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The Antics of the Crazy Stalker Durland Fish and the New Genre in "Education."
 


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1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.

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Bush's own explainer (Oct 2000) re: Iraq Oil




 

 



No response, Big shock (not)

 

July 26, 2000

To: Massachusetts Board of Registration in Medicine
ATTN: Consumer Protection Unit
10 West Street, 3rd Floor
Boston, MA 02111
CC: Massachusetts Lyme Disease Coalition
P.O. Box 1916
Mashpee, MA 02649

Re: Allen C. Steere, Jr., MD

From: Kathleen M. Dickson
Southeastern CT Support Group Facilitator
23 Garden Street
Pawcatuck, CT 06379
860-599-5451

 

I will demonstrate that Allen Steere has a nearly psychopathological indifference towards patient outcomes as demonstrated by his handling of Lyme disease patients.

I will site correspondence from him as well as some journal abstracts that demonstrate this.

The following letter was forwarded to me by friends and occurred about a week or two after I spoke with Victor Berardi, Technical Director at Imugen, asking him why they use Borelia burgdorferi (Bb) strain FRG from Germany and G39/40 from Guilford, CT., instead of the three CDC- recommended strains of B31, 297, 2591 for Western Blotting (WB). I asked if there was known antigenic variation, how likely was it that SeCT and SwRI patients would demonstrate strong antibody production against OSPs from a German strain? After much biotechnobabble about protein conformational folding and molecular weight which seemed to support my point of view, the conversation went nowhere. However, I made the point that everyone who is tested in their lab comes out with a negative result.

I think it more than coincidental that Steere and Sikand were suddenly interested in new strains (below) so soon after this conversation, but they made no offer to follow up on patients when it is known that some strains are more virulent than others (see abstracts below) to see if they could identify the more valuable strains for vaccine candidacy. Imugen is in partnership with Corixa (see CRXA on the NYSE for businessprofile) to discover new vaccine candidates and development of diagnostic test kits for tick-borne diseases.

I argue that it was careless of this team to not ask patients to come back after 6 mos or a year to determine of they had lasting effects past the Rx'd abx regimen offered. I also would like for you to investigate the professional integrity of Allen Steere as a treating physician, researcher, paid advisor to insurance companies and whether not his association with LifeSpan, as an employee constitutes a breach in medical ethics.

 

Subject: FW: CDC Lyme Disease Study
Date: 06 Jul 1999 08:17:14 -0400
From: Sssss@pfizer.com
To: kathleen.dickson@snet.net

Kathleen:

mmmm asked me to forward the message below to you.

ssss

______________________________ Forward Header

Subject: FW: CDC Lyme Disease Study
Author: mmmm at Groton-CR
Date: 7/2/1999 3:17 PM

ssss,

Would you be kind enough and forward this message to Kathleen.

Thanks

mmmm

-----Original Message-----

From: MXXX, Stacy L
Sent: Friday, July 02, 1999 2:59 PM
To: DL-CRGRO-NC-ALL; '#GRO_QAL_ALL'; #CR Groton 30 Day Notices
Subject: CDC Lyme Disease Study

This summer, Dr. Vijay Sikand and Dr. Allen Steere will be working on a CDC and NIH supported Lyme Disease study for which they are seeking patient volunteers. This study has been approved by Tufts/New England Medical Center's Institutional Review Board.

They are primarily interested in patients with physician diagnosed erythema migrans, in whom they will be studying pathogenesis of disease, histopathology, molecular and genetic diversity of Borrelia burgdorferi isolates, cell-mediated immune factors, and serology, in the setting of clinical presentation. These patients will have skin biopsy of the lesion (small enough to be dressed with a bandaid, no sutures) as well as bloodwork on the day of presentation. Lyme disease treatment will be initiated at that time. Only one follow-up visit is required for convalescent bloodwork, 2 to 4 weeks later.  The grant provides for a $100 payment to these volunteers.

Since specimens must be sent by overnight courier to Boston, patients must be seen no later than about 3:00 p.m. Monday-Friday. Dr. Sikand's office is located next door to Brooks Pharmacy at Flanders Four Corners in East Lyme.

For more information regarding this study, or if you are interested in participating in the study please contact Dr. Engelke at X-16860.

===

If you would like to verify with this Dr. EnXXX, I am pretty sure the number is 860-441-XXXX, but please protect my privacy/anonymity.

=======

========

1986 Steere (The Early Days, before the profitability of Vaccines and Diagnostic tests was evident.):

Clinical manifestations of Lyme disease.

