THE CHRONOLOGY OF
Ehrlich and Compound 606,
Syphilis, salvarsan (arsenical)
--1911, Andrew Balfour, MD, Karoum,
granules" Read- He talks about intracellular
spirochetesThe British Medical Journal April 1, 1911
The Infective Granule In Certain Protozoal Infections, As Illustrated By
The Spirochaetosis of Sudanese Fowls
PRELIMINARY NOTE: At the first meeting of the Tropical Medicine
Section of the British Medical Association in London last year I advanced
the view that, in all probability, what might be called the " infective
granule " would yet be found to play an important part in certain protozoal
infections, and more especially in spirochaetosis and trypanosomiasis. I
based this belief on the work of Leishman as regards the changes undergone
by Spirochaeta duttoni in Ornithodorus moubata, and on the
allied changes which I had found to occur in the Sudan fowl spirochaete when
ingested by Arga pericus. I have been continuing the work on fowl
spirochaetosis and have recently arrived at some most interesting and
significant results, which may yet have
considerable bearing on the view we must take of the pathology of this and
other spirochaetal diseases, and possibly also on their treatment. ...
...It will perhaps be remembered that one found intracorpuscular forms
in this fowl spirochaetosis, and that following Sambon, one had to come to
the conclusion that these endoglobular bodies represented a stage in the
lifecycle of the spirochaete -- constituted, in short, its stage of
schizogony in the fowl. Sambon, however, who expressed this view from the
study of a few slides I gave him, did not indicate how this red cell
invasion occurred. For a long time I believed the spirochaetes themselves
entered the red cells and broke up, or coiled up, within them to form these
remarkable bodies. As the parasites can and do enter and leave the
erythroblasts of the fowl, there was good ground for this supposition. Now
however, I know better.
By the use of the dark-field method, and more especially by practising
liver puncture on chicks at the crisis or on chicks which have been given a
sufficiently large dose of salvarsan, I have found that in the liver in
particular, also in the spleen and lung, the spirochaetes undergo an
astonishing change. They discharge from their periplastic sheaths spherical
granules, and it is apparently these granules which enter the red cells,
develop in them and complete a cycle of schizogony. The appearance is very
remarkable. If a well-infected chick be given a dose of salvarsan, the
peripheral blood is soon cleared , or nearly cleared of spirochaetes. If
then a drop of liver juice be examined by the dark-field method, it will be
found swarming with spirochaetes and with highly refractile granules. The
source of the latter is soon apparent, for attention will be directed to
which are not moving in the usual way, but are in a state of violent
contortion, or are, so to speak, shaking themselves to and fro. Indeed, I
cannot give a more apt comparison than by likening their movements to those
of dogs which have been in water and are shaking themselves vigorously to
dry their coats. The object of the spirochaetes, however, is to rid
the bright sphereical granules which can be seen within them and which may
or may not be aggregations of the so-called chromatin core. They are forced
along the periplastic sheath and suddenly discharged , so that they become
the medium and dance hither and thither as tiny, solid, spherical, brilliant
white particles. In process of time the spirochaete loses its activity,
becomes difficult to see, and eventually all that is left of it is the limp
and lifeless sheath drifting aimlessly in the fluid and liable to be caught
up and swept away by some still vigorous parasite. Such a sheath may still
retain one or two of the granules which it has been unable to discharge.
As may be imagined, the process is most fascinating to watch, and my
observations have been confirmed by Captain Fry and Mr. Buchanan, of these
laboratories and Captain O'Farrell, R.A.M.C. I may also say that the
first-named had previously seen a shedding off of granules by trypanosomes
in the peripheral blood of experimental animals, a phenomenon which he is
It is these spirochaete granules in the liver, spleen and lung, and
possibly also in other internal organs, which I believe, invade the red
cells. I think I have seen the penetration occur, but require to make
further observations in order to be certain as to the mode of entry. Such a
chain of events fully explains all the puzzling features which this
intracorpuscular infection has hitherto presented, and moreover, brings it
into line with the infective granules found in the ticks, for these very
closely resemble those seen in liver-juice films both when examined by
dark-field method and when stained by the Levaditi process. There are
various other points more especially as regards the peculiar staining
reactions of these granules, into which I need not enter beyond saying that
the fact that, when free, they do not appear to take on the Romanowsky stain
may explain why they have not previously been noticed. The work is also not
yet complete as it is necessary to find out if the spirochaetes ingested by
ticks behave in a similar manner and thereby produce the granules of
I see that Jowett in South Africa has recently discovered what appears
to be an identical form of fowl spirochaetosis, and I trust he will employ
the dark-field method and endeavour by liver puncture and the use of
salvarsan, for the purpose of creating an articficial crisis to follow out
the curious cycle I have indicated.
From these observations and others which will be fully detailed at a
later date I have come to the conclusion that this fowl spirochaete must be
classed as a specific entity and I am proposing for it the name
Spirochaeta granulosa penetrans, which, though lengthy, suitably
indicates its more important peculiarities. At the same time it is quite
possible--nay, even probable
--that other pathogenic spirochaetes behave in a similar manner. I have
found these granules to be resistant forms and their presence in countless
numbers in the tissues might explain part of the mechanism of relapse and
the difficulty of curing completely some of the more chronic spirochaetal
infections, as, for example, syphilis and yaws.
In conclusion, I must thank Professor Erlich for most kindly placing at
my disposal an ample supply of his new and valuable remedy.
--1932 Tuskegee "Bad Blood" (Typical CDC Handiwork)
Relapsing Fever Borrelia, Lancet, RAMC, WWII, Lybia, Borreliosis is incurable
1944, relapsing fever during WWII, Lancet, Sept 30, 1944,
COMPLICATIONS OF relapsing fever
Ronald Bodley Scott, DM, OXFD, FRCP
LIEUT.-COLONEL RAMC; OFFICER I/C A
THE campaigns in the Lybyan Desert yielded cases
of relapsing fever of a type not previously known in Egypt. They
were due to a infection with Treponema recurrentis, whose immunological
characteristics have not yet been determined, transmitted by the bite of
an unnamed argasid tick of the genus ornithodoros, bearing a close
resemblence to O. erraticus, the vector of the Tunisand strain of
treponema (Adler, 1942). This tick probably inhabited the burrows
of desert rodents and infection was commonly acquired in caves, slit
trenches and tombs.
