Contact ActionLyme; submit
CrymeTrainer; Outline for Indictments
Non-HLA-linked diseases and
Mold-Related illnesses (like LYMErix-Disease, Lyme, CFIDS/FM, and Gulf War
Older data on
the incurability of Relapsing Fever
McSweegan trashes Navy for $$$ for ALDF.com
1988, Dattwyler &
about immune-suppressing, seronegative Lyme
1990, CDC: "Diagnose Lyme as if it was Relapsing Fever."
Allen Steere "NeuroLyme won't
test positive," 1990.
CDC officer Allen Steere falsifies testing in Europe
1992, CDC patents with
SmithKline show 2 kinds of Lyme
Barbour and Fish slam Neurologic Lyme victims.
Compare the 2 kinds of Lyme in the RICO complaint
Dearborn Booklet .pdf
invitation to participate in Dearborn .pdf
Dearborn, Who Said What?
Igenex, Harris, Dearborn .pdf
Evidence Lyme criminals knew
LYMErix produced the same "multisystem disease" as "Chronic Lyme"
LYMErix Damage Coverup (short)
120302 NIH Treatments
1998, CIA Oilmen & Israelis plan to overthrow
Saddam for the oil.
ActionLyme/Kathleen Dickson predicts all of Bushie's outcomes in Oct 2000
predicts Bush will have us worshipping his bombs (Shock and Awe"), in Oct 2000
during the Gore Debates
CHAPTER 7, Lyme Crooks Report Treatment
Failure in about 20 reports
for other older references published before the Great Whore of Medicine,
Allen Steere, emerged onto the scene, and when the disease was known as
Associated Chapters: -
Russian Cysts, and
IDSA and the
The Biomarkers Chapter and
Pam3Cys chapters (will
be combined for the actual
print version of the book, CRYME DISEASE)
are about the immune suppression
the New Great Imitator, OspA-Diseases, or
This data, below, is 20+ reports of persistence past treatment and in the
brain by the ALDF/IDSA crooks, themselves (the Lyme crooks;
cabal), that they refused to turn over to the Connecticut Attorney General for a
year and a half before settling
out of court.
Says CDC officer Alan Barbour:
"We treat spirochetal
diseases with other spirochetal diseases to cause a fever because
antibiotics don't work."
This seems like an amazing!
choice of treatment for a disease that does not exist or is easily cured by
antibiotics, and especially, for a disease that has no brain residua.
By CDC Officer Alan Barbour, in 1986, The Biology of Borrelia Species.
In 1975 there was
apparently an international conference on spirochetal diseases. The
following year the conference was summarized in book form, by Russell
Johnson and was entitled, The Biology of Parasitic Spirochetes.
Sanford of the "Uniformed Services Military Hospital" in Bethesda, Maryland,
"The ability of the borrelia, especially tick-borne strains to
the brain and in the eye after treatment with arsenic or with penicillin or even
after apparent cure is well known (1). The persistence of treponemes after
treatment of syphilis is a major area which currently requires additional study
(3,5,10,11)." -Jay Sanford,
US Military Hospital, Bethesda, MD
Johnson later published for the 1989 IDSA Reviews, special supplement on
Lyme and spirochetal diseases that:
"Although spirochetes can often be detected in culture media after 3
weeks of culture, some isolates may not be visible for several months."
IDSA says we need to culture
these suckers for several months. Did Mark Klempner
do that in his bogus long term treatment "study?"
By CDC Officer Alan Barbour, in 1986,
The Biology of Borrelia Species:
"The propensity for borrelia to go to the
brain of infected mammals
suggests that the relationship between these spirochetes and neural tissues is
not trivial. Further study of this attraction and the interaction that follows
may reveal the basis for the significant nerve and brain involvement in Lyme
is self-explanatory. We have previously seen that rodent brains so
reliably were a home for spirochetes that they were actually the culture
media. We also acknowledge that ceftriaxone for brain diseases was a
treatment IDSA themselves used and offered and still use, so Lyme must not
be a self-limiting knee disease as Allen Steere and Yale's Steven Malawista
And here we have in the same CDC officer's 1986 report that there are, gasp, "gemma:"
Russell Johnson's patent for the very first Lyme vaccine
"The chronic forms of the disease such as arthritis (joint involvement),
acrodermatitis chronica atrophicans (skin involvement), and Bannwart's syndrome
(neurological involvement) may last for months to years and are associated
with the persistence of the spirochete. A case of maternal-fetal
transmission of B. burgdorferi resulting in neonatal death has been reported.
