Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine."

01 Oct 2017


File List, RICO

1988 Steere says Lyme is like a B cell leukemia

Assoc Blogs-n-Webs:




Fungal Exosomes Inhibit Apoptosis

IDSA: "Vaccines serve the mfgs, not their victims"


BlumenthalAntiTrust Lawsuit

Exosomes, Blebs


CDC Admits Fraud, 2016
Dattwyler, 1988
Golightly, 1988
Dressler, 1994
BarbourFish, 1993
Dearborn, 1994

Pathogenic Fungi

Bush's warcrimes, Oct 2000




120420; "Lyme Disease" Update from the CDC, NIH, NIAID, Yale and, none of whom, in a month's time, has been able to tell me what OspA (the Lyme vaccines) is - they all say they do not know:

Dr. Paul Auwaerter PhotoThe following is a letter sent to them regarding Paul Auwaerter, the current blowhard/mouthpiece of IDSA:


To: KMDickson <>, "Strikas,Raymond A. (Ray) (CDC/OID/NCIRD)" <>,,,,

Subject: Paul Auwaerter Does Not Know What OspA Is Either

Date: Apr 20, 2012 4:35 AM


I called Auwaerter's office (Johns Hopkins) and left a message asking if he knows what OspA is. The answer was no, he did not.

I guess he is not an expert in Lyme Disease either.

Now, all of this is amusing given the fact that Roland Martin quit the NIH's MS-Lyme group once he found out OspA was causing
the immune suppression (and seronegative, non-Dearborn Lyme) resulting in the MS form of Lyme (known to be associated with
reactivation of Epstein-Barr):
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."

as does Adrianna Marques, who publishes these
articles with him:

"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes."

That^^ all means Chronic Lyme is seronegative and the chronic exposure to OspA/Borreliosis results in NO ANTIBODIES. We now know for sure that Klempner's "study" can be thrown out since the study design was based on the fraudulent Dearborn diagnostic standard.

[Dearborn was fraudulent because there was no consensus (I have the booklet) and because Steere formulated the antibody panel via
classic research fraud, using high passage strains and recombinant OspA and B without the antibody-producing lipids attached, setting
up his own little racketeering enterprise with Dave Persing (Corixa) and Yale's Robert Schoen (L2 Diagnostics) for the intended
post-LYMErix monopoly on blood, patentable goodies in the blood, and grants.]

Secondarily, Paul Auwaerter lists as his expertise, Lyme and Epstein-Barr (known to be activated by Lyme and probably LYMErix
as shown by Marques and Martin, above).

Thirdly, Bridgette Huber at Tufts, who published with Allen Steere ("Lyme causes only the HLA-linked hypersensitivity response")
but her area of expertise also includes Lyme and Epstein-Barr (EBV activating a human HERV, the favorite of Harold Varmus
former director of the NIH:

Fourthly, Adrianna Marques lists on *HER* website, that her areas of expertise are Lyme and Epstein-Barr (and some other herpes viruses).

So, it looks to me that "Post Lyme Syndrome" is, in Marques' words, "Chronic Active Epstein-Barr."

Especially since Paul Duray (US Army and the National Cancer Institute) reported twice in IDSA's journals that the lymphocytes of
chronic Lyme victims "look like Epstein-Barr transformed cells" in 1989 and 1992.

How this happens, how these fungal antigens seem to reactive Epstein-Barr, I do now know exactly, other than the association to
exposure to OspA and subsequent production of the immunosuppressive cytokine, IL-10.  (I won't produce the references for that
because they're well known and accessible.)

Now, the problem with Adrianna Marques is that she is recruiting chronic Lyme victims to see if she can get clean ticks to become infected with Borrelia from biting post-treated Lyme victims - from the blood.

But that's not really what's going on, is it?

It's going to be mycoplasma to which we have become tolerant via exposure to OspA (blebbing is the same as autovaccination), and some kind of herpes, et al, correct?


Ticks are a great place for transkingdom plasmid-sharing according to Barbour and Burgdorfer.

You're thinking of *that* ^ when Marques tries to pharm Borrelia not known to be in the peripheral blood of human victims due to the tissue penetrating, plasminogen-hijacking OspA??


Kathleen M. Dickson