Characteristic of this fever was the high proportion of cases
in which the central nervous system was invaded. Most descriptions
of the disease include the comment that nervous system sequelae occur and
that they are more common in the tick-botne than in the louse-borne
fever; but theirt frequency and variety are not generally recognized.
This paper is concerned with a small series of cases seen in 1941 and
1942; its observations consequently apply to the disease of the
north-west Africa coast. It is likely, however, that this
reservation is not absolute" the characteristics of Tr. recurrentus
are so labile that strains from the same locality, and even from
different relapses in the same case, may show immunological variations (Ashbel
1943). This the clinician is probably justified in discounting the
importance of differences of strain and in regarding the tick-borne
relapsing fever as an entity, however heretical the immunologist may
consider this view.
NEUROTROPIC CHARACTER OF Tr. recurrentis
In the animal the neurotropism of this
treponema is well-established. Ashbel (1943), investigating 17
strains of Tr. persica, found that organisms could be isolated
from the brains of guinea pigs 117-398 days after apparent recovery from
the infection. In some cases this cerebral invasion proved fatal
and post-mortem examination showed small perivascular hemorrhages [SHOWN
IN LIEGNER AUTOPSIES] and infiltrations with lymphocytes, monocytes and
macrophages. The neurotropism of various strains has been shown to
be equally great in other animals (Heronimus 1928).
The predilection of the treponema for nervous tissue in the
animals raises the question of whether it is similarly neurotropic in
Data are not plentiful; but as long ago as 1874 Ponfick reported
petechial hemorrhages [LIEGNER] in the brains of cases dying in the
Berlin epidemic. Belezky and Umanskaja (1930) have recorded the
findings in 8 fatal cases: clinical observations are scanty in their
paper but only one had symptoms of disease of the nervous system.
In all instances microscopy showed a patchy perivascular infiltration of
the pia with monocytes, lymphocytes and plasma cells, and in places the
cerebral vessels were encircled by a similar cellular halo. In 3
cases treponemata were found in the brain substance, diffusely
distributed and in no constant relation to vessels.
More recently, Ungar has described the case of a woman dying
in the puerperium with relapsing fever and cerebral symptoms. The
post-mortem findings included a cholesteatoma of the lateral recess
edema of the cisternal pia-arachnoid and hemorrhages in the caudal part
of the pons and the floor of the 4th ventricle. Sections showed
the Virchow-Robin spaces distended with erythrocytes, lymphocytes and
monocytes; treponemata were recovered from the cerebrospinal fluid and
from the tumor...
-- 1945 Trying to cure brain relapsing fever
with intracranial infusion of penicillin:
1951: Relapse Phenomena in Rats with a Single Spirochete
Felsenfeld and Barbour
talking about the Single Spirochete Phenomena:
-- 1961, JULY
US scientists discussing spheroplast forms (click to enlarge)
-- 1965 http://mmbr.asm.org/cgi/reprint/29/1/46?view=long&pmid=14295985
Felsenfeld, Comprehensive Review of the status of the Genus Borreliae,
-- 1971 Russian Scientists on the
viability of intracellular cysts:
Polly Murray finally gets someone's attention. (It's a bad accident, Draft
Dodger, CDC EIS Officer, Steere.)
-- 1975 Jay Sanford, Uniformed
Services University School of Medicine,
Bethesda, Maryland, Page 391,
Biology of Parasitic Spirochetes,
"The ability of the
borrelia, especially tick-borne strains
to persist in the brain and in the
after treatment with arsenic or with
penicillin or even after apparent cure
is well known (1).
of treponemes after treatment of
syphilis is a major area which currently
requires additional study
(3,5,10,11). (on the right hand page below the chart)
--1976 Borreliois is
an incurable brain infection. US Military Hosp, Bethesda, MD:
Borrelia persist in the brain past
States in this book
that 1/6 to 1/3 of all Lyme patients are permanently neurologically impaired,
in neurosyphilis: http://www.emedicine.com/neuro/topic684.htm
and discusses cyst or spheroplast or starvation forms
"Ever since it was demonstrated that
the body louse (Pediculus humanus
humanus) and the African O
moubata were the vectors of the
relapsing fever spirochetes known today
as Borrelia recurrentis and B duttonii, respectively, intense
studies have been carried out on the
development of these microorganisms in
their vectors, and on the mode of
transmission to humans. Thus, in 1912,
the French worker Charles Nicolle and
coworkers studied the behavior of B
recurrentis in lice and noted that
the spirochetes had disappeared from the
midgut 24 hours after they had been
ingested; they were no longer detectable
until days 6 to 8 when they suddenly
reappeared in the hemolymph.
A similar "negative phase" had
previously been reported for B
duttonii in O moubata by
Dutton and Todd (1905-1907), Leishman
and other investigators (1907-1920),
Fantham (1911-1915), Hindle (1911), and
later also by Hatt (1929) and Nicolle
and associates (1930). According to
these investigators, ingested
spirochetes invade the gut epithelium
where they lose motility and after 3 to
4 days develop into cysts (blebs,
vesicles) that contain granules or
chromatin bodies (Fig. 4). Duton and
Todd postulated that these spherules are
formed by protuberance of the
spirochetes periplasmic membranes; they
may occur at any point along the
spirochete. At some time during their
development, these spherules or cysts
were said to burst and release their
granules. By the 10th day after
infectious feeding, Dutton and Todd no
longer found morphologically typical
spirochetes, but instead large numbers
of granules from which eventually new
spirochetes developed provided the ticks
were maintained at temperatures above
Thus, RML scientists Dave Dorward and
Claude Gron using silver staining,
transmission and scanning electron
microscopy investigated the nature of
naturally elaborated membrane blebs on
the surface of cultured B
burgdorferi or free in the medium,
and found both linear and circular DNA
(Fig. 13). The fact that his material
was packaged within the membrane-derived
vesicles suggested that it might play a
role in the protection of genetic
markers. In vivo and in
vitro exposure of B burgdorferi
to antibiotics (penicillin G,
ceftriaxon) were shown by Preac-Mursic
and associates to produce cytomorphic
atypical but motile spirochetes with
numerous membrane-derived vesicles
(spheroblast -- L-forms) (Fig. 14).