Domestic animals such as the dog also develop arthritis and lameness to this
tick-borne infection. For every symptomatic infection, there is at least one
asymptomatic infection. Lyme disease is presently the most commonly reported
tick-borne disease in the United States." --
patent also says:
"The infection may be
treated at any time with antibiotics such as penicillin, erythromycin,
tetracycline, and ceftriaxone. Once infection has occurred, however, the
drugs may not purge the host of the spirochete but may only act to control the
chronic forms of the disease. Complications such as arthritis and fatigue
may continue for several years after diagnosis and treatment."
the time of the writing of this book, CDC officer Alan Barbour has on his
website, the following statement:
"We are taking a multi-discipline
approach, including methods of genetics, cell biology, and immunology, to study
in depth two spirochetal diseases: Lyme disease and relapsing fever. These
tick-borne infections are notable for multiphasic antigenic variation through
DNA recombinations in the case of relapsing fever, the occurrence of chronic
arthritis in the case of Lyme disease, and invasion of and persistence in the
brain in the case of both diseases. ---Alan
In 1994 Alan Steere and other published a report
The Long-Term Clinical
Outcomes of Lyme Disease: A Population-based Retrospective Cohort Study
Nancy A. Shadick; Charlotte B. Phillips; Eric L. Logigian;
Allen C. Steere; Richard F.
Kaplan; Victor P. Berardi; Paul H. Duray;
Martin G. Larson; Elizabeth A. Wright; Katherine S. Ginsburg*;
Jeffrey N. Katz; and Matthew H. Liang
1994 | Volume 121 Issue 8 | Pages 560-567
"Patient 12 had had high
fever, meningeal symptoms, and subsequent arthritis in 1982. She was
noted to have a positive serologic test result for Lyme disease 4
years later and was treated with 2 weeks of parenteral penicillin.
She later developed a progressive speech disorder, bradykinesia, and
abnormal ocular motor function. Magnetic resonance imaging of the
brain showed scattered white matter lesions in the hemispheres and
pons, and she was diagnosed with supranuclear palsy. Lumbar puncture
showed no selective concentration of antibody in the spinal fluid.
Nevertheless, she was re-treated with 2 weeks of parenteral
ceftriaxone in 1989 that had no effect on her neurologic symptoms.
During the time of observation, this patient died.
lymphoid mononuclear cells were observed surrounding the
intracerebral vessels in one section. Using Dieterle silver stain, a
spirochete was present in the cortex and another was exterior to a
Steere's multiply-treated patients died anyway with spirochetes in her brain. How
amazing that that suddenly doesn't happen any more, despite no new
breakthroughs in antibiotic treatments or their delivery.
The Primer's Shell Game
is where these Lyme crooks use the wrong DNA (or RNA) primers (they use the
variable Osp primers instead of the non-variable chromosomally encoded) to
test Lyme victims. These same crooks use the correct DNA primers when they
want to find Lyme or borrelia in ticks (to patent). It's a shell game. They all know that
all that they can use for treatment outcomes is non-variable DNA or RNA.
Mark Klempner never reported his primers in his Chronic
Lyme "study," and he would not tell me, either, in an email correspondence I
had with him. Therefore, the Lyme crooks have never validly reported on the
presence of borrelia in humans, with the exception of Gary Wormser's study of
EMs in which he found 2/9 patients who had EM, who had been treated with
antibiotics still had spirochetes in their tissues. This data can be found on
HOW RICO WILL BE CHARGED PAGE
- - -
Clin Microbiol Infect.
Comparison of PCR methods and culture for the
detection of Borrelia spp. in patients with erythema migrans.
Institute of Microbiology and Immunology, Faculty of Medicin,
University of Ljubljana, Slovenia. email@example.com
The sensitivities of two PCR assays and culture were compared for
the detection of Borrelia spp. in skin specimens of 150 patients
with typical erythema migrans. In addition, the accuracy of the
methods for the identification of Borrelia spp. was compared by
analysing culture isolates and material obtained directly from skin
biopsy specimens. Borrelia burgdorferi sensu lato was isolated from
73 (49%) of 150 skin biopsy specimens. Using a nested PCR
targeting the rrf-rrl region and a PCR targeting the flagellin
gene, 107 (71%) and 36 (24%) specimens, respectively, were positive.
With both PCRs, positive results were more frequent with
culture-positive samples (67/73 (92%) and 24/73 (33%) for the nested
and flagellin PCRs, respectively) than with culture-negative samples
(40/77 (52%) and 12/77 (16%) for nested and flagellin PCR,
respectively). Pulsed-field gel electrophoresis after MluI
restriction identified 69/73 (95%) isolates, of which 58/69 (84%)
were Borrelia afzelii and 11/69 (16%) were Borrelia garinii. After
MseI restriction of PCR products amplified from the intergenic
rrf-rrl region, B. afzelii was identified in 73/107 (68%) samples,
B. garinii in 22/107 (21%) samples, and both species in 11/107 (10%)
samples. The corresponding results for culture-positive specimens
were 41/69 (59%), 14/69 (20%), and 7/69 (10%). Comparison of the
results for specimens positive according to both approaches revealed
complete uniformity in 80% of the cases. Overall, nested PCR was
the most sensitive method for the demonstration of Borrelia spp.
in erythema migrans skin lesions, followed by culture and PCR
targeting the flagellin gene. The congruence of identification
results obtained by analyzing culture isolates and material obtained
directly from skin biopsies was relatively high but incomplete.
Flagellin Master Prober: Picken, 1992:
Burgdorferi is most similar to hermsii
Mark Klempner, in his fake long term retreatment study,
did not reveal which DNA primers he used to discover no-lyme in the CSF
of previously terated Lyme victims, even when asked directly. The
NEJM had no problem with Klempner not reporting his Methods and
Materials in the Methods and Materials section of his report.
