Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine."

15 Oct 2016



Immunosuppression-its-a-thing/ - blog (There is no NIIID, hello.  Why;  Autism vaccines.)

Key Scientific Concepts - Exported fungal antigens inhibit apoptosis

Lyme Cryme: How it all Went Down

Crymes on Vimeo

DOJ protest group


Occam's Razor


IDSA says pedi-vaccines = too many, not vetted and given too young.

CDC knew OspA caused disease


BlumenthalAntiTrust Lawsuit

Exosomes, Blebs



Dattwyler, 1988
Golightly, 1988
Dressler, 1994
BarbourFish, 1993
Dearborn, 1994

Pathogenic Fungi

Predicting all of GW Bush's warcrimes, Oct 2000




New:  How to Un-Break Americans' Brains:  Keep Hitting them Over the Head With the Obvious. There is no National Institute of Post-Sepsis or Immunosuppression Diseases (NIIID), because to do so would betray the source of the Autism pandemic.  (Yet you hear these things every day on the TV commercials.)



The "Exceptional" History of Lyme Activism:

I have been around since 1996. 1999 was when the fake Lyme vaccine came out and was giving people "Lyme." ILADS had not formed yet.  ActionLyme was formed at the same time (Summer 1999).

Our group, ActionLyme, was about getting the criminals prosecuted from the beginning (reporting the crooks to their medical boards),... and getting LYMErix off the market.  (See the criminal charge sheets as they are now, the history and the chronology of the crime of saying Lyme was "just a bad knee.")

(Blowing the Whistle at the FDA, Jan 2001, exposing Dearborn and how OspA causes immunosuppression rather than, "was a vaccine.")

We had already done the Conflicts of Interest work, research and complaint by the summer of 1999 - NOT the LDA, who copied it all - which was why we called ourselves ACTIONLYME (in other words, get rid of the crooks, for once and for all).

ILADS did *nothing* but scheme about how to get more and more money and never fight for us. They STRAIGHT UP SAID that *** we victims had to do all the fighting, that they were too busy emptying our pockets.*** YOU know this is true because they STILL say this. They did not testify at the FDA about how LYMErix was causing the same disease.

*** It is OBVIOUS why they ignored all those sick people.***

ILADS did not even help the now Senator Richard Blumenthal go after the crooks, ILADS knew *that* little about the disease and the crime. These know-nothings and do-nothings are the ones so many people are angry at me for exposing. Odd.

Even if you knew nothing about me and ActionLyme and getting LYMErix off the market, you could see ILADS did not help Blumenthal go after the crooks and show him the FRAUD aspect of it all.

I write this because people are telling me *I* am the one who is doing this all wrong, and *I* am the bad guy, because they themselves make no investment in finding out how OspA causes the same disease, and that Lyme is incurable - everyone knows this, or those same people would not be prowling the internet, looking for the next famous VooDoo remedy. They would not put their hope in Charlatans who care so little about people, they dont even fight to take Medicare patients, making the disability legitimate. ILADS does not fight at all. All they do is put themselves on a pedestal.

It has been known for 100 years that spirochetes cause Great Imitator outcomes, target the lymph nodes, and that no antibiotics of any sort can dislodge them. This is not to mention that they damage the B cell germination centers, rendering you with an AIDS like disease - AKA the Great Imitator.

It is also OBVIOUS that for every person with a true HLA-linked autoimmune disease (these people make a lot of antibodies and are not too disabled by fatigue and weakness), there are 6 of us with the NO-IMMUNE outcomes called sepsis or immunoparalysis or Endotoxin Tolerance - a well known thing, and for centuries had a lot of different names.  But not since the 1960s or so, when psychiatry became "Medicine" (it was considered flakery up until that time by real doctors), did they start slandering us.

Look at the big picture. Every person owes it to themselves to take the long look. 100 years...

As it stands right now, only a handful of people in the world are dedicated to going after the CDC criminals.  The others, particularly other Americans, have no interest in fighting for the OTHER GUY.  The majority of "Americans," the "Exceptional" people, actually do not give a shit about anyone but themselves.