In their recent publication, Brorson
and Brorson reported on the "In
vitro Conversion of Borrellia
burgdorferi to Cystic Forms in
Spinal Fluid, and on the Transformation
to Mobile Spirochetes by Incubation in
Accordingly, B burgdorferi
converted rapidly to cystic forms when
transferred to spinal fluid. No normal
spirochetes were left after 24 hours of
incubation at 37° C; all were converted
to cysts. When these cystic forms were
transferred to a rich (BSK-H) medium,
the cysts were converted back to normal,
mobile spirochetes after incubation for
9 to 17 days.
These most recent findings do confirm
the development of membrane-derived
cysts, blebs, spherules, vesicles and
the potential transformation to motile,
helical spirochetes, not as part of a
complex developmental cycle -- as
postulated by Dutton and associates --
but rather as a "survival mechanism" of
spirochetes to overcome or escape
unfavorable conditions. Such conditions
prevail during early phases of infection
when spirochetes ingested into the
midgut of ticks or lice become exposed
to the vectors' digestive enzymes and
tissue barriers (peritrophic membrane,
gut epithelium). As a result, most
detectable spirochetes produce numerous
cysts often filled with granular
Other in vitro and in
vivo factors shown to induce
development of cysts include
unsatisfactory culturing conditions,
presence of antibodies and the effects
Using silver impregnations and
immunochemical staining, cystic material
has been demonstrated in every animal
and human tissue infected by B
burgdorferi. As yet, it is not
known whether these forms of Borrelia represent products of
degenerated spirochetes or of surviving
organisms capable of transforming to
typical spirochetes once the faborable
environmental conditions are restored.
It is tempting to speculate, however,
that the survival mechanism of
spirochetes is responsible for the
diverse pathology of these organisms as
well as for their ability to survive as
cystic forms thereby producing
prolonged, chronic and periodically
--1980 to 1983, Edward Eisenberg, OXFORD
is an NIH clinical researcher in infectious
diseases. 'Stated at the Blumenthal Lyme Hearing in 1999 that the
testing for Lyme is as accurate as the testing for any other infectious
disease, which is, of course, perjury, since even Gary Wormser reported that
the current testing for Lyme misses 85% of the cases.
Wormser reported that Steere's IgG method only detects 9/59 cases.
Allow me to do the math: 9/59 X 100 =
One plus eight equals nine.
Or, eight plus one equals nine.
Practice, America! You can do
Use your fingers if you have to!!
Here are more of the Dearborn results. No one reported anywhere near
100% accuracy for this method. The average was around 15%.
100 minus 15 equals 85%
The testing for Lyme misses 85% of
If you understand this math,
kindly apply for a job at the FDA since they seem to have a shortage of such
We're paying the FDA to look
at such BigPharma data, but they disclaim their obligation to look at the
data BigPharma sends them.
Proposed Life Cycle for the Reiter
Treponeme (validity of
"cysts", or spheroplasts. or
McSweegan trashes the Navy to give all the grants to his buddies,
like Yale's Durland Fish
This is otherwise known as "steering
Making it perfectly legitimate (is
that the right word?) for Blumenthal to be up their butts, since no one in
America approved of Kaiser-Permanente writing anyone's non-treatment
Kaiser and SmithKline are once
again together involved with this cabal of defrauding Uncle Sam. New
York Medical College was financially floundering. Kaiser helped.
There were, um, some "conditions..."
Steere, The Original Serodiagnosis, later falsified :
-- 1986 CDC
"officer" Alan Barbour publishes his comprehensive article on Borrelia. Mentions rodent brains as
the storage media.
By CDC Officer Alan Barbour, in 1986, The Biology of Borrelia Species:
"The propensity for borrelia to go to the
brain of infected mammals
suggests that the relationship between these spirochetes and neural tissues is
not trivial. Further study of this attraction and the interaction that follows
may reveal the basis for the significant nerve and brain involvement in Lyme
is self-explanatory. We have previously seen that rodent brains so
reliably were a home for spirochetes that they were actually the culture
media. We also acknowledge that ceftriaxone for brain diseases was a
treatment IDSA themselves used and offered and still use, so Lyme must not
be a self-limiting knee disease as Allen Steere and Yale's Steven Malawista
And here we have in the same CDC officer's 1986 report that there are, gasp, "gemma:"
--1988, Russell C. Johnson,
First vaccine patent: US Patent
Borreliosis is incurable; Rx: "Chronic Treatment for this chronic infection."
NINDS' Roland Martin (who has since moved back to Germany because he failed to prove that the
Multiple Sclerosis form of Lyme disease is caused by an autoimmune T cell
process), demonstrates that Borreliosis is a cause of "Oligoclonal bands in
the CSF" which was the former definition of Multiple Sclerosis.
Lyme is a cause of Antiphospholipid
Antibodies (Lupus)- Says Yale and Allen Steere
Infectious Disease Reviews,
TRUTH (therefore, remains, the
Truth) This is the crooks' own data that they refuse to turn over to
the Connecticut Attorney General.
of late neurologic Lyme fails in half the cases."
they, the Infectious Diseases Society of America, in 1989, do not say that
Chronic Lyme is due to hysteria or is imaginary, nor do they say that "Lyme
is easily diagnosed and cured."
