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24 May 2012
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CDC Greed
(won't answer the FOIA)
ELISA = arbitrary cutoff.
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Overview
TUSKEGEE - By Jerry Leonard
1998, CIA Oilmen & Israelis plan to overthrow
Saddam for the oil.
Bush/Gore Oil/War-(Oct,2000)
Bush's own explainer (Oct
2000) re:
Iraq Oil
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Most
popular referred page 101009
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You are gonna laugh.
Look how many times Allen Steere, John
Halperin, UCONN staff, and Yale staff, published about how serious "CHRONIC
NEUROLOGIC LYME" was, but they now say there is no such thing as chronic Lyme,
and that the "post-Lyme syndrome" is due to "catastrophising," hypochondria,
depression, "medically unexplained symptoms," "some psychiatric illness,"
"Lyme Anxiety," "Lyme Paranoia," "Delusional Parasitosis," etc., and Mark
Klempner says that people who respond positively to antibiotic treatment do so
due to the "placebo effect."
ROFTL
[Remember now, we have for years
asked friends and family to help us with the Lyme activism and they all either
blew us all off, or reported to the DCF that we're "irresponsible parents
because we go to the library"- Nancy E. Martin, 21 Redstone Way.
Farmington, Corrupticut.
←More stuff you
just can't make up. That's recorded in depositions' transcripts.
The New Haven FBI is too busy watching porn on
the internet all day to drag themselves away from their porn monitors in New
Haven, Corrupticut (at 600 State Street- an unmarked building), to help us,
even though all of this is obvious to anyone with a brain in their head and
wonders what will happen to them if a microscopic tick bites them and they
don't see it- Porn and sleaze are very big in Corrupticut. Just ask the
former head of the DCF or one of the floozies down in New Haven DCF court who
spend 3 hours a day in front of a mirror, instead of doing what I do,
protecting children from Yale's crimes. And just for the record, 75% of
women fake orgasm, so AAG Jessica Gauvin only fools "judges," with her
sex-o-metric behavior and Hollywood make-up studio get-up, since "judges" are
inherently stupid and openly express hatred toward science- Just ask
Anton Scalia.
Nancy E. Martin has been on
Bob Bransfield's MMI list since the
Fall of 1999, since I thought she wanted to help people with chronic
illnesses. It turns out, she wants to file false allegations to DCF
about them, and then perjure herself. She has no children of her own.
God works in mysterious ways. Nancy Martin is too selfish to be a
mother. She's like Gauvin. You may be wondering why there are so
many stupid people in Corrupticut...]
"Chronic
Neurologic Lyme Disease," by Allen Steere, who now calls disease this
"CRAZY."


http://www.nejm.org/doi/pdf/10.1056/NEJM199011223232102
1990 Steere using this seronegative Lyme Assay to evaluate "chronic neurologic
Lyme" cases:

=====================
"The triad
of neurological manifestations of Lyme disease," by Allen Steere and Andrew
Pachner

Jeepers. How did so many of us
telepsychogenically hypochondriactomize the exact same chronic Lyme symptoms
and the exact same markers of disease for so many years, even before we were
the "internet cult??"
We have proof that UCONN's
Louis Reik now says to patients that chronic neurologic Lyme is a non-entity
and is hypochondria:
"Demyelinating
encephalopathy in chronic neurologic Lyme Disease," by UCONN's Louis Reik,
who now calls this disease, "CRAZY."

JJ
Halperin- Now says Chronic Neurologic Lyme is hypochondria, and this is
despite knowing that the only true "seropositives" of chronic Lyme will only
have a flagellin antibody, or band 41, since chronic Lyme blood testing is not
Lyme Arthritis blood testing (Steere's knees-only disease), nor is it "Early
Lyme blood" testing, due to antigenic variation.
A person who has had Lyme
a long time will not have antibodies to a spirochete that just came out of a
tick. All the bugs continue to undergo antigenic variation and
Andrew Pachner explains here about antigenic
variation in brain-adapted spirochetes, and
Alan Barbour explains here
in his
flagella-less Borrelia patent. They all say that due to antigenic
variation, all we can use to test for Lyme is a Borrelia-specific flagellin
antibody method- the method that Yale did
not use to validate LYMErix
"Lyme
borreliosis-associated encephalopathy"-- JJ Halperin