To Wit:  "Lyme activism" mirrors the true nature of Americans:  "It's All About Me, or I Aint Playin.  I, the American, do not play for the team, I play for me."

And no "doctors" fight for us.  Take a look around, and see that that is true.  Look for and try to find a single person on earth with "MD" after their names who can tell you what OspA is, and how it caused a disease like "Chronic Lyme," and how that immunosuppression ties in all the other diseases of abuse, such as Autism, Gulf War Illness, Chronic Fatigue Syndrome, Fibromyalgia, etc. 

It's not that complicated, but true facts make no person FAMOUS!!!  And there you have the nature of the Exceptionals in Exceptional America:  "It's All About Me, Or I Ain't Playin."

Every American is a little Godlet, and wants everyone else to bow down to them.  I. Me. My. 

"I will write a famous book!!! - so tell me all your ideas...."

"I was a protestor, I told MY STORY at several events!!"

"Here's my picture at a Lyme rally; I am a famous warrior since I am sick and you should hear! about how sick I am.  No one in the world has ever suffered as much as me!!"

"I should be on TV!! for how much of a sufferer I am.  Rich people like me, that is VERY FAMOUS suffering!!!  Poor people, not so much."
 (Really, someone wrote that, recently.)

"I am a rich person, therefore I have a more dangerous and serious disease than those peasants!!!" - Marisol Thomas, wife of a fiddler.  Great.

But the OTHER GUY?

Give them nothing.  Shut them up.  It goes that way across the board, from America.  This is the greatest nation in history.  We make guns and sow violence especially in the lands where there are resources we want.  :D

We Americans even go so far as to say we have to "protect our assets in other countries."  This would be like Russia saying they "have to invade and destroy South Africa because that's where our Russian gold is."


Arrogance has no creative ability, since self-idolatry mimics Lucifer and the demons' escapades.  They are cut off from the Light (Truth).   This is clearly the reason Americans lose at everything.  We suck at medical science, we don't do "care" (there are no doctors), and we lose all the False Flag Oil Wars. 

Now Yellen (the Fed) is calling for Austerity, cutting benefits, cutting welfare.  And this when over 50% of the national budget goes to losing the oil wars to Russia, Turkey, Syria, Egypt, Khazakstan, China, Iran, India and those allies of Eurasia.  The History of America is entirely about how selfishness and greed lose you your country.  It parallels the Lyme wars.


Imagine.  'Something Rockefeller University studied and knew about in 1922: that spirochetes are permanent infections and live in the lymph nodes, destroying immunity,... and we hear about it again in 2015 by a lady scientist, Nicole Baumgarth!!
I would say that was OUTSTANDING "translational" medicine, where all the words and years in between were lost,... and we had to have the Craazy Eddie McSweegan gang and a fake FUNGAL lipoprotein Lyme vaccine.  And no one in the ENTIRE ever asked what OspA was, nor to this day will admit what OspA was (Pam3Cys).

Good translating.  Good word-saying, all around.  Needs 27 million more dollars....


University of California, Davis, says the CDC, Allen Steere, Gary Wormser, Edward McSweegan, et al must be lying their faces off:  Lyme and LYMErix cause permanent immunosuppression or is like a B cell AIDS. 



Great translating from 1922.

'Needs more money because the NIH has not donated enough to that cesspool called the Ivy League, for them to still not understand what spirochetes are/do, despite a million new cases of DISABILITY per year in the USA, alone.


"It's the perfect stealth pathogen (means: no antibodies) that causes MS, Lupus, excruciating headaches, Chronic Fatigue Syndrome, ALS, stroke, dementia, etc," says a physician associated with Department of Energy, Brookhaven, Long Island, Uconn, Yale, the NIH, Allen Steere, etc,... And spirochetes for 100 years have been known to target lymph nodes and destroy B cell germinal centers, permanently - irreversibly.  Go ahead and use PubMed and search for spirochetes and lymph nodes (You can do it!), to verify.

