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09 Feb 2012
HOME
Pharma/CDC on Brain
damage from vaccines, Fauci, Phages, Bioweapons manufacture
HHS.gov is
Incompetent; BMJ calls fraud "crime.")
Official: CFIDS and MS-Lyme are the
same disease; Epstein-Barr
CDC Greed
(won't answer the FOIA)
ELISA = arbitrary cutoff.
Disclaimer
Overview
TUSKEGEE - By Jerry Leonard
1998, CIA Oilmen & Israelis plan to overthrow
Saddam for the oil.
Bush/Gore Oil/War-(Oct,2000)
Bush's own explainer (Oct
2000):
Iraq Oil
Iraq was an oil-theft war.
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111221-120209; Table Summarizing what we
know so far about mechanisms and markers in
EBV-Borrelia co-activation.
Nearly every disease is represented here, from the mechanisms by which childhood vaccines contaminated with mycoplasma might be giving children the very diseases the vaccines were meant to prevent, to antibiotic resistance genes being transferred to the likes of Staph aureus via bacteriophage-vectored transfer of "virulence determinants" under the cover of a SLYMErix (TLR2-agonist/TLR2, stealth, tolerance) biofilm. From cancer to the failure of the MRSA-LYMErix, Tb-LYMErix, and HIV-LYMErix "vaccines"- which happened years after the falsified failure of LYMErix.
And to think Yale and SmithKline owned all that proprietary data all these years. [Yale and SmithKline (a UK company) own the proprietary antibiotic-resistance data because resistance (TLR2 tolerance) is caused by Lyme and LYMErix; they admitted they knew this at the 1998 FDA meeting!)] Yale staff are quite notorious for not understanding the meaning of the word "system." That is, Yale staff believe that one should not speak about markers of illness or illness induction by fungal antigens and that to do so means the speaker is a "terrorist like Ted Kascynski." Which, you know, how does one deal with that kind of insanity? It's like George Bush claiming, "We have interests in the Middle East." ("Wait, what?")
That kind of stupidity we know for sure is due to the "diabolical delusion" predicted to envelope Western society in the end times. "Postmodernists believe that truth is myth, and myth, truth. This equation has its roots in pop psychology. The same people also believe that emotions are a form of reality. There used to be another name for this state of mind. It used to be called psychosis." -- Brad Holland
You can't educate psychiatrists because they're True Believers in people operating solely in their own self-interest. But psychiatrists deserve to suffer a life of such cynicism, because in the end, and after all, you know it when you do something wrong. Read Hostage to the Devil and the other case histories of exorcisms. These demons admit they're crazy.
Background:
1989, Paul Duray (in IDSA's journal), Clinical pathologic correlations of Lyme disease.
“Frank signs of meningeal irritation herald stage II illness, reflected by an increase of CSF lymphocytes and plasma cells and moderate increases in total protein in CSF [9,16,17]. Immature B cells can also be seen in the spinal fluid. These cells can appear quite atypical- not unlike transformed of neoplastic lymphocytes." http://www.ncbi.nlm.nih.gov/pubmed/2814170 IDSA_CLINIPATH_DURAY.htm ("Epstein-Barr-like transformed B cells")
1992 Duray: "On occasion, these atypical-appearing large lymphocytes have been misinterpreted in biopsy by several laboratories as cells of a malignant lymphoma or leukemia. Bb antigens, then, may stimulate growth of immature lymphocytic suibsets in some target organs, as well as in the cerebrospinal fluid (Szyfelbein and Ross 1988). Usual bacterial infections do not produce such lymphocytic infiltrates in tissue. These immunoblastoid cells in Bb infections at times resemble those found in Epstein-Barr virus infections. Does Bb reactivate latent virus infections in tissues? Do some tick inocula harbor simultaneous infectious agents (ixodid ticks can harbor Rickettsiae, Babesia microti, and Ehrlichia bacteria, in addition to Bb), producing multi-agent infections in some hosts? Further studies can clarify these issues by mans of tissue-based molecular probe analysis." - Paul Duray, NCI, NIH, Ft. Detrick, at the 1992
Cold Spring Harbor Crooks' Conference, published in Steve Schutzer's Lyme Disease: Molecular and Immunologic Approaches
NOTE: You can't use antibody testing to
associate any viral and/or fungal exposures in a so-called autoimmune outcome.
Particularly not among
TLR2-agonist bearing
bacteria/fungi or with the
herpesviruses. That the NIH and CDC are not up-front about this fact
has much to do with bioweapons, since a bioweapon, to be effective, couldn't
lead to its source. It would have to be the Time-Bomb or the Trojan Horse
or the Stealth Disabler type.
