"It is well known that Borrelia burgdorferi indeed after asymptomatic infection can lurk or secrete itself in certain areas of the body, perhaps the central nervous system or perhaps the joint spaces, only to reappear months or maybe years later in the form of late stages of illness which are harder to diagnosis and treat."  - Vijay Sikand, East Lyme, CT at the 1998 FDA Meeting on LYMErix when trying to advocate for a vaccine against a disease.... they simultaneously claim does not exist.

"The fact that OspA vaccination caused all the "New Great Imitator" outcomes like MS, Lupus, RA, Cancer, etc., as well as Chronic Lyme/Fatigue was the biggest medical discovery of the last 500 years. It was a huge breakthrough in medicine (fungal-viral synergy) that Yale, et al, can't now take credit for because they vehemently and viciously lied about this, even in numerous court cases." - The Future "Khan Academy" of Lyme Cryme & HLA-Negative Diseases

 

2 New Posters for Distribution:

 

Lyme Facts:

Does someone close to you have Lyme disease? Chances are, the answer is “yes,” but due to the politically and emotionally charged climate surrounding Lyme disease, you may be unaware of their plight. In fact, many sufferers themselves are unaware of their true diagnosis.

Here is a story that will leave anyone with a conscience asking, "How can we let this continue?" It has all the elements of a best-selling modern crime novel. It has mad scientists, hungry for money and fame. It has backroom handshake deals between international corporations and corrupt government officials. It has people dying, begging for help, and being shunned, oppressed, and destroyed--physically, emotionally, socially, and financially. It has deathly ill children, and doctors losing their licenses for simply trying to help them. It has a whistleblower who was jailed and lost both her career and her children, but continues to fight 20 years later. It is tragic beyond belief, yet 100% true. It is the story of Lyme disease.


History: Prior to development of the Lyme vaccines [LYMErix and ImmuLyme, both recombinant Outer Surface Protein A or OspA (rhymes with “Wasp-ay”)], there were no “guidelines,” on the treatment of Lyme Disease. In the Infectious Diseases Society of America’s (IDSA’s) journal, Infectious Disease Reviews, in its 1989 special supplement on Lyme and Spirochetal Diseases, the status of treatment was that “the treatment endpoint is unknown.” Soon after, in 1990, the CDC’s published diagnostic standard advised to perform repeated or sequential Western Blots to look for new and expanding IgM and IgG bands because new bands meant “the bug was still alive (Allen Steere, 1986).”

[The correct name for this disease is Borreliosis or Relapsing Fever; the nature of the relapse is antigenic variation or the varying of its surface proteins, such that once an antibody was made, the bug drops that antigen and makes a new one. And therefore, you will see new IgM type (which means new antibodies) antibodies appearing over time. And therefore the idea of vaccines based on these variable outer surface proteins was counterintuitive: vaccines would do no good since antibodies do no good. The very concept vaccines is to produce antibodies against a disease such that they’re ready to be recruited to fight off an infection more rapidly and effectively.

But the essence of this Grand Lie of Lyme was to call this new disease “Lyme Disease,” such that doctors who had no education in Taxonomy would not know this was the same old Relapsing Fever written about extensively after World War II.]
 

The Spin Begins: The case definition or testing standard for Lyme Disease was falsified in 1994 at the CDC’s Dearborn, Michigan, conference so as to have the OspA vaccines appear “safe and effective.” This new, “Dearborn” case definition of the disease, or, the antibody panel suggested by Allen Steere for that farcical CDC conference, was narrowed such that only the arthritis-associated HLAs or the people with a genetic propensity to have Rheumatoid Arthritis and high antibody titers would test positive.

High antibody titers are classically the “autoimmune diseases,” like Lupus and so forth. You will see that the vast majority of people who have diseases without the classic HLA-linked high antibody responses, were actually the key to one of the greatest discoveries of the last two centuries (“post-sepsis immunosuppression” or fungal-viral synergy). All of this was exposed in the fraudulent OspA vaccine trials. If only 15% of the cases are detectable, and 5500 people were given OspA vaccines and 5500 were given placebo (the LYMErix vaccine study design), and these victims were sent out into the world to see if they got tick bites and Lyme disease,… you can guess what happened to the 85% who got Lyme anyway and whose Lyme was not the Dearborn CDC/vaccine patenteer’s version of it. Right, they were told they had an “unexplained, vague disease,” and they were not counted as vaccine failures.

If you can’t find Lyme Disease, you can’t find vaccine failure, right? This new, falsified, 1994, non-consensus “Dearborn case definition” said “only people with late, HLA-linked, autoimmune-linked Lyme arthritis could have a ‘case’ of ‘Early Lyme.’”

What about the other 85% who were formerly considered Lyme cases? They were thrown out with the vaccine-failure and vaccine-caused adverse events cases.

Amazingly, the OspA vaccines were fungal antigens (or TLR2-agonists which means high fat content or lipidated or fungal) and were, it turned out, the very thing responsible for the “protean” (Ben Luft, 1998 FDA meeting), “multi-system disease,” (Dave Persing, US Patent # 6,045,804) OspA vaccine outcomes, just like the contentious “Chronic Neurologic Lyme.” Only about 15% of the population have the arthritis HLAs, which means Lyme disease will be missed in 85% of the cases - this was the crime. The crime was committed solely to sell vaccines for a disease that defies the concept of vaccines.

Additionally, adding shock to criminal-injury-intent, not only was the Steere/Dearborn proposal for a new case definition based solely on the HLA or arthritis case outcomes, none of the labs “invited to participate in the proceedings” of the Dearborn conference agreed with Steere’s proposal for a new case definition. The only lab that agreed was MarDx, but they had been given arthritis-positive blood to qualify their Western Blot test strips; MarDx was later hired by both OspA vaccine trial companies to assess, allegedly, Western Blotting to determine whether or not the OspA vaccines prevented Lyme. The average of the other participating labs said the accuracy of the new, proposed case definition was 15%.

Again, the new, falsified, Dearborn case definition was designed to only detect the 15% of the population with the Rheumatoid Arthritis genetic predisposition - and 15% was also, not coincidentally, therefore, the average accuracy assessed by the participating labs at Dearborn when tested in the field. For example, New York Medical College’s Gary Wormser reported at Dearborn that this newly proposed (by Steere) case definition only detected 9 out of 59 cases in IgG (15%). Imugen Labs said this Dearborn proposal only detected 14% of the cases.

We got this standard anyway. No one knows why, since Dearborn was obviously not a consensus conference. It could be that because the CDC officers who ran the Dearborn conference were also owners of multiple patents that would be commercially worthless if it was known that 85% of the victims of Relapsing Fever, especially this new one with the abundant Outer Surface Protein A, which is a fungal antigen and is immunosuppressive for the other 85%, rarely produced any antibodies other than band 41 or the anti-flagellar antibody for any length of time. It could be that you cannot assess the safety and accuracy of vaccines for a disease that has essentially no antibodies due to spirochetal shedding of these very same surface proteins which turn off the antibody response in the classical way other fungal diseases do (or “cause T cell anergy,” Dattwyler, 1988). It could be that this whole DNA patenteering stunt was too outrageous for the U.S. Government to admit to approving because all the other agencies like the NIH would look incompetent for allowing it to happen. 
Willy Burgdorfer, the discoverer of the spirochete responsible for the disease said: “The controversy in Lyme disease research is a shameful affair. And I say that because the whole thing is politically tainted. Money goes to people who have, for the past 30 years, produced the same thing—nothing. Serology has to be started from scratch with people who don’t know beforehand the results of their research” (Under Our Skin crew).
http://underourskin.com/news/lyme-discoverer-willy-burgdorfer-breaks-silence-heated-controversy

Dearborn was “BS” in the common vernacular.

And the LYMErix vaccine was approved, anyway, by the FDA (the FDA panel being loaded with people like Allen Steere, Robert Schoen, and Vijay Sikand – the very people who ran the OspA trials) and came on to the market in late 1998 despite numerous “provisos.” More than 1,000 systemic adverse events were reported through the VAERS from September 1999 to November 2000, whereupon the FDA granted a public hearing, January 31, 2001. Several months later, in the fall of 2001, Karen Forschner of the Hartford, CT based Lyme Disease Foundation (Lyme.org) delivered to the FDA – in person -, a patent owned by Bridgitte Huber at Tufts where it was declared that OspA was technically a “toxin,” right in the abstract (US Patent 6,689,384). The FDA then gave SmithKline and Yale (the assignee of the LYMErix patent), an ultimatum: “Either you remove LYMErix voluntarily or we will order it off the market.” SmithKline chose to avoid the embarrassment and pulled their own non-vaccine.

We’re still stuck with this bogus Dearborn case definition, despite numerous attempts at lawsuits against IDSA, SmithKline, and filing complaints to the U. S. Department of Justice. It is still very dangerous for the public to be unaware that the average person, or 85% of us – who are the “seronegative patients are the sickest ,“ according to Raymond Dattwyler at the 1994 FDA meeting on Lyme and LYMErix (which preceded Dearborn) - have no chance of testing positive to this criminal CDC’-Dearborn standard, because the actual disease is one of immunosuppression, or is an Acquired Immune Deficiency,… or is similar to AIDS with all the opportunistic infections that the Lyme and LYMErix victims cannot control.

It was said at the time LYMErix was still on the market that this vaccine, via its claimed mechanism of disinfecting ticks with human antibodies (yes, if you can believe it), that LYMErix would turn humans into walking cannisters of tick disinfectant, when in fact, LYMErix turned people into walking “cesspools of disease.” The same is true for Chronic Lyme. Chronic Lyme victims’ immune systems are “overwhelmed”- a term used by CDC officer Alan Barbour, when describing what antigenic variation in spirochetes does to humans (US Patent 6,719,983). This is a term you want to remember in case you hear it again: “overwhelmed” immune system means: “turned off.” “Turned off” is the complete opposite of an “inflammatory” or “autoimmune disease.”

 

The 300,000. The CDC recently claimed that Lyme disease was only 10% reported making their 30,000 cases reported per year, really 300,000. However, since Lyme is only 15% DETECTABLE because of the Dearborn crime, the number of new cases per year is really closer to 2 million.

Instead of being treated for Lyme disease, the 85% are given garbage pail diagnoses of Fibromyalgia, Chronic Fatigue Syndrome and psychosomatic disorders, for which there are no cures--only expensive drugs to mask some of the symptoms. The technical definition of a somatoform or psychogenic disease is that a person has all the valid, scientific, laboratory-confirmed signs of disease, but that this is not due to an actual disease, but magical powers, such as conjuring BigFoots or UFOs or conjuring anything, really. Mothman. The Jersey Devil. The Creature from the Black Lagoon. No one in the medical community ever questions why so many conjurers are allowed to run loose with such paranormal powers. None even ask if the person complaining of her/his imaginary disease is a victim of a hex, rather than the self-perp.

As you can see, this is obviously something more than a mere Conflict of Interest issue. These same “scientists” (and goofball “MDs” who believe in magic and BigFoots) make up the majority of the IDSA. CDC officers are merely using the IDSA as their free PR firm for selling vaccines- some of which are CDC’s own vaccines. Some CDC officers hold leadership positions at major research institutions such as Yale, UCal, Irvine, and Johns Hopkins.

Senator Richard Blumenthal sued the IDSA for Anti-Trust” in 2006. The result of this crime is homicide and permanent maiming and disability. Some participants of this scam have “Disclaimers” on their websites stating that they’re not liable for “medical negligence” if any doctor takes their advice and recommendations (the ALDF.com and the EUCALB have the exact same language in their disclaimers).

The "Dearborn and LYMErix-causes-immunosuppression" Whistleblower (see the FDA, Jan 2001), a former Pfizer analytical chemist, filed a RICO and Fraud complaint with the U.S. Department of Justice in 2003. For more than 10 years there has been a total failure by the USDOJ to prosecute this crime and protect U.S. Citizens.

Activist patients are organizing and mobilizing with national efforts to #OccupyTheUSDOJ. Though we are extraordinarily ill, we have passion and truth on our side. We also have all the necessary research and documentation to prove this crime. Those responsible will be prosecuted. We will get justice.

This is one of the biggest corruption cases in U.S. history. It involves medical research fraud, and organized crime between government agencies, academia, and the health care industry. As a bonus, the prosecution of the Lyme criminals also will expose the key to unlocking the autism epidemic and Gulf War Syndrome. Your assistance in bringing attention to our movement has the potential to help millions of people suffering needlessly all over the world.

The time is now.

#OccupyTheUSDOJ - Anonymous

 

Borreliosis Basics – Version 3

Borreliosis is a “multi-system disease” caused by a spirochetal parasite. A spirochete is a spiral-shaped parasite with a unique mechanism for movement that features an internal bundle of flagella (think of it as an internal bungie). Borrelia and Treponema (close cousins) have never been known to be curable with antibiotics “or even arsenic” (Jay Sanford, U.S. Uniformed Services Hospital, 1975, and Andrew Balfour, U.K. Wellcome Research, BMJ, April 1911). They typically reside in the brain/nervous system and are found in the brains of Alzheimer’s victims at nearly a universal rate as compared to controls or non-Alzheimer’s victims. (Riviere, Oregon Health and Sciences University, 2002).

Borreliosis is transmitted primarily through the bite of an infected tick, but also may be transmitted by insect vectors and transplacentally to an unborn fetus, according to Yale and the NIH (Paul H. Duray). If not treated immediately with antibiotics, the infection can persist for years and cause neurological diseases such as MS, Lupus, cancer, Chronic Fatigue/Myalgic Encephalomyelitis, ALS (Lou Gehrig's Disease), Cancer, and Alzheimer’s, according to IDSA and the CDC. Thus, borreliosis is commonly referred to as “The Great Imitator” or “New Great Imitator.” As you will later see, it would be more correct to call Lyme and Syphilis, the Great Detonators.

Borreliae parasites have the ability to shed (bleb off) their outer membrane lipoproteins to both evade detection by –, and disable- the immune system. They modify their numerous different surface proteins in a mechanism commonly known as antigenic variation. These lipoproteins are called “outer surface proteins” (Osps, rhymes with wasps) and “variable major proteins” (Vmps, rhymes with wimps). The blebbing, or shedding, of OspA and similar antigens by the spirochete sends the immune system after the sloughed-off or pinched off antigens like OspA (much like flak confuses radar), allowing the spirochete to hide from immune attack and persist in the host (Barbour and Barthold, The Scientist, 1996). This mechanism renders antibodies useless (note: no vaccines can therefore be made against these surface antigens).

These outer surface lipoproteins, such as OspA (the Lyme "vaccine"), are TLR-2 agonists or fungal antigens, meaning they essentially disable the immune system. They are very similar to toxins in this regard (see post-sepsis or post-septic shock survivors, Washington University, St, Louis, 2014). They do this by way of tolerance to other fungal antigens (TLR2s) as well as tolerance to other antigen types, managed by other TLRs, such as viruses (TLR7/9) and other bacterial TLR4-agonists (“cross-tolerance”). [(Clifford Harding, Case Western Reserve): "Because IRAK1 is required for TLR7/9-induced IFN-I production, we propose that TLR2 signaling induces rapid depletion of IRAK1, which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2 inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes that express ligands for both TLR2 and TLR7/TLR9, or responses to bacteria/virus coinfection." http://www.ncbi.nlm.nih.gov/pubmed/22227568 ]

Tolerance means the immune system will stop responding to the antigen. Cross tolerance means spreading or expanding normal disease-combating insufficiency to other antigen types such as TLR4 agonists or TLR7/9 agonists (viruses). Exposure to shed borrelial lipoproteins causes fungal tolerance in the blood and the inability to get rid of mycoplasma/eperythrozoons (also bearers of fungal antigens like OspA) from the blood, especially from the red blood cells. It is well known that fungi infections in the blood, particularly these blood parasites, the eperythrozoa or mycoplasma, causes fatigue, because they, by attaching to the surfaces of- and within- red blood cells hijack sugar metabolism and change the osmotic potential of the red cell membrane, rendering Oxygen unable to cross the red cell membrane, starving the body cells of Oxygen.

This generalized immunosuppression as a result of exposure to borreliae and their shed varying surface antigens creates an environment where common opportunistic infections are reactivated, such as latent Epstein-Barr, and cause chronic disabling disease. We know Lupus and MS are generally accepted as being caused by the herpesviruses (Epstein-Barr, HHV-6, or Cytomegalovirus, etc., or some combination of them). We have called chronic Lyme and LYMErix Disease: Tick-, or Lyme-, or LYMErix-Acquired Immune Deficiency (TAIDS or LAIDS).

Since we knew Lyme was formerly called the New Great Imitator because it caused Lupus, MS, etc, it now appears that the TLR2-agonists shed by these spirochetes cause the tolerance and cross-tolerance that reactivates latent herpes viruses, particularly Epstein Barr Virus (EBV). This came to light because the OspA vaccines caused the same systemic, “multi-system” (Dave Persing, Mayo), “protean” (Ben Luft, SUNY-SB) disease in those victims. Reactivation occurs thru IL-10, the inhibition of apoptosis of OspA or EBV infected cells, the mechanism shown above re TLR2-signaling and the inhibition of IFN-I, and apparently generalized immune activation or common cytokines. One group of researchers simply reported:
“Certain cytokines, particularly TGF -beta, can also induce lytic viral infection in a subset of Burkitt lymphoma lines in vitro, and could potentially reactivate EBV in vivo (Fahmi et al., 2000; Adler et al., 2002). The interaction between CD 4 T cells and EBV -infected B cells has also been reported to induce lytic infection (Fu and Cannon, 2000). Finally, there is increasing evidence that severe host cell stress in response to many different toxic stimuli (including chemotherapy and irradiation) can induce lytic EBV infection (Feng et al.,2002a, 2004; Westphal et al., 2000; Roychowdhury et al., 2003).” – Kenney, 2007, from the book, Human Herpesviruses, Biology, Therapy, Immunoprophylaxis, chp 25.

Recently, it was reported in the New York Times by Jane Brody quoting the National Institutes of Health’s Adrianna Marques, who now manages the Lyme-MS Division of NINDS (her boss, Roland Martin, quit the NIH and returned home to Germany once he found out OspA was responsible for the MS outcome of Lyme), "Complicating the picture is the fact that some people with PTLDS symptoms apparently never had Lyme disease in the first place, Dr. Marques said in an interview. There are other infectious organisms — Epstein-Barr virus, for example — that can produce similar symptoms and may be the real culprits." - When Lyme Disease Lasts and Lasts, July, 2013.

Chronic EBV is probably the main driver of all these New Great and Great Imitator diseases. Inasmuch as the Infectious Diseases Society of America, Yale, the CDC and related participants in the OspA vaccine fiasco vehemently oppose antibiotic treatment for Lyme for whatever [false] reason, the truth is that they worry antibiotic resistance will be acquired by the OTHER infections such as the fungal types, such as mycobacteria and mycoplasma for which we now have become carriers.

