The fraud and racketeering (an intentional
monopoly on vector-borne diseases testing for Yale and Allen Steere) in the
testing for Lyme, and the fraud over illness signs (valid markers of disease vs
psychiatric bullshit)
EXPLAINER ON THE BOGUS
TESTING FOR LYME:
1) Invitations to "PARTICIPATE IN
THE PROCEEDINGS" The Dearborn FARCE
2) Who was in the "work group," who
then threw out all the recommendations of the invited labs:
http://www.actionlyme.org/Dearborn_Who_Approved.htm
3) What the invited labs said (see
last three pages, which are in the wrong order):
http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf
4) June 1994 FDA Meeting, Dattwyler
recommends serial Western Blots to look for changing and expanding IgM and IgG
()because this is a relapsing fever bug which does antigenic variation):
http://www.actionlyme.org/Dattwyler_Luft_Bb_DNA_in_CSF.htm
5) Steere recommends serial Western
Blots to look for changing and expanding IgM in 1986:
http://www.ncbi.nlm.nih.gov/pubmed/3531237
6) CDC in 1990 adopts Steere's
Relapsing Fever and Changing bands, serial Western Blotting schema (because Lyme
is a relapsing fever organism that does antigenic variation):
http://www.actionlyme.org/CDC_DOCUMENTS_1990.htm
7) Weinstein tries again to
validate the complete and utter nonsense that high antibody concentration is a
component of method validation, and even tries to come up with his own bogus
detector (any chromatographer would die laughing at this):
http://www.nymc.edu/intouch/spr98/lyme.htm
(Google chromatography detectors-
you will see that Weinstein is insane)
8) Weinstein tries to pass
off a bogus validation where high antibody responses are a good thing, whereas
in real life, you are trying to develop a method which detects the lowest amount
of something, reliably:
http://www.ncbi.nlm.nih.gov/pubmed/8053960
This is the
"It's-not-an-elephant-unless-it-weighs-40-million-tons" "validation." Most
of us can detect 200 pound elephants and would prefer to. BushCo applied
the Weinstein Rule to Al Quaeda in 2001: "It's not a terrorist threat unless
they actually *do* fly planes into the WTC."
9) Yale and
several other labs develop a flagellin-specific antibody test because they have
figured out that Lyme is a borreliosis:
http://www.ncbi.nlm.nih.gov/pubmed/1894359
http://www.ncbi.nlm.nih.gov/pubmed/2341173
http://www.ncbi.nlm.nih.gov/pubmed/2380361
Here is Yves Lobet of SmithKline
(Google his name and mine in the same google search):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=1356932%5BUID%5D
identifying spirochete species in
ticks, using the non-varying DNA products, which should always be in use because
not everyone has burgdorferi- osis. Not everyone has
borreliosis with the OspA in it. In fact, we may not have
known for a long time that the arthritis that was a new epidemic in Lyme was due
to a spirochete, unless the US had drafted Willy Burgdorfer from Switzerland to
work on the US bioweapons program.
The fact that the NIH's Rocky
Mountain Labs was surrounded by a moat, tells you this is what they did there.
'Same vector-pathogen competence studies that they have done for years on
Plum Island. If you had done the research I recommended on
Yale's Shope and Tully, you would have found that Yale and Plum Island and
animal diseases and vector borne disease are all part of the same bioweapons
mixing pot:
http://www.actionlyme.org/LYME_CORRUPTICUT.htm
and that is why Steere was looking
for a virus as a cause of Lyme arthritis. These guys mainly
worked with weird viruses, that is IF YOU DO THE RESEARCH REPORTED BY TULLY AND
SHOPE and knew anything about WWII, the OSS, the CIA, and Yale (Yale is the OSS'
Bletchley Park II, the former would-be brainiac center, which is now missing all
of their brainiacs, and hires only RE-tards, like Durland Fish and Allen
Steere):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=tully%20jg%5BAuthor%5D
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=shope%20re%5BAuthor%5D
The Rockefeller Institute also worked on the US
bioweapons program. That would likely be because the
Rockefeller family actually helped the US during WWII, unlike many other
companies, like the Bush Bankers, Harriman Brown who instead aided the NAZI
party.
http://www.google.com/search?hl=en&lr=&q=averill+harriman++brown+bush+union+banking+corporation+thyssen&btnG=Search
The OSS was based in New York at the Rockefeller
Center. The OSS was the British Intelligence group which
became the CIA, much to the temper tantrums, and interference of yet another
queer, like Edward McSweegan, J Edgar Hoover.
