IDSA's Secrets:

Guardian: "New World Disorder"
IDSA's Persistence "Cryme Disease" book Klempner's Fraud USDOJ RICO Myco-Viral Synergy Bioweapons Attributes Kissinger NWO Beast
Relapsing Fever Dearborn Quotes Plum Island Corixa RICO Epstein▲Borreliosis Borrelia & B-cells Rx Brain Damage
Steere Falsifies Test Dearborn Booklet Russians & NYMC RICO Patents GarthNicolson-GWI Despite NIH CD20 Hell/NDEs
IDSA's Imitators Yale/SKB admit crime IDSA: "Cyst Viable" CDCs Patents w/SKB CT Med Board Grants Search "TLR2" Psychiatry
IDSA's ShellGame Schoen-LYMErix LYMErix ►Imitators DARPA Boots CDC 3 Kinds Lyme-MS DCF's-Penisbiter
IDSA's Biomarkers Weinstein's Frauds UConn's KidTuskegee Plum Stupid Fraud With Intent PubMed Updates: TLR2   DCF's Entrapment
IDSA's Stupid Rx
 
Dickson FDA Yale Yale's Congen Lyme
 
IDSA ▲ self-indicts
 

 
Penisbiter Update
 


09 Feb 2012 

HOME

Pharma/CDC on Brain damage from vaccines, Fauci, Phages, Bioweapons manufacture

HHS.gov is Incompetent; BMJ calls fraud "crime.")

Official: CFIDS and MS-Lyme are the same disease; Epstein-Barr 


CDC Greed (won't answer the FOIA)

ELISA = arbitrary cutoff.

Disclaimer

Overview
 


TUSKEGEE - By Jerry Leonard


1998, CIA Oilmen & Israelis plan to overthrow Saddam for the oil.

Bush/Gore  Oil/War-(Oct,2000)  

Bush's own explainer (Oct 2000): Iraq Oil

Iraq was an oil-theft war.




 

 


111221-120209; Table Summarizing what we know so far about mechanisms and markers in EBV-Borrelia co-activation

Nearly every disease is represented here, from the mechanisms by which childhood vaccines contaminated with mycoplasma might be giving children the very diseases the vaccines were meant to prevent, to antibiotic resistance genes being transferred to the likes of Staph aureus via bacteriophage-vectored transfer of "virulence determinants" under the cover of a SLYMErix (TLR2-agonist/TLR2, stealth, tolerance) biofilm.  From cancer to the failure of the MRSA-LYMErix, Tb-LYMErix, and HIV-LYMErix "vaccines"- which happened years after the falsified failure of LYMErix.

And to think Yale and SmithKline owned all that proprietary data all these years.
[Yale and SmithKline (a UK company) own the proprietary antibiotic-resistance data because resistance (TLR2 tolerance) is caused by Lyme and LYMErix; they admitted they knew this at the 1998 FDA meeting!)]   Yale staff are quite notorious for not understanding the meaning of the word "system."  That is, Yale staff believe that one should not speak about markers of illness or illness induction by fungal antigens and that to do so means the speaker is a "terrorist like Ted Kascynski."  Which, you know, how does one deal with that kind of insanity?  It's like George Bush claiming, "We have interests in the Middle East."   ("Wait, what?")

That kind of stupidity we know for sure is due to the "diabolical delusion" predicted to envelope Western society in the end times.  
"Postmodernists believe that truth is myth, and myth, truth. This equation has its roots in pop psychology. The same people also believe that emotions are a form of reality. There used to be another name for this state of mind. It used to be called psychosis." -- Brad Holland

You can't educate psychiatrists because they're True Believers in people operating solely in their own self-interest.  But psychiatrists deserve to suffer a life of such cynicism, because in the end, and after all, you know it when you do something wrong.  Read Hostage to the Devil and the other case histories of exorcisms.  These demons admit they're crazy.

 

Background:

1989, Paul Duray (in IDSA's journal), Clinical pathologic correlations of Lyme disease.

“Frank signs of meningeal irritation herald stage II illness, reflected by an increase of CSF lymphocytes and plasma cells and moderate increases in total protein in CSF [9,16,17].  Immature B cells can also be seen in the spinal fluid.  These cells can appear quite atypical- not unlike transformed of neoplastic lymphocytes."  http://www.ncbi.nlm.nih.gov/pubmed/2814170
IDSA_CLINIPATH_DURAY.htm ("Epstein-Barr-like transformed B cells")