--- USDOJ OCCUPY, All Abused Groups, Summer, 2014

Feel free to distribute the above 2 posters.  There were many contributors so we all agreed to attribute to the OCCUPY the USDOJ group.

Mid-October, 2014

Lyme Cryme Update:  We're getting a lot more traffic to the OCCUPY JUSTICE Facebook group from all over, including Europe and that means we need help from the OspA-Disease-Literate among us,... and that does not include anyone from ILADS.org to my knowledge. So let's review the overview and the State of the Science here, such that people can see that there is no available treatment for this yet, unless you got an MS or CLL diagnosis (or use the wustl.edu words, "post-sepsis immunosuppression"):

This information is even in the mainstream USA university news (See # 11 in Section 3 of the Khan Academy of Lyme Cryme) so that's progress for America, but not at the practice level; as I have been saying for years, Lyme victims should try to get an MS diagnosis:

Get the full text report  (wustl.edu, "post-sepsis") - and READ IT before you proceed, here - and see the Related articles linked from this page:
http://www.ncbi.nlm.nih.gov/pubmed/24919177

As far as "autoimmunity" in Chronic Fatigue Syndrome, no, that is the incorrect term, or else you would have lots of antibodies, and these abused and neglected diseases are famous for their no-antibodies.  See near the top of 101016.htm to discover you might not have (higher) antibodies against the herpes viruses while they're still reactivated:
"Benefit of B cell depletion in CFIDS"
http://www.ncbi.nlm.nih.gov/pubmed/22039471

There is no way around this, you, the patient, are going to have to try to read these articles.  I can't digest it simpler for you;  This is America - no one can help you:
http://www.ncbi.nlm.nih.gov/pubmed/?term=alemtuzumab+and+valganciclovir

Something like Alemtuzumab or Rituximab and anti-virals together may help you, but as shown in # 18 of the "20 Reports" (below and section 3 of the Kahn Academy of Lyme Cryme), only the Chinese are trying to look at how to reverse TLR2-agonist (Lyme fungal antigens) tolerance are looking at how to reverse this:

(A20 is critical for the induction of Pam3CSK4-tolerance in monocytic THP-1 cells.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3905037/  )

This is the State of the Science and I don't know what the treatment is for post-Lyme Sepsis, I am just an analytical chemist.  I can only re-report to you what the science says.  The cryme has to be prosecuted in order for treatments like these to open up.

---

2015 is going to be very different for the CDC, the incompetent "MDs" who believe the CDC's malarkey without true analysis (the MMR "data" was even worse than the LYMErix "data"), and for their victims (Gulf War Illness, Autism Parents, Psych Rights, Parents Rights, ME/CFIDS, Fibro, Lyme etc). 

Not only are we having an All Groups OCCUPY JUSTICE next Spring, we're going to create a Competency Test that if your MD can't pass, not only are you not paying them, you're going to charge THEM for wasting your time, and you're going to report them to their medical boards.  Scroll down past the following data on the status of Lyme cryme prosecution regarding the July 31, 2014 reply to the Senators by the FDA (you can now report your adverse event to the CDC's falsified "Lyme Disease" "case definition" to the FDA's Adverse Events section re Medical Testing).

There is some pretty straightforward data that has come out recently about shed Borrelial fungal antigens of the Pam3Cys type and how they cause the typical post-sepsis reactivation of latent herpes viruses from UConn, if you can believe it, - but then again, they recruited the Russian scientist Medvedev from Duke.  Scroll down to the "20+ Reports."  This data will be the basis of the Competency Test we create for your "MD."

You can also join us on the OCCUPY JUSTICE page for all groups:
USDOJ Protest for All Abused Groups(Autism,GWI,CFIDS,Lyme,Parents,Psych,..)
https://www.facebook.com/#!/groups/271420453060565/

https://www.facebook.com/#!/notes/kathleen-dickson/20-reports-show-autism-lyme-ospa-and-fungal-immunosuppression-competency-trainin/822596964439192

Note re Vaccines and Autism - it's finally catching on...

"The “deactivated” viruses of the measles, mumps and rubella (MMR) shot have also, it has been claimed, to cause autism spectrum disorders, too."

Read more: http://www.law.com/sites/jamesgrundvig/2014/09/22/whistleblowers-dent-the-deep-state-of-the-u-s-vaccine-program/#ixzz3EKuqQ36y
 

You will see that the CDC knows the kids are getting the viruses instead of the "protection."

 

= = = = =

Lyme Cryme Prosecution Status, September 2014:

Recall that LYMErix was ordered off the market by the FDA because it was never proven to be a vaccine, ... because the current "Two-Tiered" Dearborn case definition was falsified, not to mention, not even a consensus.  The LYMErix vaccine was a fungal antigen of the Pam3Cys type and as such was an immune-suppressing TLR2/1 agonist and caused systemic "protean" disease just like chronic Lyme (probably because it activates herpesviruses as is now a well known outcome in sepsis).  ILADS.org and the LymeDisease.org have no idea what they are talking about, or treating, and have said they do not care to know or do anything about the falsified case definition or why exposure to fungal antigens like shed borrelial lipoproteins cause immunosuppression and the activation of latent herpesviruses.

As a result, ILADS has come under attack by the IDSA/Yale/CDC Lyme crooks (see The Daily Beast, again) and can't defend themselves, whereas they do not attack ActionLyme and associates since we are simply repeating what the NIH says about Lyme being a disease of immunosuppression and reactivated herpesviruses, LMAO.  The United States cannot produce a single science journalist, so no one picks up this whole hilarious series of events and runs with it straight to a Pulitzer or at least Comedy Central.

When Senator Richard Blumenthal sued the IDSociety.org Lyme criminals for "Anti-Trust," yet his staff lawyers in CT when he was Attorney General referred me to the USDOJ to file the RICO and FRAUD complaint, specifically because they found out about the falsified "case definition" (which we call "Dearborn"), why didn't Blumenthal sue for FRAUD, too?

Because the State of CT Department of Public Health and the University of Connecticut participated in the vaccines crimes with Yale et al (ALDF.com).  The State of CT could be sued by the other 49 States.  One State of CT employee was actually listed as an inventor on the ImmuLyme OspA vaccine, which meant the State of CT would get some Royalties, not to mention furthering their monopoly on grants and testing for Vector Borne Diseases in cahoots with Yale.

Now, many of you will remember that a one CDC officer Mark Klempner got a 4.7 million dollar grant to "re-treat" Lyme disease with 30 extra days of intravenous ceftriaxone, when in fact, Klempner's study was based on the Dearborn case definition (fraud, therefore rendering the "study" also a fraud and recoverable in damages by the USDOJ times 3).  One of the biggest problems with this "study" in the first place was that a one Phyllis Mervine of the antagonistic "Lymedisease.org" or "lymediseaseassociation.org" was chosen with another person to be sort of the patient "reviewer" of the "study" "protocol."  So, they idiotically approved this waste of 4.7 million dollars.

Neither was qualified, nor did they know that the Dearborn case definition was research fraud (I didn't either, I only got ahold of the Dearborn booklet in the summer of 1999), but still to this day, ILADS.org and the Lymedisease.org/LDA absolutely refuse to discuss the Dearborn falsified case definition and how the OspA vaccines caused systemic disease via immumnosuppression.

This is while Senator Blumenthal is now going after the FDA via the OPM, by FORCING the FDA to look into the falsified Dearborn cased definition as you see here, in this very latest response by the FDA to the furious Senators:
This is from the new announcement regarding the FDA now about to ENFORCE their validation rules:
http://www.fda.gov/downloads/MedicalDevices/ProductsandMedicalProcedures/InVitroDiagnostics/ucm407409.pdf


 

And now that the Senators are trying to overturn the falsified Dearborn "case definition" by forcing the FDA to finally do their jobs, the LDA is running around hysterically trying to promote their "Lyme bill," which AGAIN will stall and obstruct the impending prosecution.

No Dearborn - No IDSA "Guidelines," People.  No Klempner.  The IDSA Guidelines are based on Klempner's bogus research fraud "retreatment study" that was invalid because it used invalid assessment tools, did not retreat 2/3rds of the patients, and was based on the fraudulent Dearborn "case definition" that Blumenthal and the Senators are trying to overturn.

We don't need no "Lyme Bills."  The Senators are forcing the FDA to retract Dearborn and hopefully prosecute it or send it to the USDOJ to prosecute.

---

Uncle Henry's "Exceptional" America, LMAO: 

Don't ya mean SPECIAL ED America, Henry?  IBM now has to reprogram Watson with this same TERRORIST!!! information about how OspA causes immunosuppression and activates latent viruses, explaining also the Autism pandemic (see below in the very next data-set).

 

20+ REPORTS for Doctor Competency Evaluations - Show OspA and the 2 kinds of Autism are linked (and not spirochetes):
 

Raymond Dattwyler in his European patent for an inhalation form of OspA says OspA is Pam3Cys and a TLR2-agonist (everyone else like NIH, CDC, even Yale say they don't know what OspA is, but ILADS and the LDA would not even ask, duh, "Cause Known"); it means OspA could never have been a human injectable vaccine because it is fungal and causes immunosuppression as you will see:

" A lipidation/processing reaction has been described for the intact OspA gene of B. burgdorferi. The p...rimary translation product of the full-length B. burgdorferi OspA gene contains a hydrophobic N-terminal sequence, of 16 amino acids, which is a substrate for the attachment of a diacyl glyceryl to the sulflhydryl side chain of the adjacent cysteine (Cys) residue (at position 17). Following this attachment, cleavage by signal peptidase II and the attachment of a third fatty acid to the N-terminus occurs. The completed lipid moiety, a tripalmitoyl-S-glycerylcysteine modification, is termed Pam3Cys (or is sometimes referred to herein as Pam(3)Cys or Pam3Cys). It has been suggested that the lipid modification allows membrane localization of proteins, with polypeptide portions exposed as immune targets. In addition to serving as targets for the immune response, Pam3Cys-modified proteins, such as OspA, have been reported to act as potent inflammatory stimulants though the toll-like 2 receptor mechanism (TLR2).

http://patentscope.wipo.int/search/en/detail.jsf?docId=US42934470&recNum=9&maxRec=30&office=&prevFilter=&sortOption=Pub+Date+Desc&queryString=tripalmitoyl+
cysteine+or+Pam3Cys+and+Epstein-Barr&tab=NationalBibliohttp://patentscope.wipo.int/search/en/detail.jsf?docId=US42934470&recNum=9&maxRec=30&office=&prevFilter=&sortOption=Pub+Date+Desc&queryString=tripalmitoyl+
cysteine+or+Pam3Cys+and+Epstein-Barr&tab=NationalBiblio


All data here was also moved to the "Khan Academy" of Lyme

The 20+ REPORTS that show OspA/fungal antigens act like BCL2 class genes and are the causes of the 2 kinds of Autism (and cancer, MS, ALS, etc);

History: Use the following page and search for the term apoptosis, http://www.actionlyme.org/101016.htm  to see how well known it is that fungal antigens inhibit apoptosis. Additionally, go to PubMed and search for P38 and apoptosis and p53 and apoptosis and find that these are the apoptosis enzymes whose functions are inhibited by bacterial lipoproteins like OspA (re the report here, on reprogamming Watson, below).

One kind of Autism is ***Genetic [reverse duplication of a BCL2-class gene, resulting in inhibition of apoptosis and resultant inhibition of "normal synaptic pruning" (see Einstein, Grandin, Tesla)],*** … and the other, the Brain-Damage kind, results from contaminated vaccines (with mycoplasma/fungi), and the subsequent immunosuppression and reactivation of the live, attenuated viruses in the vaccine vial. Both are related to LYMErix (fungal / acts like BCL2, inhibiting apoptosis), or shed Lyme/spirochetal fungal antigens and not spirochetes, per say.

Herpesviruses also have a human BCL2 analog, and that is the main cause of leukemias, including childhood leukemias. BCL means B Cell Lymphoma (Clue).
 

1) MARCH 2002: Organisation of the pericentromeric region of chromosome 15: at least four partial gene copies are amplified in patients with a proximal duplication of 15q.

"We identified a fourth pseudogene, BCL8A [a BCL-2 class molecule, which inhibits apoptosis is reverse-duplicated and therefore overexpressed- KMD], which maps to the pericentromeric region and is coamplified along with the NF1 sequences. Interphase FISH ordering experiments show that IgH D lies closest to the centromere, while BCL8A is the most distal locus in this pseudogene array;"   http://www.ncbi.nlm.nih.gov/pubmed/11897815 

Reversed duplications imply the anti-apoptosis gene is probably over-expressed. The New York Times recently (summer, 2014) ran an article about another mechanism of inhibition of apoptosis leading to the genetic kind of autism, as if that was news, given what we know about the co-occurance of Einstein kind of Autism in Neurofibromatosis. This connection was written up in all the older books on “psychiatry,” duh.

Compare to:
http://www.nytimes.com/2014/08/22/health/brains-of-autistic-children-have-too-many-synapses-study-suggests.html?_r=0

Duh.

============================

2) – BigPharma: Patent, US # 7,632,510

Methods of inducing flavivirus immune responses through the administration of recombinant flaviviruses comprising an engineered japanese encephalitis virus signal sequence

"Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the vaccine may actually cause disease."

http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7%2C632%2C510.PN.&OS=PN%2F7%2C632%2C510&RS=PN%2F7%2C632%2C510 

===========================

3) CDC: Measles, Mumps, and Rubella -- Vaccine Use and Strategies for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and Control of Mumps: Recommendations of the Advisory Committee on Immunization Practices (ACIP)

"Updated information on adverse events and contraindications, particularly for persons with severe HIV infection, persons with a egg allergy or gelatin allergy, persons with a history of thrombocytopenia, and persons receiving steroid therapy [are immunosuppressed - KMD]." 
http://www.cdc.gov/mmwr/preview/mmwrhtml/00053391.htm 
 

Fauci just announced (Sep, 2014) huge new funding for adjuvants for vaccines for immunosuppressed children, duh, why would he do that if this was not a problem.

"NIH awards seven new vaccine adjuvant discovery contracts"
"The goal of this research is to identify novel adjuvant candidates that safely and selectively boost vaccine-induced immune responses,” said NIAID Director Anthony S. Fauci, M.D. “Such adjuvants could be used to improve current vaccines, extend the vaccine supply or enhance vaccine efficacy in people with immature or weakened immune systems, such as infants and the elderly.”
http://www.nih.gov/news/health/sep2014/niaid-29.htm

=========================

4) CDC: Human Exposure to Brucella abortus Strain RB51 -- Kansas, 1997

http://www.cdc.gov/mmwr/preview/mmwrhtml/00051495.htm 

In the above, an immunosuppressed pregnant cow was given a Brucella (“LYME”rix-like) "live attenuated" vaccine and the baby cow ended up with the disease, which then was transferred to the humans handling the cow and her dead baby. This parallels what is happening to children who are vaccinated while immunosuppressed, or who receive mycoplasmally (“LYME”rix-like) contaminated vaccines.

=========================

5) In the case of pandemic MRSA, as we have seen, the vaccine didn't work because TLR2 agonists (lipoproteins) suppress the immune system. We also learned from the MRSA vaccine patent, that (US patent 7,771,728)

Method for identification, isolation and production of antigens to a specific pathogen

"Several established vaccines consist of live attenuated organisms where the risk of reversion to the virulent wild-type strain exists. In particular in immunocompromised hosts this can be a live threatening scenario. Alternatively, vaccines are administered as a combination of pathogen-derived antigens together with compounds that induce or enhance immune responses against these antigens (these compounds are commonly termed adjuvant), since these subunit vaccines on their own are generally not effective."

http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7%2C771%2C728.PN.&OS=PN%2F7%2C771%2C728&RS=PN%2F7%2C771%2C728

=======================

6) The effect of exogenous corticosterone on West Nile virus infection in Northern Cardinals (Cardinalis cardinalis)

“Corticosterone was administered at levels that individuals enduring chronic stressors (i.e ., long-term inclement weather, food shortage, anthropogenic pollution) might experience in the wild. Corticosterone greatly impacted mortality: half of the corticosterone-implanted cardinals died between five - 11 days post-inoculation whereas only one of nine sham-implanted (control) birds died.

”No differences were found in viral titer between corticosterone- and sham-implanted birds. However, cardinals that survived infections had significantly higher average body temperatures during peak infection than individuals that died.

”In sum, this study indicates that elevated corticosterone could affect the survival of WNV-infected wild birds, suggesting that populations may be disproportionately at-risk to disease in stressful environments.”

http://7thspace.com/headlines/410671/the_effect_of_exogenous_corticosterone_on_west_nile_virus_infection_in_northern_cardinals_cardinalis_cardinalis.html

The same is true for humans and cortisol and the activation of latent herpesviruses, therefore it’s not “psychiatric” or “psychosomatic,” it’s EBV due to cortisol, meaning real and not due to having the powers of a witch. (Later we add this data, in this project, and you can see it now in the next section.)

======================

7) NYTimes; Doctors admit Thimerosal is put in vaccines to prevent fungi:

Vaccine Rule Is Said to Hurt Health Efforts (Dec, 2012)

They say:

"But a proposal that the ban include thimerosal, which has been used since the 1930s to prevent bacterial and fungal contamination in multidose vials of vaccines, has drawn strong criticism from pediatricians.

"They say that the ethyl-mercury compound is critical for vaccine use in the developing world, where multidose vials are a mainstay.

"'Banning it would require switching to single-dose vials for vaccines, which would cost far more and require new networks of cold storage facilities and additional capacity for waste disposal, the authors of the articles said.'"

http://www.nytimes.com/2012/12/17/health/experts-say-thimerosal-ban-would-imperil-global-health-efforts.html?_r=2& 

^^ They are revealing why so few doctors' own kids get vaccines-acquired brain damage. Their kids probably get the first shot out of the vial.

====================

8) Fungi Activate Viruses, Rockefeller, 1953 [Go back to the 1950s to see Rockefeller Bioweapons Inc experimenting with mycoplasma antigens like OspA - Enhancing Mouse Leukemia Virus, just like fungally-contaminated vaccines do, which was the reason they put Thimerosal in vaccines, to prevent fungi from doing this (Clue: HIV and "Lyme" bearing the same immunosuppressing triacyl lipopeptide fungal antigen, OspA]:

ACUTE HEPATITIS ASSOCIATED WITH MOUSE LEUKEMIA

IV. THE RELATIONSHIP OF EPERYTHROZOON COCCOIDES TO THE HEPATITIS VIRUS OF PRINCETON MICE

"In Swiss mice, animals with high natural resistance to hepatitis virus, the pathogenicity of this agent was markedly enhanced by combined infection with eperythrozoa. Eperythrozoa were maintained throughout 18 successive passages in normal Princeton and Swiss weanlings with intact spleens. The combined infection of Princeton mice with eperythrozoa and the virus component of Gledhill, Dick, and Andrewes, which is nearly inactive when injected alone, resulted in acute hepatitis with fatal outcome."

