120420; "Lyme Disease" Update from the CDC, NIH, NIAID, Yale and IDSociety.org, none of whom, in a month's time, has been able to tell me what OspA (the Lyme vaccines) is - they all say they do not know:
The following is a letter sent to them regarding Paul Auwaerter, the current blowhard/mouthpiece of IDSA:
(UPDATE ON PAUL AUWAERTER - HE DOES NOT KNOW What OspA IS):
To: KMDickson <email@example.com>, "Strikas,Raymond A. (Ray) (CDC/OID/NCIRD)" <firstname.lastname@example.org>, email@example.com, firstname.lastname@example.org, email@example.com, firstname.lastname@example.org
Subject: Paul Auwaerter Does Not Know What OspA Is Either
Date: Apr 20, 2012 4:35 AM
I called Auwaerter's office (Johns Hopkins) and left a message asking if he knows what OspA is. The answer was no, he did not.
I guess he is not an expert in Lyme Disease either.
Now, all of this is amusing given the fact that Roland Martin quit the NIH's MS-Lyme group once he found out OspA was causing the immune suppression (and seronegative, non-Dearborn Lyme) resulting in the MS form of Lyme (known to be associated with reactivation of Epstein-Barr): "Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression." http://www.ncbi.nlm.nih.gov/pubmed/16783164
as does Adrianna Marques, who publishes these articles with him:
That^^ all means Chronic Lyme is seronegative and the chronic exposure to OspA/Borreliosis results in NO ANTIBODIES. We now know for sure that Klempner's "study" can be thrown out since the study design was based on the fraudulent Dearborn diagnostic standard.
[Dearborn was fraudulent because there was no consensus (I have the booklet) and because Steere formulated the antibody panel via classic research fraud, using high passage strains and recombinant OspA and B without the antibody-producing lipids attached, setting up his own little racketeering enterprise with Dave Persing (Corixa) and Yale's Robert Schoen (L2 Diagnostics) for the intended post-LYMErix monopoly on blood, patentable goodies in the blood, and grants.]
How this happens, how these fungal antigens seem to reactive Epstein-Barr, I do now know exactly, other than the association to exposure to OspA and subsequent production of the immunosuppressive cytokine, IL-10. (I won't produce the references for that because they're well known and accessible.)
Now, the problem with Adrianna Marques is that she is recruiting chronic Lyme victims to see if she can get clean ticks to become infected with Borrelia from biting post-treated Lyme victims - from the blood.
But that's not really what's going on, is it?
It's going to be mycoplasma to which we have become tolerant via exposure to OspA (blebbing is the same as autovaccination), and some kind of herpes, et al, correct?
Ticks are a great place for transkingdom plasmid-sharing according to Barbour and Burgdorfer.
You're thinking of *that* ^ when Marques tries to pharm Borrelia not known to be in the peripheral blood of human victims due to the tissue penetrating, plasminogen-hijacking OspA??