Why Yale had a "Lyme and
Lupus Clinic" before that became the biotech spinoff, "L2 Diagnostics," a
central entity in the Lyme scientific fraud and racketeering organization.
Most people (except MDs)
know Lupus is a "neuropsychiatric" illness since antiphospholipid antibodies
are also anti-brain antibodies. Thus, Chronic Neurologic Lyme can't be
hysteria or hypochondria, according to Allen Steere and Yale University, and
it is malpractice to diagnose anyone with a DSM disorder without looking for
these particular markers of real disease:
Anticardiolipin antibodies in neurologic Lyme, by Allen Steere:

http://www.geocities.com/kmdickson0308/lyme-dilemma.html
All of the scientifically valid
markers of real disease were discovered by the Lyme crooks, themselves,
but what they are and how to treat them are not listed in the IDSA Guidelines.
Gary Wormser was featured in "Ticks- The Real Vampires,"
a Discovery Channel Special, and in it he said, they do not know what are all
the infections carried by ticks.
But they just know how to treat
WE DON'T KNOW, and if you don't recover
from WE DON'T KNOW disease treatment, Wormser et al say, "You never
had
WE DON'T KNOW disease."
Scientific fraud
with intent to cause harm-
deploying the idiots of psychiatry and the
Munchausen's
accusations, which is clearly a criminal act -
a complaint to the United Nations Human Rights
Commission in Geneva.
Four of us on ActionLyme performed the "Pick the Perps
Operation," and recorded all the false statements made in the press by these
criminals, and on the Perps Team was Barb Fitzmaurice who passed away 2
summers ago. Barb was a great person and will always be
remembered by me as one of the true heroes of The Lyme Wars. I hate it
when the Lyme people, especially the players like Barb, croak before we've
won this war. Gayla Davies passed away recently and she too was a big
player for us.
Makes me FURIOUS.
"Telling women and girls inaccurately
that they have Lyme disease
"condemns patients to long-term,
untreated debility and useless,
toxic
and expensive courses of antibiotics,"
Sigal wrote in an editorial in
the May 15 issue of the journal
Hospital
Practice."--Emerging
diseases // Ticks carry multitude of
threats //
Maybe they have actual scientists in Eurasia, Africa,
and South America, who can query MedLine and see whether or not Roche claims
these are pseudogallstones and therefore do not require surgery, since not a
single MD in America apparently knows how to use MedLine.
We female hypochondriactomizers have too much Twilight Zone
and therefore rely on all the men to perform such research for us. Maybe
I can find a male around here to type in ceftriaxone and pseudolithiasis
since I don't know how to spell them, myself. ...
Thanks, smart male hanging around. I
can't do this research myself because I have
hyper-catastrophiziosis and therefore can't
concentrate on anything other than my imaginary symptoms.
The IDSA
Lyme guidelines are based on a "positive" test for Lyme (Klempner's Bogus
Chronic Lyme "study"), when they all know the
testing for Lyme is bogus and most of the crooks have reported that this
testing is bogus themselves.
(Blumenthal has all their older reports which
shows IDSA has committed scientific fraud, so what IDSA is excluding in the
development of their guidelines is their own published data).
But the
central perp behind the bogus testing is Allen Steere, whose father worked in
the insurance industry. Steere dared to published that Lyme was "overdiagnosed"
(sic) when he knows he pulled off the biggest medical scam in human history.
This is
what he did:
Steere
in Europe, using illegally
high-passage strain G39/40. Click the "Steere in Europe" link to see all
of these reports and follow what he did.
Why
did Steere go all the way to Europe to imaginate there should be no OspA and B
antibodies in "Lyme Disease" after saying "Lyme disease" was
too many antibodies
against OspA ???
http://iai.asm.org/cgi/reprint/56/8/1831?view=long&pmid=3397175
NIH Rocky Mountain Labs, including
Willy Burgdorfer, say to use low passage strains to Western Blot because high
passage strains drop plasmids- yet this is the high passage strain from which
we got the Dearborn CDC criteria.
This is the CDC's Dearborn method:
http://www.cdc.gov/mmwR/preview/mmwrhtml/00038469.htm
Notice
there is no band 31 (OspA) or 34 (OspB) listed.
That was the scam.