And while the CDC admits to 300,000 cases of "Lyme disease" annually, the falsified Dearborn case definition ("only an HLA-linked hypersensitivity or arthritis in a knee response") means only 15% of the cases are reportable, which means there 2 million Lyme infection cases a year, half of whom remain disabled (UCSF).    That means there are 1 million new cases of disability per year in America alone, and an untold number in the greater world.  The cryme of falsifying the disease definition and description by CDC officers Alan Barbour and Barbara Johnson has never been prosecuted because this is America - Land of Oppression and Abuse, that jails and tortures whistleblowers.  The CDC claims the cyst or spheroplast form of Borreliae are not viable, while in 1964 wrote instructions for freeze-drying your borrelia for later use as intact spirochetes (weaponizing).

And while all of this is easily discoverable, not one single "MD" in America or any staff of the CDC or NIH or any medical society will admit to it all publicly.  None will tell you that the OspA/LYMErix vaccine not only never prevented spirochetes in any animal, it is the same fungal endotoxin that is responsible for ruining the immune system and causing post-sepsis syndrome, which is the real name and definition of Chronic Fatigue Syndrome/ME or "Chronic Lyme."  Certainly no "LLMDs" talk about the mechanisms by which spirochetes cause disease or explain to you why LYMErix, alone, caused the chronic, irreversible illness, with the secondary, opportunistic infections, such as Candida, other fungi, mycoplasma, etc., and especially the reactivated herpes viruses that are ACTUALLY responsible for the MS, Lupus, etc outcomes.

Do they?  And how many "MDs" in America specialize in Infectious Diseases?  You can see, they're worthless.  None even ask how LYMErix could have caused a "multi-system disease indistinguishable from later presentations of Lyme disease" - Mayo Clinic's former employee, David Persing


Remember, people:  The LAST thing you want to be known as or remembered as, is as a COWARD.  Cowards are revolting, repulsive, disgusting, and grotesque.  They are the very lowest life forms.  Even bathroom slime has more integrity because it does not willingly try to sell itself as perfume or scented body-wash.  I am talking mainly about people with "MD" after their names. 

Think of how many "doctors" were wrong about everything, how they did not know about fungal-viral synergy, about how fungal antigens cause immunosuppression, how this immunosuppression plays a role in the Autism pandemic (kids are GETTING the viruses, and not the "protection").  Think about how in the last 10-15 years psychiatry was debunked as hocus-pocus and how BigPharma is worried that no one wants to be a psychiatrist out of "medical school" any more (so who will push their bogus, brain-damaging "drugs?"). 

These facts - these truths we bring you that you can verify independently -, very clearly have zero to do with people who have "MD" after their names.  "Doctors" are useless, all around.  Their function is limited to writing drug prescriptions, but most do not know what goes into a validating a  lab test or are aware of the role of insurance companies writing diagnostic and treatment "guidelines."  Most "doctors" don't even know MS and Lupus are caused by some combination of the herpesviruses, as are most other illnesses and most cancers.  They don't even know they can use PubMed to search for this data and these associations!!

Now, what causes such stupidity?  Arrogance, which is the result of cowardice.  'The fear of appearing to be "less-than" the other guy. 

How in the world could Lyme be a "Great Imitator" causing all kinds of outcomes like ALS, Lupus (Yale used to have a "Lyme and Lupus Clinic, but now Yale says Lupus is most likely due to Epstein-Barr),
and multiple sclerosis (NIH had an "MS-and-Lyme Clinic," until they found out the fungal OspA or LYMErix vaccine caused the MS outcome of Lyme - also due to reactivated latent herpesviruses),
cancer, ME/CFS, stroke, etc, if spirochetes were not screwing up peoples' immune systems, permanently?

We patients have to explain and show this data to you, because there really are no "doctors" in America.  Think about it.  All there are, are sleazebags, whores and greedy douches.  And they're all STUPID - which is the result of their arrogance. 

Pity.  It's a pity that nowadays, we victims who figured out this 500 year old mystery of the Great Imitator, are treated like trash across the board, by the US Government and all the people in America with "MD" after their names.  That's the best America can do.  Torture their victims.   Think about it.  Who's ever thanked us?  Who's ever admitted we were right, about Lyme and OspA, formally?

NO ONE.  None have the balls. 