Isn't that kinda the same thing Brian
Fallon discovered 15 years later?
That Lyme is just a relapsing fever, permanent brain infection that requires
the same thing that Russell Johnson published in his first patent for a Lyme
-- 1989 IDSA Reviews-
the rest of the full text articles, scanned in.
(Go ahead and read
them all. You'll be shocked, and will understand why IDSA refuses to
turn this data over to Mr. Blumenthal)
Benach, Early Central Nervous System
Invasion I remind that the central nervous system is neither a knee
nor the genitals, requiring nasal surgery to cure sexually repressed women of
excessive masturbation- which is what Fraud, (sorry, Freudian slip) Freud did.
-- 1990, CDC Adopts
Allen Steere's method for diagnosing Lyme as a relapsing fever borreliosis, by performing serial Western Blots to look for changing and
expanding IgM and IgG antibodies, since the production of NEW IgM means the
bug is still alive and not killed by antibiotics.
This criteria is later changed at the fake
Dearborn consensus conference, which was designed around changing Lyme into an
untreatable autoimmune arthritis in a knee.
ALDF.com founded. Is a "front" or
a false non-profit, since they publish bogus articles.
suspect is a front for Kaiser-Permanente.
ALDF BOARD OF
DIRECTORS, 1999 Insider Investors and Israeli insider bioweaponeers,
Hank Greenberg, Mortimer Zuckerman, and now www.otaotr.com which is an international, illegal Jewish spying firm.
--1990, Sigal begins his Spin at Robert Wood Johnson
LymeRIX coming along (Fikrig, animal studies; LYMErix fails to prevent Lyme)
Connolly, Gary Wormser, and Durland Fish decide to capitalize on
Tick Borne Infections and control
treatment and outcomes of them (ALDF.com was born). Establish the
"entrepreneurial trio." Kaiser helps establish the
cabal, and are still at New York Medical College, training MDs.
Kaiser-trained MDs become "experts" at the OPMC witch trials.
-- 1991, Yale's Erol Fikrig develops the only
scientifically valid test for Lyme:
THE RICO PATENTS
Yale's Bb Flagellin Patent,
5.618,533 94.4% accurate,
earliest, and likely nearly 100% specific, or is the best antibody test for Lyme
The Yale report that goes with the patent (1991):
Steere, sorta doing fine before the BrainWobble...
Steere in convinced this entity "seronegative
Lyme" exists, because he used the seronegative Lyme lymphoproliferative
assay on his lab workers and found that 4/9 had inhaled spirochetes.
See Chapter 3 for those reports.
Alan Barbour publishes an article that is
no longer available on the web in full text, in which he describes how
to select for spirochetes that don't have certain surface antigens on them-
by using something like a vaccine, which is a rather direct way of saying,
NO VACCINE IS POSSIBLE, SINCE LYME IS A RELAPSING FEVER ANTIGEN and the
nature of the relapse is that antibodies "select for new mutants"
Mark Klempner and his INTRACELLURITY /fibroblasts/Ceftriaxone
-- 1992 Steere at the
of Insurance Medicine"
knew there was an "Academy of Insurance Medicine?")
Formation of Multiple Treponemes
The spirochetes clump together and form a common outer envelop.
According to the crooks, because this happened in Europe it did not happen,
when the truth of the matter is that whatever happens according to the CDC
should not be believed in Europe.
--1992, Formation of
KAISER?? and John Connolly to limit
Managed Care's payment for treatment
with ceftriaxone. 1989 Infectious
Disease Reviews, Dattwyler:
"Treatment remains speculative."
If we like had a sort of federal bureau of
investigation, or something like that who had authority across state
lines, that would be like cool because they could find out whether or not
this is a Kaiser ghost company, you know like in the old days when the
Rockefellers had numerous companies that everyone thought were independent
of each other. Like I think it like used to be illegal, you know in
the last century. The one before the Fourth Reich.
Steere has a major BrAiNwArP (Same time as "Steere in Europe") >> Lyme is
actually FM, CFIDS, and Hypochondria?
Don'tchya hate it when that happens?
The man thought he discovered a disease,
and he didn't after all? (He used
strain G39/40, which detects Lyme
disease 14% of the time, according to
Imugen's submission to Dearborn).
CastleConnolly and Steere having a
brainwarp occur at the same time.
Steere, 1991: Lyme is "perilously
close to FM, CFIDS, and Depression."
Spirochetes suddenly start cooperating
with Managed Care after millions of
years of evolution.
It's quite miraculous and
we would hope Managed Care can help with the others of the world's maladies,
like, Can they kindly turn seawater into oil?
(Gasoline is better.
You know, skipping the refining process.)
Steere Brainwarp Abstract A:
Steere Brainwarp Abstract B:
1992- Steere goes to Europe with a
"bogus" borrelia, one that is "high
passage" (which is illegal) and with recombinant OspA and B, lacking the
lipid end, which is likeliest to produce antibodies.
Harbor labs hold a conference.
Duray, Sigal, Dattwyler.
Dattwyler says, expanding IgM and IgG
demonstrates the persistence of the
illness. (However, antibodies to
just lipoproteins are not the only
antibodies-- There are antibodies to
other membrane components, such as: New
GLYCOLIPID-- Neuroborreliosis and Not
LymeRIX disease. Structure seen on
page 34 of the .pdf GC-MassSpec)
See Pathologies Anti-glycolipid
Joe Burrascano complains to Congress.
Then immediately becomes a target of persecution.
Nothing is done because
Dodd and Kennedy know Plum Island is a
bioweaponeering facility and don't actually care about their
constituents, since the self-jerking off "military men are dumb, stupid
animals to be used as pawns for foreign policy"-- Heiney Kissinger.
Dreaded Dressler/Steere Report:
Full Text leaves OspA and B out of the standard,
because they used a high passage strain,
which dropped expression of OspA and B.
