game:

When looking for spirochetal DNA in ticks to patent, these criminals use the correct primers.

When they want to find “No Lyme,” they use the wrong primers; the look for a DNA sequence that they know is unlikely to be there in a patient.

You can see the BigInsurance influence in this game.  Their bottom line is all about not identifying and then being obligated to treat anything.

Here are the PubMed articles and patent numbers that prove UConn, et al, know which are the correct primers to use to find Borrelia spirochetes- but have never been used in treatment outcomes for humans:

US PATENTS

5, 618, 533  - Yale’s Flagellin method, which is the only scientifically valid test for Lyme Borreliosis; it is not in use, licensed by no one, and Yale did not use this patented method to assess their other patent, LYMErix, because they knew LYMErix did not prevent Lyme.

6, 045, 804  - The Central RICO patent and the associated report worked on by Yale’s Robert Schoen; in this patent Schoen et al, demonstrate that they know which RNA primers to use to detect OspA-Borrelia.  The patent was applied for in 1996, developed in at least 1995, and in it they describe 1) How they can’t read their Western Blots in LYMErix-vaccinated people (meaning, they have no way to prove LYMErix prevented Lyme), and 2) vaccine failure is indistinguishable from chronic neurologic Lyme Borreliosis.

But they never told anyone.  And this is important, because we later found out the reason chronic neurologic Lyme was the New Great Imitator was the result of Pam3Cys or OspA-induced immune suppression and the activation of latent viruses of all kinds (MS and Cancer) in addition to tolerance to fungal antigens (ALS and Chronic Fatigue Syndrome).

In other words, “LYMErix Disease” – the diseases set caused by chronic Lyme and LYMErix - is the Yuppie AIDS, or acquired immune deficiencies.

It’s a huge crime.  The Nobel Prize winner for the discovery of the HIV virus, Luc Montagnier, is now following up on why the LYMErix Pam3Cys vaccine failed, because it is the same antigen as the HIVgp120 failed vaccine, generally, Pam3Cys (different amino acids, but the general structure is the same).

Imagine what would have happened if Yale and UConn had been honest about this 10 years ago?

5, 932, 220  B. Theileri-Come-Mastersi-Come-Barbouri.   This is a patent for Borrelia found in Lone Star Ticks, which is also known as Southern Lyme Disease or Master’s Disease, or STARI.  Gary Wormser of New York Medical College says this is not “Lyme Disease,” not informing people that this patent is for a cow-relapsing fever that infects humans through a Lone Star Tick.  The patent is for flagellin, because these crooks know that differences in flagellin are the demarcation for different species. 

However, they- Yale, UConn, and the cabal - say repeatedly in public, that the human victims of this disease do not have “Lyme Disease” and are therefore not sick and need no treatment.   They’re ALL Relapsing Fever organisms.  “Lyme Disease” is just chapter one of probably a 50 year roll-out plan where we only learn about a new disease after the vaccine has been fraudulently qualified.  In the meanwhile these crooks say we’re CRAZY – denying us medical care and long-term disability coverage from our employers - when in fact, we do have a real brain disease.  ‘One that needs intravenous ceftriaxone, relapsingly.

EUROPEAN PATENTS: WO9324145
CDC staff members own patents in Europe with SmithKline, in which they prove that they know that the arthritis-only kind of Lyme or UConn’s and Yale’s fraudulent definition of Lyme or the Dearborn kind of Lyme is not the only kind of Lyme:

http://v3.espacenet.com/inpadoc?submitted=true&DB=EPODOC&CC=WO&NR=9324145&KC=&F=8&OREQ=0&textdoc=TRUE&FT=E

 
Summary of the Invention In one aspect, the invention provides isolated B. burgdorferi antigens which are regulated and differentiated by growth of the B. burgdorferi in a tick vector. Novel antigens of the invention are listed below in Table I.

Certain of these antigens are characterized as being B. burgdorferi B31 strain specific and major histocompatibility complex (MHC) nonrestricted. Certain other of these antigens are characterized as being MHCrestricted. Sera generated to these antigens (B31 MHC nonrestricted and B31 MHC restricted) are further characterized by the ability or lack of ability to react with B. burgdorferi JD-1 strain; the antigens themselves (B31 MHC nonrestricted and B31 MHC restricted) are further characterized by being homologous or heterologous with B. burgdorferi JD-1 strain antigens. The most preferred antigens of this invention, because of their ability to induce cross-strain immunity to B. burgdorferi in different animal haplotypes, are characterized by being B31 MHC nonrestricted, JD-1 crossreactive, and JD-1 nonrestricted.Other antigens are also useful in vaccine compositions and as diagnostics.