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2136329/?tool=pubmed 

====================

9) OspA acts like BCL2, inhibiting apoptosis reactivating EBV/Similars:

Targeting proteins computationally

"Apoptosis is regulated by both pro- and anti-apoptotic members of the B cell lymphoma-2 (Bcl-2) family of proteins. Epstein Barr virus (EBV), the causative agent of Burkitt’s lymphoma, encodes a pro-survival mimic of these Bcl-2 proteins called BHRF1 that inhibits cellular apoptosis during infection and is thought to contribute to lymphomagenesis."
http://www.fredhutch.org/en/news/spotlight/imports/targeting-proteins-computationally.html 

=======================

10) This is the NIH (NINDS’s MS-Lyme Group) group that discovered that *** OspA *** was the cause of the MS/New Great Imitator outcome of Lyme reporting in the New York Times in the summer of 2013 (Martin and Marques, 2006):

When Lyme Disease Lasts and Lasts – Jane Brody

"Complicating the picture is the fact that some people with PTLDS symptoms apparently never had Lyme disease in the first place, Dr. Marques said in an interview. There are other infectious organisms — Epstein-Barr virus, for example — that can produce similar symptoms and may be the real culprits."

http://well.blogs.nytimes.com/2013/07/08/when-lyme-disease-lasts-and-lasts/ 

Here are the NIH's 2 reports that say OspA (TLR2-agonist) is the cause of the MS/CFIDS/EBV-reactivated kind of Lyme (that also causes humoral immunosuppression),... and that as a result of exposure to OspA-like antigens (shed constantly in a process called blebbing, as revealed by CDC officer Alan "Stealth Bomber" Barbour), you might not even have anti-flagellar antibodies (TLR5-agonists):

Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression.
http://www.ncbi.nlm.nih.gov/pubmed/16783164 

and

Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes.
http://www.ncbi.nlm.nih.gov/pubmed/16479520 

=======================

11) Wustl.edu discover (?- wow, thanks) that sepsis is lyke Lyme, in that the survivors of it are likely to have survived via the immunosuppression (TLR2-agonist tolerance/Endotoxin tolerance), but the result is the reactivation of latent viruses:

Dormant viruses re-emerge in patients with lingering sepsis, signaling immune suppression

"Patients with lingering sepsis had markedly higher levels of viruses detectable in the blood, compared with the healthy controls and critically ill patients without sepsis. Among the sepsis patients, for example, the researchers found that 53 percent had Epstein-Barr virus, 24 percent had cytomegalovirus, 14 percent had herpes-simplex virus, and 10 percent had human herpes simplex virus-7.

"These viruses generally don’t lead to significant illness in people who are healthy but can cause problems in patients who are immune-suppressed. "

http://news.wustl.edu/news/Pages/27015.aspx 

FULL JOURNAL REPORT, snippet…

Reactivation of Multiple Viruses in Patients with Sepsis

“Sepsis is the host's non-resolving inflammatory response to infection that leads to organ dysfunction [1], [2]. A current controversial hypothesis postulates that if sepsis pursues a protracted course, it progresses from an initial primarily hyper-inflammatory phase to a predominantly immunosuppressive state [3]–[7]. Experimental therapeutic approaches in sepsis have almost exclusively focused on blocking early inflammation or host-pathogen interaction and failed [8]–[10]. Recently, immuno-adjuvant therapies that boost host immunity, e.g., GM-CSF and interferon-γ, have been successful in small clinical trials thereby supporting the concept that reversing immunosuppression in sepsis is a plausible strategy to improve outcome [11], [12]. However, several issues have limited this approach including lack of consensus that immunosuppression is a clinically important phenomenon [5], [6], [13]. Also, difficulty in identifying patients with impaired immunity as well as determining optimal timing for administration pose significant challenges to pursuing this approach [14]. While immuno-adjuvant therapies might improve sepsis survival if administered during the later immunosuppressive phase, these agents might worsen outcome if given during the early hyper-inflammatory phase [4], [14]. Thus, a means to distinguish these two contrasting phases of sepsis is needed not only to verify the hypothesis that sepsis progresses to an immunosuppressive state but also to guide use of potential agents which boost immunity.

“Latent viruses such as cytomegalovirus are normally held in abeyance by cellular and immune surveillance mechanisms which if impaired, for example by immunosuppressive medications, often result in viral reactivation, replication, and virally-mediated tissue injury [15]–[20]. Sepsis impairs innate and adaptive immunity by multiple mechanisms including apoptosis-induced depletion of immune effector cells and induction of T-cell exhaustion thereby possibly predisposing to viral reactivation and dissemination [21]–[23]. …”
http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0098819 

========================

12) NCI and US Army Ft Detrick Pathologist Paul Duray on the CSF cells looking like "Epstein-Barr-like transformed cells" in IDSA's 1989 Reviews Supplement on Spirochetal Diseases:

Rev Infect Dis. 1989 Sep-Oct;11 Suppl 6:S1487-93.
Clinical pathologic correlations of Lyme disease.
"Immature B cells can also be seen in the spinal fluid. These cells can appear quite atypical- not unlike those of transformed or neoplastic lymphocytes." --
http://www.ncbi.nlm.nih.gov/pubmed/2814170 
Full Text:  http://www.actionlyme.org/IDSA_CLINIPATH_DURAY.htm 

NEW, by the NIH:  "Surviving Sepsis: Detection and Treatment Advances"

By Carolyn Beans for the National Institutes of Health   |   August 18, 2014 08:43am ET
http://www.livescience.com/47387-sepsis-diagnosis-treatment-research-nigms.html

"Preventing Secondary Infections

"Some people who survive sepsis can develop secondary infections days or even months later. A research team that included Richard Hotchkiss, Jonathan Green and Gregory Storch of Washington University School of Medicine in St. Louis suspected that this is because sepsis might cause lasting damage to the immune system. To test this hypothesis, the scientists compared viral activation in people with sepsis, other critically ill people and healthy individuals. The researchers looked for viruses like Epstein-Barr and herpes simplex that are often dormant in healthy people but can reactivate in those with suppressed immune systems. [Sepsis Has Long-Term Impact for Older Adults, Study Finds]"

========================

13) Duray again in 1992, in Steve Schutzer's review of the 1992 Cold Spring Harbor Conference on Lyme:

"On occasion, these atypical-appearing large lymphocytes have been misinterpreted in biopsy by several laboratories as cells of a malignant lymphoma or leukemia. Bb antigens, then, may stimulate growth of immature lymphocytic suibsets in some target organs, as well as in the cerebrospinal fluid (Szyfelbein and Ross 1988). Usual bacterial infections do not produce such lymphocytic infiltrates in tissue. ****These immunoblastoid cells in Bb infections at times resemble those found in Epstein-Barr virus infections.**** Does Bb reactivate latent virus infections in tissues? Do some tick inocula harbor simultaneous infectious agents (ixodid ticks can harbor Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing multi-agent infections in some hosts? Further studies can clarify these issues by mans of tissue-based molecular probe analysis." -

Paul Duray, NCI, NIH, Ft. Detrick, at the 1992 Cold Spring Harbor Crooks' Conference, published in Steve Schutzer's Lyme Disease: Molecular and Immunologic Approaches. - book.

=======================

14) Anthony Fauci on how the LYMErix vaccine (Pam3Cys) was apparently (?) used as an HIV vaccine failed in the same way LYMErix failed, causing immunosuppression and greater susceptibility to disease (HIV's gp120 is Pam3Cys in triplicate at least in the vaccine attempt, apparently - DeFoort):

An HIV Vaccine — Challenges and Prospects

‎"The initial empirical approach of immunizing with VaxGen's AIDSVax, a recombinant form of the outer glycoprotein-120 (gp120) portion of the HIV envelope, which was based on a strategy that was successful with hepatitis B, failed to protect volunteers from infection, apparently because the vaccine did not induce broadly neutralizing antibodies.3"

"Unexpectedly, post hoc analyses of the STEP trial also found a trend toward a greater number of new infections among vaccine recipients than among placebo recipients."

http://www.nejm.org/doi/full/10.1056/NEJMp0806162#t=article 

[DEFOORT Pam3Cys Quadrivalent OspA-HIV, see]

AND, ODDLY…

Distinction between HIV-1 and HIV-2 infection using novel synthetic lipopeptide conjugates as antigens in enzyme immunoassays.

Böltz T1, Hummel RP, Tröger W, Rübsamen-Waigmann H, Biesert L, Müller-Lantzsch N, Koch P, Bessler W, Jung G.

"A novel immunoassay technique using synthetic lipopeptide (Pam3Cys-Ser) linked to immunodominant peptide domains of HIV-1 and HIV-2 envelope proteins as an antigen adsorbent has been developed. Attachment of peptides to microtiter plates can be considerably improved with this method by employing the hydrophobic properties of lipopeptide. From the sera of 121 HIV-1 infected patients 117 reacted with Pam3Cys-Ser-[HIV-1(598-609)cyclic disulfide]. Five of 5 HIV-2 positive sera were positive with Pam3Cys-Ser-[HIV-2(593-603)cyclic disulfide]. Control sera failed to react with these conjugates.
http://www.ncbi.nlm.nih.gov/pubmed/?term=2464607 
 

So, what does that mean? They made an HIV vaccine out of the same structure as the fungal OspA- LYMErix?,... and did not know this would not work because Yale lied to the FDA and the journals about the outcome of LYMErix? How did these patients test positive to the new Pam3Cys antigen while everyone with Lyme does not, especially later in the disease.  Were these HIV patients all NEW HIV patients?  No way to know at present, since there is no available structure of HIV's gp120 that we can find.

The Koreans found Pam3Cys sticks to itself? >> http://newjournal.kcsnet.or.kr/main/j_search/j_download.htm?code=B961118 

That OspA sticks to itself explains why the Western Blots in LYMErix-and ImmuLyme-vaccinated people had unreadable Western Blots with the Blot-Smudging?

PERSING WITH SIGAL EXPLAINING THAT THE WESTERN BLOTS WERE UNREADABLE due to the blot smudging (probably due to OspA sticking to itself, which Alan Barbour reported a long time ago, yet he is the owner of the ImmuLyme OspA vaccine patent), 2000:
Detection of Multiple Reactive Protein Species by Immunoblotting after Recombinant Outer Surface Protein A Lyme Disease Vaccination
http://www.journals.uchicago.edu/doi/pdf/10.1086/313920 
 

Anthony Fauci's patent for the treatment of immune suppression outcomes of bacterial, viral, and fungal infections:
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=5,696,079.PN.&OS=PN/5,696,079&RS=PN/5,696,079

"FIELD OF THE INVENTION

"The present invention pertains to a method for activating the immune system of a patient by intermittently administering interleukin-2 (IL-2) to that patient. Such administration of IL-2 can optionally be combined with other therapies, such as anti-retroviral, anti-bacterial or anti-fungal therapies, suitable for treatment of the patient's condition. This invention also relates to an approach to gene therapy that entails administering IL-2 to a patient so as to facilitate in situ lymphocyte transduction by a retroviral vector also administered to the patient.

"BACKGROUND OF THE INVENTION

"....Illustrative of specific disease states in treatment of which the present invention can be applied are HIV infection and other diseases characterized by a decrease of T-cell immunity, for example, mycobacterial infections like tuberculosis and fungal infections such as cryptococcal disease. This method also can be used in the treatment of secondary infections that occur in patients with suppressed immune systems, such as the opportunistic infections that occur in AIDS patients. ..."
 

You can't make this stuff up.

=======================

15) EVERYONE READ THIS, AND THE RELATED ARTICLES ON THE PLOS WEBSITE LINKED ON THE RIGHT in this article; THIS IS LYMERIX-DISEASE (TLR2-agonist, or bacterial lipoprotein and NOT LPS) OR POST-LYME SEPSIS,

MicrRNA-146a Is Upregulated by and Negatively Regulates TLR2 Signalling

"Pre-exposure of monocytes/macrophages (in vitro) or the host (in vivo) to LPS or endotoxin induces a transient state of cellular hyporesponsiveness to a secondary LPS challenge with diminished production of proinflammatory cytokines, thereby conferring protection against LPS-induced lethality and resulting in a significant survival advantage [8]–[10]. This phenomenon is well established and termed endotoxin tolerance. Furthermore, emerging evidence has revealed that modulation of the TLR4-mediated signal transduction pathway is involved in the development of endotoxin tolerance [9], [10]. For example, down-regulated cell-surface expression of TLR4, reduced interleukin-1 receptor-associated kinase 1 (IRAK-1) expression and myeloid differentiation factor 88 (MyD88)-IRAK immunocomplex formation, and attenuated nuclear factor (NF)-κB activation and mitogen-activated protein kinase (MAPK) phosphorylation are all considered as significant markers for endotoxin tolerance. Therefore, endotoxin tolerance may represent a protective mechanism, whose primary function is to prevent excessive inflammatory response induced by overactivation of the TLR4 signaling pathway. It has been shown that endotoxin tolerance occurs in several disease settings including sepsis, trauma, surgery and pancreatitis [9]; however, acquisition of endotoxin tolerance may contribute to an increased incidence of secondary bacterial infection in hospitalized patients due to development of an immunesuppressed state [9], [10]."

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3639252/ 

=============================

16) THIS IS UCONN's Medvedev (fiormerly with Duke?) talking about Immunosuppression from Post-Lyme and Post-LYMErix Sepsis (TLR2-agonists or Pam3Cys is OspA or shed borrelial antigens)

IRAK4 kinase activity is not required for induction of endotoxin tolerance but contributes to TLR2-mediated tolerance

"Development of endotoxin tolerance following the initial “cytokine storm” phase of sepsis is thought to protect the host from an overexuberant immune response and tissue damage but at the same time, may render the host immunocompromised and more susceptible to secondary infection [18,–20]. ...

"Reprogramming [21] of TLR4 signaling in endotoxin-tolerant monocytes and macrophages does not occur as a result of decreased TLR4 expression but involves altered recruitment, tyrosine phosphorylation, and K63-linked polyubiquitination of proximal receptor-adapter-kinase complexes [22,–27] and induction of negative regulators IRAK-M, SHIP1, and A20 [24, 25, 28]. Although a few studies have sought to dissociate kinase and adapter functions of IRAK4 in IL-1R/TLR signaling, albeit with conflicting results [13,–16, 29,–31], it is unclear how IRAK4 kinase activity affects induction of TLR2 and TLR4 homo- and heterotolerance. To address these questions, we used IRAK4KDKI mice to determine the impact of kinase deficiency of IRAK4 on the induction of TLR tolerance. Our data showed comparable induction of endotoxin tolerance in WT or IRAK4KDKI PMs and BMDMs, as judged by attenuated MAPK phosphorylation, inhibited expression of proinflammatory cytokines and chemokines, and up-regulation of negative TLR regulators, A20 and IRAK-M. Notably, IRAK4 kinase activity was found to be a prerequisite for conferring inhibition of LPS-inducible JNK and p38 MAPK activation following prior exposure to Pam3Cys. These results represent the first systematic analyses of the role of IRAK4 kinase activity in TLR homo- and heterotolerance and pave the way for improved understanding of how IRAK4 kinase dysregulation may underlie immunocompromised states in late sepsis."

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3714565/ 

====================

17) RE-PROGRAMMING WATSON - with the OspA data, LOL; This is funny. p53 is the apoptosis generator/enzyme/kinase, that is inhibited in the first step of nearly all disease, and the very thing I am screaming about for what 10 years? OspA stops the normal activity of p53/p38, LOL. Then read this and the lead-in into it. Yer gonna laff:

IBM Watson Ushers in a New Era of Data-Driven Discoveries

"In a retrospective, peer reviewed study released this week by Baylor College of Medicine and IBM, scientists demonstrated a possible new path for generating scientific questions that may be helpful in the long term development of new, effective treatments for disease. In a matter of weeks, biologists and data scientists using the Baylor Knowledge Integration Toolkit (KnIT), based on Watson technology, accurately identified proteins that modify p53, an important protein related to many cancers, which can eventually lead to better efficacy of drugs and other treatments. A feat that would have taken researchers years to accomplish without Watson's cognitive capabilities, Watson analyzed 70,000 scientific articles on p53 to predict proteins that turn on or off p53's activity. This automated analysis led the Baylor cancer researchers to identify six potential proteins to target for new research. These results are notable, considering that over the last 30 years, scientists averaged one similar target protein discovery per year.

"On average, a scientist might read between one and five research papers on a good day," said Dr. Olivier Lichtarge, the principal investigator and professor of molecular and human genetics, biochemistry and molecular biology at Baylor College of Medicine. "To put this in perspective with p53, there are over 70,000 papers published on this protein. Even if I'm reading five papers a day, it could take me nearly 38 years to completely understand all of the research already available today on this protein. Watson has demonstrated the potential to accelerate the rate and the quality of breakthrough discoveries."

http://www.stockhouse.com/news/press-releases/2014/08/28/ibm-watson-ushers-in-a-new-era-of-data-driven-discoveries#F16SfxTHczjPCGJ4.99 

=========================

18) This is a Chinese group trying to figger out how to reverse the well-known phenomenon of OspA-, fungal-antigen- induced immunosuppression or OspA-induced fungal antigen tolerance

A20 is critical for the induction of Pam3CSK4-tolerance in monocytic THP-1 cells.

"A20 functions to terminate Toll-like receptor (TLR)-induced immune response, and play important roles in the induction of lipopolysacchride (LPS)-tolerance. However, the molecular mechanism for Pam3CSK4-tolerance is uncertain. Here we report that TLR1/2 ligand Pam3CSK4 induced tolerance in monocytic THP-1 cells. The pre-treatment of THP-1 cells with Pam3CSK4 down-regulated the induction of pro-inflammatory cytokines induced by Pam3CSK4 re-stimulation. Pam3CSK4 pre-treatment also down-regulated the signaling transduction of JNK, p38 and NF-κB induced by Pam3CSK4 re-stimulation. The activation of TLR1/2 induced a rapid and robust up-regulation of A20, suggesting that A20 may contribute to the induction of Pam3CSK4-tolerance. This hypothesis was proved by the observation that the over-expression of A20 by gene transfer down-regulated Pam3CSK4-induced inflammatory responses, and the down-regulation of A20 by RNA interference inhibited the induction of tolerance. Moreover, LPS induced a significant up-regulation of A20, which contributed to the induction of cross-tolerance between LPS and Pam3CSK4. A20 was also induced by the treatment of THP-1 cells with TNF-α and IL-1β. The pre-treatment with TNF-α and IL-1β partly down-regulated Pam3CSK4-induced activation of MAPKs. Furthermore, pharmacologic inhibition of GSK3 signaling down-regulated Pam3CSK4-induced A20 expression, up-regulated Pam3CSK4-induced inflammatory responses, and partly reversed Pam3CSK4 pre-treatment-induced tolerance, suggesting that GSK3 is involved in TLR1/2-induced tolerance by up-regulation of A20 expression. Taken together, these results indicated that A20 is a critical regulator for TLR1/2-induced pro-inflammatory responses."