Steere AC, Bartenhagen NH, Craft JE, Hutchinson GJ, Newman JH, Pachner AR, Rahn DW, Sigal LH, Taylor E, Malawista SE

 Zentralbl Bakteriol Mikrobiol Hyg [A] 1986 Dec 263:1-2 201-5

 Abstract

Lyme disease typically begins with a unique skin lesion, erythema chronicum migrans (ECM) (stage 1). Patients with this lesion may also have headache, meningeal irritation, mild encephalopathy, multiple annular secondary lesions, malar or urticarial rash, generalized lymphadenopathy and splenomegaly, migratory musculoskeletal pain, hepatitis, sore throat, non-productive cough, conjunctivitis, periorbital edema, or testicular swelling.  After a few weeks to months (stage 2), about 15% of patients develop frank neurologic abnormalities, including meningitis, encephalitis, cranial neuritis (including bilateral facial palsy), motor or sensory radiculoneuritis, mononeuritis multiplex, or myelitis. At this time, about 8% of patients develop cardiac involvement--AV block, acute myopericarditis, cardiomegaly, or pancarditis. Throughout this stage, many patients continue to experience migratory musculoskeletal pain in joints, tendons, bursae, muscle, or bone. Months to years after disease onset (stage 3), about 60% of patients develop frank arthritis, which may be intermittent or chronic. Recently evidence suggests that Lyme disease may also be associated with chronic neurologic or skin involvement. Thus, Lyme disease occurs in stages with different clinical manifestations at each stage, but the course of the illness in each patient is highly variable.

 

1994 Steere (This report is inconsistent with the next):

 

N Engl J Med 1994 Jan 27;330(4):229-34
Detection of Borrelia burgdorferi DNA by polymerase chain reaction in synovial fluid from patients with Lyme arthritis.

Nocton JJ, Dressler F, Rutledge BJ, Rys PN, Persing DH, Steere AC

Division of Rheumatology/Immunology, New England Medical Center, Boston, MA 02111.

BACKGROUND. Borrelia burgdorferi is difficult to detect in synovial fluid, which limits our understanding of the pathogenesis of Lyme arthritis, particularly when arthritis persists despite antibiotic therapy.

METHODS. Using the polymerase chain reaction (PCR), we attempted to detect B. burgdorferi DNA in joint-fluid samples obtained over a 17-year period.  The samples were tested in two separate laboratories with four sets of primers and probes, three of which target plasmid DNA that encodes outer-surface protein A (OspA).

 RESULTS. B. burgdorferi DNA was detected in 75 of 88 patients with Lyme arthritis (85 percent) and in none of 64 control patients. Each of the three OspA primer-probe sets was sensitive, and the results were moderately concordant in the two laboratories (kappa = 0.54 to 0.73). Of 73 patients with Lyme arthritis that was untreated or treated with only short courses of oral antibiotics, 70 (96 percent) had positive PCR results. In contrast, of 19 patients who received either parenteral antibiotics or long courses of oral antibiotics (> or = 1 month), only 7 (37 percent) had positive tests (P < 0.001).  None of these seven patients had received more than two months of oral antibiotic treatment or more than three weeks of intravenous antibiotic  treatment. Of 10 patients with chronic arthritis (continuous joint inflammation for one year or more) despite multiple courses of antibiotics, 7 had  consistently negative tests in samples obtained three months to two years after treatment.

CONCLUSIONS. PCR testing can detect B. burgdorferi DNA in synovial fluid. This test may be able to show whether Lyme arthritis that persists after antibiotic treatment is due to persistence of the spirochete.

 

1999 Steere:

 Arthritis Rheum 1999 Dec;42(12):2705-9
 Lack of Borrelia burgdorferi DNA in synovial samples from patients with antibiotic treatment-resistant Lyme arthritis.

 Carlson D, Hernandez J, Bloom BJ, Coburn J, Aversa JM,  Steere AC

Tufts University School of Medicine, New England Medical  Center, Tupper Research Institute, Boston, Massachusetts 02111, USA.

OBJECTIVE: To determine whether Borrelia burgdorferi DNA may be detected in synovial tissue from patients with Lyme arthritis who have persistent synovial inflammation after antibiotic treatment.

METHODS: Synovial specimens obtained at synovectomy from 26patients with antibiotic treatment-resistant Lyme arthritis and from 10 control subjects were tested for B burgdorferi DNA using 3 primer-probe sets that target genes encoding outer surface proteins A or B or a flagellar protein (P41) of the spirochete. RESULTS: The 26 patients with Lyme arthritis, who had received antibiotic therapy for a mean total duration of 8 weeks prior to synovectomy, and the 10 control subjects each had negative polymerase chain reaction (PCR) results in synovial samples. When the samples were spiked with approximately 1-10 B burgdorferi, all but 1 had positive PCR results, suggesting that spirochetal DNA could have been detected in most of the unspiked samples if it had been present.