Additionally, in the prospective study,
only late Lyme arthritis have this
presentation at 72%.
Dressler/Steere does not agree at all
with Steere's first serodiagnosis
--1993, Fish and Barbour begin the spin.
Barbour, re OspA "we don't know what it does" (Re: the OspA)
MEDLINE "The biological and
social phenomenon of Lyme disease."
Now Lyme is a Character Flaw.
Phase II Vaccine trials underway.
Barbour's hometown wants to erect a
statue of him.
That would be like Russia erecting a statue of Hitler in Red Square.
--1994, MARCH, Connaught rOspA "vaccine"
(ImmuLyme, CDC officer Alan Barbour's patent) trial
begins (Where did the standard come
from?? Dearborn hadn't happened
Later Lenny Sigal, who headed the Connaught
ImmuLyme trial, reported that he could not read his Western Blots using
MarDx test strips, in a report with Molloy and Persing. So, although
Sigal reported that ImmuLyme was 92% effective at preventing Lyme, he later
said the Western Blots were unreadable and that he could not really tell if
ImmuLyme prevented Lyme - neglecting to mention that LYMErix and ImmuLyme
used the same MarDx test strips to assay vaccine outcomes.
The Sigal ImmuLyme report where he says
ImmuLyme is 92% effective and that he was using MarDx Blot strips:
(New England Journal of BigInsurance Propaganda and BigPharma Disease
http://content.nejm.org/cgi/content/full/339/4/216 full text
"A total of 10,305 subjects (age range,
18 to 92 years of age) were enrolled at 14 study sites in areas
of the United States in which Lyme disease was endemic and were
randomly assigned to receive two doses of either 30 µg of OspA
vaccine or placebo, given one month apart, according to a preset
randomization schedule. Randomization was balanced at each study
center in treatment blocks of 10. The first injection was given
between March 1, 1994, and April 30, 1994, [BEFORE
THE DEARBORN CONFERENCE, WHICH TOOK
PLACE IN OCTOBER 1994, SO WE WONDER WHO SAID THEY COULD USE THE BOGUS
DRESSLER/STEERE CRITERIA TO ASSESS OUTCOMES???] and the second
injection was given approximately one month later, between April
1, 1994, and May 31, 1994. At the request of the Food and Drug
Administration, we obtained data on a third dose of vaccine given
12 months after the first injection. A total of 7515 subjects
received a third (booster) injection of placebo or vaccine
between March 1, 1995, and April 30, 1995. The subjects were
observed during the two seasons in which the risk of disease
transmission was greatest, irrespective of whether they had
received the booster dose.
"Subjects in whom Lyme disease was
suspected returned to the study center, where a clinical
evaluation was performed by the investigator and serum samples
were obtained during the acute and convalescent phases of the
illness. Western blot analyses (MarDx Western blot kit, MarDx
Diagnostics, Carlsbad, Calif.) to determine whether IgG or
IgM antibodies to B. burgdorferi were present were performed for
all subjects with suspected cases of Lyme disease by the central
laboratory (Division of Infectious Diseases, New York Medical
College, Valhalla), according to the manufacturer's instructions.
Results were considered positive if the serum sample contained
IgM antibodies that reacted with at least 2 of the following 3
bands: 25 kd (corresponding to band 23, which indicates OspC), 39
kd, and 41 kd within the first 60 days after the onset of
erythema migrans, symptoms, or both or with IgG to at least 5 of
the following 10 bands: 21, 25 (23), 28, 30, 39, 41, 45, 58, 66,
and 83 (corresponding to band 93) kd at any time after the onset
of symptoms. The reports of the results of these tests were sent
to the investigators and did not include specific information on
reactivity to OspA bands.
The Sigal-Persing Molloy Unreadable Blots
Everyone in the world ought to read this full text report, because it seems
like what I told the FDA Vaccine
Committee was more true than I even knew at the time about LYMErix
appearing to activate latent infection in people who had Lyme previously and
believed they had recovered.. I was just observing the empirical data (the
victims of LYMErix called me, very ill, looking for help)
BLOT SMUDGING-- Unreadable blots in OspA vaccinated people, alleviated by
using a strain of Bb with no OspA-B plasmid in it. Here Persing is talking
about LYMErix, when in fact, his co-author, Lenny Sigal ran the ImmuLyme
trial, did not report blot smudging, and Persing already knew about the blot
smudging since that's the RICO patent that Persing developed with Schoen and
the reason OspA and B were left out of the diagnostic standard!! See 1)
above for the journal report that goes with the Persing RICO patent:
Unreadable blots graphics.
--1994 JUNE Vaccine Meeting Minutes:
Dattwyler and Luft, 2/3 of EM patients
have Bb DNA in their CSF,
Dattwyler *says* to use serial
or sequential Western Blots to assess outcomes.
This is ignored.
I will explain the words
serial for any AmeriTards who may be reading:
It means, "one followed by
another." Or "one after an another." Or, 'In order, like 1, 2, 3,
Steere and Long Term Outomes:
"Patient 12 had had high fever, meningeal symptoms, and subsequent
arthritis in 1982. She was noted to have a positive serologic
result for Lyme disease 4 years later and was treated with 2 weeks
of parenteral penicillin. She later developed a progressive speech
disorder, bradykinesia, and abnormal ocular motor function. Magnetic resonance imaging of the brain showed scattered white
matter lesions in the hemispheres and pons, and she was diagnosed
with supranuclear palsy. Lumbar puncture showed no selective
concentration of antibody in the spinal fluid. Nevertheless,
was re-treated with 2 weeks of parenteral ceftriaxone in 1989 that
had no effect on her neurologic symptoms. During the time of
observation, this patient died. At autopsy, lymphoid mononuclear
cells were observed surrounding the intracerebral vessels in one
section. Using Dieterle silver stain, a spirochete was present in
the cortex and another was exterior to a leptomeningeal vessel."
(One of Steere's multiply treated
patients died anyway with spirochetes in her brain.)