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3905037/ 

===================

19) Ray Dattwyler (SUNY-SB) says in 1988 that exposure to Lyme or even just its supernatants (OspA-like molecules) turn off the NK cell activity (or is immunosuppressive): http://actionlyme.org/TRAINER_2012SUMMER.htm 

Modulation of natural killer cell activity by Borrelia burgdorferi. - 1988

Golightly M, Thomas J, Volkman D, Dattwyler R.Department of Pathology, State University of New York, Stony Brook 11794.PMID: 3056196 [PubMed - indexed for MEDLINE]Ann N Y Acad Sci. 1988;539:103-11.

"Effect of B burgdorferi Culture on Normal PBL

"..when lymphocytes are cultured in the presence of growing Bb there is a marked inhibition ( p < .0005 ) of NK activity on days 3, 5, and 7 when compared to lymphocytes cultured in BSKII media in the absence of spirochetes. This effect is not due to a selective depletion or or toxicity to endogenous NK since viability studies and monoclonal antibodies demonstrate no significant changes after culture with the organism.

"The inhibition is directly attributable to the organism or its supernatants (data not shown)."

http://www.ncbi.nlm.nih.gov/pubmed/?term=3056196 

http://www.actionlyme.org/DATTWYLER_NK_SUPPRESSION.htm 

===================

Gary Wormser's peer-reviewed article about how OspA was immunosuppressive

http://www.ncbi.nlm.nih.gov/pubmed/10865170 

Gary Wormser's peer reviewed article about how Dearborn "case definition" detected only 15% of the cases in IgG (9 out of 59):

http://www.pubmedcentral.nih.gov/picrender.fcgi?artid=266355&blobtype=pdf 

It's pretty obvious why the majority of the participants at the 1994 Dearborn conference (see below) said Steere's testing proposal that said Lyme was only an HLA-linked arthritis, SUCKED!!!

========================

20) Here is Clifford Harding, the discoverer of the mechanisms of both outcomes of OspA Disease or Lyme Disease [immunosuppression and no antbodies, and the other one, where the HLA-molecule together with the antigen is released as an entirely new antigen called an exosome or a vessicle, but is really a complex against which partial auto-/self- antibodies will be made; and in the case of alyme, particularly Allen Steere's version of autoimmunity against Human Leukocyte antigen (I assume)] talking about how if you have been exposed to Borrelial lipid TLR2-liktigens, you will end up with a null or non-response to other TLR agonists, such as viruses:

TLR2 signaling depletes IRAK1 and inhibits induction of type I IFN by TLR7/9.
J Immunol. 2012 Feb 1;188(3):1019-26. doi: 10.4049/jimmunol.1102181. Epub 2012 Jan 6.
Liu YC1, Simmons DP, Li X, Abbott DW, Boom WH, Harding CV.
Author information
1Department of Pathology, Case Western Reserve University/University Hospitals Case Medical Center, Cleveland, OH 44106, USA.

Abstract

"Pathogens may signal through multiple TLRs with synergistic or antagonistic effects on the induction of cytokines, including type I IFN (IFN-I). IFN-I is typically induced by TLR9, but not TLR2. Moreover, we previously reported that TLR2 signaling by Mycobacterium tuberculosis or other TLR2 agonists inhibited TLR9 induction of IFN-I and IFN-I-dependent MHC-I Ag cross processing. The current studies revealed that lipopeptide-induced TLR2 signaling inhibited induction of first-wave IFN-α and IFN-β mRNA by TLR9, whereas induction of second-wave IFN-I mRNA was not inhibited. TLR2 also inhibited induction of IFN-I by TLR7, another MyD88-dependent IFN-I-inducing receptor, but did not inhibit IFN-I induction by TLR3 or TLR4 (both Toll/IL-1R domain-containing adapter-inducing IFN-β dependent, MyD88 independent). The inhibitory effect of TLR2 was not dependent on new protein synthesis or intercellular signaling. IL-1R-associated kinase 1 (IRAK1) was depleted rapidly (within 10 min) by TLR2 agonist, but not until later (e.g., 2 h) by TLR9 agonist. Because IRAK1 is required for TLR7/9-induced IFN-I production, we propose that TLR2 signaling induces rapid depletion of IRAK1, which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2 inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes that express ligands for both TLR2 and TLR7/TLR9, or responses to bacteria/virus coinfection."

http://www.ncbi.nlm.nih.gov/pubmed/22227568

============================

21) Poland 2013 The influence of toll-like receptor stimulation on expression of EBV lytic genes.

Siennicka J¹, Trzcińska A², Cześcik A², Dunal-Szczepaniak M², Lagosz B².

• ¹Department of Virology, National Institute of Public Health - National Institute of Hygiene, Warsaw, Poland. jsiennicka@pzh.gov.pl
• ²Department of Virology, National Institute of Public Health - National Institute of Hygiene, Warsaw, Poland.

Abstract

Epstein-Barr virus (EBV) establishes latency in the resting memory B-cell compartment. It has been recently suggested that maintenance of chronic infection is dependent on periodic reactivation. Although the stimuli for EBV reactivation in vivo during natural infections are largely unknown, there is evidence indicating that heterologous infections could trigger herpesviruses reactivation. The purpose of this work was to identify the influence of Toll-like receptors stimulation on EBV replication in EBV latently infected Burkitt lymphoma cells (P3HR-1, Raji and Namalwa). The cells were stimulated with Pam3CSK4 (synthetic triacylated lipoprotein), PolyI:C (synthetic analog of dsRNA), LPS (lipopolysaccharide from E.coli), measles virus (MeV) and PMA (phorbol myristate acetate). Non-stimulated cells (NS) served as control. EBV expression was investigated at mRNA level for three viral lytic genes: BZLF1 (immediate early, ZEBRA), BALF2 (early, EA) and BcLF1 (late, VCA). Additionally, the effect of stimulation on NF-kBp65 and inflammatory cytokines (IL-lb, IL-6, IL-8, IL-10, IL-12p70, and TNF) was investigated. Stimulation of TLRs led to limited changes in EBV expression manifesting as increase of ZEBRA at mRNA level in cells treated with PolyI:C and Pam3CSK4. Stimulation with PolyI:C, Pam3CSK4 and LPS also lead to considerable increase of NF-kBp65, while increased levels of inflammatory cytokines were observed for IL-8, TNF and IL-6 in cells treated with PMA and MeV. In conclusion, the results of our experiments support the suggestion that TLRs stimulation with microbial ligands influences EBV virus replication.

http://www.ncbi.nlm.nih.gov/pubmed/24459828
Full text pdf  : http://www.pjm.microbiology.pl/archive/vol6232013237.pdf  <<< Get the full text, seriously, it clarifies much

 

 

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Aug 26, 2014 - On How "MDs" typically have no science training, and comparing Allen Steere to Clifford Harding (a real scientist)

Most "doctors" were trained by BigPharma. More recently BigInsurance butted their fat heads into the picture to insist on "Guidelines"- that they influence. You rarely see a "doctor" working at the things BigPharma does. So, they really have no clue. People have to seriously change their way of thinking about "doctors." What "doctors" do now could be done with machines (and computers) and a lab tech to draw the blood or run the diagnostic imaging.

We really need to work on this socially ingrained misconception. The only time "doctors" work for BigPharma is they run the clinical trials. They're next-to-never in the labs, working on Drug Metabolism or any Analytical or Animal studies. Only a very small percent do real research.

Look at Allen C. Steere's Wikipedia page. He's still trying to figger out how Lyme causes arthritis in 15% of the HLA-linked cases, when this was solved years ago by Clifford V. Harding, http://www.ncbi.nlm.nih.gov/pubmed/?term=Harding+CV+and+exosomes

the same dude who also showed us how Lyme and LYMErix causes immunosuppression and no antibodies in the other 85% of the cases:
http://www.ncbi.nlm.nih.gov/pubmed/?term=Harding+CV+and+radolf+jd

J Immunol. 2001 Jul 15;167(2):910-8.

Toll-like receptor 2-dependent inhibition of macrophage class II MHC expression and antigen processing by 19-kDa lipoprotein of Mycobacterium tuberculosis.

Noss EH¹, Pai RK, Sellati TJ, Radolf JD, Belisle J, Golenbock DT, Boom WH, Harding CV.

Abstract

Mycobacterium tuberculosis (MTB) induces vigorous immune responses, yet persists inside macrophages, evading host immunity. MTB bacilli or lysate was found to inhibit macrophage expression of class II MHC (MHC-II) molecules and MHC-II Ag processing. This report characterizes and identifies a specific component of MTB that mediates these inhibitory effects. The inhibitor was extracted from MTB lysate with Triton X-114, isolated by gel electroelution, and identified with Abs to be MTB 19-kDa lipoprotein. Electroelution- or immunoaffinity-purified MTB 19-kDa lipoprotein inhibited MHC-II expression and processing of both soluble Ags and Ag 85B from intact MTB bacilli. Inhibition of MHC-II Ag processing by either MTB bacilli or purified MTB 19-kDa lipoprotein was dependent on Toll-like receptor (TLR) 2 and independent of TLR 4. Synthetic analogs of lipopeptides from Treponema pallidum also inhibited Ag processing. Despite the ability of MTB 19-kDa lipoprotein to activate microbicidal and innate immune functions early in infection, TLR 2-dependent inhibition of MHC-II expression and Ag processing by MTB 19-kDa lipoprotein during later phases of macrophage infection may prevent presentation of MTB Ags and decrease recognition by T cells. This mechanism may allow intracellular MTB to evade immune surveillance and maintain chronic infection.
 

Technically speaking, CDC officer Allen Steere works for an insurance company - an HMO. His email address is @partners.org - an insurance company that basically is taking over all the Boston hospitals.

Yall follow? Very little serious science is happening. That is why only this week did the NYTimes report about the genetic kind of Autism happening via a similar mechanism by which OspA/fungal antigens cause immunosuppression - inhibition of apoptosis (and therefore an inhibition of "normal synaptic pruning"). I was screaming about this 12 years ago because that data was already out:

MARCH 2002: "We identified a fourth pseudogene, BCL8A [a BCL-2 class molecule, which inhibits apoptosis is reverse-duplicated and therefore overexpressed- KMD], which maps to the pericentromeric region and is coamplified along with the NF1 sequences. Interphase FISH ordering experiments show that IgH D lies closest to the centromere, while BCL8A is the most distal locus in this pseudogene array;"
http://www.ncbi.nlm.nih.gov/pubmed/11897815

http://www.nytimes.com/2014/08/22/health/brains-of-autistic-children-have-too-many-synapses-study-suggests.html?_r=0

duh.

There are no "authorities" in this country, so, you're on your own, but you'll be the better for it.

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Aug 25, 2014,  On the ALS Ice-Bucket Challenge and how that is all going to ALS-Lyme (See the Lyme Crooks Own data on that)

What are we going to do with all those 50+ millions of dollars from the ALS-Lyme Ice Bucket Challenge?  That, in combination with all of Yale's properties, capital assets, patents, and Endowment Fund (right now, what? $20 billion?)?  We're going to have to set up a committee to deal with all those bootles in a grand Trust for all the victims of Yale's various crimes.  Join me over on the OCCUPY the USDOJ Facebook page if you would like to participate in the proceedings.  https://www.facebook.com/#!/groups/271420453060565/

All Things Considered

Let's back up for a moment.  Recall that the whistle was officially blown in 2001 over the falsified case definition and who that benefitted (see below the double horizontal line below- it was Yale who owned the only validated test for Lyme but did not deploy this FDA-validated testing to assess their other patent, LYMErix, so that was a FALSE CLAIM or "a FRAUD on the Government").  Then recall that in July 2003, Senator Blumenthal's office when he was CT AG referred me to the USDOJ in New Haven to file the RICO and FRAUD claim once they had seen that there were 2 different diagnostic standards for Lyme and that the testing for Lyme had CHANGED??!!! to facilitate the fake OspA vaccine trials. Next, in 2006, Blumenthal himself sued IDSA for "AntiTrust."

And GUESS WHO tried to pull an end-run around the Blumenthal subpoena for all of IDSA's documents that went into the so called "guidelines" for "Lyme Disease" by publishing secondary fake "guidelines" from some Neurology group?

JJ Halperin, the author of the "Lyme causes ALS" report.  It's pretty hard to claim that Lyme Disease is merely an HLA-linked arthritis in a knee (the current false, Dearborn "case definition") when it is known to cause MS, Lupus, and ALS.

So, what is to consider?  If, as the report claims,








6 patients were positive only to the cell mediated immunity test, what is that test, and what does that mean exactly, in terms of the current research fraud "case definition?"  It means a murder charge.  But it also means, if there is cell proliferation upon exposure to Borrelial antigens, could they have been SIMILAR fungal antigens, such as from mycoplasma?

I say so because Lyme and LYMErix tolerize to fungal antigens, which means Lyme appears to have set up the condition where fungal antigens and subsequent immunosuppression / activation of latent herpesviruses lead to ALS, MS, Lupus, all of IDSA's New Great Imitator outcomes...

Fortunately for us, there is no one either responsible, accountable or intelligent who works for the "Government" so, it is a free for all in terms of research that shows the relatedness between Lyme, LYMErix, the herpesviruses, mycolasma/fungi, vaccines-acquired and genetic Autism (both occur via the same mechanism, basically; one is a reverse duplication of a BCL2-class gene, inhibiting apoptosis and subsequent inhibition of "normal synaptic pruning," and the other is fungally contaminated vaccines that result in immunosuppression and the resultant he activation of the live, attenuated viruses from the vaccine vials; LYMErix also inhibits apoptosis, since that's what fungal antigens do), and the Great Imitator outcomes like ALS, various cancers, leukemia, MS, RA and Lupus.

---

---

The Food and Drug Administration wants us to report non-FDA-validated testing for "Lyme Disease" when we already did, 13+ years ago, with the supporting documentation from the Dearborn booklet:
See page 4, here: http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf  (there were actually 30 pages;  19 of the pages were out of the Dearborn booklet, so the FDA was given the evidence of this crime on Jan 31, 2001)

The FDA wants us to report our adverse events (missed diagnosis and timely treatment) due to the fraudulent "two-tiered" CDC-Dearborn testing schema [20140820];

Report if you were told you don't have Lyme Disease based on the Dearborn criteria but later found out you had at least band 41 (anti-flagellar antibody; see CDC officer Alan Barbour approving: http://www.ncbi.nlm.nih.gov/pubmed/3516878, although Yale more completely validated a Flagellin Method, as shown in Chapter One of Cryme Disease) on a Western Blot and all the triad symptoms (cognitive, fatigue, musculoskeletal):

https://www.accessdata.fda.gov/scripts/medwatch/index.cfm?action=consumer.reporting1

The FDA said they will now ENFORCE the correct validation criteria for the the Lyme test detection method, which is Chapter One of Cryme Disease: "What *is* Valid?"

This is from the new announcement regarding the FDA now about to ENFORCE their validation rules:
http://www.fda.gov/downloads/MedicalDevices/ProductsandMedicalProcedures/InVitroDiagnostics/ucm407409.pdf


 

I will provide you here with the documentation you will need to file your complaint to the FDA, and there is more information below on this homepage and here in this referenced report: http://www.actionlyme.org/PLUMSTUPID.htm about how Steere falsified the testing with bogus high-passage strains and left OspA and B out of the standard for his later Imugen monopoly on testing with Corixa and Yale's L2 Diagnostics, by using recombinant OspA and B proteins without the lipids attached - you need the lipids to be immunogenic or to cause antibodies.  Dearborn was about falsifying the OspA vaccines/LYMErix outcomes and for the later monopoly on post-LYMErix-approval testing for Tick Borne Diseases afterwards. (You don't test for a disease with the same antigens that are in the vaccine.)

Notice that this meeting at the FDA in JUNE 1994 where Ray Dattwyler spoke about how the sickest patients were seronegative PRECEDED the Dearborn stunt, which took place in October of the same year:

<< June 1994 FDA Meeting Minutes. See the date stamp.  This happened BEFORE Dearborn which was October 1994.

So, the FDA never approved the Dearborn proposal.

The FDA would have access to all their own FDA Meeting Minutes for the 1994 and 1998 Lyme and LYMErix vaccine meetings and can see that a significant portion of the people on the FDA committees for Lyme and LYMErix were CDC officers.  The others objected to the testing schema (Luft and Dattwyler) The current bogus schema for Lyme testing is therefore CDC-approved and not FDA-approved.

Here is the INVITATION!! to "participate in the proceedings!!" at Dearborn, MI, held October, 1994, which was after June 1994, the FDA meeting where Dattwyler said arthritis was the exception rather than the rule and that the seronegative patients were the sickest.

DEARBORNINVITATION.pdf

The Dearborn Booklet issued by the CDC:
http://www.actionlyme.org/DEARBORN_PDF.pdf

Who was actually at this bogus Dearborn conference and approved of Steere's (CDC ofcicer) bogus proposal based on scientific fraud (see STEERE_IN_EUROPE.htm) despite none of the labs who participated in the proceedings agreeing?

http://www.actionlyme.org/Dearborn_Who_Approved.htm  [Alan Barbour (CDC officer), Allen Steere (CDC officer), Edward McSweegan (crook who defrauded the Govt with a fake whistleblower complaint about the Navy to Senator Goldwater), Arthur Weinstein who was in charge of "data safety monitoring" in the ImmuLyme trial but never reported that the Western Blots in that vaccine trial were, in fact, unreadable: PERSING WITH SIGAL EXPLAINING THAT THE WESTERN BLOTS WERE UNREADABLE, 2000:
http://www.journals.uchicago.edu/doi/pdf/10.1086/313920
, and "The CDC Lyme Disease Group"]

MarDx Labs were given Lyme arthritis-positive blood to qualify their Western Blot test strips for their Dearborn submission.  In the vaccine trials, it turned out that the blots were unreadable and both ImmuLyme and LYMErix trials used MarDx test strips:
PERSING WITH SIGAL EXPLAINING THAT THE WESTERN BLOTS WERE UNREADABLE, 2000:
http://www.journals.uchicago.edu/doi/pdf/10.1086/313920


Here is the fraudulent Dearborn criteria (Oct, 1994):
http://www.cdc.gov/mmwr/preview/mmwrhtml/mm5405a6.htm

You can see that the CDC approved their own bogus method, despite none of the participants at Dearborn agreeing with the Steere research fraud proposal,... and not the FDA.  ...