1992 Duray:
"On occasion, these atypical-appearing large lymphocytes have been misinterpreted in biopsy by several laboratories as cells of a malignant lymphoma or leukemia.  Bb antigens, then, may stimulate growth of immature lymphocytic suibsets in some target organs, as well as in the cerebrospinal fluid (Szyfelbein and Ross 1988).  Usual bacterial infections do not produce such lymphocytic infiltrates in tissue.  These immunoblastoid cells in Bb infections at times resemble those found in Epstein-Barr virus infections.  Does Bb reactivate latent virus infections in tissues?  Do some tick inocula harbor simultaneous infectious agents (ixodid ticks can harbor Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing multi-agent infections in some hosts?  Further studies can clarify these issues by mans of tissue-based molecular probe analysis." - 
 Paul Duray, NCI, NIH, Ft. Detrick, at the 1992 Cold Spring Harbor Crooks' Conference, published in Steve Schutzer's
Lyme Disease: Molecular and Immunologic Approaches
 

NOTE:  You can't use antibody testing to associate any viral and/or fungal exposures in a so-called autoimmune outcome.  Particularly not among TLR2-agonist bearing bacteria/fungi or with the herpesviruses.  That the NIH and CDC are not up-front about this fact has much to do with bioweapons, since a bioweapon, to be effective, couldn't lead to its source.  It would have to be the Time-Bomb or the Trojan Horse or the Stealth Disabler type.

This table is working.  We see the hijacking of the Life of a Cell by 2 organisms ...  Or as Gary Wormser declared in 2001, while OspA was still on the market, "a blocking of the cell cycle progression."   It's like immunological paralysis.  To go with the NIH's intellectual paralysis.

Herpes Mouse Herpes/ XMRV Fungal/Viral Spirochetes Myco/OspA EBV/HERVs Flagellin MRSA/Chlamydia Tuberculosis Cysts Q Fever
Stevil Straus: Characterization & treatment of chronic active EBV disease: 28yrs, USA (CFIDS is Chronic EBV)

 

Mouse Herpes 68 hijacks MAVS and IKKbeta TLR2 Signaling Depletes IRAK1 and Inhibits Induction of Type 1 by TLR7/9  -- Harding, 2012 "Bacterial/Viral Coinfections" Duray at Ft. Detrick in 1992 and reporting to IDSA about EBV- transformed lymphocytes in 1989 Chronic Lyme is seronegative because Lyme is chronic
(downregulation of MHC-II; Justin Radolf) due to (blebbing)
Harold Varmus, Denise Huber
Huber says there is 2 kinds of Lyme: the EBV evoking kind and Steere's "knees"
Formation of cysts within cells results in the release of flagellin (See H. pylori&EBV Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involved. 101016.htm 111106.htm
US Army: Spirochetal cysts are in dried animal urine.
Q-Fever/Chronic Lyme (activation of viruses) TLR agonism becomes seronegative.
Update, Mayo Clinic on RA and Cytomegalovirus (120202)

A profile of immune response to herpesvirus... [Arthritis Res Ther. 2012] - PubMed - NCBI

NCI/XMRV  and NF-kB prostate cancer from the imaginary XMRV ?? "Conclusions: EBV DNA was often found together with other microbial findings in CSF of immunocompromised [LYMErix- or Lyme-Disease]  patients."  "Pam3Cys keeps the precursors in a more immature stage"  (IL-17) Tolerance induced  Pam3Cys (Yale's LYMErix "vaccine") is due to ablation of IL-1R-associated kinase-1. 15294992   Flagellin activates Mouse Gammaherpes PubMed: Chlamydia and TLR2 EBV transformed cells help present fungal antigens Steere's lab workers inhaled Borrelial cysts. Coxiella agonizes TLR2 (would be seronegative)

"Conclusions: EBV DNA was often found together with other microbial findings in CSF of immunocompromised [LYMErix- or Lyme-Disease]  patients." 

 

Flagellin activates Mouse Gammaherpes TLR2 agonists synergistically increase the proliferation of EBV

101016.htm Epstein-Borreliosis datapage; Note: Harding explains Steere's Knees (HLA-antigen complex is shed)

OspA induces IL-10

IL10>>CD4- = EBV

EBV & IL-10  

OspA delays apoptosis through inhibition f caspase-3 activity:  CD14&TLR-2.  (Ireland,  BLP (OspA) inhibits neutrophil mitochondrial membrane depolarization"

"It has been demonstrated that LPS inhibits PMN apoptosis preferentially through stabilization of the mitochondrial membrane and subsequent inhibition of caspase-3 (33)."

    PubMed: Chlamydia and Epstein-Barr  

 

Mario Philipp's monkey study = transfer of cysts.  Called the MIT. Gulf War Illness Q Fever

No one can be diagnosed with anything that's  a bioweapon.