Of course, then David
Rockefeller went off the deep end and joined the Council on Foreign Relations.
This RICO-separated Standard Oil recombined into Exxon Mobile, which was
invited to the White House in Jan 2001, as regards Iraq's oil:
http://www.youtube.com/watch?v=-uMpGefb3ok&mode=related&search=
But I diverge. The problem is
Yale University and their bioweapons program:
http://www.actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm
"Chuck" goes particularly off the deep end when I
talk about this, and that would be because his good buddy Durland Fish is
involved in Yale-Plum Island bioweapons program, as you can see for a fact from
those published articles.
Here is the US Army admitting
ticks could be used to deploy bioweapons:
http://www.actionlyme.org/LYME_A_BIOWEAPON.htm
And here is Plum Island identifying
some of the mycoplasmas they worked with there (mycoplasmas cause plant rots and
smuts, as well as causing arthritis and inhibition of milk production in
livestock, so they have triple bioweapons usefulness in addition to being
stealth, like borreliosis, and this is from where we got OspA):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=6190898%5BUID%5D
And here is Durland Fish performing
the same vector-pathogen competence studies which lead to the accidental release
of a Relapsing Fever Borrelia which has acquired the mycoplasmal antigen OspA
(Tully found numerous mycoplasmas and spiroplasmas in ticks):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=9499019%5BUID%5D
OspA is similar to antigens found in mycoplasma:
http://www.jimmunol.org/cgi/content/full/173/4/2683
VALID MARKERS OF DISEASE
1) Klempner determines that ceftriaxone does not kill all the spirochetes:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=1634816&query_hl=47&itool=pubmed_docsum
"Thus, several eukaryotic cell types provide the Lyme disease spirochete with a
protective environment contributing to its long-term survival."
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=8486939&query_hl=47&itool=pubmed_docsum
"These observations suggest that B. burgdorferi can adhere to, penetrate, and
invade human fibroblasts in organisms that remain viable."
Klempner's Matrix-Metalloproteinases in the spinal fluid of Lyme patients which
he thinks are contributing to the dementia:
http://www.actionlyme.org/Retro_Klempnerization.htm
(You have to print out one jpg file at a time by selecting it first and copying
it to a Word document)
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=9466528&query_hl=52&itool=pubmed_docsum
Klempner later publishing that there are no valid markers of illness in CNS Lyme
patients and is an author of the IDSA guidelines:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=12821733&query_hl=64&itool=pubmed_docsum
=========
2) SIGAL AND QEEG:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_t
erm=7554300%5BUID%5D
"Quantitative EEG, flash visual evoked potentials, auditory evoked potentials to
common and rare tones, and median nerve somatosensory evoked potentials were
obtained from 12 patients with active CNS Lyme disease and from 11 patients
previously treated for active CNS Lyme disease. Abnormal QEEG and/or EPs were
found in 75% of the active Lyme disease patients and in 54% of the post CNS Lyme
disease patients. Three different types of neurophysiological abnormality were
observed in these patients including QEEG slowing, possible signs of cortical
hyperexcitability, and focal patterns indicating disturbed interhemispheric
relationships. In patients tested before and after treatment QEEG and EP
normalization was associated with clinical improvement."