Klempner states in his report that people who were
positive were excluded from the study because it would be unethical not
to treat patients who were DNA positive for Borrelia, and so they were
excluded from the start. We know of at least one patient who
joined Klempner's study because she/he was Borrelia burgdorferi
For Klempner to claim publicly that there were no Bb DNA
positive patients at all, is research fraud.
Recall from the
Russell Johnson Culturing article published in the 1989 IDSA Reviews
special supplement on Lyme and spirochetal diseases, it could take
months to regrow intact spirochetes in BSK media from the cystic form:
In 1994 and in 1996, Steere and Nocton published a reports of treatment
failure and brain invasion using RNA and DNA methods and they found
treatment failed in a third of the patients with their OspA primer sets.
In the first report, in 1994, published in the New England Journal of
Medicine, that they used 4 primer sets. Three were for the OspA gene
(which we know undergoes antigenic variation and therefore these are the
wrong probes). The fourth set was for 16S RNA intragenic spacer
(remember now, for Borrelia burgdorferi, ignoring all the other
possibilities). These were rather valuable reports, overall, since
they prove that Allen Steere and the ALDF cabal is saying something much
different today about the what the disease actually is (brain infection) and
about treatment outcomes.
Detection of Borrelia
burgdorferi DNA by polymerase chain reaction in cerebrospinal
fluid in Lyme neuroborreliosis.
"of 73 patients with Lyme arthritis who were
untreated or treated with short courses of oral antibiotics
before testing, 70 (96 percent) had positive PCR results. In
contrast, of 19 patients who received either parenteral
antibiotics or long courses of oral antibiotics, only 7 (37
percent) had positive test results after treatment (P<0.001).
In the 29 patients for whom serial samples were available,
all pretreatment samples were positive."
About a third of the patients still had spirochetes in their
knees after antibiotic treatment.
- - - -
The following is not a treatment outcomes assessment, but merely demonstrates
that Allen Steere acknowledges the brain disease called "chronic
neuroborreliosis" and that he previously had proven with the synovial fluid
DNA post-treatment analysis, that treatment failed in a third of the cases.
As I do not have the full text of this report, and the probes were not
reported, I can't comment on whether or not they were the correct ones.
As the correct ones by this time had been used by Gary Wormser and Robert
Schoen when assessing tick bite treatment outcomes and whether or not the
spirochete missing the OspA-B plasmid belonged to this new New England deer
tick relapsing fever group, respectively, we can guess that Allen Steere
maybe knew which primer probes to use: 16S and 23S RNA, but better
from more from the genera:
Detection of Borrelia burgdorferi DNA by
polymerase chain reaction in cerebrospinal fluid in Lyme
J Infect Dis.
Division of Rheumatology/Immunology, New England
Medical Center, Boston, Massachusetts02111, USA.
A polymerase chain reaction (PCR) assay that detects
Borrelia burgdorferi DNA in cerebrospinal fluid (CSF) was evaluated as a
diagnostic test for acute or chronic Lyme neuroborreliosis. In one
laboratory, 102 samples were tested blindly, and 40 samples were retested in
a second laboratory. In the first laboratory, B. burgdorferi DNA was
detected in CSF samples in 6 (38%) of 16 patients with acute
neuroborreliosis, 11 (25%) of 44 with chronic neuroborreliosis, and none of
42 samples from patients with other illnesses. There was a significant
correlation between PCR results and the duration of previous intravenous
antibiotic therapy. The overall frequency of positive results was similar in
the second laboratory, but concordance between the laboratories and among
primer-probe sets was limited because many samples were positive with only
one primer-probe set. Thus, PCR testing can sometimes detect B. burgdorferi
DNA in CSF in patients with acute or chronic neuroborreliosis, but with
current methods, the sensitivity of the test is limited.
PMID: 8769624 [PubMed - indexed for MEDLINE]
Perhaps the best way to prove
antibiotic efficacy is with monkeys treated with ceftriaxone and then
subject to the Mouse (monkey) Infectivity Test.
As reported in the previous
chapter, and in the introduction of this one,
Mark Klempner says the IV drug ceftriaxone, which is used
for meningitis (and not knee-only diseases), does not kill all the
spirochetes (click here for full text journal article)
protected B. burgdorferi for at least 14 days of exposure to ceftriaxone. Mouse
keratinocytes, HEp-2 cells, and Vero cells but not Caco-2 cells showed the same
protective effect. Thus, several eukaryotic cell types provide the Lyme disease
spirochete with a protective environment contributing to its long-term
HERE on Medline (same report as above)
Mark Klempner here describes a special nerve
and brain degrading enzyme, Matrix-metalloproteinase 130 that he found in
chronic Lyme patients. Be sure to read why he performed this study.
10) In 1999, Mark
Klempner reported with Denise Huber at Tufts, "Autoimmunity; Is is Me or
Klempner reported that
OspA or LYMErix could cause brain disease as if that needs an explainer.
"T cells that react to OspA, OspC and p 22 epitopes also recognize
myelin basic protein, SST-R1m and IL-1R, respectively, possibly leading
to encephalopathy and radiculopathy..."