Steere's Imugen Lab, partners with Phil Molloy, and Yale's L2 Diagnostics,
intended to be the only labs licensed to use the Dave Persing RICO method
where they Western Blot with a strain with no OspA plasmid in it. In
this way, Steere and Yale and Corixa had a monopoly on the testing for Lyme,
as well as the LYMErix vaccine royalties.
YouTube
12:50 mins
The Lyme and LYMErix RICO Patents - The
Monopoly was set up around Yale's bogus LYMErix vaccine.
The Yale scientifically valid way to test for Lyme is US
Patent
5,618,533 The patent says the vast majority of all Lyme
cases have this antiobody, band 41, or flagellin, so Yale made it SPECIFIC
to Lyme borrelia, which is what one does in a method development and
validation.
That means since 1991, Yale knew what was the
scientifically valid way to diagnose Lyme, but we are still not allowed to
use this method, which is
MEDICAL NEGLIGENCE- the very thing
disclaimed by ALDF/IDSA.
http://patft1.uspto.gov/ dot RICO dot monopoly dot 6045804
STEERE's ORIGINAL SERODIAGNOSTIC
OBSERVATIONS:
Antigens of Borrelia burgdorferi recognized during Lyme
disease. Appearance of a new immunoglobulin M response and expansion of the
immunoglobulin G response late in the illness.
Using immunoblots, we identified proteins
of Borrelia burgdorferi bound by IgM and IgG antibodies during Lyme disease.
In 12 patients with early disease alone, both the IgM and IgG responses were
restricted primarily to a 41-kD antigen. This limited response disappeared
within several months. In contrast, among six patients with prolonged illness,
the IgM response to the 41-kD protein sometimes persisted for months to years,
and late in the illness during arthritis, a new IgM response sometimes
developed to a 34-kD component of the organism. The IgG response in these
patients appeared in a characteristic sequential pattern over months to years
to as many as 11 spirochetal antigens. The appearance of a new IgM response
and the expansion of the IgG response late in the illness, and the lack of
such responses in patients with early disease alone, suggest that B.
burgdorferi remains alive throughout the illness.
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=3531237
Get the full text and read
every word. I went through all of this step by step with the FBI but
they could not take their noses away from all the porn they watch down there
at 600 State Street, New Haven, CT. I happen to think corporate crime
is more interesting that porn, but then the Department of Justice is set up
to protect corporate crime since they're republicans.
Go here to MedLine and look at all of Steere's
earlier reports. He clearly said Lyme disease was an arthritis in a knee
caused by OspA-B
If you, the reader, have any self respect, you will look
at all of those reports and wonder how we ended up with a standard test for
Lyme which says if you have an antibody to OspA, you don't have Lyme disease.
The next question is, "Well, if OspA is specific enough to prevent
Lyme, why isn't it specific enough to diagnose Lyme?"
% specificity of each
band is equal to the accuracy of the test for each individual. In order
to increase the reliability of the test, they could go to capillary gel
electrophoresis, since Western Blotting is so outdated, but you won't hear
that from any MDs. MDs are as bad as republicans. It's a gang
mentality. A herd mentality.
You see, this is the crux of the Lyme crime.
The testing for Lyme is bogus, and they all know it, but they are preventing
anyone from being treated for Lyme if the person doesn't have the positive
bogus test (for Lyme arthritis). This latest outburst by the very
frightened of jail John Halperin is about a distraction over treatment
outcomes, when the biggest bogus treatment outcome was Klempner's 2001 report.
I will be back later to debunk Klempner (again), line by line, word for word,
exposing in particular the insane nonsense by Arthur Weinstein, since I did it
for www.ILADS.org which has a Klempner
debunker page on their website. In other words, this crime has been a
well-articulated crime since 2001, but nothing has been done because the
FBI and USDOJ prefer porn watching.
The Truth about Lyme Disease by the
Infectious Diseases Society of America (IDSA)
BRAIN PARENCHYMA (tissue) INVOLVEMENT- 1989. ANDREW
PACHNER
Title Page of Reviews of Infectious Disease, Supplement 6, 1989