This is while the Yale Flagellin Method was validated per the FDA rules in 1991, but was not used to qualify their other patent, LYMErix, because they knew LYMErix did not prevent Lyme... and THAT is the simple False Claims Act case.  Yale made the false claims. They owned both patents, LYMErix and the FDA-validated Flagellin Method.

1) Yale's Bb Flagellin Patent, 5.618,533 
2) Yale's LYMErix Patent, 5,747,294
 

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USA INVOLVED IN ISRAELI WAR CRIMES IN GAZA - "PROVIDING FINANCIAL AND MATERIAL SUPPORT FOR TERRORISTS" (and more than that, as you will see)
=================

GLENN GREENWALD: The Snowden documents demonstrate an extremely close relationship between the NSA and U.S. intelligence agencies, in general, and the Israelis. It demonstrates that there are large amounts of cash, unaccounted-for cash, being transferred from the NSA to Israeli operatives, and that the specific information that the Israelis have and that they use to target people in Lebanon or in Gaza or the means that they have to assert occupation and control over the West Bank come in large part directly from the U.S. government and from the intelligence gathered by the NSA. It also shows that the crucial information that the U.S. government gathers about the Palestinians and about people in Gaza come from the Jordan monarchy, the Jordanian monarchy, as well as the Palestinian Authority, which of course receives lots of funds from the U.S. government and then, in turn, spies on people in Gaza for the benefit of the U.S. and Israel.
http://www.democracynow.org/2014/8/13/glenn_greenwald_us_intel_agencies_provide

And

1. CIA is much like the CDC and their relationship with Israelis, Kaiser-Permanente, and IDSA, LOL
   =========================================
   "Greenwald says the NPR report erred in failing to mention that ***the firm is funded by the CIA.*** "This was such a pure and indisputable case of journalistic malpractice and deceit," Greenwald charges. "NPR radically misled millions of people with this report."
   http://www.democracynow.org/2014/8/13/glenn_greenwald_criticizes_npr_for_relying
    

Dear Pentagon, These are not the Aryans you're looking for:

The CDC is currently (Aug, 2014) going around telling labs in the country that they can't run Western Blots without running an ELISA first (ELISAs only detects late Lyme arthritis, the HLA-linked hypersensitivity case), when no one agreed with the CDC's case definition at the FDA meetings in 1994 and 1998 (leading to the next one Jan, 2001 which came as a result of the 1100 systemic adverse events reported thru the VAERS), when CDC staff were the ones who falsified this case definition in the first place; when CDC staff own most of the patents for Vector-Borne Diseases; when CDC staff were on the FDA committees for OspA vaccines; and when the CDC staff were the ones who approved their own bogus CDC, Dearborn, MI, 1994 case defintion, despite none of the labs participating in Dearborn meeting agreeing, either !!!  See STEERE_IN_EUROPE for how Steere came up wth that bogus, current criminally falsified case defintion that only detects late Lyme arthritis, an alleged autoimmune disease that has no other symptoms.

The USA Govt does not go after these people because most of them are Israelis: Gary Wormser, Durland Fish, Alan Barbour, Mark Klempner, Lenny Sigal, Eugene Shapiro, Larry Zemel, Henry Feder, Robert T. Schoen, John Nowakowski, JJ Halperin, Stanley Plotkin, Kent Sepkowitz, Simon Wessely, Paul Auwaerter, (Allen Steere, Barbara Johnson, Edward McSweegan)...

◄ Use the left navigation bar to follow the OspA scam.  Every single thing this gang has done since Dearborn and the fake OspA "vaccine" "trial" has been about promoting the idea that they all believed the Dearborn case definition was real and valid.  Nope - that is what we have been complaining about all along.  This test does not detect Lyme Borreliosis, which was good for not detecting Lyme in vaccinated people and for saying therefore, that the vaccines "prevented Lyme."

In fact, these suckers were so dumb and un-scientifically-inclined, OspA was the very fungal antigen responsible for Chronic Lyme," the "New Great Imitator," the Activation of Epsrtein-Barr/Similars, and for TLR2-agonist tolerance, that results in an AIDS-like disease.

I guess we can say the Israelis are not the Aryan supermen the USA Einsatzgruppen are looking for (see more about that below, next).  They're lying cowards, certainly when it comes to medical science.  And there is only one worse thing than being bad at medical science, and that is telling people you're an intellectually superior race without any "science-based evidence." 

They fecked up research in every major disease: AIDS (one of the AIDS vaccines that failed was LYMErix, actually, Pam3Cys), hospital acquired infections, sepsis, Tuberculosis, Leukemia, other cancers, Multiple Sclerosis, Lou Gehrig's Disease, Lupus, Rheumatoid Arthritis, ... you name it - because they lied about PamCys as a "vaccine," when it was an immune suppressing fungal antigen that helps activate Epstein-Barr and similar latent viruses.

I don't know if it is possible for one's scientific or intellectual reputation to be lower, really.  I guess being a self-aggrandizing asshole is a superboomeranger.

Update - You're going to wonder why Yale, the CDC staff and their dual-citizen Israelis et al are not being prosecuted for falsifying the LYMErix data because here is the NIH agreeing with us once again on the mechanisms of illness in immunosuppression/inhibtion of apoptosis cause by OspA - which acts like a BCL-2 class molecule, inhibiting apoptosis - in sepsis (think, that asshole Kent Sepkowitz), ... and in reversing the OspA- like/BCL-2- like inhibition of apopstosis as a cure for CANCER !!!

and

Surviving Sepsis: Detection and Treatment Advances

By Carolyn Beans for the National Institutes of Health   |   August 18, 2014 08:43am ET
http://www.livescience.com/47387-sepsis-diagnosis-treatment-research-nigms.html

"Preventing Secondary Infections

"Some people who survive sepsis can develop secondary infections days or even months later. A research team that included Richard Hotchkiss, Jonathan Green and Gregory Storch of Washington University School of Medicine in St. Louis suspected that this is because sepsis might cause lasting damage to the immune system. To test this hypothesis, the scientists compared viral activation in people with sepsis, other critically ill people and healthy individuals. The researchers looked for viruses like Epstein-Barr and herpes simplex that are often dormant in healthy people but can reactivate in those with suppressed immune systems. [Sepsis Has Long-Term Impact for Older Adults, Study Finds]"

There ya have it.  How OspA caused systemic disease was the key to nearly all disease - including CANCER - and these assholes associated with Yale and IDSA had it all right in their laps with the immunosuppression results of the OspA vaccine trials that they decided to lie about -, causing the same systemic, protean (Ben Luft), "multi-system " (Dave Persing) disease as Chronic Lyme and the New Great Imitators.

The truth about the Lyme crymes was put out in its entirety in ONE DAY (today, Aug 19, 2014) - a day after some asshole Lyme activists in the UK trashed me and called all this "politically driven double speak" (which of course is CRAZY to say since real science has ZERO to do with politics - politics is for fairies or the unmanly),  and "despicable."

All Truth comes from God.  The devil has no creative abilities and all their ops are about lies (Lucifer) and destruction (Satan) of Creation. Now you know for whom ALL these assholes are working. The Truth would only be "despicable" to the demonic.

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Die Einsatzgruppen der Arbeit für die Nazis, und sie sind immer noch auf der Suche nach Ariern, sie zu führen. 

(LOL.  Ur gonna laff... keep reading...)

1.  

    

    

    

    

    

    

    

    

    

    

   
   
   
   And...

   THE CDC... WHO ADMITS THEY DON'T KNOW WHAT rOSP-A IS, LOL:
   ================================================
   "The result is a small ruling cabal, able to control policy on a national scale not through any conscious or deliberate conspiracy, but rather owing to a continuing sequence of ostensible emergencies; their power is, in theory, contingent on extraordinary security circumstances, yet in practice it has functioned as a self-sustaining, shadow legal system, commensurate to a bloodless coup. The bedrock of the insidious executive power Arkin illustrates is a complacent and overarching trust in the omniscience of apparent experts, the *** technocrats in federal bureaucracies who supposedly know better than the rest of us. *** They have managed to gradually wrest governing power, to concentrate it in that formless space of regulatory authority between actual, black-letter laws. Their appeal to discretion as experts is the source of their ill-defined power. All of that Arkin calls “the Program,” “an entity and group” he analogizes to Wall Street, a synecdoche we use to mean not only the place itself, but “also allusively the entirety of interests” we associate with it."
   http://fff.org/explore-freedom/article/a-treacherous-undertow/

   And...

   
    

   The self-appointed Intellectual Elite is still looking for their Aryan supermen because everything they've touched so far has turned to shit. 

   Hmmm.

   Lemme think, where's the threat, here?  LOL.

   ---

    

   There are several people who whore for the gubbamint whose specialties are *ONLY* Lyme and Epstein-Barr:
   
   Paul Auwaerter, Adrianna Marques, Bridgette Huber (worked with Allen Steere on "Bad Knees"), and Roland Martin, a German scientist hired by the NIH, who discovered Lyme caused MS, who went back home to Germany once he found out LYMErix was the cause of the MS version of Lyme (activates EBV/Similars).
   
   So, we should be calling all of these non-diseases (CFIDS, ME, Fibro, Chronic Lyme) "Chronic Active Epstein-Barr," as Straus does/did, and the NIH admitted last year:
   
   "Complicating the picture is the fact that some people with PTLDS symptoms apparently never had Lyme disease in the first place, Dr. Marques said in an interview. There are other infectious organisms — Epstein-Barr virus, for example — that can produce similar symptoms and may be the real culprits."
   http://well.blogs.nytimes.com/2013/07/08/when-lyme-disease-lasts-and-lasts/
   
   If the NIH says there are 4 million people in USA with CFIDS, and 8 million with Fibro - and throws no money at it -, you KNOW they know what it is, and it is not any new kind of disease. They just do not want to talk about immunosuppression (because that reveals the source of the Autism pandemic).

   If they ignore CFIDS, Lyme and Fibro, it's because they know what it is. And it could only be something common and something that everyone has. It could only be something they do not worry about.

   The lies are JUST about Autism (failed vaccines) and bioweapons, which target HLA-negative victims with stealth disablers, which are of the fungal type. Nothing else is reliably stealth, but fungal antigens.

   Do you see this? (Steve Schutzer)
   What does this mean?
   
   Same Disease (Chronic Active EBV), different (probably fungal) Initiator:
   http://www.ncbi.nlm.nih.gov/pubmed/21383843

   

   Distinct cerebrospinal fluid proteomes differentiat... [PLoS One. 2011] - PubMed - NCBI

   www.ncbi.nlm.nih.gov

    

   "ANTIBODY NEGATIVE REACTIVATED EBV IS PROBABLY CHRONIC FATIGUE SYNDROME" AND COULD BE *THE* MARKER OF IT:
   
   ttp://www.ncbi.nlm.nih.gov/pubmed/24454857

   

   Deficient EBV-specific B- and T-cell response in pa... [PLoS One. 2014] - PubMed - NCBI

   
   And on this page, there are 4 other reports re EBV being antibody negative:

   http://www.actionlyme.org/101016.htm

   You will find the fungal-associated / TLR2-agonist tolerance / immunosuppression is like losing 1/3 of your blood in an accident.  The Oxygen is there but the epERTHRYOzoons (former name of mycoplasma) render the O2 unable to cross the membrane.

    

   You can believe Uncle Sam does not care about the 20% or more of the USA population being disabled because all the gubbamint cares about is if you contribute to their NAZI-like Military machine.  It's the exact same NAZI ideology.  You are a useless eater and not necessary if you are no use to the Military either as a soldier or working for a weapons contractor.

    

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If you want to understand why Israelis appear to be so completely insane, read their Talmudic doctrine.  We Gentiles are not even human according to the Talmud.  Thus, you can kill your donkey, no problem.
 

In parallel, see Henry Kissinger speaking to Charley Rose about creating chaos so people will beg for the New World Order.  Therefore, because Hillary Clinton is Henry's parrot, if she is "voted" in, we will have an Israeli as president of the United States.

Most importantly, you would wonder if there is a more doctrinal relationship to the New World Order with its Petrodollar Sign of the Beast (calculate in, and exchange in), and all the chaos being conducted by the New World Orderites?

See the next snippet, below about the Messiah:

 

 

http://www.jewfaq.org/mashiach.htm

 

You can see that all of AmerIsrael's insane carrying-on and the nutcases who think they own/run America and Earth do in fact, have an overall plan in mind. 

At the same time, from most of the Lyme criminals and their spokespersons who were not involved in the LYMErix scam, like Joseph Alpert, you can how see the Talmudic/Jews-as-the-Messiah non-humans, and "talking animals", cockroaches and donkeys DOCTRINE/theory of Gentiles' endowment applies.

Why are these criminal Israelis of the IDSA and the ALDF.com never prosecuted? 

It's no crime to murder and and otherwise abuse a Gentile.  Jewish Law is mainly about how that's all okay, since Gentiles are not human.  Palestinians are not Jews, therefore to kill them is legal. 
 

Israelis are also terrified of Russia, because they would know about the other theology, which is substantiated, scientifically, in our time (Fatima).

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Stunning Boomeranger - You're not going to believe this.  IDSA kills off one of their own, and of all things, the INTELLECTUAL PROPERTY Director at Johns Hopkins!!

 

 

 

 

 

 

 

 

Auwaeter is @ Johns Hopkins

Says he does not know what OspA is.

 

 

 

 

 

Click on these images for the links out





 

 

 

 

 

 

<< Stanley Plotkin committed FRAUD with the ImmuLyme OspA trial. That was why he's been such an asshole towards us.
 

The Lyme criminals could not even read their Western Blots in LYMErix or ImmuLyme-vaccinated people, yet they claimed to have used the Dearborn Method to assess their vaccines outcomes: 
Here are those 4 "we can't read our OspA vaccine results" reports:

1) SCHOEN and PERSING, with JOHN ANDERSON,1996 - the RICO report:
http://jcm.asm.org/cgi/reprint/35/1/233?view=long&pmid=8968914
2) SCHOEN AND PERSING IN THEIR 1996 RICO MONOPOLY ON BLOOD TESTING METHOD PATENT:
The Dave Persing, Mayo Clinic FRAUD Patent-6,045,804 
3) PERSING WITH SIGAL EXPLAINING THAT THE WESTERN BLOTS WERE UNREADABLE, 2000:
http://www.journals.uchicago.edu/doi/pdf/10.1086/313920
4) Yale's ROBERT SCHOEN in the 1998 Munchausen's Book, instructing MDs to blow off LYMErix systemically
injured people ("but send the post-vaccination blood to the Yale L2 Diagnostics RICO lab if you must bother to be
a physician").

This ▲ is a FALSE CLAIM or a QUI TAM or FRAUD on the GOVERNMENT case:
http://www.usdoj.gov/usao/eousa/foia_reading_room/usam/title9/crm00921.htm

They, Yale, Schoen, knew they could not read their Western Blots in LYMErix-vaccinated people, but they wanted all the national bloodwork sent to their labs (L2 Diagnostics and PolyGenomics), so they could patent, and pharm, and be the only ones with access to the new vector borne diseases to patent, and be the only ones with access to - nationally - genetic information about disease susceptibilities (this has bioweapons applications).   The reason we can't have this, is because Yale is too stupid to be managing diseases and disease susceptibilities, because they missed out on the real reason Pam3Cys or LYMErix, or HIV vaccines did not work, or why Lyme was the New Great Imitator- IMMUNE SUPPRESSION due to tolerization to fungal antigens. (Yale employees were never smart enough to be in charge of such data.  In fact, there are no intelligent people left in Connecticut at all.)







The ^^ Dave Persing, Mayo Clinic FRAUD Patent-6,045,804  << USPTO.gov link.  It's for a strain of Borrelia that does not have the OspA-B plasmid it it.  Was intended to be the only testing anyone could use in America or Canada once LYMErix was on the market since LYMErix is OspA and similar to OspB (since they are encoded on the same plasmid, they had to take the whole plasmid out).  You don't usually test for a disease with the same antigen as the vaccine - that was why OspA and B were left out of the CDC's Dearborn case definition.  Simply for this bogus vaccine trial and especially for what was to happen afterwards - the monopoly on VBDs testing and vaccines and money and grants and ownership of ALL Vector Borne Disease DNA and anything else found interesting in America's blood, such as HLA-related diseases susceptibility or "racially specific bioweapons" :)




 

 

 

 

 

  << click to enlarge and see the "data" for yourself.  I spoke about this

 FRAUD and gave the FDA Vaccine Committee (on Jan 31, 2001), all this data on vaccine failure in a 30 page booklet, and on what happened at Dearborn, who said what (no one agreed at that Dearborn conference with the new and bogus testing proposal which was again, RESEARCH FRAUD COMMITTED BY ALLEN STEERE IN EUROPE).
 

IDSA/JJ Halperin reports that Lyme causes ALS: 
http://www.actionlyme.org/ALSLYME47.htm


IDSA reports that Lyme causes MS


Mark Klempner reported that OspA caused MS:
 





and so did the NINDS in 2006, say OspA caused the MS outcome of Lyme:


Adrianna Marques and Germany's Roland Martin (NIH or NINDS) on LYMErix or exposure to OspA blebbing as the source of the MS version of Lyme and LYMErix Disease (2 reports, the second one shows you might not even have antibodies to flagellin as a result of exposure to OspA/TLR2-agonists/fungal antigens):
"Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression."
http://www.ncbi.nlm.nih.gov/pubmed/16783164
AND
"Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes."
http://www.ncbi.nlm.nih.gov/pubmed/16479520

 

Continuing, IDSA reported that Lyme caused Lupus (turns out that was a result of the herpsviruses that OspA exposure activates), Dementia, was an incurable brain infection, spirochetes are associated with Alzheimer's (a dentists group, actually, see # 22 here), .. turns out all of that Great Imitator stuff was caused by spirochetal fungal antigens like OspA activating the latent herpesviruses.

Turns out, they knew LYMErix causes systemic disease as shown here, above, in those LYMErix trial results.

Turns out Yale's claim that "LYMErix prevents Asymptomatic Lyme" was code language for "LYMErix causes all the Great Imitators by being an immunosupressive fungal antigen that causes TLR2 agonist tolerance and activates the latent herpesviruses" - outcomes they were able to throw out because they re-defined "Lyme Disease" as "just a bad knee, hypersensitivity response" at the criminal CDC's 1994 Dearborn RI, MI, RICO crime scene.


Turns out those assholes at the NEW HAVEN USDOJ has been sitting on all this data - 5 computer-paper boxes worth-  since July 2003.