We're not too impressed with the T cell data to support that hypothesis, but
we are impressed with Mark Klempner's non-reporting the possible association
between OspA vaccination and brain damage, since in 1999, LYMErix had come
onto the market. We're also impressed with the fact that in 2003 Mark
Klempner said there was no such thing as cognitive impairment associated
with Lyme infection. We claim that myelin has something to do with
brains and nerves, and that the spinal fluid was from whence Mark discovered
the very special MMP-130.
NINDS' Roland Martin did not prove T cell autoimmunity,
and so he went home to Germany. Martin failed to prove that the Multiple Sclerosis
version of "Lyme Disease" is caused by T cell autoimmunity, just as Allen Steere
failed to find T cell autoimmunity was the cause of the knee kind of Lyme.
If Lyme-Knee and Lyme-Brain are not really T cell autoimmunity diseases,
then what are they ya think?
Read more about that on this page:
Klempner's nice flow chart (more detailed version,
Pathology and the Congenital Brain Infection of Newborn Resulting in Death
"The death of the newborn was probably due to respiratory failure as a
consequence of perinatal brain damage."
child and mother were seronegative, they were treated, and there was
remarkably "no inflammation." Yale's Eugene Shapiro claimed in PBS'
Life on Earth Series that the only way you can have a disease is if you have
baby died of congenital Lyme brain damage, anyway. This is not the
only such report, and they report on several babies who died from congenital
12) When trying to strike fear into the hearts of the New England Munchausers,
hypochondriacs, trophy-wives who co-suffer Viagra-deficit syndrome,
Fibrofemzalgiacs, "what I call Lyme Paranoia, " antibiomaniacs, members of
the "Lyme internet cult," and other delusional newsgroup posters, Yale's
Robert Schoen said about the LYMErix vaccine:
In late-stage disease [a disease now called Munchausen's by
proxy, etc], the central nervous system [which we know to be a complicating
variable and should be thrown out] may be involved. A new diagnostic
test [not ever to be used, since it is a scientifically valid marker of real
illness and we Lyme crooks never go there, preferring the services of the
professional, perpetual pee-pee whacking Voo-Dooists of medicine,
psychiatry] measuring glial fibrillary acidic protein in the cerebrospinal
fluid may prove to be a useful tool in measuring such involvement.
"Oh," we say.
"My, my. A new disease for us bored housewives to
wear. GFAp-itis. We can talk about it at cocktail parties..."
had the nearly dead dumped on him repeatedly, the very famous and brilliant
(truly) Kenneth B. Liegner of Armonk, New York, and having had the
experience and training to go with the bodies, decided to send samples of
multiply-treated human bodies to the crooks, to have THEM determine
whether or not Lyme infection persists past treatment and VIOLA!
participates in (Liegner) autopsy and identifies persisting DNA in samples of
patients treated many times for Lyme, which therefore proves antibiotic
treatment does not completely eradicate the Lyme infection.
SUNY, Tulane, CDC, The Mayo Clinic...
"POSITIVE FOR BORRELIA" either by staining or by DNA methods.
And then the New
York Medical Board tortured Ken what else is new.
Kaiser supplies the
New York Medical Board's "experts" from New York Medical College. It's
sorta like the George Bush, Karl Rove, US Attorneygate, Alberto Gonzales and
John Yoo scenario. Stack the deck, then commit the crime.
in a repeat of Robert Schoen's scare-mongering of the already mortally
scared of having nothing to talk about at cocktail parties, East Lyme,
Connecticut's Vijay Sikand said at the 1998 FDA LYMErix Vaccine Meeting
"...the specter of asymptomatic infection is something that troubles
me a great deal and troubles a great number of my colleagues who need to treat
Lyme disease. The obvious analogy with syphilis infection with Treponema
pallidum is there to consider. It is well known that Borrelia burgdorferi
indeed after asymptomatic infection can lurk or secrete itself in certain areas
of the body, perhaps the central nervous system or perhaps the joint spaces,
only to reappear months or maybe years later in the form of late stages of
illness which are harder to diagnosis and treat." ---Vijay
"Do that again!!!"
<squeal of delight>
["Harder to diagnose and treat?" I thought Lyme was "easily diagnosed and
cured?" Sikand sounds remarkably like myself when I say "TROJAN
HORSE." Or maybe it's the other way around.]
15) The combined National Institutes say: --
"8. Infectious diseases
of the CNS mediated through immune mechanisms, including acute and chronic
Lyme disease and neuroAIDS;"
Heavens! Who are
we to argue with all of the combined
Early Brain Invasion
by Lyme crook, Jorge Benach.
Benach later sent a letter to the editor of the New York Times stating that
Allen Steere was right, that we Lyme victims are delusional, and that "Allen
Steere has the science on his side," when you can clearly see that we have Jorge
Benach's science as well as Allen Steere's on our side.
Early Brain Invasion by Ray
Dattwyler is now the author of the new IDSA "guidelines,"
where Lyme is a non-disease.
But this is what
he told to the FDA in 1994:
Hence, Lyme is not a knee disease, and
the Klempner report is bogus, so there is no data to support the new IDSA
guidelines on Lyme as an imaginary, self-limited disease that is cured by
the placebo effect of antibiotics as Mark Klempner asserted.