Stress steroid hormones literally activates EBV Which means psych.org is trashed, once again   "Internal"  Lyme spirochetes increase EBV  replication. Structure of Leptospira TLR2 agonists  They say not to use mice TLR2. TLR2 and nf-kappa        
 
Q Fever &  inhibition of apoptosis Could be missed.  Crooks  playing RNA/DNA ShellGame
Psych.org is trashed (NYT)   Interaction of Borrelia burgdorferi sensu lato with Epstein-Barr virus in lymphoblastoid cells Lyme causes CLL Leukemia

(Look up the symptoms of that)

IL-10 (induced by OspA)

IL10>>CD4- = EBV

 

          RML:  Sustained of Akt and Erk1/2 is required for Q Fever anti-apoptotic activity
15950179
EBV alters mitochondrial membrane

CDC: EBV wrecks mitochondria.

  H. pylori works with EBV   TLR2 activation inhibits embryonic neural progenitor cell proliferation EBV transformed cells help present fungal antigens   H. pylori works with EBV     C. burnetii inhibits activation  apoptosis through a mechanism that involves preventing cytochrome c release from mitochondria
EBV transformed cells help present fungal antigens   H. Pylori/EBV affect NF-kappa-B   Systemic stimulation of TLR2 (such as in vaccination with OspA) impairs neonatal mouse brain development     H. Pylori/EBV affect NF-kappa-B    

 

 
EBV LMP1 reduces p53 protein levels independent of the PI3K-Akt pathway. Dec 21, 2011

111222: HSV-1 Infection Activates the Epstein-Barr Virus Replicative Cycle via a CREB-Dependent Mechanism

     

 

Treatment of Throat Cancer Caused by Epstein-Barr is to Reverse Dattwyler's NK-Cell Suppression:              
Despite the NIH anti-CD20 datapage      

NIH: says "OspA suppresses & reprograms immunity" (wins Nobel; means Dearborn is a lie as are "Lyme" "guidelines"

           
Seronegative EBV (downregulation of MHC-II)

BCL-2 Homolog BHRF1

     

PubMed: "Endotoxin Tolerance and TLR2" [means LYMErix (OspA) was not a vaccine, Dearborn is a lie, and Borrelial antigens are immunosuppressive as TLR2 agonists.]

           
EBV & LMP1          
 
 
       
Oncogene BCL2-like in EBV          

 

 
       
EBV &  mycoplasma                    
EBV inhibits p53 auto-kill kinase                    
NF-Kappa B and Herpes (PubMed, General search)      

 

             
Herpes Simplex Immunosuppression (NF-kappa)                    
Epstein-Barr and anti-CD20 (Rituximab)
(PubMed, 500+)(and CFIDS)
                   
IL10>>CD4- = EBV                    
EBV & IL-10                    
 

 

Three times the CDC and BigPharma reported that childhood vaccines should not be given to immunosuppressed children because they might then get the disease the vaccines were intended to prevent - and how that immunosuppression might be acquired by mycoplasmal contamination in the vaccine batch/vial...

"Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the vaccine may actually cause disease."
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7%2C632%2C510.PN.&OS=PN%2F7%2C632%2C510&RS=PN%2F7%2C632%2C510

and

Measles, Mumps, and Rubella -- Vaccine Use and Strategies for Elimination of Measles, Rubella, and Congenital Rubella Syndrome and Control of Mumps: Recommendations of the Advisory Committee on Immunization Practices (ACIP) http://www.cdc.gov/mmwr/preview/mmwrhtml/00053391.htm

"Updated information on adverse events and contraindications, particularly for persons with severe HIV infection, persons with a history of egg allergy or gelatin allergy, persons with a history of thrombocytopenia, and persons receiving steroid therapy." [are immunosuppressed- KMD]

and

in the case of pandemic MRSA, as we have seen, the vaccine didn't work because TLR2 agonists (lipoproteins)
suppress the immune system.  We also learned from the MRSA vaccine patent, that:

"Several established vaccines consist of live attenuated organisms where ***the risk of reversion to the virulent wild-type strain exists. In particular in immunocompromised hosts this can be a live threatening scenario.*** Alternatively, vaccines are administered as a combination of pathogen-derived antigens together with compounds that induce or enhance immune responses against these antigens (these compounds are commonly termed adjuvant), since these subunit vaccines on their own are generally not effective."
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7,771,728.PN.&OS=PN/7,771,728&RS=PN/7,771,728

We learned that this was the reason for the pandemic of vaccine brain damaged children:  This is the "Mumps without the Lumps, the Measles without the Spots,"... the Post-natal instead of Prenatal Rubella-induced "autism" (the reason for the Rubella vaccine in the first place).

I would even guess that swine were involved in the ferret pandemic experiment, somewhere along the line.  (Either it's true that ferret/human/swine receptors are similar or swine are somewhere in the experimental ferret-flu picture and that data is being left out of the publications). 

Swine flu and probably human flu, together, in the same animal, is what was claimed (below).  That would mean the ferrets or the swine, when creating H9N2 or H9N5 in a swine/ferret, might have been simultaneously injected with a human flu vaccine??
 