CONCLUSION: These results indicate that synovial inflammation may persist in some patients with Lyme arthritis after the apparent eradication of the spirochete from the joint with antibiotic therapy.

 

So, which is it? 37% or none?

 

1999 Ben Luft, et al, identify strain variation playing a role in antigenicity and pathology:

 

 Genetics 1999 Jan;151(1):15-30

 Genetic diversity of ospC in a local population of Borrelia burgdorferi sensu stricto.

 Wang IN, Dykhuizen DE, Qiu W, Dunn JJ, Bosler EM, Luft BJ

 Department of Ecology and Evolution, State University of New York, Stony Brook, New York 11794-5245, USA.

 The outer surface protein, OspC, is highly variable in Borrelia burgdorferi sensu stricto, the agent of Lyme disease. We have shown that even within a single population OspC is highly variable. The variation of ospA and ospC in the 40 infected deer ticks collected from a single site on Shelter Island, New York, was determined using PCR-SSCP. There is very strong apparent linkage disequilibrium between ospA and ospC alleles, even though they are located on separate plasmids. Thirteen discernible SSCP mobility classes for ospC were identified and the DNA sequence for each was determined. These sequences, combined with 40 GenBank sequences, allow us to define 19 major ospC groups. Sequences within a major ospC group are, on average, <1% different from each other, while sequences between major ospC groups are, on average, approximately 20% different. The tick sample contains 11 major ospC groups, GenBank contains 16 groups, with 8 groups found in both samples. Thus, the ospC variation within a local population is almost as great as the variation of a similar-sized sample of the entire species. The Ewens-Watterson-Slatkin test of allele frequency showed significant deviation from the neutral expectation, indicating balancing selection for these major ospC groups. The variation represented by major ospC groups needs to be considered if the OspC protein is to be used as a serodiagnostic antigen or a vaccine.

 

1999 (July) Ray Dattwyler, et al Demonstrate that strain variation plays a role in antigenicity and pathology:

 

Infect Immun 1999 Jul;67(7):3518-24
Four clones of Borrelia burgdorferi sensu stricto cause invasive infection in humans.

Seinost G, Dykhuizen DE, Dattwyler RJ, Golde WT, Dunn JJ, Wang IN, Wormser GP, Schriefer ME, Luft BJ

Department of Medicine, State University of New York at Stony Brook, Stony Brook, New York 11794, USA.

Lyme disease begins at the site of a tick bite, producing a primary infection with spread of the organism to secondary sites occurring early in the course of infection. A major outer surface protein expressed by the spirochete early in infection is outer surface protein C (OspC). In Borrelia burgdorferi sensu stricto, OspC is highly variable. Based on sequence divergence, alleles of ospC can be divided into 21 major groups. To assess whether strain differences defined by ospC group are linked to invasiveness and pathogenicity, we compared the frequency distributions of major ospC groups from ticks, from the primary erythema migrans skin lesion, and from secondary sites, principally from blood and spinal fluid. The frequency distribution of ospC groups from ticks is significantly different from that from primary sites, which in turn is significantly different from that from secondary sites. The major groups A, B, I, and K had higher frequencies in the primary sites than in ticks and were the only groups found in secondary sites. We define three categories of major ospC groups: one that is common in ticks but very rarely if ever causes human disease, a second that causes only local infection at the tick bite site, and a third that causes systemic disease. The finding that all systemic B. burgdorferi sensu stricto infections are associated with four ospC groups has importance in the diagnosis, treatment, and prevention of Lyme disease.

 =====

I checked for nucleotide sequence similarity of major OSPs in B31, 297, 2591 and FRG and/or G39/40 in the Pasteur Institute Molecular Genetics Server for Bb and did not find these latter two strains listed.

I think there is enough evidence to say that identification of new strains (DNA patent) and a follow up on patient outcomes past the 2-4 week serological look (test kit) would have been prudent and that this statement reflects a broad and general attitude of intended negligence.

 

Berardi: Persistent Lyme Athritis is due to persistent infection.

Ann Intern Med 1986 Jun;104(6):798-800

Borrelia burgdorferi in joint fluid in chronic Lyme arthritis.