He later said we have fibromyalgia and that fibromyalgia is catastophizing:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_t
erm=sigal%20LH%5BAuthor%5D%20AND%20%28%22fibromyalgia%22%5BMeSH%20Terms%5D%20OR%
20fibromyalgia%5BText%20Word%5D%29
He also said we're poisoning ourselves and our children, as a cause of chronic
Lyme disease:
http://www.actionlyme.org/MUNCHAUSENS.htm
====
3) Steere, Sigal, Rahn and Schoen report Lyme treatment fails in 1/2 the cases:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=3109144&query_hl=44&itool=pubmed_docsum
===========
4) Halperin and Quin as a neurotoxin
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Search&db=pubmed&term=quinolin
ic+acid+and+borrelia&tool=fuzzy&ot=quinolonic+acid+and+borrelia
TODAY (They don't know what Chronic Lyme is? see more below):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=17029130&query_hl=66&itool=pubmed_docsum
Halperin is a contributor to the report that shows that Lou Gehrig's Disease is
associated with Lyme in 47% of the cases:
http://www.actionlyme.org/ALS_&_Lyme_47%25.htm
Why doesn't Halperin, an author of the new IDSA guidelines mention this as an
outcome of Lyme?
===============
5 & 6 ) GFAp, Yale, Schoen, and Congenital Lyme:
Yale's Robert Schoen mentioning Gliosis (degradation of glial cells, or the
energy producing cells of the brain) as a serious outcome of NeuroLyme:
http://www.annals.org/cgi/reprint/132/8/661.pdf
Who said what derogatory/mysogynistic remark and where it is published:
http://www.actionlyme.org/UN_PETITION.htm
"LYME PARANOIA"-- Robert Schoen, MD, Yale Rheumatology
Robert Schoen tells me I have "I don't know."
http://www.actionlyme.org/Schoen.htm while pregnant, having given Lyme and
probably ehlichiosis to my first two kids, and Yale knows Lyme congentially
confers, since Yale pathology contributed to this autopsy of a dead congenital
Lyme newborn
http://www.actionlyme.org/Congenital_Brain_Infection_of_Newborn_Resulting_in_Dea
th.htm
See more reports about cogenital newborns, Lyme and maternal antibodies - a
report by Allen Steere.
The Publication of GFAp as a marker of brain/nerve degradation that Schoen
references:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_t
erm=12938230%5BUID%5D
==============
6) NEOPTERIN (This report is not by a known US bad guy but is a valid marker of
infection)
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=7865624&query_hl=33&itool=pubmed_docsum
====
7) ANTIGANGLIOSIDE ANTIBODIES, Benach (who later published in the New York Times
that Steere was right, and Steere had the science on his side, but that the
patients don't, in a letter to the editor):
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=7558329&query_hl=32&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?itool=abstractplus&db=pubmed&cmd=R
etrieve&dopt=abstractplus&list_uids=8410057
'Benach demonstrated that antibodies to gangliosides inhibit nerve function in
vitro.
Benach saying to the TIMES that we patients are idiots and Steere is a genius:
http://query.nytimes.com/gst/fullpage.html?res=9F05EFDE173CF933A05756C0A9669C8B6
3
=====
8) ANTIPHOSPHOLIPID ANTIBODIES (LUPUS), Steere
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra
ctPlus&list_uids=3408508&query_hl=34&itool=pubmed_docsum
======
9) LYME AND ALL BORRELIOSES ARE PERMANENT BRAIN INFECTIONS
by Bergstrom, the business partner (See the US and European Espacenet patent
databases) of Alan Barbour, CDC EIS Officer and CDC Level IV Bioweapons lab head
at UCAL Irvine;
Alan Barbour says Lyme and Relapsing Fever permanently infect the brain on his
website:
http://www.ucihs.uci.edu/microbio/index.html?top.html&menu.html&facultyResearch/
faculty/barbour.html
"Lyme disease and relapsing fever. These tick-borne infections are notable for
multiphasic antigenic variation through DNA recombinations in the case of
relapsing fever, the occurrence of chronic arthritis in the case of Lyme
disease, and invasion of and persistence in the brain in the case of both
diseases."