This has other indictment value as
regards Steere and what he did in Europe, since clearly we want to diagnose
the earliest case of Lyme to possibly prevent brain invasion. (I know.
Steven Malawista and the Yale Psychiatry Department would not agree with
Antigenic variation in the
This means you can't use the Dressler-Steere antibody method to
determine late Lyme in the brain and neither can you say what Mark Klempner now
says. This is just one more well-established report on Lyme as a brain
disease by a well-acknowledged authority. This report adds nothing as
regards permanence of the infection, since it is not a treatment study, but
it's simply the pursuit of the obvious to anyone who cares about reality.
IDSA REVIEWS 1989
IDSA reviews demonstrate
that the REAL expert, Russell Johnson claims that some spirochete cultures
could take months to re-grow intact spirochetes from the spheroplast form
(serum-starvation or survival in harsh conditions form). This negates
the Klempner report.
fails in more than half the cases"- Dattwyler, Luft, Sigal and
RUSSELL JOHNSON, CULTURING
"spheroplasts could take months to revert to intact spirochetes in BSK
NEW "GREAT IMITATOR," PACHNER
(We have enough data to work this up now. We had it over a decade
ago. The creeps kept the lid on the truth and now American scientists
are known as "stupid and incomprehensible" in Europe. The CT Attorney
General had to sue the bastards, they refused to turn over their own
self-incriminating data to him, and the settlement looks very much like they
want to avoid criminal charges. They threw a fit when we laughed at
their response to the settlement with Richard Blumenthal. We laughed
some more. Now they say nothing, except for Edward McSweegan who has
become more virulent against myself, in particular, which is in itself a
clue as to what he doesn't want the world to see. We say "Grow the Eff
up and accept responsibility for what you have done, since there's no better
example of a coward than he who blames the victim and the world is obviously
DURAY, CLINICAL PATHOLOGICAL CORRELATES
ABNORMALITIES OF THE NERVOUS SYSTEM, HALPERIN
DATTWYLER and LUFT,
TMT of SYPHILIS, CEFTRIAXONE RECOMMENDED, ENDPOINT UNKNOWN
evaluations of ceftriaxone for early syphilis therapy are promising; however,
the optimal dose and duration of therapy are unknown."
CDC's GEORGE SCHMID on LYME and SYPHILIS'
Thirty days of IV ceftriaxone was
in the first place, an arbitrary decision by the crooks, themselves,
that was because they thought it was a brain disease, and the same for
Then, again, came Mark Klempner.
Dunn at Brookhaven et al, say that Lyme is "the
perfect stealth pathogen" that can "mistaken for MS, Lupus, can cause
It's always nice to have the Department of Defense and
the Department of Energy on your side :)))
*21) Alan Barbour on what happens if you wait long than 7 days to treat
Lyme very aggressively
Remember, now, there is the issue with mice not being the best
human brain example
[fill in the blank self-ass-biting comment]:
Antimicrob Agents Chemother. 1996 Nov;40(11):2632-6
In vivo activities of ceftriaxone and
vancomycin against Borrelia spp. in the mouse brain and other sites.
Department of Medicine (Infectious Diseases), University
of Texas Health Science Center at San Antonio 78284, USA.
Borrelia burgdorferi, the agent of Lyme disease, and B.
turicatae, a neurotropic agent of relapsing fever, are susceptible to vancomycin
in vitro, with an MIC of 0.5 microgram/ml. To determine the activity of
vancomycin in vivo, particularly in the brain, we infected adult immunocompetent
BALB/c and immunodeficient CB-17 scid mice with B. burgdorferi or B. turicatae.
The mice were then treated with vancomycin, ceftriaxone as a positive control,
or normal saline as a negative control. The effectiveness of treatment was
assessed by cultures of blood and brain and other tissues. Ceftriaxone at a dose
of 25 mg/kg of body weight administered every 12 h for 7 to 10 days eliminated
cultivable B. burgdorferi or B. turicatae from all BALB/c or scid mice in the
study. Vancomycin at 30 mg/kg administered every 12 h was effective in
eliminating infection from immunodeficient mice if treatment was started
within 3 days of the onset of infection. If treatment with vancomycin was delayed
for 7 days or more, vancomycin failed to eradicate infection with B. burgdorferi
or B. turicatae from immunodeficient mice. The failure of vancomycin in
eradicating established infections in immunodeficient mice was associated with
the persistence of viable spirochetes in the brain during antibiotic treatment.
PMID: 8913478 [PubMed - indexed for MEDLINE]
See the Chronology of the Lyme crimes for the
Identify the disease early and treat it aggressively, but always remember
the Trojan Horse, so avoid psychiatry, because they, in their hysteria and
hissy-fitting response to this website and the proofs that Lyme is a
permanent brain infection, dared to say, "There will be no more
which we reply, "We think it's a free-for-all, now, and anyone can make up
any diseases they want, as long as you psychiatrists claim to have the
magical ability to diagnose us with anything without the use of any
scientifically valid tests."
say, "RETROCHONDRIA!!" in your general direction.
say, "The AMA has FLACCITUDE!"