This table is working. We see the
hijacking of the Life of a Cell by 2 organisms ... Or as Gary Wormser
declared in 2001, while OspA was still on the market, "a
blocking of the cell cycle progression." It's like immunological
paralysis. To go with the NIH's intellectual paralysis.
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Herpes |
Mouse Herpes/ XMRV |
Fungal/Viral |
Spirochetes |
Myco/OspA |
EBV/HERVs |
Flagellin |
MRSA/Chlamydia |
Tuberculosis |
Cysts |
Q Fever |
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Stevil Straus:
Characterization & treatment of chronic
active EBV disease: 28yrs, USA
(CFIDS is Chronic EBV) |
Mouse Herpes 68 hijacks
MAVS and IKKbeta |
TLR2 Signaling Depletes
IRAK1 and Inhibits Induction of Type 1
by TLR7/9
-- Harding, 2012 "Bacterial/Viral
Coinfections" |
Duray at Ft. Detrick in 1992 and
reporting to
IDSA about EBV- transformed
lymphocytes in 1989 |
Chronic Lyme is
seronegative because Lyme is chronic
(downregulation
of MHC-II; Justin Radolf) due to (blebbing) |
Harold Varmus, Denise Huber
Huber says there is 2 kinds of Lyme: the
EBV evoking kind and Steere's "knees" |
Formation of cysts within cells
results in the release of flagellin
(See H. pylori&EBV |
Lipoteichoic acid (LTA)
of Streptococcus pneumoniae and
Staphylococcus aureus
activates immune cells via Toll-like
receptor (TLR)-2,
lipopolysaccharide-binding protein (LBP),
and CD14, whereas TLR-4 and MD-2 are not
involved. |
101016.htm |
111106.htm
US Army: Spirochetal cysts are in dried
animal urine. |
Q-Fever/Chronic Lyme
(activation of viruses)
TLR agonism becomes seronegative. |
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Update, Mayo Clinic on RA
and Cytomegalovirus (120202)
A profile of immune response to herpesvirus... [Arthritis Res Ther. 2012] - PubMed - NCBI
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NCI/XMRV and NF-kB prostate
cancer from the imaginary XMRV ?? |
"Conclusions: EBV DNA
was often found together with other
microbial findings in CSF of
immunocompromised
[LYMErix- or
Lyme-Disease]
patients."
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"Pam3Cys keeps the precursors in a more
immature stage" (IL-17) |
Tolerance induced
Pam3Cys (Yale's
LYMErix "vaccine") is due to
ablation of IL-1R-associated kinase-1.
15294992 |
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Flagellin activates Mouse Gammaherpes |
PubMed: Chlamydia and
TLR2 |
EBV transformed cells
help present fungal antigens |
Steere's lab workers inhaled Borrelial
cysts. |
Coxiella agonizes TLR2
(would be seronegative) |
"Conclusions: EBV DNA
was often found together with other
microbial findings in CSF of
immunocompromised
[LYMErix- or
Lyme-Disease]
patients."
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Flagellin activates Mouse Gammaherpes |
TLR2 agonists
synergistically increase the
proliferation of EBV
101016.htm
Epstein-Borreliosis datapage; Note:
Harding explains Steere's Knees (HLA-antigen
complex is shed) |
OspA induces IL-10
IL10>>CD4- = EBV
EBV & IL-10 |
OspA delays apoptosis
through inhibition f caspase-3 activity:
CD14&TLR-2.
(Ireland, BLP (OspA) inhibits
neutrophil mitochondrial membrane
depolarization"
"It has been demonstrated
that LPS inhibits PMN apoptosis
preferentially through stabilization of
the mitochondrial membrane and
subsequent inhibition of caspase-3 (33)." |
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PubMed: Chlamydia and
Epstein-Barr |
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Mario
Philipp's monkey study = transfer of
cysts. Called the MIT. |
Gulf War Illness Q Fever
No one can be diagnosed
with anything that's a bioweapon.