In America, in the near future - what's left of us when the rest of the world abandons us to environmental and financial disasters (when the rest of the world realizes they do not need us or our petrodollar  Monopoly money)-, the ones remembered for courage will not include any "doctors."  There will be no former "public servants," or cops, or jail guards - no govt employees or sluts for the new world order - of any kind, among us.  No bureaucrats, no CEOs, no one who ever wore a uniform or had a state or federal govt badge of any sort.   No employee of the military at any level. 

Not a single person whose livelihood was dependent upon inflicting suffering on others will be among us.


Why doesn't the CDC want you to know that Post-Lyme and Chronic Fatigue/ME is really Post-SEPSIS-Syndrome?


Because the mechanisms of illness - acquired immune deficiency particularly from the likes of TLR2-agonists or fungal-antigen-bearing/shedding infections or even contamination of childhood vaccines with the likes of a fungal antigen like LYMErix -, are the same in Post-Sepsis and Vaccine-Damage Autism.  

The reason the babies are actually GETTING those live, attenuated, brain-tropic viruses from the vaccine vials is due to immunosuppression and/or contamination causing the immunosuppression. 

The reason someone came up with the idea for a Rubella vaccine in the first place was because Rubella causes the
neurodevelopmental brain damage we call "Autism."  Think about it. The kids are GETTING the VIRUSES and they're all brain-tropic.

The Infectious Diseases Society of America says these vaccines are given too many, too young, and are not properly vetted - please see for yourself, their exact language; the adverse events are a lot like sepsis events.  <<< See all 3 videos, they are all related, and you'll see - finally - that this is simple, and regarding models of disease you know about and have seen before (dual infections and synergy).

The Autism pandemic is the result of babies getting the actual brain-tropic viruses that are in the vaccine vials, either because those children are immunosuppressed or because the vaccines are contaminated with the likes of fungal antigens like LYMErix. 

Yes, the *ENTIRE* is that stupid.  Add to that, everyone with "MD" after their names.  YES, ALL OF THEM are THAT STUPID.

You have seen the MRIs of the heads of the ZIKA brain damaged children in the news.  The scientific literature says Borna virus is the model for the "neurodevelopmental brain damage" we call Autism.  The IDSA says vaccines are given too many, and too young, and none of them are properly vetted, and this was true since the 1970s.  The scientific literature on those MMR qualifications shows those children were never followed longer than a few days.  The MMR vaccine qualifications were even more criminal than the LYMErix stunt.  Go look for yourself on Pubmed.  You'll do it if you give a sh*t about other people.  But if you don't, you won't bother. 

It is the same with activism, in general.

If you are looking for the very simplest explainer on the Lyme crymes, see the RICO complaint already filed with the USDOJ.  I was talking to lawyers after all, so I had to really dumb it down.


Cryme Disease, the movie:    

The fungal-ish OspA vaccine caused the same post-sepsis, immunosuppression disease as the one thrown out of the case definition at Dearborn.

Steere used a seronegative Lyme assay to assess chronic neuro Lyme, a disease he later falsified and now claims never existed.  Steere recently claimed in a radio interview that (June 14, 2016) he never knew Lyme caused a chronic neuro outcome.  The CDC would like to throw Allen Steere under the bus,... and Steere is trying to avoid being thrown under the bus.  Obviously not happening.




IF you are the kind of person who wants a simple one-liner, single model, single, simple general idea to explain something, this would be it (Dattwyler and Fauci, below).  OspA is a known immune-suppressor, not a "vaccine."

We are not about to prove that OspA is a triacyl lipoprotein also called a basic Pam3Cys molecule type, is fungal, is a more serious endotoxin than LPS, a TLR2/TLR1 agonist, and that other spirochetal lipoproteins are triacylated, if not Pam3Cys exactly.  We can assume anyone knows how to use the National Library of Medicine or else the United States and European patent databases or even just pull the one arm of one-armed information bandit named Google and discover that, for instance, State University of New York, Stony Brook’s, Raymond Dattwyler has a patent for an inhalation form of OspA. 

It, the OspA in Dattwyler’s OspA patent, is intended to attenuate (lessen, or dampen) any suffocating inflammatory response people might suffer from the likes of the dual infections of, for example, influenza and pneumonia
, such as what happened in the 1918 Spanish Flu epidemic [FAUCI].  During that pandemic, the healthy people succumbed to the secondary pneumonia.  That is, the strong, mucus-y inflammatory response is what killed 20+ million people back then. They choked to death.  (One infection invites another, this is well-known).

