24 May 2012 

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CDC Greed (won't answer the FOIA)

ELISA = arbitrary cutoff.

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Overview

TUSKEGEE - By Jerry Leonard

1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.

Bush/Gore  Oil/War-(Oct,2000)  

Bush's own explainer (Oct 2000) re: Iraq Oil

The Primer's Shell Game is where these Lyme crooks use the wrong DNA (or RNA) primers (they use the variable Osp primers instead of the non-variable chromosomally encoded) to test the outcomes of treated Lyme victims.

These same crooks use the correct DNA primers when they want to find Lyme or borrelia in ticks.  It's a shell game. They all know that all that they can use for treatment outcomes is non-variable DNA or RNA. 

Mark Klempner never reported his primers in his Chronic Lyme "study," and he would not tell me, either, in an email correspondence I had with him.  Therefore, the 180s have never validly reported on the presence of borrelia in humans, with the exception of Gary Wormser's study of EMs in which he found 2/9 patients who had EM, who had been treated with antibiotics still had spirochetes in their tissues.  This data can be found on the HOW RICO WILL BE CHARGED PAGE

 

1.)

This is a very important article by Mark Klempner.  You may copy the whole thing from my website and keep it, since it belongs to you anyway.  It is Mark Klempner's attempt to discover why ceftriaxone does not cure everyone.  He found that the spirochetes are intracellular and are protected from ceftriaxone. 

That spirochetes are intracellular and hide out and hang our and go into a dormant stage, also known as a serum starvation stage is well- known.  'Among the crooks.

2.)

Allen Steere says:  http://www.annals.org/cgi/content/full/121/8/560   "Patient 12 had had high fever, meningeal symptoms, and subsequent arthritis in 1982. She was noted to have a positive serologic test result for Lyme disease 4 years later and was treated with 2 weeks of parenteral penicillin. She later developed a progressive speech disorder, bradykinesia, and abnormal ocular motor function. Magnetic resonance imaging of the brain showed scattered white matter lesions in the hemispheres and pons, and she was diagnosed with supranuclear palsy. Lumbar puncture showed no selective concentration of antibody in the spinal fluid. Nevertheless, she was re-treated with 2 weeks of parenteral ceftriaxone in 1989 that had no effect on her neurologic symptoms. During the time of observation, this patient died. At autopsy, lymphoid mononuclear cells were observed surrounding the intracerebral vessels in one section. Using Dieterle silver stain, a spirochete was present in the cortex and another was exterior to a leptomeningeal vessel."

(One of Steere's multiply treated patients died anyway with spirochetes in her brain.)

3.)

http://www.ncbi.nlm.nih.gov/s   8769624

Detection of Borrelia burgdorferi DNA by polymerase chain reaction in cerebrospinal fluid in Lyme neuroborreliosis.

Nocton JJ, Bloom BJ, Rutledge BJ, Persing DH, Logigian EL, Schmid CH, Steere AC.

Division of Rheumatology/Immunology, New England Medical Center, Boston, Massachusetts02111, USA.

A polymerase chain reaction (PCR) assay that detects Borrelia burgdorferi DNA in cerebrospinal fluid (CSF) was evaluated as a diagnostic test for acute or chronic Lyme neuroborreliosis. In one laboratory, 102 samples were tested blindly, and 40 samples were retested in a second laboratory. In the first laboratory, B. burgdorferi DNA was detected in CSF samples in 6 (38%) of 16 patients with acute neuroborreliosis, 11 (25%) of 44 with chronic neuroborreliosis, and none of 42 samples from patients with other illnesses. There was a significant correlation between PCR results and the duration of previous intravenous antibiotic therapy. The overall frequency of positive results was similar in the second laboratory, but concordance between the laboratories and among primer-probe sets was limited because many samples were positive with only one primer-probe set. Thus, PCR testing can sometimes detect B. burgdorferi DNA in CSF in patients with acute or chronic neuroborreliosis, but with current methods, the sensitivity of the test is limited.