The Primer's Game
these Lyme crooks use the wrong DNA (or RNA) primers (they use the variable
Osp primers instead of the non-variable chromosomally encoded) to test Lyme
victims. These same crooks use the correct DNA primers when they
want to find Lyme or borrelia in ticks. It's a shell game. They all know
that all that they can use for treatment outcomes is non-variable
DNA or RNA.
Mark Klempner never reported
his primers in his Chronic Lyme "study," and he would not tell me, either, in
an email correspondence I had with him. Therefore, the Lyme crooks have
never validly reported on the presence of borrelia in humans, with the
exception of Gary Wormser's study of EMs in which he found 2/9 patients who
had EM, who had been treated with antibiotics still had spirochetes in their
tissues. This data can be found on the
HOW RICO WILL BE CHARGED PAGE
Steere and Nocton (2 articles, both showing
persisting infection past treatment, even using the wrong DNA or RNA primers)
Detection of Borrelia burgdorferi DNA by polymerase chain
reaction in cerebrospinal fluid in Lyme neuroborreliosis.
Division of Rheumatology/Immunology, New
England Medical Center, Boston, Massachusetts02111, USA.
A polymerase chain reaction (PCR) assay
that detects Borrelia burgdorferi DNA in cerebrospinal fluid (CSF) was
evaluated as a diagnostic test for acute or chronic Lyme neuroborreliosis. In
one laboratory, 102 samples were tested blindly, and 40 samples were retested
in a second laboratory. In the first laboratory, B. burgdorferi DNA was
detected in CSF samples in 6 (38%) of 16 patients with acute neuroborreliosis,
11 (25%) of 44 with chronic neuroborreliosis, and none of 42 samples from
patients with other illnesses. There was a significant correlation between PCR
results and the duration of previous intravenous antibiotic therapy. The
overall frequency of positive results was similar in the second laboratory,
but concordance between the laboratories and among primer-probe sets was
limited because many samples were positive with only one primer-probe set.
Thus, PCR testing can sometimes detect B. burgdorferi DNA in CSF in patients
with acute or chronic neuroborreliosis, but with current methods, the
sensitivity of the test is limited.
In the following report, Steere found persisting DNA in joints
for up to ten years, after treatment, even using the wrong primers.
Detection of Borrelia burgdorferi DNA by polymerase chain
reaction in synovial fluid from patients with Lyme arthritis.
Division of Rheumatology/Immunology, New
England Medical Center, Boston, MA 02111.
BACKGROUND. Borrelia burgdorferi is
difficult to detect in synovial fluid, which limits our understanding of the
pathogenesis of Lyme arthritis, particularly when arthritis persists despite
antibiotic therapy. METHODS. Using the polymerase chain reaction (PCR), we
attempted to detect B. burgdorferi DNA in joint-fluid samples obtained over a
17-year period. The samples were tested in two separate laboratories with four
sets of primers and probes, three of which target plasmid DNA that encodes
outer-surface protein A (OspA). RESULTS. B. burgdorferi DNA was detected in 75
of 88 patients with Lyme arthritis (85 percent) and in none of 64 control
patients. Each of the three OspA primer-probe sets was sensitive, and the
results were moderately concordant in the two laboratories (kappa = 0.54 to
0.73). Of 73 patients with Lyme arthritis that was untreated or treated with
only short courses of oral antibiotics, 70 (96 percent) had positive PCR
results. In contrast, of 19 patients who received either parenteral
antibiotics or long courses of oral antibiotics (> or = 1 month), only 7 (37
percent) had positive tests (P < 0.001). None of these seven patients had
received more than two months of oral antibiotic treatment or more than three
weeks of intravenous antibiotic treatment. Of 10 patients with chronic
arthritis (continuous joint inflammation for one year or more) despite
multiple courses of antibiotics, 7 had consistently negative tests in samples
obtained three months to two years after treatment. CONCLUSIONS. PCR testing
can detect B. burgdorferi DNA in synovial fluid. This test may be able to show
whether Lyme arthritis that persists after antibiotic treatment is due to
persistence of the spirochete.
PMID: 8272083 [PubMed - indexed for MEDLINE]
HERE, in Chapter 3 of the Cryme Disease
--- 1995, The Persing Schoen RICO
Patent and Report:
In this report - and this is the RICO report
associated with the main RICO patent
- Yale demonstrates that they know how to diagnose any kind of
Lyme or borreliosis anywhere in the US.
--1995, Shapiro and Sigal = "The Twins" Spindoctors-- NOT TRUE →
--1995- 1996 --
Schoen and Persing develop and patent the RICO method of
testing for Lyme with a spirochete that has no OspA-B plasmid in it.
They determine it is a Lyme Borrelia by using the correct RNA primers.
Persing applies for the patent before the
scientific report is sent for publication.
First things first:
"Get the money."
--1997 Klempner and
Fallon start their Chronic Lyme Trials. Klempner finishes first by ending his
bogus protocol & "study" without ever enough participants, using invalid
methods, lying about his previous findings, and lying about finding "no DNA
positive patients" among the previously treated.
The Intramural study on the MS form of Lyme is kept all hush-hush.
Roland Martin basically found nothing as regards the production of autoimmune
T cells recently, just like he really had no data on autoimmune T cells back in
Vaccine Meeting Minutes...
Vijay Sikand says
Lyme spirochetes can go dormant in the brain and is later "harder to
diagnose and treat."
Schoen says Lyme is 10 times under-reported.
The truth is that it is 10 times under reported times only 15% reportable.
If the CDC says there are 20,000 cases per year, the real number is 200,000
divided by 0.15, or 1,333,333. A million and a third new
permanent infections per year.
1/6 to 1/3 will be
--1998 LymeRIX "results"
(A "76% safe and effective vaccine" in a trial where no one could tell whether
or not LYMErix prevented Lyme due to the unreadable Western blots.)
--1998, December, LYMErix "approved" by the FDA.
