Welcome to ActionLyme - the Lyme Cryme Whistleblower's Website

Borreliosis Basics (poster) USDOJ RICO Complaint (2003) The ALDF/IDSA RICO Patents Dattwyler on Seronegative Lyme Dearborn Booklet (pdf) ALDF.com files
Lyme Facts (poster) Blumenthal Sues (AntiTrust) CDC's Patents with GSK Steere on Seronegative Lyme Dearborn Invitation (pdf) Fish @ Internat.Spy Firm
Primers Shell Game (report
 your adverse events to FDA)
History of Relapsing Fever as  
 permanent brain infection
UConn Assaults Czech children
 with fake vaccine
AIDS-like Epstein-OspA- Borreliosis;
 
vaccine scam clinched it
Imugen (Steere owns?) on
 
Dearborn as 14% Accurate
Khan Academy of Cryme &
 HLA-negative diseases
Post-Lyme-Sepsis & Autism;
 
20 reports
IDSA: Lyme is permanent
   brain infection
Congenital Lyme Autopsies by Yale Steere in Europe, falsifying testing
 1992
,(2 reports, 1 not in Dearborn)
Antics of Edward
 McSweegan & Durland Fish
5 "Axes" of brain compromise
Yale's Lyme Vaccine Scam IDSA's & CDC's Biomarkers JJ Halperin on Lyme & ALS Steere on Lyme causing Lupus Mark Klempner's Crymes Cryme Trainer 2012
Intent to Cause Harm (UN,2003) Russians@NYMC HLA-pharming Plum Island Evidence Plum Stupid, referenced formal report Munchausen's Accusations Jan-Nov 2014 hom
Tolerance from OspA/Pam3Cys NIH says CLD is active herpes et al ME/CFS is chronic active Mono,etc

Pam3Cys/OspA activates EBV

Lyme as a bioweapon? Army says Bioweapon


12/18/2014 07:05:04

Index

/Home

Site Navigation

Pandora: ME/CFS related to AutismVaccines


Cryme Disease
Norway Site
(new)



Follow the Lyme/LYMErix Scam in the order it occurred, below:

Older data on the incurability of Relapsing Fever

1986, McSweegan trashes Navy for $$$ for ALDF.com

1988, Dattwyler & about immune-suppressing, seronegative Lyme

1990, CDC: "Diagnose Lyme as if it was Relapsing Fever."   

Allen Steere  "NeuroLyme won't test positive," 1990.

1992, CDC officer Allen Steere falsifies testing in Europe

1992, CDC patents with SmithKline show 2 kinds of Lyme

1993, Barbour and Fish slam Neurologic Lyme victims.

Compare the 2 kinds of Lyme in the RICO complaint

1994, FDA LYMErix Meeting

1994, CDC's Dearborn Booklet .pdf

CDC's invitation to participate in Dearborn .pdf

Dearborn, Who Said What?

Igenex, Harris, Dearborn .pdf

Evidence  Lyme criminals knew LYMErix produced the same "multisystem disease" as "Chronic Lyme"

LYMErix Damage Coverup (short)
 

1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.

ActionLyme/Kathleen Dickson predicts all of Bushie's outcomes in Oct 2000

ActionLyme/Kathleen Dickson predicts Bush will have us worshipping his bombs (Shock and Awe"), in Oct 2000 during the Gore Debates    



Steere's "Seronegative Lyme" Hysteria about OspA-induced Antibody Inhibition
Read about it also in the 1998 FDA LYMErix meeting transcript:
http://www.fda.gov/ohrms/dockets/ac/98/transcpt/3422t1.rtf


1990
; Steere using Dattwyler and Volkman's Seronegative Lyme Assay (developed because Dattwyler knew fungal antigens produced immunosuppression) to evaluate "Chronic Neurologic Lyme" cases:


http://www.nejm.org/doi/pdf/10.1056/NEJM199011223232102


See Also, CRYMEDISEASE_CHP3_B.htm  where we show Steere in 1991 actually used this seronegative Lyme assay on his own lab
workers, 101016.htm (
http://www.ncbi.nlm.nih.gov/pubmed/1883122)  where we see Steere knowingly falsely advising the Academy
of Insurance Medicine a year later (1992), and the Plum Island Chapter of Cryme Disease where Justin Radolf demonstrates in 2001
that OspA vaccination (or autovaccination via blebbing), results in inhibition of antibody production via downregulation of HLA.

N  1988 Dec 1;319(22):1441-6.

Seronegative Lyme disease. Dissociation of specific T- and B-lymphocyte responses to Borrelia burgdorferi.

Source

Department of Medicine, State University of New York, School of Medicine, Stony Brook 11794-8161.

Abstract

The diagnosis of Lyme disease often depends on the measurement of serum antibodies to Borrelia burgdorferi, the spirochete that causes this disorder. Although prompt treatment with antibiotics may abrogate the antibody response to the infection, symptoms persist in some patients. We studied 17 patients who had presented with acute Lyme disease and received prompt treatment with oral antibiotics, but in whom chronic Lyme disease subsequently developed. Although these patients had clinically active disease, none had diagnostic levels of antibodies to B. burgdorferi on either a standard enzyme-linked immunosorbent assay or immunofluorescence assay. On Western blot analysis, the level of immunoglobulin reactivity against B. burgdorferi in serum from these patients was no greater than that in serum from normal controls. The patients had a vigorous T-cell proliferative response to whole B. burgdorferi, with a mean ( +/- SEM) stimulation index of 17.8 +/- 3.3, similar to that (15.8 +/- 3.2) in 18 patients with chronic Lyme disease who had detectable antibodies. The T-cell response of both groups was greater than that of a control group of healthy subjects (3.1 +/- 0.5; P less than 0.001). We conclude that the presence of chronic Lyme disease cannot be excluded by the absence of antibodies against B. burgdorferi and that a specific T-cell blastogenic response to B. burgdorferi is evidence of infection in seronegative patients with clinical indications of chronic Lyme disease.

Comment in

http://www.ncbi.nlm.nih.gov/pubmed/3054554