4.)

In the following report, Steere found persisting DNA in joints for up to ten years, after treatment, even using the wrong primers.

http://www.ncbi.nlm.nih.gov/s8272083

Detection of Borrelia burgdorferi DNA by polymerase chain reaction in synovial fluid from patients with Lyme arthritis.

Nocton JJ, Dressler F, Rutledge BJ, Rys PN, Persing DH, Steere AC.

Division of Rheumatology/Immunology, New England Medical Center, Boston, MA 02111.

BACKGROUND. Borrelia burgdorferi is difficult to detect in synovial fluid, which limits our understanding of the pathogenesis of Lyme arthritis, particularly when arthritis persists despite antibiotic therapy. METHODS. Using the polymerase chain reaction (PCR), we attempted to detect B. burgdorferi DNA in joint-fluid samples obtained over a 17-year period. The samples were tested in two separate laboratories with four sets of primers and probes, three of which target plasmid DNA that encodes outer-surface protein A (OspA). RESULTS. B. burgdorferi DNA was detected in 75 of 88 patients with Lyme arthritis (85 percent) and in none of 64 control patients. Each of the three OspA primer-probe sets was sensitive, and the results were moderately concordant in the two laboratories (kappa = 0.54 to 0.73). Of 73 patients with Lyme arthritis that was untreated or treated with only short courses of oral antibiotics, 70 (96 percent) had positive PCR results. In contrast, of 19 patients who received either parenteral antibiotics or long courses of oral antibiotics (> or = 1 month), only 7 (37 percent) had positive tests (P < 0.001). None of these seven patients had received more than two months of oral antibiotic treatment or more than three weeks of intravenous antibiotic treatment. Of 10 patients with chronic arthritis (continuous joint inflammation for one year or more) despite multiple courses of antibiotics, 7 had consistently negative tests in samples obtained three months to two years after treatment. CONCLUSIONS. PCR testing can detect B. burgdorferi DNA in synovial fluid. This test may be able to show whether Lyme arthritis that persists after antibiotic treatment is due to persistence of the spirochete.

PMID: 8272083 [PubMed - indexed for MEDLINE]

 

 

5)

Alan Barbour on what happens if you wait long than 7 days to treat Lyme very aggressively:

  http://www.ncbi.nlm.nih.gov/   8913478

Antimicrob Agents Chemother. 1996 Nov;40(11):2632-6

In vivo activities of ceftriaxone and vancomycin against Borrelia spp. in the mouse brain and other sites.

Department of Medicine (Infectious Diseases), University of Texas Health Science Center at San Antonio 78284, USA.

Borrelia burgdorferi, the agent of Lyme disease, and B. turicatae, a neurotropic agent of relapsing fever, are susceptible to vancomycin in vitro, with an MIC of 0.5 microgram/ml. To determine the activity of vancomycin in vivo, particularly in the brain, we infected adult immunocompetent BALB/c and immunodeficient CB-17 scid mice with B. burgdorferi or B. turicatae. The mice were then treated with vancomycin, ceftriaxone as a positive control, or normal saline as a negative control. The effectiveness of treatment was assessed by cultures of blood and brain and other tissues. Ceftriaxone at a dose of 25 mg/kg of body weight administered every 12 h for 7 to 10 days eliminated cultivable B. burgdorferi or B. turicatae from all BALB/c or scid mice in the study. Vancomycin at 30 mg/kg administered every 12 h was effective in eliminating infection from immunodeficient mice if treatment was started within 3 days of the onset of infection. If treatment with vancomycin was delayed for 7 days or more, vancomycin failed to eradicate infection with B. burgdorferi or B. turicatae from immunodeficient mice. The failure of vancomycin in eradicating established infections in immunodeficient mice was associated with the persistence of viable spirochetes in the brain during antibiotic treatment.  PMID: 8913478 [PubMed - indexed for MEDLINE]