BRITISH_PSYCHIATRY- How Psychopaths
Running the Asylum began.
BLAME THE VICTIM- that's the nature of evil; psychiatry.
http://actionlyme.org/Psychiatric_MumboJumbo.wmv
VIDEO Psychiatry;
bogus MDs, and their brain damaging "medicine" - DCF
Scientific journal references for the "Psychiatric MumboJumbo video
http://www.traumacenter.org/van_der_Kolk_Complex_PTSD.pdf
The worst trauma is the removal from parents.
All people, and all parents,
especially, should read this book, because the
American Psychiatric Association doesn't want you to What they don't want you to know, you
definitely want to know.
Don't be a negligent parent. Know what you
are dealing with when you are dealing with Yale University.
"Shrink" a
reality:
Permanent brain shrinkage associated
with long term use of psychotropics
SmithKline Hiding Bad Data Again-- Paxil
The Elsie Figueroa Amicus- The extraordinary dangerousness of living in
Corrupticut. A must-read for all CT residents.
People have no idea what a chamber of horrors they are getting in to when
they mess with psychiatry.
READ-->
http://www.benzodiazepine.org/
Psychiatrists blame their victims for
the brain damage and related agitation
they cause with their non-medicine.
They are also liable for the murder
suicides like Diaz and McDermott, and
they are liable for the pediatric
suicides in DCF's "care." The scientific published articles below
is the evidence that they are fully
aware of their liabilities.
Criminal Violence and suicide is
mitigated in every single case of
psychotropics's use. That's the
main DCF/DMHAS scam or racket- They
don't want it known, that they are
liable for this malpractice-
Psychiatric LIARS, PERJURERS, and
COWARDS
See
also:
http://www.breggin.com See
also the Andy Vickery video on akathisia:
http://justiceseekers.com/
See also, Sheller and Brooks:
http://www.sheller.com/
============
Was Diaz taken care of as regards his
complaints of agitation on antidepressants?
J Forensic Sci. 2003
Jan;48(1):187-9.
Neuroleptic-induced
akathisia and violence: a review.
Leong GB,
Silva JA.
Center for Forensic Services, Western
State Hospital, Tacoma, WA 98498-7213,
USA. leonggb@dshs.wa.gov
Surprisingly, the association of
neuroleptic-induced akathisia and
aggressive behavior was not formally
recognized until nearly two and one-half
decades of antipsychotic prescribing had
passed. Using a search of the
anglophonic literature, this phenomenon
is reviewed. Advances in
psychopharmacology have reduced
neuroleptic-induced akathisia and hold
promise to eliminate it altogether.
Nonetheless, important clinical and
forensic aspects of neuroleptic-induced
akathisia and aggression remain and are
explored. PMID:
12570226 [PubMed - indexed for MEDLINE]
Pfizer describing how
to treat the agitation and violence that
they simultaneously deny exists:
1:
J Psychopharmacol.
1998;12(2):192-214.
SSRI-induced
extrapyramidal side-effects and
akathisia: implications for treatment.
Lane RM.
Pfizer Inc., New York, NY 10017, USA.
laner@pfizer.com
The selective serotonin reuptake
inhibitors (SSRIs) may occasionally
induce extrapyramidal side-effects (EPS)
and/or akathisia.This may be a
consequence of serotonergically-mediated
inhibition of the dopaminergic system.
Manifestations of these effects in
patients may depend on predisposing
factors such as the presence of
psychomotor disturbance, a previous
history of drug-induced akathisia and/or
EPS, concurrent antidopaminergic and/or
serotonergic therapy, recent monoamine
oxidase inhibitor discontinuation,
comorbid Parkinson's disease and
possibly deficient cytochrome P450 (CYP)
isoenzyme status. There is increasing
awareness that there may be a distinct
form of melancholic or endogenous
depression with neurobiological
underpinnings similar to those of
disorders of the basal ganglia such as
Parkinson's disease. Thus, it is not
surprising that some individuals with
depressive disorders appear to be
susceptible to developing drug-induced
EPS and/or akathisia. In addition, the
propensity for the SSRIs to induce these
effects in individual patients may vary
within the drug class depending, for
example, on their selectivity for
serotonin relative to other monoamines,
affinity for the 5-HT2C receptor,
pharmacokinetic drug interaction
potential with concomitantly
administered neuroleptics and potential
for accumulation due to a long
half-life. The relative risk of EPS and
akathisia associated with SSRIs have yet
to be clearly established. The potential
risks may be reduced by avoiding rapid
and unnecessary dose titration.
Furthermore, early recognition and
appropriate management of EPS and/or
akathisia is required to prevent the
impact of these effects on patient
compliance and subjective well-being. It is important that the rare
occurrence of EPS in patients receiving
SSRIs does not preclude their use in
Parkinson's disease where their
potentially significant role requires
more systematic evaluation.PMID: 9694033
[PubMed - indexed for MEDLINE]
"Loss of
striatal cholinergic neurons as a basis
for tardive and L-dopa-induced
dyskinesias, neuroleptic-induced
supersensitivity psychosis and
refractory schizophrenia."
Miller R, Chouinard G., Biol
Psychiatry. 1993 Nov 15;34(10):713-38.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=7904833&query_hl=9
Psychatric
research in the 21st Century;
Opportunities and Limitations,
GR, Heninger, Millenium Article,
Molecular Psychiatry (1999) 4, 429-436 (Psychiatry is beginning to consider
thinking like scientists--KMD)
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=10523815&query_hl=12
"The
Pathophysiology of Agitation", Jearn-Pierre
Lindenmayer, J Clin Psychaitry 2002;61 (suppl
14_
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=11154018&query_hl=16
"Akathisia and
Exacerbation of Psychopathology; A
Preliminary Report", Dunca, et al.
Clinical Neuropharmacology, Vol 23, No.
3, pp. 169-173
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=10895402&query_hl=18
"Subjective
Emotional Experience and Cognitive
Impairment in Drug-Induced Akathisia",
Jong-Hoon Kim, et al, Comprehensive
Psychiatry, Vol.43, No. 6 (November/DEcember),
2002: pp 456-462
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=12439833&query_hl=20
"Correlations
Between Akathisia and Residual
Psychopathology: A By-product of
Neuroleptic-Induced Dysphoria",
Newcomer, et al. Br J
Psychiatry. 1994 Jun;164(6):834-8.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=7952993&query_hl=22
"Cardiovascular
Effects of Antipsychotics Used in
Bipolar Illness", Piepho, Robert W.,
J Clin Psychiatry 2002;63 [suppl
4]:20-23
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=11913671&query_hl=24
Movement
Disorders Associated With Atypical
Antipsychotic Drugs, Caroff, SN, et
al, J Clin Psychiatry 2002;63[suppl 4]
12-19
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=11913670&query_hl=26