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Stress steroid hormones
literally activates EBV
Which means psych.org is
trashed, once again |
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"Internal" Lyme
spirochetes increase EBV
replication. |
Structure of Leptospira TLR2 agonists
They say not to use mice TLR2. |
TLR2 and nf-kappa |
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Q Fever & inhibition of apoptosis
Could be missed. Crooks
playing
RNA/DNA
ShellGame |
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Psych.org is trashed (NYT) |
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Interaction of
Borrelia
burgdorferi sensu lato with Epstein-Barr
virus in lymphoblastoid cells |
Lyme causes CLL Leukemia
(Look up the symptoms of
that) |
IL-10 (induced by OspA)
IL10>>CD4- = EBV
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RML: Sustained of Akt and Erk1/2 is required for Q Fever anti-apoptotic activity |
15950179
EBV alters
mitochondrial membrane
CDC: EBV wrecks mitochondria. |
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H. pylori works with EBV |
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TLR2 activation inhibits embryonic neural progenitor cell proliferation |
EBV transformed cells help present fungal antigens |
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H. pylori works with EBV |
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C. burnetii inhibits activation
apoptosis through a mechanism that involves preventing cytochrome c
release from mitochondria |
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EBV transformed cells help present fungal antigens |
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H. Pylori/EBV
affect NF-kappa-B |
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Systemic stimulation of TLR2 (such as in vaccination with OspA) impairs neonatal mouse brain development |
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H. Pylori/EBV affect NF-kappa-B |
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EBV LMP1 reduces p53
protein levels independent of the PI3K-Akt pathway. Dec 21, 2011
111222:
HSV-1 Infection Activates the
Epstein-Barr Virus Replicative Cycle via a CREB-Dependent Mechanism |
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Treatment of Throat Cancer Caused by Epstein-Barr is to Reverse Dattwyler's NK-Cell Suppression:
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Despite the NIH anti-CD20 datapage |
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NIH: says "OspA suppresses & reprograms immunity" (wins Nobel; means Dearborn is a lie as are "Lyme" "guidelines" |
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Seronegative EBV (downregulation of MHC-II)
BCL-2 Homolog BHRF1 |
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PubMed: "Endotoxin Tolerance and TLR2" [means LYMErix (OspA) was not a vaccine, Dearborn is a lie, and Borrelial antigens are immunosuppressive as TLR2 agonists.] |
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| EBV & LMP1 |
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Oncogene BCL2-like in EBV |
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EBV & mycoplasma |
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EBV inhibits p53
auto-kill kinase |
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NF-Kappa B and Herpes (PubMed, General search) |
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| Herpes Simplex Immunosuppression (NF-kappa) |
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Epstein-Barr and anti-CD20 (Rituximab)
(PubMed, 500+)(and CFIDS) |
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IL10>>CD4- = EBV |
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EBV & IL-10 |
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Three times the CDC and BigPharma reported that childhood vaccines should not be given to immunosuppressed children because they might then get the disease the vaccines were intended to prevent - and how that immunosuppression might be acquired by mycoplasmal contamination in the vaccine batch/vial...
"Finally, there is the risk that the virus may not be fully or completely inactivated or attenuated and thus, the vaccine may actually cause disease." http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7%2C632%2C510.PN.&OS=PN%2F7%2C632%2C510&RS=PN%2F7%2C632%2C510
and
Measles, Mumps, and Rubella -- Vaccine
Use and Strategies for Elimination of
Measles, Rubella, and Congenital Rubella
Syndrome and Control of Mumps:
Recommendations of the Advisory
Committee on Immunization Practices (ACIP)
http://www.cdc.gov/mmwr/preview/mmwrhtml/00053391.htm
"Updated information on adverse events
and contraindications, particularly for
persons with severe HIV infection,
persons with a history of egg allergy or
gelatin allergy, persons with a history
of thrombocytopenia, and persons
receiving steroid therapy." [are
immunosuppressed- KMD]
and
in the case of pandemic MRSA, as we have
seen, the
vaccine didn't work because TLR2
agonists (lipoproteins)
suppress the immune system. We
also learned from the MRSA
vaccine patent, that:
"Several established vaccines consist of
live attenuated organisms where ***the
risk of reversion to the virulent
wild-type strain exists. In particular
in immunocompromised hosts this can be a
live threatening scenario.***
Alternatively, vaccines are administered
as a combination of pathogen-derived
antigens together with compounds that
induce or enhance immune responses
against these antigens (these compounds
are commonly termed adjuvant), since
these subunit vaccines on their own are
generally not effective."
http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=7,771,728.PN.&OS=PN/7,771,728&RS=PN/7,771,728
We learned that this was the reason for
the
pandemic of vaccine brain damaged
children:
This is the "Mumps without the
Lumps, the Measles
without the Spots,"... the Post-natal
instead of
Prenatal Rubella-induced "autism" (the
reason
for the Rubella vaccine in the first
place).
I would even guess that swine were involved in the ferret pandemic experiment, somewhere along the line. (Either it's true that ferret/human/swine receptors are similar or swine are somewhere in the experimental ferret-flu picture and that data is being left out of the publications).
Swine flu and probably human flu, together, in the same animal, is what was claimed (below). That would mean the ferrets or the swine, when creating H9N2 or H9N5 in a swine/ferret, might have been simultaneously injected with a human flu vaccine?? Regardless, here is what we know about what is the requirement for a pandemic human strain to take off:
Chinese: Characterization of H9 subtype influenza viruses from the ducks of southern China: a candidate for the next influenza pandemic in humans? http://www.ncbi.nlm.nih.gov/pubmed/12768017
Don Wiley, 2001, before he was murdered "changing a flat tire":
"α2,6-Linked sialosides bind in a cis conformation, exposing the glycosidic oxygen to solution and nonpolar atoms of the receptor to Leu-226, a human-specific residue. ...