Turns out I was right about those NEW HAVEN FBI assholes, too - they sit around all day looking at porn, NSA's stolen sexting.

 

Greetings Justice Fans (140718),

Finally it is time to announce the OCCUPY the USDOJ for next year, May, 2015 until the farcically named "Independence Day," July 4, 2015. (Many of the participants are sick and disabled and cannot do an occupy in DC in the heat of the summer; details are forthcoming and managed by the CFIDS community.)

This is an All Groups Occupy: Chronic Fatigue, Autism Parents, Psych Rights advocates, Gulf War Illnesses, "Lyme Disease," Fibromyalgia, etc, all who are captives of the Government-Whoring-for-their-Corporate-Pimps Complex.  (Call them whatever you like for short. Go'-Hoes works for me.)

The singular message for the Chronic Illness Groups (Gulf War Illness, Vaccines-brain damaged children, "Lyme" and CFIDS/Fibro) is that the CDC does not want to admit to diseases of immunosuppression because the mechanisms of those betray the source of the Autism pandemic (fungally contaminated vaccines, or the vaccination of children who are immunosuppressed and who should have been pre-screened for immune status prior to vaccination).

For Psych Rights and Parents Rights (and this clear has ties to the abused medical groups), it is that the Director of the National Institute of Mental Health, Thomas Insel, has declared psychiatry to be scientifically invalid and that fact has 2 important vectors that need to be refined: What *is* valid in in framing brain compromise, and what legal tools are now available to you in fighting psychiatric abuse?

I will be developing this ActionLyme homepage to make it All Groups Friendly, so stand by.  Meanwhile you can join the Facebook, Occupy the USDOJ page.  The only rule for that group is that you can't talk about yourself or your personal case:

USDOJ Protest for All Abused Groups (Autism,GWI,CFIDS,Lyme,Parents,Psych..)
https://www.facebook.com/#!/groups/271420453060565/

It's bad enough that our morally and cognitively pathological "elected representatives" have no interest in our rights or complaints.  But it is quite another thing when you're a paid employee of the Justice Department, not to do your job.  Therefore, the USDOJ will either do their jobs and protect us against corporate crime, or they will show the WHOLE WORLD that the United States is not what it claims to be.  The whole world can see how the United States treats their sick and disabled, sick and disabled because of corporate crime.

You can see that we absolutely have to take this stand and this action.
 

---

CDC Is an Organized Crime, Corrupt Practices and Racketeering Enterprise - dirty-mouthed Durland Fish-ish thugs who literally carry around hit-lists, and all - in cahoots with Yale University:

Scroll down and see how this happened, and started with Edward McSweegan trashing the US Navy (and see the US Navy's response), in 1986, in a letter to Senator Goldwater to steal the Navy's Vector Borne Diseases funding to funnel into their own little cabal, as if Govt funding was their Free Venture Capital.  Normally people take out loans or raise other money for such a broad enterprise as vaccines for all vector borne diseases discovered and yet to be discovered. Not this gang - not with McSweegan doing Durland Fish's bidding, even to go so far as to stalk, wiretap and harass the Lyme Disease Foundation.

Here is something hilarious (more Attack-Pattern "Science") from the CDC, who falsified the case definition for their own personal patents' bottom line :)   CDC is quite terrified of being prosecuted for falsifying the testing for Lyme.  Scroll down this homepage for more of the exact details on how CDC officers Allen Steere, Barbara Johnson, Alan Barbour, et al, performed this scam in Germany (1992) and in Dearborn, Michigan (1994).

Article: http://politicalnews.me/?id=29174&keys=DIAGNOSES-CONDITIONS-MEDICAL-OBAMACARE

You're not going to sell vaccines and test kits for a disease known to be largely seronegative (and fungal) right?   

CDC is scared to death of being prosecuted for this, so they attack their victims and call us crazy, when we all know it is crazy to say a fungal antigen could be a vaccine.  Thimerosal is put in vaccines to prevent fungi, duh.

Consider the cost of the HIV-LYMErix vaccine trial.  CDC never told anyone this HIV-LYMErix vaccine, Pam3Cys, would not work.  So, the damages include not only all the money the NIH wasted on the Lyme crooks,... and not only how the LYMErix scam inhibited discovery in every major disease, the CDC cost taxpayers money with the fake OspA-HIV vaccine trial (also Pam3Cys), ... which Anthony Fauci admitted gave the same immunosuppression results as Lyme and LYMErix!!
 

---

The new NAZI ideology adopted by "America" and Israel has to do with alleged moral or cultural superiority rather than racial. 



Noam Chomsky: "America's Real Foreign Policy: Global Corporatism by Force"
Norman Pollack: "US Govt Intimidated by its own Mercenaries"
Mark Dankof: "US Government a criminal enterprise"
Paul Craig Roberts/Vladimir Putin: "The West is Turning the Planet into a Global Barracks"

However, how moral or cultural is it for the US Govt to be so assholish as to wreck the lives of 12 million Americans (with CFIDS or Fibromyalgia, alone), not to mention "Lyme Disease," Gulf War Illnesses, Dead and Disabled soldiers from the bogus 911-Thermate-Oil Wars, and all the kids brain damaged for life by "vaccines"?  A good 20% of us are disabled, but the Mafioso THUGS who whore for the "government" never lose any sleep over it.  As long as they get their paychecks for a job not done and all the bennies... :)

Just take a look at this dick:
 

WHY are the CDC and NIH such a bunch of lying assholes, what don't they want you to know, and what is in common with all the Abused and Neglected Diseases like Gulf War Illness, Failed Childhood Vaccines (we call it Autism, but it's really just plain brain damage and ADHD might be too), "Chronic Lyme," LYMErix Disease, Chronic Fatigue Syndrome, and Fibromyalgia?
 

The answer has to do with the failed childhood vaccines and how they fail (kids end up with the viruses instead of the protection or antibodies). 

 

CDC does not recognize diseases without antibodies, right?

And Simon Wessely (above) was hired to trash Gulf War Illness Vets, Chronic Fatigue and Chronic Lyme victims, correct? 

A man who knows nothing scientific, was not trained in anything having to do with a validation... and psychiatry has been officially debunked as scientifically invalid (I filed a RICO case against psych.org in Jan 2004 claiming it was scientifically invalid, but it took 9.5 years for the NIMH to finally admit that that was true, psychiatry is not a valid medical or scientific practice; psychiatry does not meet the criteria of "Scientific Validity")... and the same asshole Wessely was deployed against ALL OF US?  [Maybe Wessely can write his memoirs and tell us about how PRIDE got in the way of reality for him.]
 

Childhood Immunizations and the brain damage that is currently called autism also fits this model – CDC’s and BigPharma’s own data (5 reports), plus the NYT revelation that the thimerosal was intended to prevent mycoplasmal or fungal growth within the vials:

 
A) – BigPharma: "Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the
vaccine may actually cause disease."
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7%2C632%2C510.PN.&OS=PN%2F7%2C632%2C510&RS=PN%2F7%2C632%2C510


B) CDC:
”Measles, Mumps, and Rubella -- Vaccine Use and Strategies for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and Control of Mumps: Recommendations of the Advisory Committee on Immunization Practices (ACIP) 
 
"Updated information on adverse events and contraindications, particularly for persons with severe HIV infection, persons with a egg allergy or gelatin allergy, persons with a history of thrombocytopenia, and persons receiving steroid therapy [are immunosuppressed - KMD]."
http://www.cdc.gov/mmwr/preview/mmwrhtml/00053391.htm
 

C) CDC: Human Exposure to Brucella abortus Strain RB51 -- Kansas, 1997 
http://www.cdc.gov/mmwr/preview/mmwrhtml/00051495.htm

In the above, an immunosuppressed pregnant cow was given a Brucella (“LYME”rix-like) "live attenuated" vaccine and the baby cow ended up with the disease, which then was transferred to the humans handling the cow and her dead baby.  This parallels what is happening to children who are vaccinated while immunosuppressed, or who receive mycoplasmally (“LYME”rix-like) contaminated vaccines.


D) in the case of pandemic MRSA, as we have seen, the vaccine didn't work because TLR2 agonists (lipoproteins) suppress the immune system.  We also learned from the MRSA vaccine patent, that:

"Several established vaccines consist of live attenuated organisms where the risk of reversion to the virulent wild-type strain exists. In particular in immunocompromised hosts this can be a live threatening scenario. Alternatively, vaccines are administered as a combination of pathogen-derived antigens together with compounds that induce or enhance immune responses against these antigens (these compounds are commonly termed adjuvant), since these subunit vaccines on their own are generally not effective." 
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7,771,728.PN.&OS=PN/7,771,728&RS=PN/7,771,728
 

E) "The effect of exogenous corticosterone on West Nile virus infection in Northern Cardinals (Cardinalis cardinalis)
“Corticosterone was administered at levels that individuals enduring chronic stressors (i.e ., long-term inclement weather, food shortage, anthropogenic pollution) might experience in the wild. Corticosterone greatly impacted mortality: half of the corticosterone-implanted cardinals died between five - 11 days post-inoculation whereas only one of nine sham-implanted (control) birds died.

”No differences were found in viral titer between corticosterone- and sham-implanted birds. However, cardinals that survived
infections had significantly higher average body temperatures during peak infection than individuals that died.

”In sum, this study indicates that elevated corticosterone could affect the survival of WNV-infected wild birds, suggesting that
populations may be disproportionately at-risk to disease in stressful environments.”
http://7thspace.com/headlines/410671/the_effect_of_exogenous_corticosterone_on_west_nile_virus
_infection_in_northern_cardinals_cardinalis_cardinalis.html

 

WHY do they put Thimerosal in vaccines?  To prevent LYMErix or Fungal antigens or TLR2-agonists, since they cause immunosuppression and the activation of the live, attenuated viruses.  They took the Thimerosal out and what happened?  'Autism" cases exploded.


Now, here are some doctors writing in the NYT about "BRING BACK THE THIMEROSAL" due to contaminated vaccines and the INCREASE in vaccine brain damage events:
http://www.nytimes.com/2012/12/17/health/experts-say-thimerosal-ban-would-imperil-global-health-efforts.html?_r=0

They say:

"But a proposal that the ban include thimerosal, which has been used since the 1930s to prevent bacterial and fungal contamination in multidose vials of vaccines, has drawn strong criticism from pediatricians.

"They say that the ethyl-mercury compound is critical for vaccine use in the developing world, where multidose vials are a mainstay.

"'Banning it would require switching to single-dose vials for vaccines, which would cost far more and require new networks of cold storage facilities and additional capacity for waste disposal, the authors of the articles said.'"

^^^  They are revealing why so few doctors' own kids get vaccines-acquired brain damage.  Their kids probably get the first shot out of the vial.
 

---

Given the treatment of Veterans and Whistleblowers, it's no stretch to consider to how "the government" treats the vast majority of their sick like crap.  Consider the 12 million (NIH's own stats) with Chronic Fatigue Syndrome and Fibromyalgia alone.  These 12 million are abused because to treat them would be to identify the cause of the illness, which reveals the cause of the AUTISM epidemic.  "The government" is no more than a Corrupt Practices Organization, just like NAZI Germany - it's a RICO.  They're the street thugs of the Corporate Mafia.  Take vaccines:  "Here, you are buying this 'protection' or else we'll have the CPS Gestapo bag you and your kids."

 

Remember the Logo. 

It's CRYME DISEASE. There is no such thing as "Lyme Disease."  Allen Steere invented that in Europe in 1992 and was intended to go with the fake OspA (fungal, caused immunosuppression) vaccine trials.  Remember, Allen Steere is a CDC officer, and the CDC's strict relationships include vaccine manufacturers and Kaiser-Permanente.  CDC staff, when they leave the CDC, often go to work for Kaiser-Permanente as "advisors." 

 

Here is how it ALL started...

Says Crazy Eddie McSweegan to Senator Barry Goldwater in 1986:

 

 

 

 

 

 

 

 

Porton Down staff admitted ^^ Lyme was given over to industry, presumably after they and the CDC messed with it.

TRANSCRIPT OF THE ABOVE Sweeg Letter to Senator Barry Goldwater:

"I'm certain that the DoD would attempt to make a case for its continued in-house research programs.  Yet, the defense department does not feel the need to make its own fighter planes, or Christmas fruitcakes, or nerve gas, or acetylcholinesterase [nerve agent antidote, revealing what a nerve agent is].  Why then to they feel that they need to have their own programs to study the mechanisms of campylobacter diarrhea, or lectin-ligand interactions, or the cloning of rickettsial surface proteins genes [means making Lyme vaccines]?  These are projects for university graduates, not Navy and Army lieutenants.  If the DoD thinks these are topics of military importance they should be willing to fund the research in the form of grants to universities, and contracts to businesses [like the ALDF.com which then parasitized the IDSociety.org].  The end result would be a leaner, cheaper defense department and stronger universities and biotechnical businesses.  Everyone would benefit."  - Eddie Da Sweeg


Also, read carefully the 1995 email exchange between Crazy Eddie and Durland Fish, where Durland flatters Sweeg and then asks about the status of his money - this is how to work a psychopath.  They planned on stalking and harassing the Lyme Disease Foundation as you will see.  And then Sweeg did.  The lawsuits followed...
 

 

 

 

CDC is at the center of the scam.  We do not know who gave them permission to try to change the diagnostic standard to suit their own personal patents bottom line.

 

 

 

 

 

 

 

 

 

 

 

 

Steere (CDC officer) goes to Europe to falsify testing in 1992, leaving out all neurological cases of Lyme (and OspA and B), since by 1989, "50%" of those cases were known to be "incurable," according to Steere, Sigal, Dattwyler and Luft (1989, IDSA Reviews).

In 1992 Allen Steere went to Germany, with, as he claimed, bogus strains and recombinant OspA-B without the lipid attached.  You need the lipid aspect of the Pam3Cys molecules or else you won't make antibodies.

“The group 1 strain of B. burgdorferi, G39/40, used in this study and in the previous study of US patients was isolated from an Ixodes damini tick in Guilford, Connecticut [21].  The group 2 strain, FRG [Federal Republic of Germany], was isolated from Ixodes ricinus near  Cologne [22].  The group 3 strain, IP3, was isolated from Ixodes persulcatus near Leningrad [23].  All three strains used in this study were high passage isolates [which drop plasmids :) ], which were classified by Richard Marconi (Rocky Mountain Laboratory, Hamilton, MT) using 16S ribosomal RNA sequence determination as described [11, 24].  The recombinant preparations of OspA and OspB used in this study were purified maltose- binding protein-Osp fusion proteins derived from group 1 strain B31 [25].  The fusion proteins contained the full-length OspA or OspB sequence without the lipid moiety or the signal sequence -"

-- illegal, plasmid-dropping, antigen-and-antibody dropping, “high passage strains” and OspA and B with no lipids attached (not likely to produce antibodies), resulting in the new, 1994, Centers for Disease Control (CDC), “case definition” of “Lyme Disease (8, 9)."

This report of Allen Steere's was NOT contained in the Dearborn booklet, whereas the other one found on this page was.

 

CDC's former criteria for a positive test was this:

 

No ELISAs, only ^^^ Western Blots to look for New, IgM bands.


Here, next, you can see that once again, Gary Wormser and CDC officer Mark Klempner show that only HLA-linked arthritis is a case of "Lyme Disease," (only they test positive) and that those arthritis-only people and those HLA-people have no other symptoms besides arthritis:



FROM ...   http://www.journals.uchicago.edu/doi/pdf/10.1086/432733

The reason they did this was because they found OspA vaccination caused the same former "systemic disease" known as "Chronic Lyme" and the "New Great Imitator."  CDC officer Alan Barbour and Durland Fish published a Chronic-Systemic-Lyme-victim-trashing article in 1993 (here, pdf) and it it they revealed that the Phase I and Phase II OspA trials were underway.  1993 is before 1994 or Dearborn.  Clue.

But we have a lot more evidence that they knew OspA vaccination caused a "multi-system disease" indistinguishable from the former "Chronic Lyme."

See More At:  "Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure.... - Dave Persing who with his company CORIXA in his RICO-Monopoly patent listed formally with the Securities and Exchange Commission as "Partners" with Imugen (Steere) and Yale's L2-Diagnostics (Schoen).



They left OspA and B out of the diagnostic standard because of what was to happen AFTER LYMErix was on the market (The MONOPOLY on Vector Borne Diseases Testing to be monopolized by Corixa, Imugen and Yale's L2 Diagnostics)

"The present invention provides a method useful to detect a B. burgdorferi infection in a subject. The method provided by the invention is particularly useful to discriminate B. burgdorferi infection from OspA vaccination, although it is sufficiently sensitive and specific to use in any general Lyme disease screening or diagnostic application. Thus, the method of the invention is particularly appropriate for large scale screening or diagnostic applications where only part of the subject population has been vaccinated or where the vaccination status of the population is unknown. "

(Or, appropriate for a monopoly on vaccines, royalties, grants, and testing for all VBDs here and in Canada, as they claimed.)
 

All of this data is already in the hands of the New Haven USDOJ since 2003.


Another View of the CDC's Dearborn Falsified Testing (Western Blots) - filed with the USDOJ RICO Complaint

 

This is how methods are REALLY validated:
http://www.labcompliance.com/tutorial/methods/default.aspx

 

Compare to the 1991 Yale Flagellin Method, where they make the test more SENSITIVE (LoQ) or detect lower concentrations of the SPECIFIC (Bb's Fla region, "G" they call it) and even go so far as to spike more of the G fragment in to ELISAs:

"Comparable sensitivities (88 to 94%) were noted when sera from 17 persons who had erythema migrans and antibodies to whole-cell B. burgdorferi were tested against the p41-G antigen. In tests of a second study group of 36 persons who had erythema migrans but no detectable antibodies to whole-cell B. burgdorferi, 3 (8%) were positive when the p41-G antigen was used. Assay specificity likewise increased when the p41-G fragment was included in an ELISA with human sera containing treponemal antibodies. Recombinant flagellar proteins of B. burgdorferi, such as the p41-G antigen, can be used in an ELISA and may help confirm Lyme borreliosis during early stages of infection and improve specificity."
http://www.ncbi.nlm.nih.gov/pubmed/1280650

So, if they knew how to validate a method as shown above, and developed a valid one to detect very low concentrations of the specific flagellin for Borrelia burgdorferi (US Patent 5, 618,533), why did the CDC and their front "non-profit", the ALDF.com change the diagnostic standard at Dearborn in 1994? 

The CDC staff own most of the patents for vector-borne disease vaccines and test kits (Alan Barbour, Barbara Johnson, and Allen Steere through the Mayo Clinic's former David Persing with this RICO-Monopoly-on-Post-LYMErix-Blood-Testing Patent for Corixa, Imugen and Yale's L2 Diagnostics), and they knew OspA caused systemic disease from the early Phase I and II trials of OspA. 