Snydman DR, Schenkein DP, Berardi VP, Lastavica CC, Pariser KM

Although indirect evidence suggests that chronic Lyme arthritis is caused by persistent infection with Borrelia burgdorferi, direct visualization has been lacking. We report the demonstration of B. burgdorferi from synovial fluid aspirated from the right knee of a 31-year-old man with Lyme arthritis for more than 1 year. After 6 days, culture medium inoculated with synovial fluid showed one motile and several nonmotile spirochetes. Direct immunofluorescence staining showed reactivity with anti-B. burgdorferi serum. Spirochetes were not seen in subcultured material. The patient's arthritis improved with high-dose intravenous penicillin. Identification of B. burgdorferi from the joint fluid of a patient with long-standing arthritis supports the concept that the arthritis is due to persistent infection.

 

More Berardi, no abstract available:

 

Zentralbl Bakteriol Mikrobiol Hyg [A] 1986

Dec;263(1-2):288

Demonstration of Borrelia burgdorferi in a patient with chronic Lyme arthritis.
Lastavica CC, Snydman DR, Schenkein DP, **Berardi VP**, Pariser KM
PMID: 3554842, UI: 87208551

Berardi and Steere: Degree of cross-reactivity with antibody-capture method.

J Infect Dis 1988 Oct;158(4):754-60

Serodiagnosis of early Lyme disease: analysis of IgM and IgG antibody responses by using an antibody-capture enzyme immunoassay.

Berardi VP, Weeks KE, Steere AC ,  Department of Virology, Center for Disease Control, Boston, Massachusetts 02130.

We used an antibody-capture enzyme immunoassay (EIA) to evaluate the early antibody responses to Borrelia burgdorferi in paired sera from 30 patients with erythema chronicum migrans. During acute disease, 20 (67%) patients had elevated specific IgM responses, and by convalescence (one to four weeks after treatment), 28 (93%) patients had increased IgM or IgG responses. In acute specimens, elevated IgM responses correlated with disseminated infection; however, by convalescence, most patients with either localized or disseminated disease had positive tests. Among 133 control subjects, IgM cross-reactivity was observed in 4 of 37 patients with either Epstein-Barr virus or rickettsial infections, and false-positive IgG tests were seen in 8 of 28 patients with syphilis. With antibody-capture EIA, the diagnosis of Lyme disease can be confirmed in the majority of acutely ill patients and in almost all patients by convalescence.

 

Steere and Berardi: Observing but not treating patients in the USSR.

 

J Infect Dis 1988 Oct;158(4):748-53

Lyme borreliosis in the Soviet Union: a cooperative US-USSR report.

Dekonenko EJ, Steere AC, Berardi VP, Kravchuk LN Poliomyelitis Institute, Moscow, Union of Soviet Socialist Republics.
 

We identified 90 patients with tick-borne erythema migrans in the Union of Soviet Socialist Republics (USSR) in areas from the western Baltic Republics to the Maritime Territory on the Pacific Ocean. Symptoms associated with the erythema included fever, malaise and fatigue, headache, myalgias, arthralgias, or regional lymphadenopathy. Within two weeks to four months, 58 (64%) of the patients developed neurological abnormalities, particularly radicular pain, cranial neuritis, or lymphocytic meningitis, and four (4%) patients developed monoarticular or oligoarticular arthritis. We tested the sera from 35 Soviet patients by using an isolate from the United States. The serological data showed elevated IgM and/or IgG antibody titers to Borrelia burgdorferi in 2 of 10 patients with erythema migrans, 15 of 21 with neurological abnormalities, and 2 of 4 with arthritis. Our observations suggest that Lyme borreliosis occurs in diverse areas of the USSR.

 

They brought their own Borrelia, and it looks like they only observed and did not treat patients. This demonstrated the accuracy of using US strains in the USSR. It is not an accurate test.] We would like you to investigate whether Allen Steere is using the CDC-recommended strains fo Borreia in his curtrent 4.5 million dollar grant study of Chronic Lyme Disease.

 

More Berardi:

N Engl J Med 1989 Jan 19;320(3):133-7

Rapid emergence of a focal epidemic of Lyme disease in coastal Massachusetts.

Lastavica CC, Wilson ML, Berardi VP, Spielman A, Deblinger RD

Department of Community Health, Tufts University School of Medicine, Boston, MA 02111.