"Your ideas are Steereborn!!"
say, "Meet me at the Yale Center for the Study of Erections, and we can
have a discussion about all your uncomplicated variables ."
An independent study on spirochetes in the brain
from dentists and they say:
Molecular and immunological
evidence of oral Treponema in the human brain and their association with
http://www.ncbi.nlm.nih.gov...11929559 medline link to verify
Department of Pediatric Dentistry, School of Dentistry,
Oregon Health and Sciences University, Portland, OR 97201-3097, USA.
The purpose of this investigation was to use molecular and
immunological techniques to determine whether oral Treponema infected the human
brain. Pieces of frontal lobe cortex from 34 subjects were analyzed with
species-specific PCR and monoclonal antibodies. PCR detected Treponema in 14/16
Alzheimer's disease (AD) and 4/18 non-AD donors (P < 0.001), and AD specimens
had more Treponema species than controls (P < 0.001). PCR also detected
Treponema in trigeminal ganglia from three AD and two control donors. Cortex
from 15/16 AD subjects and 6/18 controls contained Treponema pectinovorum and/or
Treponema socranskii species-specific antigens (P < 0.01). T. pectinovorum
and/or T. socranskii antigens were also found in trigeminal ganglia and pons
from four embalmed cadavers, and 2/4 cadavers also had Treponema in the
hippocampus. These findings suggest that oral Treponema may infect the brain via
branches of the trigeminal nerve.
PMID: 11929559 [PubMed - indexed for MEDLINE]
"PCR detected Treponema in 14/16 Alzheimer's disease (AD)
and 4/18 non-AD donors."
"Cortex from 15/16 AD subjects and 6/18 controls contained
Treponema pectinovorum and/or
Treponema socranskii species-specific antigens."
We wondered why this information never makes breaking
headline news as regards new developments in Alzheimer's but then we
remember the American Psychiatric Association has become like the Pope in
his treatment of Galileo. Such is heresy against the dogma that "no
more spirochete-like discoveries will be made."
Despite the history and syphilis and the insane asylums and
Ehrlich's Compound 606, the arsenic bullet and so on and so forth...
Mousehausen's is now
admits ceftriaxone fails to eradicate all spirochetes.
Persistence of Borrelia burgdorferi
Following Antibiotic Treatment in Mice
Sunlian Feng, Kevin
Holden, Kimberly J. Freet, and Stephen W. Barthold*
Center for Comparative Medicine, Schools of Medicine and Veterinary
Medicine, University of California at Davis, One Shields Avenue, Davis, CA
The effectiveness of antibiotic treatment was examined in a
mouse model of Lyme borreliosis. Mice were treated with ceftriaxone
or saline for one month, commencing during the early (3 weeks)
or chronic (4 months) stages of infection with Borrelia burgdorferi.
Tissues from mice were tested for infection by culture, polymerase
chain reaction (PCR), xenodiagnosis, and transplantation of
allografts at 1 and 3 months after completion of treatment.
addition, tissues were examined for spirochetes by immunohistochemistry.
In contrast to saline-treated mice, mice treated with antibiotic
were consistently culture-negative, but tissues from some of
the mice remained PCR-positive, and spirochetes could be visualized
in collagen-rich tissues. Furthermore, when some of the antibiotic
treated mice were fed upon by Ixodes scapularis ticks (xenodiagnosis),
spirochetes were acquired by the ticks, based upon PCR, and
ticks from those cohorts transmitted spirochetes to naïve
SCID mice, which became PCR-positive, but culture-negative.
Results indicated that following antibiotic treatment, mice
remained infected with non-dividing but infectious spirochetes,
particularly when antibiotic treatment was commenced during
chronic stage of infection.
Ya gonna argue with the CDC, now?
spirochetes were acquired by the ticks
Culture negative means nothing, as we know. It could
take months in vitro. They should have completed the
Mouse Infectivity Test.
CDC on intracellular nerve and brain borrelia spirochetes
We all wonder how the hell the CDC can talk about this phenomenon and not
come right and say "Lyme is a relapsing fever organism and is incurable due to
their intracellularity and the formation of dormant cyst or spheroplast forms,
which are also inhalable," - since this is all either their own data, or the
NIH's data, or the US Army's data.
Willy Burgdorfer discussing
the cyst or the spheroplast form. Combine that with the
reality that CDC officers discuss intracellular spirochetes (Mark Klempner
and the one above). While Willy Burgdorfer is discussing European
articles, however - disclaimed as invalid by the CDC except when Allen
Steere does it (Chapter 3) - recall that Willy Burgdorfer was himself
recruited by the NIH Rocky Mountains Bioweapons Lab (which had a moat dug
around it hmmm strangely like a mini island, like a mini Plum Island) from
Switzerland, and the NIH also recruited the German scientist
Roland Martin to head up the NINDS-Multiple Sclerosis group.
So, everyone can discount CDC's discount of
foreign research since obviously no matter what they say, it's a lie.