Clinical Pathological Correlations of Lyme Disease



Immunological Aspects of Lyme Borreliosis- Ray Dattwyler and Ben Luft



How Lyme Borreliosis and LYMErix cause immune
suppression illness, and why that has
Bioweapons potential:
"Signaling through TLR-2 by
lipoproteins may represent a double-edged sword for host
responses to chronic intracellular pathogens such as M.
tuberculosis. Short-term signaling through TLR-2 activates
macrophages and initiates acute inflammation that may help control
initial infection. In contrast, prolonged TLR-2 signaling in
macrophages results in down-regulation of certain critical immune
functions, such as MHC-II Ag processing. M. tuberculosis
infects, survives, and persists in macrophages. The ability of M.
tuberculosis to survive acute inflammation positions the bacilli
to take advantage, through secretion of lipoproteins such as LprG and
LpqH, of this down-regulation of macrophage immune function."
http://www.jimmunol.org/cgi/content/full/173/4/2660
TRANSLATION:
Downregulation of MHC-II Ag (antigen) processing means no antibodies are
produced after a time (the chronic infection becomes "seronegative").
JJ Halperin served as an expert witness for SmithKline
(committed perjury)
Halperin is seriously desperate
about not going to jail since he knows he has published more than anyone,
how serious Lyme disease is. He knows he cannot retract all his previously
published reports. ...
This is the CDC's Dearborn method:
http://www.cdc.gov/mmwR/preview/mmwrhtml/00038469.htm
Notice there is no band 31 (OspA) or 34
(OspB) listed.
Amazing, right?"
First Steere said, "Lyme is a disease of
too many antibodies to OspA that cross-react with knee tissue," and then he
said, "Oops, Um, No, I mean Lyme is everything BUT antibodies to OspA, so please
take this OspA vaccine, since OspA antibodies is Lyme disease."
And he dares to say *WE* have "some
psychiatric illness?"
Here's the old 1990 CDC standard test for Lyme- which is Allen Steere's
first standard - changing and expanding IgM and IgG antibodies, or that one
should perform serial Western Blots, since Lyme is a relapsing fever borreliosis
It was
because of this fact, that the testing for Lyme changed to suit the
false-positive outcome of LYMErix, that AG
Richard Blumenthal's staff lawyers referred me to
US Attorney Kevin O'Connor
to file the scientific fraud and racketeering complaint against Yale in July
2003.
What I told the FDA on January 31, 2001:

http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf
Jan 31, 2001, KMDickson


YouTube Movie- Listen to Mark Klempner, himself, discuss
his "unpublished and not for large dissemination" HLA data re the genetic link
between Lyme and Multiple Sclerosis
(HLA-DQB1*0602)
You may be wondering how the nitrous oxide got into Allen
Steere's neonatal rat brains so that Steere would publish in 1994 what he thinks
is a scientifically valid explanation for ....
"Induction of NO synthase
by bacterial agents within the brain may represent a common pathway of CNS
inflammation and neurotoxicity. PubMed ID # 7513330"
And then Steere later said we had "some
psychiatric illness," rather than a real brain disease.
Ol' Vijay Sikand of East Lyme, Corrupticut tawkin to da FDA 'bout how serious
Lyme disease is at the 1998 FDA meeting about LYMErix. Later these crooks all
claimed Lyme is imaginary:
http://www.fda.gov/ohrms/dockets/AC/98/transcpt/3422t1.rtf
"It is well known that Borrelia burgdorferi indeed after asymptomatic infection
can lurk or secrete itself in certain areas of the body, perhaps the central
nervous system or perhaps the joint spaces, only to reappear months or maybe
years later in the form of late stages of illness which are harder to diagnosis
and treat.
"It is probably worth noting, since I have learned a lot, that we don't have the
clinical luxury in private practice that we had in the SmithKline Beecham trial
in which we had baseline sera on all the patients who enrolled so that when they
presented with symptoms, we could draw acute and convalescent serologies so as
to compare them with each other and with baseline to better understand what
symptoms they are presenting with.
"Finally, there are indeed many dilemmas in therapy. In particular, untreated or
inadequately treated Lyme disease may lead to the chronic morbidity with which
we are very familiar. Most commonly arthritis and the not common but complex
neurological syndromes are what often result and which confront the primary care
physician in the office diagnostically and therapeutically.
"These particular outcomes result in much more intensive, long-term expensive
therapy, often in the form of long-term intravenous antibiotics. These are the
patients who often are refractory to treatment. Indeed, these are the patients
in whom symptoms seem to persist despite what we have given in terms of adequate
antibiotic therapy by any known measure.
"In conclusion, we need a vaccine for Lyme disease because it is increasing in
incidence and geographic spread. We need a vaccine for Lyme disease because
there are problems in clinical diagnosis, its laboratory evaluation, and its
treatment. We need a vaccine for Lyme disease because preventive measures are
unfortunately ineffective. Lyme disease is indeed vaccine preventable.
Availability of this vaccine would lead to a significant reduction in chronic
sequelae and substantive morbidity. Lyme vaccine is thus a critical new public
health approach to the primary prevention of Lyme disease in the United States.
Thank you very much."--
Vijay Sikand, EAST LYME, CT
"But I,
Vijay Sikand, also did not report adverse events to the FDA because Yale
told me not to. We're all secretly pretending spirochetes only infect
knees.