In 1993, CDC officer Alan Barbour and Yale's Durland Fish together wrote an article trashing Neurologic Lyme patients, and in it they reveal that the OspA trials were underway :)    1993, Barbour and Fish slam Neurologic Lyme victims, .pdf

1993 is before 1994 (Dearborn)

Standard practice in America:
Blame the Victim or the Victim's parents. 
Deploy psychiatry, call the Gulf War Illness Vets fairies and cowards (Simon Wessely).
Label Pharma and CDC Whistleblowers "Terrorists" and "Threats to Public Health"
Recycle the idea that people who psychogenically produce real, verifiable, physically detectable illness signs in themselves without having any real illness are also >>> the same people who believe in Bigfoots and the Jersey Devil, when the reverse is true.  Whoever thinks "psychogenic" is anything different than paranormal apparitions and apports - conjuring - would have to be either insane or retarded.

---

Memorial Day, Monday, May 26, 2014:

This just in - this fantastic summary by a new patient who I only met about a week ago; this is how much she has learned in about a week, seriously.

Borreliosis Basics – Version 3 (updated Oct, 2014)

Borreliosis is a “multi-system disease” caused by a spirochetal parasite. A spirochete is a spiral-shaped parasite with a unique mechanism for movement that features an internal bundle of flagella (think of it as an internal bungie). Borrelia and Treponema (close cousins) have never been known to be curable with antibiotics “or even arsenic” (Jay Sanford, U.S. Uniformed Services Hospital, 1975, and Andrew Balfour, U.K. Wellcome Research, BMJ, April 1911). They typically reside in the brain/nervous system and are found in the brains of Alzheimer’s victims at nearly a universal rate as compared to controls or non-Alzheimer’s victims. (Riviere, Oregon Health and Sciences University, 2002).

Borreliosis is transmitted primarily through the bite of an infected tick, but also may be transmitted by insect vectors and transplacentally to an unborn fetus, according to Yale and the NIH (Paul H. Duray). If not treated immediately with antibiotics, the infection can persist for years and cause neurological diseases such as MS, Lupus, cancer, Chronic Fatigue/Myalgic Encephalomyelitis, ALS (Lou Gehrig's Disease), Cancer, and Alzheimer’s, according to IDSA and the CDC. Thus, borreliosis is commonly referred to as “The Great Imitator” or “New Great Imitator.” As you will later see, it would be more correct to call Lyme and Syphilis, the Great Detonators.

Borreliae parasites have the ability to shed (bleb off) their outer membrane lipoproteins to both evade detection by –, and disable- the immune system. They modify their numerous different surface proteins in a mechanism commonly known as antigenic variation. These lipoproteins are called “outer surface proteins” (Osps, rhymes with wasps) and “variable major proteins” (Vmps, rhymes with wimps). The blebbing, or shedding, of OspA and similar antigens by the spirochete sends the immune system after the sloughed-off or pinched off antigens like OspA (much like flak confuses radar), allowing the spirochete to hide from immune attack and persist in the host (Barbour and Barthold, The Scientist, 1996). This mechanism renders antibodies useless (note: no vaccines can therefore be made against these surface antigens).

These outer surface lipoproteins, such as OspA (the Lyme "vaccine"), are TLR-2 agonists or fungal antigens, meaning they essentially disable the immune system. They are very similar to toxins in this regard (see post-sepsis or post-septic shock survivors, Washington University, St, Louis, 2014). They do this by way of tolerance to other fungal antigens (TLR2s) as well as tolerance to other antigen types, managed by other TLRs, such as viruses (TLR7/9) and other bacterial TLR4-agonists (“cross-tolerance”). [(Clifford Harding, Case Western Reserve): "Because IRAK1 is required for TLR7/9-induced IFN-I production, we propose that TLR2 signaling induces rapid depletion of IRAK1, which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2 inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes that express ligands for both TLR2 and TLR7/TLR9, or responses to bacteria/virus coinfection." http://www.ncbi.nlm.nih.gov/pubmed/22227568 ]

Tolerance means the immune system will stop responding to the antigen. Cross tolerance means spreading or expanding normal disease-combating insufficiency to other antigen types such as TLR4 agonists or TLR7/9 agonists (viruses). Exposure to shed borrelial lipoproteins causes fungal tolerance in the blood and the inability to get rid of mycoplasma/eperythrozoons (also bearers of fungal antigens like OspA) from the blood, especially from the red blood cells. It is well known that fungi infections in the blood, particularly these blood parasites, the eperythrozoa or mycoplasma, causes fatigue, because they, by attaching to the surfaces of- and within- red blood cells hijack sugar metabolism and change the osmotic potential of the red cell membrane, rendering Oxygen unable to cross the red cell membrane, starving the body cells of Oxygen.

This generalized immunosuppression as a result of exposure to borreliae and their shed varying surface antigens creates an environment where common opportunistic infections are reactivated, such as latent Epstein-Barr, and cause chronic disabling disease. We know Lupus and MS are generally accepted as being caused by the herpesviruses (Epstein-Barr, HHV-6, or Cytomegalovirus, etc., or some combination of them). We have called chronic Lyme and LYMErix Disease: Tick-, or Lyme-, or LYMErix-Acquired Immune Deficiency (TAIDS or LAIDS).

Since we knew Lyme was formerly called the New Great Imitator because it caused Lupus, MS, etc, it now appears that the TLR2-agonists shed by these spirochetes cause the tolerance and cross-tolerance that reactivates latent herpes viruses, particularly Epstein Barr Virus (EBV). This came to light because the OspA vaccines caused the same systemic, “multi-system” (Dave Persing, Mayo), “protean” (Ben Luft, SUNY-SB) disease in those victims. Reactivation occurs thru IL-10, the inhibition of apoptosis of OspA or EBV infected cells, the mechanism shown above re TLR2-signaling and the inhibition of IFN-I, and apparently generalized immune activation or common cytokines. One group of researchers simply reported:
“Certain cytokines, particularly TGF -beta, can also induce lytic viral infection in a subset of Burkitt lymphoma lines in vitro, and could potentially reactivate EBV in vivo (Fahmi et al., 2000; Adler et al., 2002). The interaction between CD 4 T cells and EBV -infected B cells has also been reported to induce lytic infection (Fu and Cannon, 2000). Finally, there is increasing evidence that severe host cell stress in response to many different toxic stimuli (including chemotherapy and irradiation) can induce lytic EBV infection (Feng et al.,2002a, 2004; Westphal et al., 2000; Roychowdhury et al., 2003).” – Kenney, 2007, from the book, Human Herpesviruses, Biology, Therapy, Immunoprophylaxis, chp 25.

Recently, it was reported in the New York Times by Jane Brody quoting the National Institutes of Health’s Adrianna Marques, who now manages the Lyme-MS Division of NINDS (her boss, Roland Martin, quit the NIH and returned home to Germany once he found out OspA was responsible for the MS outcome of Lyme), "Complicating the picture is the fact that some people with PTLDS symptoms apparently never had Lyme disease in the first place, Dr. Marques said in an interview. There are other infectious organisms — Epstein-Barr virus, for example — that can produce similar symptoms and may be the real culprits." - When Lyme Disease Lasts and Lasts, July, 2013.

Chronic EBV is probably the main driver of all these New Great and Great Imitator diseases. Inasmuch as the Infectious Diseases Society of America, Yale, the CDC and related participants in the OspA vaccine fiasco vehemently oppose antibiotic treatment for Lyme for whatever [false] reason, the truth is that they worry antibiotic resistance will be acquired by the OTHER infections such as the fungal types, such as mycobacteria and mycoplasma for which we now have become carriers.

--- USDOJ OCCUPY, All Abused Groups, Summer, 2014

And the FBI doesn't know any chemists, LOL...

FACTOID re Petrodollar Fags of the New World Order:

A dude from the FBI, New Haven, CT, told me on the phone that the FBI does not have any chemists and they do not know how to contact or hire any chemists.  'Completely true, and you know it is true, because you can't make something like that up (we all watch TV crime shows where they flaunt their analytical chemistry skills).  

The idea was to find someone to inform the FBI about the INVALID Dearborn method to detect Lyme, vs the Yale-patented, valid method, US Patent # 5,618,533-; valid per the FDA's rules on the validation of an analytical method -, and then prosecute it.

I thought it would be important for America to know that the FBI would dare to say such a thing to an American citizen.  "We don't have any chemists," and "We don't know any chemists," and "We don't know how to contact any chemists," they said.  LOL.

You can't say "fag" on Facebook or they put you in FB jail, but we all know it means only cowardice - attacking truth-tellers (everyone knows the standard practice is America is to jail whistleblowers) rather than taking on corporate fraud and defending the people against mass-murdering criminals.

That's straight up cowardice.  That's the FBI, USDOJ, and NSA and DHS.  Straight up cowards.  Disgusting.  Even more revolting than the anti-gay closet-gay Republicans.  Even more revolting than the "Christian" politicians with 3 wives...
 

"Woe to you scribes and pharisees; hypocrites.  You have left out the weightier elements of the law; justice and mercy.  You strain out a gnat, while swallowing a camel."
 

"Government" "employees" debase all professions, not just their own.  They're low-lives.  Inverted manhood.

Francis Collins (NIH Director) caught in a scandal over hiding information about yet another a sicko study on children in Alabama. 

This was AFTER that asshole Collins threatened me with "sending the police" when I asked what OspA was!

The latest VA scandal - which of course is old news and involved hiring Simon Wessely to trash sick war veterans.

FBI stalking Occupiers...

FBI pretending to be Black terrorists, carrying copies of To Kill a Mockingbird...   ◄You can't make this shit up!!!

Woe to you.  China and Russia now have an energy and geopolitical alliance against the Hegemon.  The Great Pervert.  The Great Whore, Babylon.  The Petrodollar Beast in whose "sign" we calculated (head) and was the former medium of international exchange (hand).

And did you know, coincidentally, that the surviving Fatima witness said we were in Revelation now?  Chapters13-18?  What does that section talk about?  The Great Whore being abandoned and burning up...

 

---

THIS JUST IN (From the CDC, 1964):  How to dessicate and weaponize your Borrelia :)

And now you know why the NIH Rocky Mountain Bioweapon Lab recruited Willy Burgdorfer, a spirochete expert from Switzerland - so he would sign a piece of paper keeping his mouth shut when he ran across this :)

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC277387/pdf/jbacter00438-0287.pdf

Add that to the other data we have and of course all their Effin LIES about the viability of the cyst form :)

This was a bioweapon, little doubt now.

See more at:

 

 

21. Objectives
Biological antipersonnel agents are those which are effective directly against man and are selected based on their ability to cause death or disability through disease.  While these agents might be employed against selected individuals, their main value appears to lie in producing mass casualties over large areas with resulant physical and psychological effects that would weaken or destroy our [?] ability to resist aggression.

 

 

 

http://books.google.com/books?id=kqQXAAAAYAAJ&pg=PA21&lpg=PA21&dq=antipersonnel+and+fungi+and+viruses&source=bl&ots=bvZ-i0BCEA&sig=sqZkG2Lr9zAV0iV1mXX39wkcRbs&hl=en&sa=X&ei=dfZzU7_uDKGosASEuIGYAQ&ved=0CCcQ6AEwAg#v=onepage&q=antipersonnel%20and%20fungi%20and%20viruses&f=false

And see more at http://www.actionlyme.org/BRITISH_PSYCHIATRY.htm
 where the point of stoner drugs like marijuana is to make the population docile and not willing to fight.  And then consider that they wanted to give LYMErix to everyone and you have a picture of Uncle Sam/NWO wanting to make the whole world as stupid as they are.  To Wit:  The Rockefeller family never produced any kind of an academic.

" Then in the spring of 1943, they perfected the right mix of marijuana and tobacco to produce a “state of irresponsibility” in the subject.
... The Overholser group gave marijuana to U.S. soldiers at Army bases throughout the country, supposedly to aid in the search for subversives. Later, during the 1950s and 1960s, the strategists of the MK- Ultra project would utilize the same channels of influence with U.S. security agencies to let them transform a generation of youth into dope users."
 

Deploy a bioweapon like Lyme or LYMErix, then deploy psychiatry to stupidly blame the victim.  You get this.

===

See the chapter of Cryme Disease on Allen Steere, where the US Army has a manual for soldiers re avoiding inhaling dried animal urine, as it might contain spirochetal cyst forms, here. 

http://www.afpmb.org/pubs/dveps/haiti.pdf  ← US Army tells soldiers to be careful not to inhale desiccated spirochetes, LOL (page 13 of the .pdf):

                           

 

New - Borrelia listed as as bioweapon, in both the tick-delivery and weaponized/aersolized form :)

http://entomology.montana.edu/historybug/insects_as_bioweapons.htm

"In 1951, BW and chemical warfare were incorporated into official strategic planning by the armed forces of the U.S. Brig. General Rothchild, chemical officer of the Far East command, in 1953 wrote that BW could have played a vital role in the Korean War, by distributing anthrax or yellow fever pathogens into the cold air flows that travel from Siberia through the populated areas of China.

"Clearly, BW received strong support among the brass in the U.S. and British armed forces. By the end of the 50's the Fort Detrick labs were set up to breed 130 million yellow fever mosquitoes a month, infect them with yellow fever, and deliver them to the enemy via cluster bombs or from the warheads in a Sergeant Missile. Also, the facilities could accommodate the breeding of 50 million fleas per week. By 1960, the labs were experimenting with malaria, dengue, cholera, anthrax, and dysentery, relapsing fever [Lyme Disease], tularemia...

"In the early 60's, insects as BW vectors fell out of favor with the scientists and planners. This was due in large part to the successful development of dry biological formulations of toxins and microbes.

"With dry formulations of BW agents, the practicality and ease of disseminating diseases was greatly increased. It became easy for pneumonic plague, botulinum toxin, q-fever, and other diseases to be spread reliably and efficiently without the need for insects.

Yep.  :)   They tried to hide it for years, removing the terms Borrelia and Relapsing Fever, but it can still be found.

And just think, if those assholes Sweeg and Durland had not been deployed on us, the ruse might have been maintained longer.  So, RULE:  Never deploy obvious psychopaths when you have a bioweapons crime you want covered up :)
 

---

"God removes His Grace" from the Pervert Nation... that can't get anything right and all of whose institutions have clearly failed.  So, what's that all about?  Why does the United States suck at everything?

Why isn't anyone in Boston (or CT, even stupider) able to handle the chronic fatiguing/mitochondrial dysfunction illness of a child? 

The answer reveals the mechanisms of disease in the epidemics of vaccines-acquired "Autism" and Childhood Leukemia, not to mention the CDC's infamous "stealth disablers" - which are FUNGAL (produce no antibodies)... It's all meant to be a secret.  :)

So, do like the "doctors" do:  Make sure your kid has the first shot out of the vial, at the very least.  Thimerosal was put in vaccines to prevent fungi or LYMErix since these fungal antigens injected together with live, attenuated viruses result in the child getting the virus instead of the antibodies or the protection:

"But a proposal that the ban include thimerosal, which has been used since the 1930s to prevent bacterial and fungal contamination [like LYMErix-KMD] in multidose vials of vaccines, has drawn strong criticism from pediatricians.

"They say that the ethyl-mercury compound is critical for vaccine use in the developing world, where multidose vials are a mainstay.

"'Banning it would require switching to single-dose vials for vaccines, which would cost far more and require new networks of cold storage facilities and additional capacity for waste disposal, the authors of the articles said.'"

http://www.nytimes.com/2012/12/17/health/experts-say-thimerosal-ban-would-imperil-global-health-efforts.html?_r=0

^^^  They are revealing why so few doctors' own kids get vaccines-acquired brain damage.  Their kids probably get the first shot out of the vial.

CDC does not recognized diseases without antibodies.  Clue.

There is no National Institute of Immunosuppression and Infectious Diseases, there is only ALLERGY (the opposite) and Infectious Disease (NIAID).  Clue.

The NIH themselves have stated that there are 8 million Americans with "Fibromyalgia" (no-antibody disease) and the NIH has said there are 4 million people in this country with "Chronic Fatigue Syndrome" (no-antibody disease).  Yet they do not throw a bloody red nickel at these diseases. 

Clue.

---

---

The NIH has officially debunked psychiatry and their bullshit, which means that the people who kidnapped Justina Pelletier have some explaining to do. 

Here we see a psychiatrist in Fox News not even question the illogic of claiming that people can produce valid medical illness biomarkers with their psychogenic powers alone, when, we know if anyone had such abilities they would not inflict the disease on themselves, but upon people like the DCF or psychiatry, ... while at the same time claiming they have no scientific evidence for how someone could produce such scientifically valid illness signs in themselves with their magical brains:

Follow:    "... causing her to believe she is medically ill, when she is not—that they have kindled in her a 'somatoform disorder' in which bodily symptoms actually have purely psychological roots, not anatomic ones..."

And: "First, we lack sufficient research data to back up my clinical experience and professional opinion (which some psychiatrists would agree with and some would disagree with)."
http://www.foxnews.com/health/2014/03/28/dr-ablow-sure-parents-can-make-their-kids-sick/

So, we see ▲ that one can assert that another person has magical powers... without explaining how this magic is done, while at the same time discounting all the scientifically valid medical markers.  It's the same thing as saying Justina or her parents are witches.  Now, among the important data missing from the analysis is that Justina's mother has "Chronic Fatigue Syndrome."  That would indicate there might be mold in the house, or else someone got a tick bite  :)

CDC themselves have published that mold or Lyme or LYMErix exposure could cause the reactivated EBV to produce mitochondrial dysfunction.  They, the CDC and the NIH both know Chronic Fatigue/Lyme are chronic active Epstein-Barr and TLR2-agonist (fungal) Tolerance (similar to Endotoxin Tolerance)...

Note also that this crazy psychiatrist would dare to assert that there could be opinions about whether there are facts ("we lack sufficient research data... which some psychiatrists would agree with and some would disagree with").  That's too funny for words.  Apparently none of these psychiatrists know what a fact is, or that science and facts are intended to get rid of opinions.  I would point out that NASA and the Jet Propulsion Labs do not consult with psychiatrists, and neither does General Motors or Toyota.  They're all quite confident in the Laws of Physics and similar physical realities.

REALITY REBOOT

The 5 Valid "Axes" of Mental Compromise - (discard the current DSM Matrix-esque framework, says the NIH)

• IN THE BEGINNING...