..."Evidently, the “closed” geometry of the avian H5 HA, which prefers α2,3 linkages, results from the Gln-226/Gly-228 pair. This geometry appears optimal for positioning Gln-226 to hydrogen-bond to the α2,3 trans motif composed of the 4-OH of Gal-2 and the glycosidic oxygen (Fig. (Fig.22c). The human H3 HA with the Leu-226/Ser-228 pair is at the opposite extreme, more “open” at both 228 and 226, which may be optimal for Leu-226 to make nonpolar contacts to α2,6 cis linkages. Swine H9 HA (Leu-226/Gly-228) and the L226Q variant of human H3 HA (Gln-226/Ser-228) appear to be intermediate, with partial avian and partial human character and the nonstandard Leu/Gly and Gln/Ser pairs (Fig. (Fig.22f)." http://www.ncbi.nlm.nih.gov/pmc/articles/PMC58807/?tool=pubmed
CDC, Jan 2012: In vitro evolution of H5N1 avian influenza virus toward human-type receptor specificity.
"Acquisition of α2-6 sialoside receptor specificity by α2-3 specific highly-pathogenic avian influenza viruses (H5N1) is thought to be a prerequisite for efficient transmission in humans. By in vitro selection for binding α2-6 sialosides, we identified four variant viruses with amino acid substitutions in the hemagglutinin (S227N, D187G, E190G, and Q196R) that revealed modestly increased α2-6 and minimally decreased α2-3 binding by glycan array analysis. However, a mutant virus combining Q196R with mutations from previous pandemic viruses (Q226L and G228S) revealed predominantly α2-6 binding. Unlike the wild type H5N1, this mutant virus was transmitted by direct contact in the ferret model although not by airborne respiratory droplets. However, a reassortant virus with the mutant hemagglutinin, a human N2 neuraminidase and internal genes from an H5N1 virus was partially transmitted via respiratory droplets. The complex changes required for airborne transmissibility in ferrets suggest that extensive evolution is needed for H5N1 transmissibility in humans." http://www.ncbi.nlm.nih.gov/pubmed?term=22056389 Pandemic flu and the "Unknown Intermediate" host being a pig: "Reassortant viruses appear to have caused the pandemics of 1957 and 1968; the 1957 H2 virus differed by three genes, those for HA, NA and the RNA polymerase subunit PB1, from the H1 virus that infected humans between 1918 and 1957; the 1968 H3 virus differed by two genes, those for HA and PB1, from the H2 virus that infected humans between 1957 and 1968 (Kawaoka et al., 1989). In both cases, the genes for the H2 and H3 HAs are proposed to have been contributed by avian viruses, ***during infection of an unknown host that was infected simultaneously by the prevalent human virus."*** http://www.ncbi.nlm.nih.gov/pmc/articles/PMC125880/?tool=pubmed
RECENT ACTIONLYME
HOMEPAGES
120209.htm
More of the same. News and Updates
related to how the real science got away
from anyone associated with Connecticut
and now the new CT Gov wants Education
and State Employee pensions reform.
Hilarious. Payback time. Now
Connecticut wonders how to pay for the
incompetence and crimes of Corrupticut
since the early 1990s while not bringing
in any revenue because of the
unmarketability of the Peniscentrical
Dogma of Yale.
120129.htm
Fungal-Viral Synergy Drives Autoimmunity
and Vaccines' Brain Damage, & the Plum
Island Method of Bioweapons (Pandemic
Flu) Production.
111221.htm
The Chinese, the CDC, and Don Wiley
reveal that Pandemic Human Flu will
likely be an H9N5 Swine Flu, and this
H9N5 type seems to be what was created
in US/Danish Labs this month, causing
all the hysteria; the US Government
deliberately lies in the journals.
More on Bioweapons, like lipoproteins
being "needed for efficient spore
formation." Tuberculosis and the
value of Molecular Mice (no good). ◄
This is funny because UConn, being
denied grants for years - becuase
they're lying assholes, intends to go
into business as a Mouse Factory
(Slate); LOL, that would be par for the
Clueless Course for UConn. Irish
Lady Scientist.
Update:
State Grants Final Approval to Spend
$291 Million On Jackson Laboratory
Research Center
LMAO! That is so predictable,
LOLlolololzz..
111106.htm
Varmus, Rituximab, Ben Luft and Dennis
Parenti 1998 FDA transcript where SKB
admits LYMErix causes chronic Lyme-like
illness.
110822.htm
110625.htm
110518.htm
110316.htm
TOC1.htm
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