We describe a focal epidemic of Lyme disease, which spread from a nature preserve and affected an adjacent community of permanent residents in coastal Massachusetts. The attack rate from 1980 through 1987 was 35 percent among 190 residents living within 5 km of the nature preserve and was greatest (66 percent) among those living closest to the preserve. The risk of infection bore little relation to sex or age. Late Lyme disease, which clustered near the preserve, occurred mainly in residents infected early in the epidemic who did not have a history of erythema migrans and did not receive antibiotic therapy. All the residents with serologic evidence of infection had early or late clinical  manifestations of Lyme disease, or both, during the period of study. The seasonal risk of infection was bimodal--greatest in June, with a secondary peak in October--and corresponded to periods of increased transmission. In the nature preserve, the density of the vector tick, Ixodes dammini, exceeded that in other New England sites. The zoonosis rapidly became endemic, and the severity of its impact correlated with the abundance of deer. This epidemic of Lyme disease demonstrated that outbreaks can be focal and can spread rapidly within a community of permanent residents.

 

[This does not mean that patients with early or late clinical manifestations of Lyme disease were seropositive, just that the seropositive patients had clinical signs. There is an important difference: Seronegative Lyme exists. See Next:]

 

"Seronegative Lyme disease. Dissociation of specific T- and B-lymphocyte responses to Borrelia burgdorferi [see comments]

*Dattwyler RJ*, Volkman DJ, Luft BJ, Halperin JJ, Thomas J, Golightly MG N Engl J Med 1988 Dec 1 319:22 1441-6

ABSTRACT: The diagnosis of Lyme disease often depends on the measurement of serum antibodies to Borrelia burgdorferi, the spirochete that causes this disorder. Although prompt treatment with antibiotics may abrogate the antibody response to the infection, symptoms persist in some patients. We studied 17 patients who had presented with acute Lyme disease and received prompt treatment with oral antibiotics, but in whom chronic Lyme disease subsequently developed. Although these patients had clinically active disease, none had diagnostic levels of antibodies to B. burgdorferi on either a standard enzyme-linked immunosorbent assay or immunofluorescence assay. On Western blot analysis, the level of immunoglobulin reactivity against B. burgdorferi in serum from these patients was no greater than that in serum from normal controls. The patients had a vigorous T-cell proliferative response to whole B. burgdorferi, with a mean ( +/- SEM) stimulation index of 17.8 +/- 3.3, similar to that (15.8 +/- 3.2) in 18 patients with chronic Lyme disease who had detectable antibodies. The T-cell response of both groups was greater than that of a control group of healthy subjects (3.1 +/- 0.5; P less than 0.001).  

We conclude that the presence of chronic Lyme disease cannot be excluded by the absence of antibodies against B. burgdorferi and that a specific T-cell blastogenic response to B. burgdorferi is evidence of infection in seronegative patients with clinical indications of chronic Lyme disease."

 

1990 Steere in West Germany     [Half the US patients had no intrathecal antibody response. This study have been the source of FRG- Berardi may have been there]

J Infect Dis 1990 Jun;161(6):1203-9

Evaluation of the intrathecal antibody response to Borrelia burgdorferi as a diagnostic test for Lyme neuroborreliosis.

Steere AC, Berardi VP, Weeks KE, Logigian EL, Ackermann R

Division of Rheumatology/Immunology, New England Medical Center, Tufts University School of Medicine, Boston, MA 02111.

The intrathecal antibody response to Borrelia burgdorferi was evaluated in American and West German patients with Lyme neuroborreliosis. By an antibody capture enzyme immunoassay, 12 (92%) of 13 patients from the USA with Lyme meningitis were found to have intrathecal antibody production to B. burgdorferi, usually of multiple isotypes, most commonly IgA. Of 12 patients with putative late central nervous system manifestations of Lyme disease, 5 (42%) had local production of IgG or IgA spirochetal antibody, but cerebrospinal fluid (CSF) abnormalities could not be demonstrated in 6 patients with late peripheral nervous system manifestations of the disorder. Compared with American patients, 30 European patients with neuroborreliosis had significantly higher CSF:serum ratios of specific antibody both early and late in the illness. Intrathecal antibody determinations are the most specific diagnostic test currently available for Lyme neuroborreliosis, but local antibody production in CSF is an inconsistent finding in American patients with late neurologic manifestations of the disorder.

 

1996 Berardi on Labs NO ABSTRACT

 

Arch Pathol Lab Med 1996 Apr;120(4):323-5

Proficiency testing program for Lyme disease.

Seder RH, Berardi VP

Publication Types: Comment Letter

 

Comment on: Arch Pathol Lab Med 1994 May;118(5):501-5

[The comment is not available.]

 

Long term Outcome Study Number 1 of 2

Conflicting results.

 

Ann Intern Med 1994 Oct 15;121(8):560-

The long-term clinical outcomes of Lyme disease. A population-based retrospective cohort study.