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_t
erm=%28%22borrelia%22%5BMeSH%20Terms%5D%20OR%20borrelia%5BText%20Word%5D%29%20AN
D%20%28%22brain%22%5BMeSH%20Terms%5D%20OR%20brain%5BText%20Word%5D%29
Barbour says Lyme and Relapsing fever are nearly the same thing, and both infect
the brain, which used to be the culture media-rodent brains, says Barbour in
1986:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=3540570
http://www.actionlyme.org/LYME_IS_A_PERMANENT_BRAIN_INFECTION.htm
'All the other older reports where it was "well-known" that borrelia permanently
infect the brain
1: Microbes Infect. 2006 Jul;8(8):2213-9. Epub 2006 May 30.
Persistent brain infection and disease reactivation in relapsing fever
borreliosis.
* Larsson C,
* Andersson M,
* Pelkonen J,
* Guo BP,
* Nordstrand A,
* Bergstrom S.
Department of Molecular Biology, Umea University, SE-901 87 Umea, Sweden.
Relapsing fever, an infection caused by Borrelia spirochetes, is generally
considered a transient, self-limiting disease in humans. The present study
reveals that murine infection by Borrelia duttonii can be reactivated after an
extended time as a silent infection in the brain, with no bacteria appearing in
the blood and spirochete load comparable to the numbers in an infected tick. The
host cerebral gene expression pattern is indistinguishable from that of
uninfected animals, indicating that persistent bacteria are not recognized by
the immune system nor cause noticeable tissue damage. Silent infection can be
reactivated by immunosuppression, inducing spirochetemia comparable to that of
initial densities. B. duttonii has never been found in any host except man and
the tick vector. We therefore propose the brain to be a possible natural
reservoir of the spirochete. The view of relapsing fever as an acute disease
should be extended to include in some cases prolonged persistence,
a feature characteristic of the related spirochetal infections Lyme disease and
syphilis. PMID: 16782384 [PubMed - in process]
VIJAY SIKAND SAYING LYME IS LATENT IN THE BRAIN LIKE SYPHILIS AT
THE FDA MEETING IN 1998 WHEN THEY WERE TRYING TO PASS OFF A BOGUS VACCINE:
http://www.fda.gov/ohrms/dockets/AC/98/transcpt/3422t1.rtf
"In particular, the specter of asymptomatic infection is something that troubles
me a great deal and troubles a great number of my colleagues who need to treat
Lyme disease. The obvious analogy with syphilis infection with Treponema
pallidus is there to consider. It is well known that Borrelia burgdorferi
indeed after asymptomatic infection can lurk or secrete itself in certain areas
of the body, perhaps the central nervous system or perhaps the joint spaces,
only to reappear months or maybe years later in the form of late stages of
illness which are harder to diagnosis and treat..."
LYMErix was never demonstrated to prevent Lyme, since the testing for Lyme is
bogus:
http://www.fda.gov/ohrms/dockets/ac/01/slides/3680s2_11.pdf
The testing is bogus; Yale's
RICO plan to get all the national blood See:
http://www.actionlyme.org/LYME_CORRUPTICUT.htm
was foiled, so NIH just gave them a 31.4 million dollar grant to assimilate batches of patients (they are going to open a clinical center since they're
totally clueless and intellectually uncoordinated). I wonder who could
have approved this grant, since McSweegan is in
charge of approving grants?
In three instances these insane IDSociety.org and Yale, SmithKline and
Kaiser-Permanente biotech racketeers revealed that the know all the testing for
Lyme is bogus and that you can't use any Osp primers, or Osp primer products for
antibody testing, so take note:
1) Target Imbalance by Dave Persing (get the whole text at a medical library):
http://www.ncbi.nlm.nih.gov/...8158048
2) Firstly, in 1990, by Andrew Pachner, sampling bugs from brains, (and note
that this was an invitro study, so he never proved antibiotics cleared the
spirochetes in the brain (they don't, which is what the Pfizer scientists told
me, and Pfizer's researchers belonged to the Jay Sanford ID group, which was not IDSociety):
http://www.ncbi.nlm.nih.gov...2215944
And 3) Fikrig at Yale says
the same thing, for a third time:
http://www.ncbi.nlm.nih.gov/...14671329
The only primers you can use are the intragenic spacers RNA and flagellin DNA
and product and NEVER any DNA from any varying Osps.
