14 Feb 2012
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Pharma/CDC on Brain damage from vaccines, Fauci, Phages, Bioweapons manufacture
HHS.gov is Incompetent; BMJ calls fraud "crime.")
Official: CFIDS and MS-Lyme are the
same disease; Epstein-Barr
ELISA = arbitrary cutoff.
Disclaimer
1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.
Bush/Gore Oil/War-(Oct,2000)
Bush's own explainer (Oct
2000):
Iraq Oil
Iraq was an oil-theft war.
See the BIOMARKERS2.htm Chapter of Cryme Disease
Munchausen's Accusations:
by Lenny Sigal
FDA Rules FDA rules on method validations
National Library of Medicine:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?CMD=Display&DB=pubmed
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?CMD=search&DB=taxonomy
http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=138 BORRELIA
Genetically organized on the NLM taxonomy database by Flagellin:
THE_CENTRAL_LYME_RICO_PATENTS.htm
Borreliosis is permanent brain infection
Klempner's Matrix-Metalloproteinases in the spinal fluid of Lyme victims (which means they're not imaginating their symptoms)
Alan Barbour's Lonestari patent
Pachner Brains
Barbour's website
The Biology of Parasitic Spirochetes- Boreliosis is an incurable brain infection."
Datapackage to US Corrupticut Attorney and a copy was stolen off Blumenthal's desk by Jessica Gauvin (Oct 4, 2003)
BIOMARKERS OF DISEASE- Discovered by the very people who now deny this valid evidence that we're really sick
John J. Connolly and the Kaiser sell-out to the RICO enterprise... See Letter to Spitzer.
Schoen and Me- my congenitally infected kids
Rapid Cycling Bipolar, Lyme and Children, Clinical Trial
47% of men with ALS in Lyme endemic areas have Lyme
Antiganglioside antibodies
Antiphospholipid antibodies
Antiheat shock protein antibodies
L2 Diagnostics (formerly Yale's Lyme and Lupus Clinic)
OspA, ABC transporters, porins (Barbour and p66)
Lymphocytes are spirochete's second favorite flavor
Antiglycolipids in Lyme
Don Wiley and MS likelihood for myelin to be an "antigen"
Western Blotting is chromatography, gel electrophoresis does not identity all markers of disease, but this is the only test we are allowed to use to prove that we are really ill and don't have self-poisoning, or Munchausen's or hysteria, or Aproria, or catastrophizing, whatever is Today's Flavor of Bullshit that we get from the racketeers.
Steere's HLA
HLA molecules graphical explainer
Klempner's HLA-DQB1*0602 secret Multiple Sclerosis kind of Lyme disease
Kinetics, Superantigens, Tolerance
Antibody concentration differences between Steere's imaginary "Lyme Disease" patients and everyone else:
There is no diagnostic code for neuroborreliosis, so no one is allowed to have it.
Spirochetes are stealth pathogens (UCONN's Justin Radolf, Andrew Pachner)
Munchausen's accusations (demonstrates intent to harm, and such accusations are critical to prove this is a CRIME and not a "controversy"
Psychiatry never performs any scientifically valid testing. They're fools/tools.
Don Wiley's attempt to prove autoimmunity (10 amino acide fragment of myelin and a GRASP generated image of an HLA molecule).
Wiley's demonstration that a pandemic influenza likelier from a swine flu mutation
Superantigen/toxin binds tightly HLA molecules
MS and mouse hepatitis virus
SARS = mouse hepatitis and ,.... forget
Anti-Heat Shock antibodies in MS and Lyme borreliosis
Sigal and anti-41 = anti-Heat Shock protein, bind neurons ??
Benach, New York Times. "Lyme patients are crazy and Steere is a genius," Benach's antiganglioside IgM antibodies in Borreliosis bind to human neurons and possibly inhibit nerve function. (Nodes of Ranvier)
Klempner's Matrix Metalloproteinase 130 in spinal fluid of borreliosis patients vs later report that "Cognitive effects of Lyme = imaginary" (this is scientific fraud).