Shadick NA, Phillips CB, Logigian EL, Steere AC, Kaplan RF, Berardi VP, Duray PH, Larson MG, Wright EA, Ginsburg KS, et al

Department of Rheumatology-Immunology, Brigham & Women's Hospital, Boston, MA 02115.

OBJECTIVE: To ascertain the prevalence of and risk factors for long-term sequelae from acute Lyme disease.

DESIGN: Population-based, retrospective cohort study.

SETTING: A coastal region endemic for Lyme disease.

PARTICIPANTS: Patients with a history of Lyme disease who were previously treated with antibiotics were compared with randomly selected controls.

MEASUREMENTS: A standardized physical examination, health status measure (Short Form 36), psychometric test battery, and serologic analysis.

RESULTS: Compared with the control group (n = 43), the Lyme group (n = 38; mean duration from disease onset to study evaluation, 6.2 years) had more arthralgias (61% compared with 16%; P < 0.0001); distal paresthesias (16% compared with 2%; P = 0.03); concentration difficulties (16% compared with 2%; P = 0.03); and fatigue (26% compared with 9%; P = 0.04), and they had poorer global health status scores (P = 0.04). The Lyme group also had more abnormal joints (P = 0.02) and more verbal memory deficits (P = 0.01) than did the control group. Overall, 13 patients (34%; 95% CI, 19% to 49%) had long-term sequelae from Lyme disease (arthritis or recurrent arthralgias [n = 6], neurocognitive impairment [n = 4], and neuropathy or myelopathy [n = 3]). Compared with controls, patients who had long-term sequelae had higher IgG antibody titers to the spirochete (P = 0.03) and received treatment later (34.5 months compared with 2.7 months; P < 0.0001).

CONCLUSIONS: Persons with a history of Lyme disease have more musculoskeletal impairment and a higher prevalence of verbal memory impairment when compared with those without a history of Lyme disease. Our findings suggest that disseminated Lyme disease may be associated with long-term morbidity.

 

Comments:

 

Comment in: Ann Intern Med 1995 Jun 15;122(12):960-1; discussion 961-2

 

Comment in: Ann Intern Med 1995 Jun 15;122(12):961; discussion 961-2

 

Long Term Outcome Study Numbr 2 of 2

Ann Intern Med 1999 Dec 21;131(12):919-26

Musculoskeletal and neurologic outcomes in patients with previously treated Lyme disease.

Shadick NA, Phillips CB, Sangha O, Logigian EL, Kaplan RF, Wright EA, Fossel AH, Fossel K, Berardi V, Lew RA, Liang MH

 Harvard Medical School and Brigham and Women's Hospital, Boston, Massachusetts 02115, USA.

 

BACKGROUND: Previous follow-up studies of patients with Lyme disease suggest that disseminated infection may be associated with long-term neurologic and musculoskeletal were treated for Lyme disease in the late 1980s.  DESIGN: Population-based, retrospective cohort study.  SETTING: Nantucket Island, Massachusetts.  PARTICIPANTS: 186 persons who had a history of Lyme disease (case-patients) and 167 persons who did not (controls).  MEASUREMENTS: Standardized medical history, physical examination, functional status measure (Medical Outcomes Study 36-item Short Form Health Survey [SF-36]), mood state assessment (Profile of Mood States), neurocognitive tests, and serologic examination.  RESULTS: The prevalence of Lyme disease among adults on Nantucket Island was estimated to be 14.3% (95% CI, 9.3% to 19.1%). In multivariate analyses, persons with previous Lyme disease (mean time from infection to study evaluation, 6.0 years) had more joint pain (odds ratio for having joint pain in any joint, 2.1 [CI, 1.2 to 3.5]; P = 0.007), more symptoms of memory impairment (odds ratio for having any memory problem, 1.9 [CI, 1.1 to 3.5]; P = 0.003), and poorer functional status due to pain (odds ratio for 1 point on the SF-36 scale, 1.02 [CI, 1.01 to 1.03]; P < 0.001)  than persons without previous Lyme disease. However, on physical examination, case-patients and controls did not differ in musculoskeletal abnormalities, neurologic abnormalities, or neurocognitive performance. Persons with previous Lyme disease who had persistent symptoms after receiving treatment (n = 67) were more likely than those who had completely recovered to have had fever, headache, photosensitivity, or neck stiffness during their acute illness (87% compared with 13%; odds ratio, 2.4 [CI, 1.0 to 5.5]; P = 0.045); however,  the performance of the two groups on neurocognitive tests did not significantly differ.  