Schoen reveals the RICO in Lyme Disease, ACP Key Diseases Series
textbook, or the Munchausen's book.
February, Blumenthal Hearing:
--1999, March, Vijay Sikand, East Lyme, CT:
adverse events, East Lyme, CT, USA,
March 10, 1999, "New London Day",
Letter to the Editor
--Letters to Holc
Noble and the New York Times: May, 2000, The Times
ACTIONLYME founded. We Support Group Leaders started receiving phone
calls from patients who had Lyme "again." They had received the vaccine.
I called Dennis Parenti at SmithKline in Philadelphis and visited Karen Forschner of the Lyme Foundation...
-- 2000 Reporting
Robert Schoen to the CT Medical Board.
http://mic.sgmjournals.org/cgi/reprint/146/1/119 (This was published
in the United States in recent time so the CDC can't reject it which is what
they always do with data they don't like from Europe.)
Reversion of cyst form to intact spirochetes within one minute of
addition of rabbit blood (serum-starved means the L-form):
NY DOH OPMC
CORRUPTION, December, 2000
This report later
becomes "Pat Smith's Big Books," when it was re-written and
added to by Pam Weintraub. Art Doherty summarized grants for me.
--2001, January 31,
FDA Vaccine Meeting: Chronic Disability, and 4/13 spontaneous abortions
(miscarriages) from rOspA (LymeRIX):
2001 FDA LymeRIX Vaccine Meeting Minutes
4/13 miscarriages-extra copy)
".....as soon as
Asymptomatically infected individuals
got the vaccine, they (became ill) moved
into the "Unconfirmed Lyme" group.
The vaccine did not "Prevent
Asymptomatic Infection" as SmithKline
claimed. It just made people
sick." See: BARBOUR
"The absence of
B. burgdorferi in " Treatment Resistant
Lyme Arthritis" joint is still debated"
--Spontaneous Abortions? 'Perhaps
shedding some light on undetected Lyme
and associated infections on the fact of
CT having the highest rate of Attention
Deficit Disorder in the country, and the
Behavorial Disorders in Young Children
Maternal antibodies and Learning
Disorders, e.g., Lupus
also, Steere's report
maternal borreliosis infection and the
fetus, and his report on Anticardiolipin
which also asks, is there an increase in
the number of miscarriages,
corresponding to the epidemic of
neurodevelopmental problems in the USA
May and June, highest rate.
results suggest that maternal immune
system products can modulate the immune
response of the offspring."
(again), July 12, 2001, NEJM,
ILADS Rebuttal to
July 12, 2001 NEJM, Klempner's Chronic
Lyme "long term treatment" study:
'Summary of Klempner's
"study": invalid inclusion/inclusion
criteria, assessments, FIQ not valid,
CDC "Lyme disease" case definition not
valid. Study not
completed. MS hapotype found
and not reported in the "seronegatives"
HLA-DQB1*O602. Some patients never
had ceftriaxone in the first place, and
therefore the "results" did not
confirm the current "standard of care."
Wrong DNA primers were used.
did not culture for more than 3 weeks which is needed to allow the
spheroplast form to start growing again, as
Russell Johnson explains in his article
in the 1989 REVIEWS OF INFECTIOUS DISEASES about culturing.
--2001, July, RI
Diseases of Summer Conference, not disclosing the Haplotype data associated with
Klempner DQB1*0602. Compare
Mark Klempner discusses his HLA secret (MS and Lyme Disease)
03:03 minutes YouTube
Klempner's Secret Haplotype:
Klempner says: "Chronic Neurologic Lyme could be due
to either LYMErix vaccination (OspA) autoimmunity or antibiotic treatment
says: the IV drug ceftriaxone, which is used for meningitis (and not
knee-only diseases), does not kill all the spirochetes (click here
for full text journal article)
carefully what he says are the criteria for performing this study in the
first place. This is full text report
It sorta conflicts with what he says in the
More On Mark Klempner's Boston Biowarfare Lab. Think
about it: Mark got promoted by the CDC for lying in the journals and to the
public about "Lyme disease."
That's a pretty good endorsement of my claims that he committed research
FRAUD, right? Being promoted by the CDC? It's like Kevin J. O'Connor, the
Corrupticut "US Attorney," who was promoted by Constitution
Gonzo, while all the good US Attorneys were axed.
Klempner at the NIH "Rare Diseases" Conference
with the DQB1*0602 data:
Lyme is clinically indistinguishable
See Roland Martin, Cellular Immunology
also: Lyme and MS: NIH CLINICAL
MS and "Lyme disease" (the
CDC/Dearborn/LYMErix disease kind)
The ALS form of Lyme is ALS-like (seems to affect nerve roots, like
The MS form of Lyme is MS-like (produces mini-brain hemorrhages, oligoclonal
bands in the CSF, but not really autoimmune T cells in the CSF).
The Lupus form of Lyme is Lupus-like (Steere found that auto-immune
phospholipids were only of two types in the Lupus form of Lyme.
Damage to lymphocytes could be mistaken for leukemia (Duray).
Criminally underdeveloped data: relatedness of badly cloned B
cells, cross-reacting antibodies, and immune suppression as related
to all these outcomes, because all these outcomes could then be
applied to LYMErix Disease®.
We would like to know who is entitled to this information, since
clearly we are not??
from rOspA not reported to WHO by Yale.
: Abstract, MEDLINE
The other events are mainly related to blood vessel blockage; heart attacks
the lawsuits against The Yale
Corporation, Glaxo-SmithKline, Aventis,
The State of New Jersey, the State of
Connecticut, the State of New York, L2
Diagnostics, Possibly Mayo (Persing),
Corixa (Persing), Imugen (Molloy and
possibly Steere), New York Biotech http://www.nyba.org, ALDF.com Board
of Directors, and CastleConnolly.com are
GEN*NY*SIS (Kemp Hannon), New York State
filed with the local FBI/DOJ morons and henchmen for corporate crime, New Haven.