­ That’s long for:  “There are 2 kinds of Lyme.  Yale’s hypersensitivity reaction to OspA, over which SmithKline was sued in two class actions and Yale, et al, was later sued by the CT AG, and Neuroborreliosis, a disease denied because we, the CDC, are also profiteering, lying crooks and are in on the scam.”

THE PUBMED REPORTS ASSOCIATED WITH THE PRIMERS SHELL GAME:

The following reports are by members of the RICO cabal that show that they know that they’re playing a DNA shell game, and that they KNOW which are the correct, unchanging, Borreliae-specific DNA or RNA primers.

PubMed ID:  8968914; Borrelia burgdorferi enzyme-linked immunosorbent assay for discrimination of OspA vaccination from spirochete infection. -- Yale’s Robert Schoen, and Dave Persing of the Mayo Clinic (at that time), members of the RICO cabal.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=8968914[uid]

­ This 1996 report by this RICO gang pretty much knows how to detect any kind of Borrelia anywhere.  But no one is allowed to have a “disease” unless they have a bad knee from a hypersensitivity response to OspA, the vaccine.  Here they state that they distinguish Borrelia from species specific 23S ribosomal RNA, or the intragenic spacers that are unique to Borrelia burgdorferi.

Such a method has never been used on treatment outcomes in humans.  No one who has this devastating disease, Borreliosis or Relapsing Fever, happens to care if they have Yale’s patented OspA kind of “Lyme Disease.”  (I don’t care if I have Borrelia outermongolii or Borrelia antarcticii or Borrelia whopperburgerii, or Borrelia gimmeabreakii, if you know what I mean.)  In the National Library of Medicine Taxonomy database, there are about 160 strains of Relapsing Fever listed, 30 specific to California alone.  But, according to IDSA, “there is no Lyme Disease in California.”


Taxonomy Database (look how many strains say "CA" for California in front of them)
http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=138
About 40 "CA" strains listed.

This would be particularly true because Kaiser-Permanente basically owns California, and hence the nickname, Kaiserfornia.  New York Medical College is their New England “forward base,” so to speak.

And finally, here is Gary Wormser of New York Medical College using, the one and only time, the correct RNA or DNA primers to determine that antibiotic treatment of a tick bite fails to kill all the spirochetes in 2/9 cases:

1452688; 1992; Diagnosis of early Lyme disease by polymerase chain reaction amplification and culture of skin biopsies from erythema migrans lesions.
http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=DetailsSearch&Term=1452688[uid]
 

­ In this 1992 study, Gary Wormser published that he knows which are the correct primers to use when he wants to find spirochetes.  He found through biopsy that among 9 patients who were treated with antibiotics right after tick attachment, that there was still spirochetal RNA in the skin of at least 2 of them. 

This study, where the CORRECT RNA or DNA primers were used, has never been repeated in an IDSA antibiotic treatment study, and this cabal now publicly states that no one needs to be treated upon tick attachment.

‘Despite knowing these spirochetes go right to the brain, and the infection is permanent.

So, in summary, there is plenty of reason to dispute funding a 600 million dollar hospital for crooks who work with BigInsurance against patient care and who participated in this huge, international Pam3Cys-HIV vaccine failure fiasco costing the world at least 10 years in wasted lives and money.

=======================================

4) INTRACELLULAR, VIABLE, REPLICATING CYST OR SPHEROPLAST FORMS:

Question:  If spirochetes, when driven into the cyst form, replicate, do we end up with
even more spirochetes than we would have had we not taken antibiotics?
--1983, Proposed Life Cycle for the Reiter Treponeme  (validity of "cysts," or spheroplasts or regeneration forms)

--- From the Plum Stupid Island Chapter of Cryme Disease

1911:  ...It will perhaps be remembered that one found intracorpuscular forms in this fowl spirochaetosis, and that following Sambon, one had to come to the conclusion that these endoglobular bodies represented a stage in the lifecycle of the spirochaete -- constituted, in short, its stage of schizogony in the fowl.  Sambon, however, who expressed this view from the study of a few slides I gave him, did not indicate how this red cell invasion occurred.  For a long time I believed the spirochaetes themselves entered the red cells and broke up, or coiled up, within them to form these remarkable bodies.  As the parasites can and do enter and leave the erythroblasts of the fowl, there was good ground for this supposition.  Now however, I know better.

--- From The History of Relapsing Fever Chapter of Cryme Disease