CONCLUSIONS: Because persons with previous Lyme disease exhibited no sequelae on physical examination and neurocognitive tests a mean of 6.0 years after infection, musculoskeletal and neurocognitive outcomes seem to be favorable. However, long-term impairment of functional status can occur.

 

So which is it?

 

Does this mean 67 out of 186 continued to have symptoms despite being treated (30%) and two thirds of all patients had memory impairment? This supports the conclusion that early severe neurological symptoms are associated with continued symptoms, yet, last summer Steere and Sikand did not offer to follow up on these patients. He is aware of strain variation affecting severity of illness from his own West German study. Steere has a history of psychopathological indifference to patient outcomes. I believe his results do not reflect his data.

 

A final tidbit-Addressing the issues of strain variation:

 

Anon wrote:

Hematol Oncol 1999 Sep;17(3):107-16

Positive serology for lyme disease borrelias in primary cutaneous B-cell lymphoma: a study in 22 patients; is it a fortuitous finding?

Jelic S, Filipovic-Ljeskovic I  Institut za Onkologiju i Radiologiju Srbije, Belgrade, Yugoslavia.

BACKGROUND: The historical association of acrodermatitis chronica atrophicans (ACA), now known to be a late manifestation of Lyme disease caused by Borrelia afzelii, with cutaneous lymphoma, and several small series of PCBCL with positive Lyme disease borrelial serology initiated a study of this association. Material and methods In the last 9 years, 30 patients with PCBCL have been observed and followed, 22 of them were tested for borrelial serology. The control group consisted of 85 patients with NHL (10 cutaneous T-cell, 25 extranodal B-cell non-PCBCL, 50 nodal B-cell), 30 patients with breast cancer and 60 blood donors. The screening tests were two different ELISA tests for B. burgdorferi sensu lato and sensu stricto, and reactive sera were further tested with the ELISA test for B. garinii, a Western blot (WB) test for Swiss Borrelia strains and a WB test for Bavarian Borrelia strains, ***since an immunoblot made with local strains was not available***.   Studies with a differential WB test for B. burgdorferi sensu stricto, B. garinii and B. afzelii was performed afterwards, as well as serological studies ruling out cross-reactions with Leptospiras and Treponema.  RESULTS: Fifteen of 22 patients with PCBCL were positive on the screening tests, three of them falsely.

 

This shows that it is recognized that one should ue local strains to WB patient sera.

 

Test-treatment strategies for patients suspected of having Lyme disease: a cost-effectiveness analysis.

Nichol G; Dennis DT; Steere AC; Lightfoot R; Wells G; Shea B; Tugwell P Ottawa Civic Hospital, Ontario, Canada.

Ann Intern Med 1998 Jan 1;128(1):37-48

ABSTRACT:   PURPOSE: To examine the cost-effectiveness of test-treatment strategies for patients suspected of having Lyme disease. DATA SOURCES: The medical literature was searched for information on outcomes and costs. Expert opinion was sought for information on utilities.   STUDY SELECTION: Articles that described patient population, diagnostic criteria, dose and duration of therapy, and criteria for assessment of outcomes.   DATA EXTRACTION: The decision analysis evaluated the following strategies: 1) no testing-no treatment; 2) testing with enzyme-linked immunosorbent assay (ELISA) followed by antibiotic treatment of patients with positive results; 3) two-step testing with ELISA followed by Western blot and antibiotic treatment for patients with positive results on either test; and 4) empirical antibiotic therapy. Three patient scenarios were considered: myalgic symptoms, rash resembling erythema migrans, and recurrent oligoarticular inflammatory arthritis. Results were calculated as costs per quality- adjusted life-year and were subjected to sensitivity analysis. Adjustment was made for the diagnostic value of common clinical features of Lyme disease.    DATA SYNTHESIS: For myalgic symptoms without other features suggestive of Lyme disease, the no testing-no treatment strategy was most economically attractive (that is, had the most favorable cost-effectiveness ratio). For rash, empirical antibiotic therapy was less costly and more effective than other strategies. For oligoarticular arthritis with a history of rash and tick bite, two-step testing was associated with the lowest cost-effectiveness ratio. Testing with ELISA and empirical antibiotic therapy cost an additional $880,000 and $34,000 per quality-adjusted life-year, respectively. For oligoarticular arthritis with one or no other features suggestive of Lyme disease, two-step testing was most economically attractive.

CONCLUSIONS: Neither testing nor antibiotic treatment is cost-effective if the pretest probability of Lyme disease is low. Empirical antibiotic therapy is recommended if the pretest probability is high, and two-step testing is recommended if the pretest probability is intermediate.