Klempner and ceftriaxone in fibroblasts. Ceftriaxone does not kill all the spirochetes.
Klempner says human fibroblasts provide safe home for spirochetes.
Klempner's secret HLA. Audiotaped at South Country Hospital Diseases of Summer Conference. HLA-DQB1*0602
Klempner:
"Um, some people will view this as bad news, some will view it as good news, and some people will say, well, where do we go from here?” I think that really is the question, really is to coalesce and say, ”where do we go from here?
"Um, There, these patients obviously, are very, very much interested in that question, as we are, and I just want to highlight a preliminary piece of data of where we think we’re going from here, unpublished*, and not for large, uh, dissemination, but here is the preliminary data. ...
YouTube Movie- Hear Mark Klempner discuss his "unpublished and not for large dissemination" HLA data re the genetic link between Lyme and Multiple Sclerosis (HLA-DQB1*0602) This obviously means not all of us have Steere's haplotype, which has redefined "Lyme Disease." Listen to it "straight from the horse's mouth" -Mark Klempner himself.
"And, that is, that when you look for the possibility of an autoimmune disease, the best way to look is to see if there is any genetic clustering in HLA haplotypes. The reason for that is the way antigens get presented in the context of who you are, that is, your HLA haplotype. And we can talk in some detail about that. Those diseases that I think everybody would agree are so called Autoimmune :lupus, rheumatoid arthritis, type 1 diabetes, and perhaps MS, have some clear genetic clustering that leads us to believe that these are indeed autoimmune diseases, although we do not satisfy so-called Koch’s Postulates of autoimmune disease that we’ve written[?-KMD] about. And the odds ratio for your having that particular HLA type, in the case of R.A, a DR4, or a DQB0602 to protect you from type 1 diabetes, are on the order of 3 to 6. One of the ones that is probably highest, of course, is B27, in patients with alkyloiding spondolytis and the like. It turns out that if you look at the first 51 patients with post-treatment chronic Lyme disease, the patient population that participated in our study, there was a very high incidence of DQB0602 with an odds ratio of 770%. So it may well be that exposure to THAT organism with THAT background of HLA haplotype may lead you to develop chronic symptoms. That is a hypothesis that needs to be tested. It would obviously lead to an entirely new form and approach to therapy. "
Deploy the Psychiatric Whores.
Chronic Fatigue Syndrome and Fibromyalgia are psychiatric diagnoses so all Lyme victims lose their long term disability benefits
QEEGs
GFAps
Autoreactive T cells
Orginal CDC method to diagnose Lyme- SERIAL WESTERN BLOTS, is Allen Steere's Orginal Diagnostic Method... Thrown out IgM (not allowed to report)
Dearborn- Who Approved of this bogus Lyme test, anyway?
Robert Schoen in 1998 instructs MDs not to do any valid tests on LYMErix injured people and also reveals the monopoly described HERE: LYME_CORRUPTICUT
"You can't have Lyme disease, if you have an antibody to OspA, but OspA is the vaccine." "OspA is specific enough to prevent Lyme disease, but it is not specific enough to diagnose Lyme disease."
The reason for that BullShit was the Monopoly. Only L2 Diagnostics and Imugen were licensed to use the OspA-B-Less bug, See the HOW RICO WILL BE CHARGED PAGE.
LYMErix trial administrators only reveal a year and a half after LYMErix was on the market, that they could not read their Western Blots in LYMErix-vaccinated people, but they never reported that to the FDA
http://www.journals.uchicago.edu/CID/journal/issues/v31n1/991200/991200.html
Figure 1. Results of Western blotting (MarBlot for detection of IgG antibody to Borrelia burgdorferi; MarDx, Carlsbad, CA) of serum specimens from 14 recipients of the recombinant outer surface protein A Lyme disease vaccine. Representative molecular weights (in kDa) are identified in the far left column. P, a positive control subject with PCR analysis positive Lyme arthritis; N, a negative control subject. Study subjects 1