The three main things wrong with IDSA guidelines are
1) The Dearborn/Steere antibody testing schema we have now for Lyme is bogus,
which means Klempner's study is bogus which means the IDSociety’s Guidelines are
bogus. That, we already demonstrated was invalid as soon as it came out:
http://www.ilads.org/position2.html
The testing for Lyme is bogus, therefore we have no idea what kind of patients
Klempner had, and he refused to answer me when I asked him what primers he used
to Borrelia DNA assay spinal fluid in those patients.
This CDC testing we have now is only to verify Lyme arthritis, because
Borreliosis is actually a bioweapon, as we learned from McSweegan's old
“Bioterror” website, and is a "stealth disabler"- which means low or no antibody
response, and which means, the arthritis kind of Lyme as the only kind of Lyme
is FRAUDULENT:
http://www.actionlyme.org/
verify with the published data on linked from my website.
A)
The US Army says ticks could be used as
bioweapons to spread disease,
B) The Department of Energy calls Lyme "the
perfect stealth bacteria" which means it usually does not have the Steere/Dearbor
kind of antibody response;
C) UNSCOM was looking for tick nursery equipment in Iraq before duyba dubya
dubya dot dubya's bogus 9/11 war with the bogus Niger uranium letter and the thermite
in the WTC dubya dubya three.
Lyme arthritis, is a genetically-linked outcome. Like, not everyone has seasonal
allergies, and not everyone has a genetic predisposition to have arthritis.
Klempner himself also explains it here (Windows Movie File):
http://www.actionlyme.org/Klempner-0602.wmv
The difference in the blots you see here, are clear:
http://www.actionlyme.org/USDOJ_COMPLAINT_RICO.htm
On the right, the darker bands means higher antibody concentration (is an
inflammatory/genetically linked arthritis or acrodermatitis response)
2) Any DNA testing for Lyme in a patient has to be the non-changing kind. No
OspA gene or anything like it, but only Borreliae genus and species specific
flagellin, or the same primers that Wormser et al use to look for Borreliae in
ticks or in erythema rashes that they used previously. (16-23S RNA, RNA spacers)
http://www.pubmedcentral.nih.gov/.....9986813
Barbour's lonestari flagellin patent:
http://patft.uspto.gov/.....5,932,220
Notice that these are non-varying DNA primers, and are the essential ones to be
used all the time when testing patients via a DNA method.
Flagellin or the Borreliae specific intragenic spacers.
ONLY.
Right now no one uses these primers. None of these valid genus and species
(some say Phylum and some say Order) primers were used in any treatment or
diagnois protocols, nor are they even available to the public by any lab.
3) The IDSocirty treatment on tickbite plan has never been validated, in
the manner dissemination had been checked in the past, where Ray Dattwyler says
he sampled the spinal fluid of people who showed up in his clinic with EM rashes
and found Bb DNA in 2/3 of those people.
http://www.actionlyme.org/Dattwyler_Luft_Bb_DNA_in_CSF.htm
IDSA offers no similar proof that their short treatment protocol for tick bite
is valid. They did not sample the spinal fluid of people who were treated
with one dose of doxy, 3 or 4 weeks later and, using the right primers,
determine that the brain had not been invaded.
Simon Sez: "One way of doing that is through
neuro-imaging, but we didn’t get the money to do that, so instead we have used
sophisticated neuro-psychological testing," -- NOT
SCIENTIFICALLY VALID.
"Those tablets, the NAPS
tablets, it’s just not possible to study. Pesticides, we don’t find evidence.
[The antidotes to nerve agents given to veterans are a problem- KMD]
Chemical weapons, well, we don’t think that for the British armed forces that
was a big issue. But we do think there is a relationship between a particular
pattern of protection and what happened later."-- NOT
SCIENTIFICALLY VALID.