 

It is utterly preposterous for Allen Steere to have been involved in a study in which the hypothesis was that people have no inherent privilege or right to choose whether they should be evaluated and/ or diagnosed or choose be treated. What is an academician who "studies" the effects of Lyme disease, here and abroad, doing being involved in such a ridiculous, immoral, unethical endeavor?

Was there, Is there, some financial incentive to make such a declaration which suggests that we, of the ignorant non-MD masses, without the intellectual capacity to make decisions for ourselves need to have such a decision made for us? A cost effectiveness stuyd such as this was clearly for the financial goals of insurance companies. Was the study sponsored by insurance companies? Please investigate.

 

I point out to you that the United States ratified the International Human Rights Declaration. In so doing, we U.S. citizens are guaranteed the highest level of medical care technology available to us.

 

International Covenant on Economic, Social and Cultural Rights

 http://www.unhchr.ch/html/menu3/b/a_cescr.htm

  

Adopted and opened for signature, ratification and accession by General Assembly resolution 2200A (XXI) of 16 December 1966 entry into force 3 January 1976, in accordance with article 27 http://www.un.org/Depts/Treaty/final/ts2/newfiles/part_boo/iv_boo/iv_3.html

ratified by the United States of America October 5, 1977

Preamble

The States Parties to the present Covenant,

Considering that, in accordance with the principles proclaimed in the Charter of the United Nations, recognition of the inherent dignity and of the equal and inalienable rights of all members of the human family is the foundation of freedom, justice and peace in the world,

Recognizing that these rights derive from the inherent dignity of the human person, 

Recognizing that, in accordance with the Universal Declaration of Human Rights, the ideal of free human beings enjoying freedom from fear and want can only be achieved if conditions are created whereby everyone may enjoy his economic, social and cultural rights, as well as his civil and political rights,

Article 12

1. The States Parties to the present Covenant recognize the right of everyone to the enjoyment of the highest attainable standard of physical and mental health.

 

It is the attitude of Allen Steere that Lyme disease is overdiagnosed and overtreated. In his 1993 JAMA study, he reassigns the diagnoses of musculoskeletal and late disseminated Lyme disease in patients referred to him. He had no objective proof that Lyme disease becomes and autoimmune disease such as Fibromyalgia or Chronic fatigue Syndrome, yet he arbitrarily made this reclassification and in so doing he deliberately denied them care. We ask that you investigate his laboratory notes and see what strains of Borrelia he used to test these patients.

 

He flip-flops in reporting his on his long-term outcome results.

 

At first, Lyme is a serious disease, then it is not. Then he can extract Borrelia DNA from synovial fluid, then he can not.

 

He cannot be believed regarding any statements he makes.

 

In a comment he made about patients to the press it is clear that he believes himself so superior to patients. He was quoted in the Boston Herald 11/3/99 as saying we are "Not rational."

 

This statement is discriminatory and is another violation of the International Human Rights Declaration in that it prevents Lyme patients from having future objective audience with medical professionals

 

27. Principle 1.4 includes the following provisions relating to improving the cost-effectiveness of resource allocation and health planning:

 

"With the development of relatively new methods for measuring the burden of disease on human life that constitute potential tools for guiding decisions for improving the cost-effectiveness (efficiency) of resource allocation and health planning, it is essential that the further refinement of these methods be guided by the principles of equity and non-discrimination on such grounds as age, sex, ethnic origin, personal status, etc., as well as efficiency, and that countries with an interest in applying these tools be provided with the resources for building capacities for undertaking these analyses in a manner consonant with national and local needs."

 

28. With respect to applying bioethical concepts to relevant aspects of human rights, principle 2.1 states that:

 

"important opportunities exist for applying bioethics concept in developing the content of human rights relating to health, health protection, and health care. Such rights can be clustered into three categories, viz.:

 

- rights to health care and to the benefits of scientific progress;

 

- rights relating to information, association, and freedom of action that could empower groups to protect and promote their health; and

 

- rights relating to self-determination and integrity of the

 

person, including rights to liberty and security and the right to private life."

 

 

It is a Human Rights Violation for him to reassign our state of ill physical health to be one of unfounded psychiatric preoccupation. We have the right to organize to protest his disregard for the legitimacy of our complaints.

 

You have in your State of Massachusetts, someone with a personality disorder that reflects a pathological indifference to human beings and is practicing something other than medicine from Tufts University. Please do investigate whether the attitudes and practices of Allen Steere are consistent with your present and former interpretation of the role of the physician in our society.

 

Kathleen M. Dickson

 

Southeastern CT Support Group Facilitator

23 Garden Street

Pawcatuck, CT 06379

860-599-5451