Welcome to ActionLyme - the Lyme Cryme Whistleblower's Website

Prosecuting the CDC
CDC's "Great Imitator"
USDOJ RICO filed "Cryme Disease" book Bioweapons Attributes
(See Israeli section)

Pam3Cys ►Imitators/AIDS

Immune Suppressor "vaccine"

OCCUPY USDOJ Groups
Relapsing Fever History Dearborn Booklet (pdf) Chp 1 Scientific Validity Cong. Dual Use Bioweapons Epstein-OspA-Borreliosis Myco Erythrocytes (fatigue) OCCUPY_GWI
CDC: "Tmt Fails" Dearborn Quotes Chp 2 McSweegan ALDF/IDSA's RICO Patents "LYMErix ▲ Disease" GarthNicolson-GWI OCCUPY_CFIDS
CDC Falsifies Testing UConn's Kid Tuskegee Falsified Vax & FDA Corixa-Imugen RICO Pam3Cys_ImmuSupp OCCUPY_PSYCH
IDSA's Biomarkers Schoen-LYMErix Plum Island (mycoplasma) CDCs Patents w/SKB Confronting NIH CT Med Board OCCUPY_CPS
Klempner's NumerousCrimes Weinstein's Frauds Russians at NYMC DARPA Boots CDC Auwaerter EBV UN Complaint (LIBEL) OCCUPY_LYME
IDSA Says: "Cysts Viable" Yale's Valid (real) Test Yale Congenital Lyme Reports  Plum Stupid Vaccines' Brain Damage Fraud With Intent OCCUPY_AUTISM

DNA/RNA ShellGame

Not ▲ used for LYMErix "Jews" hate us? See Talmud. IDSA ▲self-indicts
 
Durland, Gangster Queen
 

 


10/24/2014 09:31:26

Index/Home

Khan Academy of "Lyme & LYMErix Activate the Herpes.. and Dearborn was performed to hide that fact :)

Cryme Trainer; Outline for Indictments and the OCCUPY the USDOJ

Non-HLA-linked diseases = Failed Autism Vaccines, Mold-Related illnesses, LYMErix-Disease, Lyme, CFIDS/FM, & Gulf War Illness




Follow the Lyme/LYMErix Scam in the order it occurred, below:

Older data on the incurability of Relapsing Fever

1986, McSweegan trashes Navy for $$$ for ALDF.com

1988, Dattwyler & about immune-suppressing, seronegative Lyme

1990, CDC: "Diagnose Lyme as if it was Relapsing Fever."

Allen Steere  "NeuroLyme won't test positive," 1990.

1992, CDC officer Allen Steere falsifies testing in Europe

1992, CDC patents with SmithKline show 2 kinds of Lyme

1993, Barbour and Fish slam Neurologic Lyme victims.

Compare the 2 kinds of Lyme in the RICO complaint

1994, FDA LYMErix Meeting

1994, CDC's Dearborn Booklet .pdf

CDC's invitation to participate in Dearborn .pdf

Dearborn, Who Said What?

Igenex, Harris, Dearborn .pdf

Evidence  Lyme criminals knew LYMErix produced the same "multisystem disease" as "Chronic Lyme"

LYMErix Damage Coverup (short)
 

120302 NIH Treatments
 

1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.

ActionLyme/Kathleen Dickson predicts all of Bushie's outcomes in Oct 2000

ActionLyme/Kathleen Dickson predicts Bush will have us worshipping his bombs (Shock and Awe"), in Oct 2000 during the Gore Debates



Steere's "Seronegative Lyme" Hysteria about OspA-induced Antibody Inhibition
Read about it also in the 1998 FDA LYMErix meeting transcript:
http://www.fda.gov/ohrms/dockets/ac/98/transcpt/3422t1.rtf


1990
; Steere using Dattwyler and Volkman's Seronegative Lyme Assay (developed because Dattwyler knew fungal antigens produced immunosuppression) to evaluate "Chronic Neurologic Lyme" cases:


http://www.nejm.org/doi/pdf/10.1056/NEJM199011223232102


See Also, CRYMEDISEASE_CHP3_B.htm  where we show Steere in 1991 actually used this seronegative Lyme assay on his own lab
workers, 101016.htm (
http://www.ncbi.nlm.nih.gov/pubmed/1883122)  where we see Steere knowingly falsely advising the Academy
of Insurance Medicine a year later (1992), and the Plum Island Chapter of Cryme Disease where Justin Radolf demonstrates in 2001
that OspA vaccination (or autovaccination via blebbing), results in inhibition of antibody production via downregulation of HLA.

N  1988 Dec 1;319(22):1441-6.

Seronegative Lyme disease. Dissociation of specific T- and B-lymphocyte responses to Borrelia burgdorferi.

Source

Department of Medicine, State University of New York, School of Medicine, Stony Brook 11794-8161.

Abstract

The diagnosis of Lyme disease often depends on the measurement of serum antibodies to Borrelia burgdorferi, the spirochete that causes this disorder. Although prompt treatment with antibiotics may abrogate the antibody response to the infection, symptoms persist in some patients. We studied 17 patients who had presented with acute Lyme disease and received prompt treatment with oral antibiotics, but in whom chronic Lyme disease subsequently developed. Although these patients had clinically active disease, none had diagnostic levels of antibodies to B. burgdorferi on either a standard enzyme-linked immunosorbent assay or immunofluorescence assay. On Western blot analysis, the level of immunoglobulin reactivity against B. burgdorferi in serum from these patients was no greater than that in serum from normal controls. The patients had a vigorous T-cell proliferative response to whole B. burgdorferi, with a mean ( +/- SEM) stimulation index of 17.8 +/- 3.3, similar to that (15.8 +/- 3.2) in 18 patients with chronic Lyme disease who had detectable antibodies. The T-cell response of both groups was greater than that of a control group of healthy subjects (3.1 +/- 0.5; P less than 0.001). We conclude that the presence of chronic Lyme disease cannot be excluded by the absence of antibodies against B. burgdorferi and that a specific T-cell blastogenic response to B. burgdorferi is evidence of infection in seronegative patients with clinical indications of chronic Lyme disease.

Comment in

http://www.ncbi.nlm.nih.gov/pubmed/3054554