Mycoplasma and Red Blood Cells- reports published
in National Library of Medicine (formerly known as MedLine)
6190898
Among the
Mycoplasma or stealth fungal infections studied at Plum Island....
Immunogenic variation among the
so-called LC strains of Mycoplasma mycoides subspecies mycoides.
Much evidence of immunogenic
heterogeneity among the LC strains of Mycoplasma mycoides ssp. mycoides emerged
from cross-immunization and -hyper-immunization experiments in mice in which
three LC strains (Vom/Plum Island, 74/2488, and Mankefar 2833) were used
for challenge purposes. All heterologous LC-strain vaccines cross-immunized
against the three challenge strains, but protection was usually only 'partial',
i.e. significantly less than that given by homologous vaccine. Cross-hyperimmunization
with all heterologous LC but not SC strains produced protection against
challenge with Vom/Plum Island that was virtually 'complete', i.e. similar to
that produced by homologous vaccine. Challenge with 74/2488 gave generally
similar results; but against Mankefar 2833 six heterologous LC vaccines gave
complete protection and six did not. Vaccines prepared from the Smith (1423)
strain of M. mycoides ssp. capri gave some protection against Vom/Plum Island
but none against 74/2488 or Mankefar 2833. The cross-immunizing ability of three
further M. mycoides ssp. capri strains appeared to resemble that of Smith
(1423). In a cross-hyperimmunization experiment, vaccines prepared from SC
strains of M. mycoides ssp. mycoides varied greatly in their ability to protect
against challenge with strains 74/2488 and Mankefar 2833. PMID: 6190898
[PubMed - indexed for MEDLINE]
"Lipoproteins and
lipopeptides have been identified in a large number of
microorganisms, the most prominent ones being mycobacteria,
mycoplasms, and spirochetes. They have been found to exhibit both a
strong innate inflammatory response in the host and an enduring
adaptive immune response in mammalian hosts (16).
The strong proinflammatory capacities of lipoproteins were first
described for outer surface proteins A and B of Borrelia burgdorferi,
which are also highly immunogenic (17)
and have lately been the basis for a Lyme disease vaccine development
(18).
These compounds exhibit an triacylated lipid anchor structure
comprising an N-palmitoyl-S-[2,3-bis(palmitoyloxy)-(2RS)-propyl]-(R)-cysteinyl
(Pam3Cys) moiety at the N terminus (19),
a feature that was previously described for the Braun lipoprotein
from Escherichia coli (20).
Because the N-terminal Pam3Cys modification is essential
for immunoactivation caused by lipoproteins of B. burgdorferi
as well as of another spirochete, Treponema pallidum (21),
subsequent studies investigating immune responses to spirochetes used
synthetic lipopeptides (22).
The Pam3Cys moiety was also reported to be present in
cytokine-inducing lipoproteins of Mycobacterium and
Mycoplasma spp. (23,
24); thus,
it can be regarded as a highly conserved molecular motif among
different classes of bacteria. In Mycoplasma fermentans, the
presence of a macrophage stimulating lipopeptide, termed 2-kDa
macrophage-activating lipopeptide (MALP-2), was observed, being
stimulatory active at picomolar concentrations (25).
This compound, in contrast to the predominant lipopeptide structures
present in lipoproteins of E. coli, B. burgdorferi, and
mycobacteria, lacks the N-palmitoyl group, thus containing a
diacylated (Pam2Cys) lipid anchor structure at the N
terminus. Following studies revealed the presence of closely related
compounds in other Mycoplasma spp. (26).."
from:
http://www.jimmunol.org/cgi/content/full/173/4/2683
More on this type of immune suppression from
MedLine
Must read:
http://www.jbc.org/cgi/content/full/274/47/33419
(UCONN's
Justin Radolf)
"Toll-like receptors (TLRs) 2 and 4 are signal transducers
for lipopolysaccharide, the major proinflammatory constituent in the
outer membrane of Gram-negative bacteria. We observed that membrane
lipoproteins/lipopeptides from Borrelia burgdorferi,
Treponema pallidum, and Mycoplasma fermentans activated
cells heterologously expressing TLR2 but not those expressing TLR1 or
TLR4. These TLR2-expressing cells were also stimulated by living
motile B. burgdorferi, suggesting that TLR2 recognition of
lipoproteins is relevant to natural Borrelia infection.
Importantly, a TLR2 antibody inhibited bacterial lipoprotein/lipopeptide-induced
tumor necrosis factor release from human peripheral blood mononuclear
cells, and TLR2-null Chinese hamster macrophages were insensitive
to lipoprotein/lipopeptide challenge. The data suggest a role
for the native protein in cellular activation by these ligands.
In
addition, TLR2-dependent responses were seen using whole Mycobacterium
avium and Staphylococcus aureus, demonstrating that this
receptor can function as a signal transducer for a wide spectrum of
bacterial products. We conclude that diverse pathogens activate cells
through TLR2 and propose that this molecule is a central pattern
recognition receptor in host immune responses to microbial invasion."
http://www.jimmunol.org/cgi/content/full/173/4/2683
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