SAY THE IDSA GUIDELINES (GARY WORMSER, et al, or the
Original ALDF.com "entrepreneurial
trio" gang of:
are Durland Fish,
Ph.D., former director of the College's Lyme Disease Center and now
a research scientist at Yale; Gary P. Wormser, M.D., still professor
of medicine and pharmacology and chief of the Division of Infectious
Diseases at the College; and John J. Connolly, Ed.D., former College
president and current chairman of the board of the
American Lyme Disease Foundation,
Inc., which had its
genesis on the Valhalla campus in 1990."
http://www.journals.uchicago.edu/doi/full/10.1086/508667 ◄ "The
2006 IDSA Guidelines"
"The notion that
symptomatic, chronic B. burgdorferi infection can exist despite
treatment courses of antibiotics (tables
) in the absence of objective clinical signs of
disease, is highly implausible as evidenced by (1) the lack of
antibiotic resistance in this genus
310], (2) the lack of correlation of persistent symptoms with
laboratory evidence of
inflammation or with the eventual development of objective
physical signs [223,
and (3) the lack of precedent for such a phenomenon in other
spirochetal infections [315-317].
[Lyme is a "STEALTH
DISABLER" and as shown in the
Yale Congenital Lyme Autopsy
report, there was "NO INFLAMMATION." SEE THE
BIOMARKERS and the MARK
CHAPTER; NOT CAPTURING THEIR OWN IDENTIFIED BIOMARKERS of
disease and instead, deploying the invalid mumbo-jumbos
(psychiatry) is a research
"Additional compelling evidence against the hypothesis that
persistent symptoms are the result of
persistent infection is the fact that the concentrations of
antibodies against B. burgdorferi in
many of these patients diminish to undetectable levels [257,
The panel is unaware
of any chronic infection in which antibody titers diminish despite
persistence of the causative
organism. [VERY DAMNING
statement. This phenomenon is related to the "stealth
disabler" mechanisms of fungal or mycoplasmal or mycobacterial
antigens- no antibodies are made against the infection due to
TLR2 tolerization and the downregulation of HLA or
antigen-presenting molecules Wormser is aware
of the immune suppression aspects of OspA vaccination, since he
reported about it:
Gary Wormser reporting the blunting of the immune
response in vaccinated animals:
interferes with the response of lymphocytes to
proliferative stimuli including a blocking of cell cycle
"In syphilis, patients who are regarded as having
treatment failure typically have
persistent or rising titers of antibodies .
[THE IMMUNE SUPPRESSING
PlumIsland PAM3CYS LIPOPEPTIDE OSPA IS NOT FOUND IN TREPONEMA PALLIDUM]
"Finally, Lyme disease lacks characteristics of
other infections that justify longer treatment courses, such as
infections in immunodeficient hosts,
ARE REALLY GONNA BE SHOCKED when you see the Plum Island
data, the activation of latent viruses data (auto-kill turned off)
the HIV Pam3Cys immune depression data, and the Fungal Vaccines data] infections in which a
pathogen is inhibited
but not killed by antimicrobial therapy or in which available
antimicrobials are minimally
active in vitro
[JOHNSON CULTURING FROM SPHEROPLAST COULD TAKE MONTHS], infections caused by an intracellular pathogen
"cef fails"], infections
involving a biofilm, infections on a heart valve, or infections
involving a clinical site in which
there is ischemia, a foreign body, a sequestrum***, or frank pus .
The “cystic” forms of B.
burgdorferi that have been seen under certain growth conditions in
vitro have not been shown
to have any clinical significance .
[INTRACELLULAR CYSTS are the KEY TO LATENCY, well-known
historically, and this is an
application of the NEGATIVE DATA RULE, which is another research
fraud crime indictment point; THE
INFECTIVITY TEST HAS NOT BEEN PERFORMED ON TREATED MICE
CDC and NIH Rocky Mountain antibody-gold sphere method as
applied to borreliosis:
The Method Developed:
*** Applied to Borreliosis by NIH Rocky
The Dorward/Garon Gold Test Apparently
detects plenty of shed antigen. And it looks pretty
sequestery to me, not to mention we could never
determine sequestery sites in a human unless we had a lot of
brains to biopsy, although the dentists did a pretty good
job detecting oral treponemes sequestering in the brains of
Alzheimer's patients- See Item 23, below.
1975 at an international spirochetal diseases conference
for which Willy Burgdorfer was present;
"The ability of the borrelia, especially tick-borne strains to
the brain and in the eye after treatment with arsenic or with penicillin or even
after apparent cure is well known (1). The persistence of treponemes after
treatment of syphilis is a major area which currently requires additional study
(3,5,10,11)." -Jay Sanford,
US Military Hospital, Bethesda, MD
As you can see, Gary Wormser, ad
IDSA et al, are trying to deny everything that Lyme actually is.
Lyme is stealth (no antibodies late in the disease), latent,
intracellular, is resistant to antibiotics (goes into a
self-protective spheroplast form upon exposure to antibiotics
which is not an "end stage" as the crooks claim), it goes into a
"dormant" or serum-starved or spheroplast mode (known to the
crooks themselves as a "Trojan Horse"), Lyme belongs to the
class of diseases where there is typically no inflammation.