October, USDOJ in Washington, the Criminal Division forwards the RICO
complaint to US Customs, who forwards it to the head of the DHHS,
who at that time was Tommy Thompson, a good buddy of the back-slappin John
Rowland and the Bush Brothers...
Thus, since the Republican Party are all whores
for BigBusiness and they want to advance the New World Order Agenda which is
global fascism in disguise, nothing was done. Now the economy is in
the toilet and the world powers are ganging up against us to squeeze the
dollar, and set up an oil bourse in rubles.
--2006, Corrupticut Attorney General
starts a lawsuit against IDSA. Asks crooks to kindly turn over all their
self-incriminating data. They refuse for 1.5 years.
-- 2006, CDC Admits that
spirochetes are intracellular in brain and nerve cells:
Invasion of neural cells by B. burgdorferi provides a putative
mechanism for the organism to avoid the host's immune response while
potentially causing functional damage to neural cells during infection of
--2007, Lying criminal tards of the CDC cave on their own bullshit and blame
the AMA for listening to the CDC's lying bullshit.
CDC says "American
physicians are all STUPID and need to be
wonder if that announcement could be a result of my reporting the CDC's
criminal behavior to the House Oversight Committee??
Go here for the AMA-AVA-CDC announcement where the AMA
tards will now play second fiddle to the Veterinarians, since they've had to
admit they drank the Yale Kool Aid on vector-borne diseases
HEEeeyyy!! Look at all the millions in grants
going to Yale or New York Medical College !!
JHU receives $93
million grant from NIH:
Award meant to help school turn promising medical discoveries into
I file a second complaint about
Airhead O'Connor to the Mr. Potatohead of the USDOJ. ...
-- 2008, CDC admits
that 30 days of intravenous ceftriaxone fails to kill all the spirochetes in
rodent brains. (Did not use Mouse
Infectivity Test or attempt to re-grow spheroplasts in culture over a span
of months, as ordered by IDSA's Russell
Persistence of Borrelia burgdorferi
Following Antibiotic Treatment in Mice
Sunlian Feng, Kevin
Holden, Kimberly J. Freet, and Stephen W. Barthold*
Center for Comparative Medicine, Schools of Medicine and Veterinary
Medicine, University of California at Davis, One Shields Avenue, Davis, CA
The effectiveness of antibiotic treatment was examined in a mouse model of Lyme borreliosis. Mice were treated with ceftriaxone
or saline for one month, commencing during the early (3 weeks)
or chronic (4 months) stages of infection with Borrelia burgdorferi.
Tissues from mice were tested for infection by culture, polymerase
chain reaction (PCR), xenodiagnosis, and transplantation of allografts at 1 and 3 months after completion of treatment.
addition, tissues were examined for spirochetes by immunohistochemistry.
In contrast to saline-treated mice, mice treated with antibiotic
were consistently culture-negative, but tissues from some of
the mice remained PCR-positive, and spirochetes could be visualized
in collagen-rich tissues. Furthermore, when some of the antibiotic
treated mice were fed upon by Ixodes scapularis ticks (xenodiagnosis),
spirochetes were acquired by the ticks, based upon PCR, and
ticks from those cohorts transmitted spirochetes to naļve SCID mice, which became PCR-positive, but
culture-negative. Results indicated that following antibiotic treatment, mice
remained infected with non-dividing but infectious spirochetes,
particularly when antibiotic treatment was commenced during the
chronic stage of infection.
Republic Slams US on incomprehensible and stupid Lyme science.
Czech Republic: "US Research (Yale and UConn) on
Borreliosis is incomprehensible and stupid" (translation:
We're furious that we've been hoodwinked over CDC bullshit)
080426 Screening of UNDER OUR SKIN in New York,
on a Saturday.
Watch the clearly evil liar Yale's Eugene Shapiro lie his ugly face off
about congenital Lyme
in the Under Our Skin trailer:
Yale participated in this autopsy of the congenitally infected newborn
who died of congenital Lyme brain damage:
There was no inflammation. Since there was no inflammation,
according to these crooks, there was no disease; the baby died of
hypochondria because he wasn't getting enough sex:
Cabal cave to CT AG Blumenthal, then immediately regret they made such a
decision, since it looks like they want to avoid criminal charges.
Note 101022: The Chronology
or History of Relapsing Fever (since 1911) was meant to be the last
chapter of the book, Cryme Disease, however,
I am adding a new, parallel history here, to look
more closely on the immunology and consideration of other fungal infections
(OspA comes from a fungus or is a TLR2 agonist; Pam3Cys, the synthetic OspA,
was originally designed to be a physiological mimic of E. coli's Lipid A,
which is a toxin) and Epstein-Barr.
WHAT's SIMPLE and CHARGEABLE as a CRIME:
Other than that, we are
working on the indictment related to the
known inability of the Lyme
criminals to read their Western Blots in OspA-vaccinated people, and we
still are working on duh "Lyme-in-da-Cocoanut"
Lyme crook-nutcase paper. This new paper will incorporate the
Biomarkers and PrimerShellGame because we
believe the data suggests that a lot of the signs of chronic Lyme are really the result of
Epstein-Barr or/and mycoplasma/mycobacteria, or opportunistics.
Bear in mind that antibody testing is obsolete and since 1988, known
to be a hindrance to identification of TLR2-agonizing agents (Lyme or
LYMErix Disease) which turn off the antibody response. The Lyme crooks
have been using the correct RNA/DNA primers to identify spirochetes in ticks
since 1991, and we focus particularly on the year 1992, since so much data
was generated by the Lyme crooks that is extremely self-indicting-- KMDickson
Saint Michael the Archangel,
defend us in battle.
Be our protection against the wickedness and snares of the devil.
May God rebuke him, we humbly pray;
and do Thou, O Prince of the Heavenly Host -
by the Divine Power of God -
cast into hell, satan and all the evil spirits,
who roam throughout the world seeking the ruin of souls.