I will tell you what you already know, which is where to find psychopaths (these are "people" who have no conscience and enjoy seeing other people suffer; they're actually "perfectly possessed" so they have no need for frontal lobe or executive function, thus that area of the brain shrinks from disuse):

http://samfordcrimson.com/?p=4734

Careers with most psychopaths 

1. CEO
2. Lawyer
3. Media (TV/Radio)
4. Salesperson
5. Surgeon
6. Journalist
7. Police Officer
8. Clergyperson
9. Chef
10. Civil Servant

• CONFIRMING the Disuse-related BrainShrink, see the Low-IQ-and-Enlarged Amygdala results shown in Conservatives.  Do you wonder which comes first, the cowardice or the Low IQ and racism? (You'll see it is cowardice-driven.)
• .
• Let's be clear on how this develops:  What is COWARDICE and how it is related to PRIDE, particularly when PRIDE/COWARDICE attempts to trump reality with their personal will?
• .
• A person is AFRAID of truisms or even other people, when the concept or the person threatens the high esteem with which they hold themselves.  Acts of jealousy or envy come from is THE FEAR OF someone appearing to be greater than the perceiver/jealous person in some way.  And that's why some of the knuckleheads you know refuse to listen to reason.  It's ONLY fear.  Only fear.  Only cowardice.  (People like this like to attack your character rather than the merits of the facts that come through you.  Pity them, for their cowardice shines through, even though calumny, slander, perjury and libel are Capital Sins and require restitution.)
• Again, no one would object to what is true, UNLESS, they have first been tempted to feel they are above the rules or have drunk the psychiatric Kool-Aid where PUTTING ONESELF FIRST! is "normal" or "natural" because, says psychiatry, "the animals do it," or "self-interest is evolutionarily adaptive," or even that debauched behavior is "normal" because of the frequency (see "Diagnostic and STATISTICAL Manual") of said debauched behavior allegedly making it "common" or "normal."  It is not. The theory does not scale up from the individual to society.  Society only works when the social construct requries recognition that we have to take care of each other.
• .
• SECONDLY, most people don't know this, but the psych.org's "guidelines" on the diagnosis of "Bipolar" is that you have to rule out MS, first.  That is, you are REQUIRED as a psychiatrist to determine if the likes of NOT FEELING GOOD could make you crabby.  You are REQUIRED not to treat DELIRIUM with psychotropics (CFIDS, Fibro, Mito, Chronic Lyme, MS, and similar chronic encephalopathies result in chronic inflammation of vasculature and glial cells, astrocytes, and microglia), resulting in a delirium, because, their own "guidelines" state, if you treat the medical delirium with psychotropics, you will make the delirium worse.
• See the huge lawsuit against Eli Lilly for treating Alzheimer's or dementia patients with Zyprexa. ;)
• So we can take MS and Lyme or CFIDS to be the same per the current DSM and guidelines on Delirium and not Dx-ing a real medical condition as "psychiatric."  This is flat-out malpractice (see the Justina Pelletier case).

So, it is already required that no one can Dx anything psychiatric  - or "treat" it - without doing a medical rule out, first.  (And this is while the very people who determined the biomarkers of the Lyme/CFIDS delirium call us "psychiatric.")
http://www.actionlyme.org/BIOMARKERS.htm

• =============

WE KNOW the NIH has just debunked psychiatry as NOT VALID - according to the rules applied to other medical sciences (the non-application to Lyme and CFIDS notwithstanding; CFIDS, Fibro and Lyme are still officially considered "psychiatric"). 

• So, what *is* Valid ?

The 5 Valid Axes are:

• 1) Genetic (cognitive profiles, IQs, VIQ vs PIQ, for example, Autistic people have a high PIQ >> VIQ, which was why, I think, Asperger's was left out in the last DSM, since it is common for people to have VIQ>>PIQ),
• 
  2) Organic ("Lyme," HIV, Alzheimer's, Infectious Dementia, MS,... and is determined/assessed using valid biomarkers like contrast MRI imaging, PET and SPECT scans, spinal fluid analysis, QEEGs, etc),
• 
  3) Traumatic (both physical and emotional, like the abuse of telling sick people they're psychiatric or "are using their psychic powers to cause genuine, valid, biomarkers of illness in themselves"),
• 
  4) the Influence of Spirit (SEE BELOW; ie., the Fatima Miracle of the Sun Event or ... like the fake "aliens" who are actually demons just like the dudes who spoke to Mohammed and Joseph Smith),
• 
  5) and personal Free Will. 

In that order.

• If you read Hostage to the Devil by Malachi Martin and listen to his interview tapes on Coast to Coast and elsewhere you will see that he very clearly points to the fact that psychiatrists can't help the majority of people in jails and mental "hospitals" because, well, the demons reveal during an exorcism, that psychiatrists "work for us."
• http://www.amazon.com/Hostage-Devil-Possession-Contemporary-Americans/dp/006065337X
• .
• Additionally, we have verification of why psychopaths (perfectly possessed) can pass a polygraph test while lying, and this data, too is valid according to the criteria:
• "But as useful as hypocrisy can be, it’s apparently not quite as basic as the human instinct to do unto others as you would have them do  unto you. Your mind can justify double standards, it seems, but in your heart you know you’re wrong."
• http://www.nytimes.com/2008/07/01/science/01tier.html 
  At the same time, this ▲report confirms the law enforcement point of view on human evil - you know it when you do something wrong or selfish or evil-, but that might not include the perfectly possessed such as most psychiatrists who undergo the cult-like initiation of psychoanalysis, where they confess all their pervert ideas to each other, just like the Skull and Boners.

The reason a psychopath can pass a polygraph is because they're actually demons ("perfectly possessed" means the human spirit is not there any more, so there is no retrieving them via exorcism), and lying is what they do.

• CONFIRMING the existence of Intelligences without bodies is the Fatima Miracle of the Sun event where 60,000 people showed up on a hillside at the designated (by the apparition, who identified herself as "The Lady of the Rosary" which refers to an earlier apparition at Lourdes), and everyone, skeptics and newspaper reporters and photographers alike, saw something happening with the sun and this was recorded in the newspapers.  You can see evidence of this today online, on Google Images.
• .
• A SECOND form of verification would be seen among the likes of people who have verified "psychic" abilities or have been verified to be able to read Auras, like Lorraine Warren and others more commonly seen on TV in these ghost hunter shows and psychics who aid cops or detectives.
• See the Jewish NDE (you will see that this guy regretted taking the pervert advice of pervert psychiatrists):
• https://www.youtube.com/watch?v=c0Ns3D6Tpfs
• .
• The problem is with dealing with this preternatural realm is the validity of the sources.  Everyone remembers the eye-less boy apparition photographed at Amityville.  (Because the apparition of the boy was incomplete, that is a sign that it comes from the Dark Side). 
• Other people who partake in psychic readings can be misled since some of these spirit contacts come from the preternatural rather than the Supernatural world.
• .
• A THIRD source of verification that there are Intelligences without bodies would be the likes of crop circles and astronauts (one with 2 PhD's) admitting they have seen UFOs...
• .
• Psychiatrists of course do not even engage this world.  They refuse to acknowledge what the rest of us do, which is that for instance MIT deployed some scientist students to replicate crop circles using electromagnetic energy, since the science says this was the source of the bent, ALIVE, crops: Electromagnetic Energy that created these intelligent designs in the crop circles.  They do not consider that when a person "hears voices" they might be the same "voices" heard by psychics ;)  They do not consider that the astronauts who have seen UFOs would probably not be making anything up.
• .
• This is the actual audio recording of the REAL exorcism of Annaliese Michel (Emily Rose) which you can tell is real by simply trying to maintain yourself that growly voice - you soon lose your voice.  In it also, the demons allude to the fact of the problem in the Vatican being Vatican II and "Modernism" where psychiatry's influence is what corrupted the priests (the demons say the priests should be educated in the seminaries rather than the universities! since in the universities, they will be trained in the DIABOLICAL PERVERSION of pop psychology where they try to use math (statistics) to demonstrate that the frequency of debauched behavior in humans makes it "NORMAL" or "LIKE THE BEASTS."
• https://www.youtube.com/watch?v=blLJk3PqHl4 

YOU WILL SEE that if you apply this new framework, everything fits:  Drug Addiction (ceding over your will to the Dark Side), Coward/Bully cops, psychopath lawyers and judges, you'll understand why the stupid whorey bitches of the Child Protective Services are so stupid....
 

---

---

From now on, just say "Fungal-Viral Synergy" or "TLR2-agonist" if you want to see anyone associated with Uncle Sam Incompetent HHS.gov, Yale, Harvard, or anyone in the biotech field in Boston or New York City spontaneously combust. 

 

You need to be scared because...

Sloan Kettering Cancer Center doesn't know anything about "Infection Control" (MRSA as TLR2-agonist-bearer, like LYMErix), and Tufts, Harvard and Boston Children's Hospital don't know anything about Epstein-Barr, cancer, Lupus, or Multiple Sclerosis.

What really happened with the fake Lyme "vaccine" - A complaint to Sloan Kettering about Daily Beast's Kent Sepkowitz, who allegedly is in charge of "Infection Control" (be scared)  at Sloan.  (And no, OspA or the "C6" nonsense did not prevent "Lyme" spirochetes in dogs, either.  All it did was tolerize them to OspA/fungi/arthritis.) 

The Dearborn and LYMErix stunts were no more than a RICO/Monopoly scam to launch a vaccines and test kits empire for Corixa, L2 Diagnostics and Steere's Imugen.  That was why Steere falsified the case definition (leaving out OspA and B) to suit what happened after LYMErix was "approved" for the market.  All the blood testing for the USA and Canada, and all the grants, and all the DNA in that blood -  both human DNA and vector borne disease DNA found there -, would go to these DNA patenteers because those 3 companies were to be the only ones licensed to test for Lyme after LYMErix (OspA)  was on the market.  The RICO patent (6,045,804) was for a Lyme bug that had dropped the OspA-B plasmid.  Their empire depended entirely on LYMErix surviving all the FDA cuts and flak, and for McSweegan and Fish, et al, to trash all the Lyme and LYMErix victims.  It didn't work.

The fake Dearborn testing schema is still in place - the "case definition" - so that is still a viable homicide charge.  But you can see why these ALDF.com/IDSociety.org assholes persist in their assholish antics such as talking about the fake Lyme vaccine and bringing out "guidelines" (again, based on falsified Dearborn case definition).  They're wanting to appear to believe the Dearborn "case definition" event was real and not a total FRAUD UPON THE GOVERNMENT.

That's all this is about.  Pretending Dearborn was real so they don't have to face charges over it.
 

Think:  When have you ever seen a bunch of "doctors" behave so viciously?

◄ Follow the scam in the order it occurred.

---

140414:  Updates on Immunosuppression and the Activation of Epstein-Barr caused by OspA-like antgens

1) LEAVE IT TO THE CHINESE; Here's more on Pam3Cys (LYMErix and the failed HIV-LYMErix vaccines) and how it inhibits immunity and development/maturation of cell types, starting in the very beginning. Someone asked me recently about Ehrlichia and I revealed we heard they developed in the bone marrow (explaining a lot) at one of the Lyme.org's conferences in the late 1990s - by one of the crooks actually;
In the end, immunosuppression like this from these fungal antigens is responsible for failed childhood vaccines (we call it Autism), Mitochondrial Dysfunction (from activated EBV), CFIDS, Lyme, Fibro, ME.... all the waste basket Dx's:

Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2014 Feb;22(1):183-6. doi: 10.7534/j.issn.1009-2137.2014.01.035.

[Influence of TLR2 and TLR4 agonists on migration of human bone marrow mesenchymal stem cells].

"This study was aimed to investigate the influence of TLR2 and TLR4 agonists on the migration and adhesion activity of human bone marrow-derived mesenchymal stem cells (MSC) and to clarify the underlying mechanisms. The expression of TLR2 and TLR4 on MSC was detected by flow cytometry. The effects of TLR2 agonist (PAM3CSK4) and TLR2 agonist (LPS) on MSC migration and adhesion ability were evaluated with chemotaxis and adhesion test. The results indicated that expressive levels of TLR2 and TLR4 on surface of human bone marrow MSC were (24.5 ± 3.2)% and (91.3 ± 5.2)% respectively. Compared with the control group, the migration activity of MSC toward SDF-1 was decreased significantly in PAM3CSK4 group, while the adhesion activity of MSC was promoted by PAM3CSK4 exposure. However, both the migration activity toward SDF-1 and the adhesion activity of MSC were not changed significantly in LPS-treated group. Further, it was found that PAM3CSK4 [LYMErix or OspA] did not affect the expressive level of CXCR4 on MSC, however, it could inhibit the spontaneous migration of MSC in dose depenclent manner. It is concluded that activation of TLR2 can decrease the migration ability of MSC, which may associate with the decreased spontaneous migration ability and the increased adhesion activity of MSC."    http://www.ncbi.nlm.nih.gov/pubmed/24598674
 

COMPARE THE ABOVE TO what Paul Duray said about OspA/Borreliosis inhibiting maturation of immune cells:

Paul Duray publishing for IDSA (EBV and cancerous B cells)  http://www.actionlyme.org/Duray.htm  

"On occasion, these atypical-appearing large lymphocytes have been misinterpreted in biopsy by several laboratories as cells of a malignant lymphoma or leukemia.  Bb antigens, then, may stimulate growth of immature lymphocytic suibsets in some target organs, as well as in the cerebrospinal fluid (Szyfelbein and Ross 1988).  Usual bacterial infections do not produce such lymphocytic infiltrates in tissue.  These immunoblastoid cells in Bb infections at times resemble those found in Epstein-Barr virus infections.  Does Bb reactivate latent virus infections in tissues?  Do some tick inocula harbor simultaneous infectious agents (ixodid ticks can harbor Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing multi-agent infections in some hosts?  Further studies can clarify these issues by mans of tissue-based molecular probe analysis." - 
 Paul Duray, NCI, NIH, Ft. Detrick, at the 1992 Cold Spring Harbor Crooks' Conference, published in Steve Schutzer's Lyme Disease: Molecular and Immunologic Approaches.

 http://www.actionlyme.org/IDSA_CLINIPATH_DURAY.htm

"Immature B cells can also be seen in the spinal fluid. 
These cells can appear quite atypical- not unlike those
of transformed or neoplastic lymphocytes." --

--1989, IDSA's Journal

 

 

 

 

 

 

 

"Pam3Cys keeps the precursors in a more immature stage"  (IL-17)

J Immunol. 2005 Jun 1;174(11):6639-47.

Role of TLR in B cell development: signaling through TLR4 promotes B cell maturation and is inhibited by TLR2.

Hayashi EA¹, Akira S, Nobrega A.

Author information

• ¹Department of Immunology, Institute of Microbiology, Federal University of Rio de Janeiro, Brazil.

Abstract

The role of TLR4 in mature B cell activation is well characterized. However, little is known about TLR4 role in B cell development. Here, we analyzed the effects of TLR4 and TLR2 agonists on B cell development using an in vitro model of B cell maturation. Highly purified B220(+)IgM(-) B cell precursors from normal C57BL/6 mouse were cultured for 72 h, and B cell maturation in the presence of the TLR agonists was evaluated by expression of IgM, IgD, CD23, and AA4. The addition of LPS or lipid A resulted in a marked increase in the percentage of CD23(+) B cells, while Pam3Cys had no effect alone, but inhibited the increase of CD23(+) B cell population induced by lipid A or LPS. The TLR4-induced expression of CD23 is not accompanied by full activation of the lymphocyte, as suggested by the absence of activation Ag CD69. Experiments with TLR2-knockout mice confirmed that the inhibitory effects of Pam3Cys depend on the expression of TLR2. We studied the effects of TLR-agonists on early steps of B cell differentiation by analyzing IL-7 responsiveness and phenotype of early B cell precursors: we found that both lipid A and Pam3Cys impaired IL-7-dependent proliferation; however, while lipid A up-regulates B220 surface marker, consistent with a more mature phenotype of the IgM(-) precursors, Pam3Cys keeps the precursors on a more immature stage. Taken together, our results suggest that TLR4 signaling favors B lymphocyte maturation, whereas TLR2 arrests/retards that process, ascribing new roles for TLRs in B cell physiology.

OspA was like a Cancer and Autoimmunity-causing "vaccine."  No one has ever thanked us for getting it off the market.

2) POLAND WENT DIRECTLY INTO THE EXPERIMENT to find out if it was indeed OspA or the fake Lyme vaccine that caused the activation of Epstein-Barr:

Pol J Microbiol. 2013;62(3):237-42.

The influence of toll-like receptor stimulation on expression of EBV lytic genes.

Siennicka J¹, Trzcińska A², Cześcik A², Dunal-Szczepaniak M², Lagosz B².

• ¹Department of Virology, National Institute of Public Health - National Institute of Hygiene, Warsaw, Poland. jsiennicka@pzh.gov.pl
• ²Department of Virology, National Institute of Public Health - National Institute of Hygiene, Warsaw, Poland.

Abstract

Epstein-Barr virus (EBV) establishes latency in the resting memory B-cell compartment. It has been recently suggested that maintenance of chronic infection is dependent on periodic reactivation. Although the stimuli for EBV reactivation in vivo during natural infections are largely unknown, there is evidence indicating that heterologous infections could trigger herpesviruses reactivation. The purpose of this work was to identify the influence of Toll-like receptors stimulation on EBV replication in EBV latently infected Burkitt lymphoma cells (P3HR-1, Raji and Namalwa). The cells were stimulated with Pam3CSK4 (synthetic triacylated lipoprotein), PolyI:C (synthetic analog of dsRNA), LPS (lipopolysaccharide from E.coli), measles virus (MeV) and PMA (phorbol myristate acetate). Non-stimulated cells (NS) served as control. EBV expression was investigated at mRNA level for three viral lytic genes: BZLF1 (immediate early, ZEBRA), BALF2 (early, EA) and BcLF1 (late, VCA). Additionally, the effect of stimulation on NF-kBp65 and inflammatory cytokines (IL-lb, IL-6, IL-8, IL-10, IL-12p70, and TNF) was investigated. Stimulation of TLRs led to limited changes in EBV expression manifesting as increase of ZEBRA at mRNA level in cells treated with PolyI:C and Pam3CSK4. Stimulation with PolyI:C, Pam3CSK4 and LPS also lead to considerable increase of NF-kBp65, while increased levels of inflammatory cytokines were observed for IL-8, TNF and IL-6 in cells treated with PMA and MeV. In conclusion, the results of our experiments support the suggestion that TLRs stimulation with microbial ligands influences EBV virus replication.

http://www.ncbi.nlm.nih.gov/pubmed/24459828
Full text pdf  : http://www.pjm.microbiology.pl/archive/vol6232013237.pdf

3) THIS IS THE CDC MENTIONING MITO DYSFUNCTION FROM CHRONIC ACTIVE EBV (see the infamous Justina Pelletier case):

Preliminary evidence of mitochondrial dysfunction associated with post-infective fatigue after acute infection with Epstein Barr virus.  http://www.ncbi.nlm.nih.gov/pubmed/16448567