It does not belong to NIAID or Anthony Fauci's division, but the
parasitic diseases division - but it's not going to belong to
either the CDC or the NIH at all after not too long, because
when they're done being investigated we will find that that NIH
and the CDC are the same as the NEJM "peer-reviewers" - not a
one of them isn't interest conflicted. In fact, we will find out
what is the exact relationship between the CDC and Kaiser or
else the NAFTA-eers of Canada and Mexico will discover it and
At this time I recommend that
Russia and China infiltrate Canadian and Mexican business
structures such that when the NAFTA NationRape of the North
American Continent takes place, Russia can return the favor for
the Israeli Oligarchic NationRape Escapade under Yeltsin.
More of these clear rebuttals
below and in the RICOCHRON.htm page.
Remember, the IDSA keywords:
"INFLAMMATION" and "OBJECTIVE SIGNS" (meaning they are
still referring to "Lyme Disease" as the Dearborn definition of
"autoimmune bad knee" as seen in the
RICO graphics), because
that later comes in in
Congenital Lyme ("lack of inflammation") and the Biomarkers
chapter as regards the description of the immune
suppression outcomes of Lyme, which is the key to all of their
crimes, especially as regards this fungal vaccine, LYMErix or
ImmuLyme, or rOspA and the activation of latent viruses of all
The crooks' own treatment failure or
persistence articles are below.
The IDSA statement about the cysts or the spheroplasts is yet
another criminal matter, since they all
referenced Dave Nelson's "reversion
to intact spirochete form from spheroplast within one
minute of the addition of rabbit blood" report, Russell
Johnson said it could take months in culture for the cyst form
to regenerate in media, and the Mouse Infectivity Test has never
been tried as a treatment efficacy trial, ever, to my knowledge
in lab animals. They cannot make the claim that the cyst
form hasn't any clinical significance, since here we have again
the Crooks' Negative Data Rule.
At the present time, there are
a whole slew of
Russian scientists now studying these intracellular
serum-starvation ("Stringent Response") forms at New York
It's pretty hard to find a cohort nowadays of non-HIV
infected syphilis patients. From the 1989
IDSA reviews chapter on
syphilis, it does not sound at all like syphilis is
under control or even given much concern
The serology of syphilis and
borreliosis can hardly be compared anyway due to the fact that
Borrelia infect all kinds of mammals and undergo great antigenic
variation. These IDSA criminals deny the validity of the
Borrelia-specific flagellin assays, when they're the only
Anything Wormser says at all needs to be challenged.
It's quite self-evident that nothing about the seriousness of
this disease will Wormser tolerate as a fact to stand out or
rise to the surface or be memorable. Clearly that attack
pattern worked in the past as regards the Tuskegee Bad Blood
experiment and it worked for a while as regards the abuse of
Gulf War Illness Veterans, and it works fine as long at the Lyme
Disease Association's Pat Smith insists her drones do nothing
other than worship Pat Smith and pretend to be Stepford Wives -
as if it were admirable to be a brainless-but-pretty robot.
But here is an era where all of these types of abusive medical
criminals' former barriers have come down.
We all know psychiatry is not a
medical practice and that the entire genre is in an embarrassing
shambles of BigPharma pay-offs and incredibly stupid,
self-indicting statements every time they're in the news.
They undid themselves because they went exponentially overboard
with their relationships to BigPharma. The Lyme criminals
can no longer deploy psychiatry as they have in the past.
Roy Meadows was himself a fulcrum for change due to the obvious
ridiculousness of the Munchausen's accusations. Sooner or
later it would have happened that someone mentioned the word
science in the context of health, when talking about children
The survivors of the Munchausen's
Terror can take comfort in the fact that God allowed this horror
to occur, like He allowed the Child Protective Services to
happen. God intends to send at least 2/3 of all the humans to
hell. If you're a survivor of these Terrors, you've been
chosen to send dozens of people to hell for your suffering.
In your suffering, you are suffering with Christ, and He is
pleased with such people the most.
When Jesus said, "the gates of heaven are narrow," and that few
would make it, He would not be lying, would He?
Anyone who has dealt with the
Child Protective Services has no doubt that it is their personal
will to torture parents and children and that they take great
pleasure and derive great satisfaction in destroying people and
causing great suffering You can see it in their queer,
sneering, half-cocked smiles. When you know the CPS, there
is no question in your mind that these entities are demonic.
The vast majority of them are either possessed or are far along
the way towards possession. (The only time a possessed
person acts out is when they are attempting to reject the
possession. The rest are asymptomatic, like the CPS and
And given the magnitude of
this epidemic of Lyme there had to be an instance where a
tick would bite a real scientist. Perhaps it only
happened once, but it happened. A real scientist is
someone who shouts "BULLSHIT!!," in addition to being highly
sensitive to bullshit's occurrence- which would only happen
if said scientist is also a female. We have no other
data on that because none exists, or else, by definition we
would hear about it.
COMPREHENSIVE ARTICLE ON SYPHILIS:
Saint Michael the Archangel,
defend us in battle.
Be our protection against the wickedness and snares of the devil.
May God rebuke him, we humbly pray;
and do Thou, O Prince of the Heavenly Host -
by the Divine Power of God -
cast into hell, satan and all the evil spirits,
who roam throughout the world seeking the ruin of souls.