Regardless, here is what we know about what is the requirement for a pandemic human strain to take off:


Chinese: Characterization of H9 subtype influenza viruses from the ducks of southern China: a candidate for the next influenza pandemic in humans? 
http://www.ncbi.nlm.nih.gov/pubmed/12768017 
 

Don Wiley, 2001, before he was murdered "changing a flat tire":    

"α2,6-Linked sialosides bind in a cis conformation, exposing the glycosidic oxygen to solution and nonpolar atoms of the receptor to Leu-226, a human-specific residue. ...

..."Evidently, the “closed” geometry of the avian H5 HA, which prefers α2,3 linkages, results from the Gln-226/Gly-228 pair. This geometry appears optimal for positioning Gln-226 to hydrogen-bond to the α2,3 trans motif composed of the 4-OH of Gal-2 and the glycosidic oxygen (Fig. ​(Fig.22c). The human H3 HA with the Leu-226/Ser-228 pair is at the opposite extreme, more “open” at both 228 and 226, which may be optimal for Leu-226 to make nonpolar contacts to α2,6 cis linkages. Swine H9 HA (Leu-226/Gly-228) and the L226Q variant of human H3 HA (Gln-226/Ser-228) appear to be intermediate, with partial avian and partial human character and the nonstandard Leu/Gly and Gln/Ser pairs
(Fig. ​(Fig.22f)."  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC58807/?tool=pubmed

 

CDC, Jan 2012In vitro evolution of H5N1 avian influenza virus toward human-type receptor specificity.

"Acquisition of α2-6 sialoside receptor specificity
by α2-3 specific highly-pathogenic avian influenza viruses (H5N1) is thought to be a prerequisite for efficient transmission in humans. By in vitro selection for binding α2-6 sialosides, we identified four variant viruses with amino acid substitutions in the hemagglutinin (S227N, D187G, E190G, and Q196R) that revealed modestly increased α2-6 and minimally decreased α2-3 binding by glycan array analysis. However, a mutant virus combining Q196R with mutations from previous pandemic viruses (Q226L and G228S) revealed predominantly α2-6 binding. Unlike the wild type H5N1, this mutant virus was transmitted by direct contact in the ferret model although not by airborne respiratory droplets. However, a reassortant virus with the mutant hemagglutinin, a human N2 neuraminidase and internal genes from an H5N1 virus was partially transmitted via respiratory droplets. The complex changes required for airborne transmissibility in ferrets suggest that extensive evolution is needed for H5N1 transmissibility in humans."  http://www.ncbi.nlm.nih.gov/pubmed?term=22056389


Pandemic flu and the "Unknown Intermediate" host being a pig:  "Reassortant viruses appear to have caused the pandemics of 1957 and 1968; the 1957 H2 virus differed by three genes, those for HA, NA and the RNA polymerase subunit PB1, from the H1 virus that infected humans between 1918 and 1957; the 1968 H3 virus differed by two genes, those for HA and PB1, from the H2 virus that infected humans between 1957 and 1968 (Kawaoka et al., 1989). In both cases, the genes for the H2 and H3 HAs are proposed to have been contributed by avian viruses, ***during infection of an unknown host that was infected simultaneously by the prevalent human virus."***
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC125880/?tool=pubmed

 


RECENT ACTIONLYME HOMEPAGES

120209.htm More of the same.  News and Updates related to how the real science got away from anyone associated with Connecticut and now the new CT Gov wants Education and State Employee pensions reform.  Hilarious.  Payback time.  Now Connecticut wonders how to pay for the incompetence and crimes of Corrupticut since the early 1990s while not bringing in any revenue because of the unmarketability of the Peniscentrical Dogma of Yale.

120129.htm  Fungal-Viral Synergy Drives Autoimmunity and Vaccines' Brain Damage, & the Plum Island Method of Bioweapons (Pandemic Flu) Production.

111221.htm  The Chinese, the CDC, and Don Wiley reveal that Pandemic Human Flu will likely be an H9N5 Swine Flu, and this H9N5 type seems to be what was created in US/Danish Labs this month, causing all the hysteria; the US Government deliberately lies in the journals.  More on Bioweapons, like lipoproteins being "needed for efficient spore formation."  Tuberculosis and the value of Molecular Mice (no good). ◄ This is funny because UConn, being denied grants for years - becuase they're lying assholes, intends to go into business as a Mouse Factory (Slate); LOL, that would be par for the Clueless Course for UConn.  Irish Lady Scientist.
Update: State Grants Final Approval to Spend $291 Million On Jackson Laboratory Research Center

LMAO!  That is so predictable, LOLlolololzz..

111106.htm Varmus, Rituximab, Ben Luft and Dennis Parenti 1998 FDA transcript where SKB admits LYMErix causes chronic Lyme-like illness.

110822.htm

110625.htm

110518.htm

110316.htm

TOC1.htm
 


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