Let’s just go ahead and have a look at what Raymond Dattwyler says about OspA as Pam3Cys and how inhaling a fungal antigen like this, OspA or Pam3Cys or a triacyl lipoprotein might tolerize you against the deadly pneumonia part of Pandemic Flu-moniaâ:


"A lipidation/processing reaction has been described for the intact OspA gene of B. burgdorferi. The primary translation product of the full-length B. burgdorferi OspA gene contains a hydrophobic N-terminal sequence, of 16 amino acids, which is a substrate for the attachment of a diacyl glyceryl to the sulflhydryl side chain of the adjacent cysteine (Cys) residue (at position 17). Following this attachment, cleavage by signal peptidase II and the attachment of a third fatty acid to the N-terminus occurs. The completed lipid moiety, a tripalmitoyl-S-glycerylcysteine modification, is termed Pam3Cys (or is sometimes referred to herein as Pam(3)Cys or Pam3Cys). It has been suggested that the lipid modification allows membrane localization of proteins, with polypeptide portions exposed as immune targets. In addition to serving as targets for the immune response, Pam3Cys-modified proteins, such as OspA, have been reported to act as potent inflammatory stimulants though the toll-like 2 receptor mechanism (TLR2).


The patent is actually against inhalation Inhalation Bubonic Plague.
You MIGHT want to listen to us.

NIAID's Chief Anthony Fauci on the 'monia part of 1918 Spanish Flumonia
® being what causes people to choke to death -- it was inflammatory response in the lungs to the fungal-ish pneumonia:

 2008 Oct 1;198(7):962-70. doi: 10.1086/591708.

Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemicinfluenza preparedness.



Despite the availability of published data on 4 pandemics that have occurred over the past 120 years, there is little modern information on the causes of death associated with influenza pandemics.


We examined relevant information from the most recent influenza pandemic that occurred during the era prior to the use of antibiotics, the 1918-1919 "Spanish flu" pandemic. We examined lung tissue sections obtained during 58 autopsies and reviewed pathologic and bacteriologic data from 109 published autopsy series that described 8398 individual autopsy investigations.


The postmortem samples we examined from people who died of influenza during 1918-1919 uniformly exhibited severe changes indicative of bacterial pneumonia. Bacteriologic and histopathologic results from published autopsy series clearly and consistently implicated secondary bacterial pneumonia caused by common upper respiratory-tract bacteria in most influenza fatalities.


The majority of deaths in the 1918-1919 influenza pandemic likely resulted directly from secondary bacterial pneumonia caused by common upper respiratory-tract bacteria. Less substantial data from the subsequent 1957 and 1968 pandemics are consistent with these findings. If severe pandemic influenza is largely a problem of viral-bacterial copathogenesis, pandemic planning needs to go beyond addressing the viral cause alone (e.g., influenza vaccines and antiviral drugs). Prevention, diagnosis, prophylaxis, and treatment of secondary bacterial pneumonia, as well as stockpiling of antibiotics and bacterial vaccines, should also be high priorities for pandemic planning.


So, if OspA was obviously something that causes too-much inflammation-and-then-immunosuppression (endotoxin tolerance), it could not have been a vaccine.  And if OspA was not a vaccine, then certain people not only lied it onto the market, they falsified the Dearborn case definition to leave out the 85% of us who do not have a genetic background for a tendency to have Rheumatoid Arthritis (HLAs).

So, IF you are the kind of person who wants a simple one-liner, single model, single, general idea to explain something, this would be it.  OspA is a known immune-suppressor, not a "vaccine."  It does the OPPOSITE of what vaccines are intended to do, which is produce antibodies.  It is a more severe endotoxin than LPS.

"TLR2 mediates inflammatory responses to a wide variety of lipidated microbial components, including bacterial lipoproteins, atypical lipopolysaccharides, and lipomannans (). Among these microbial agonists, bacterial lipoproteins are by far the most potent (, )."


See the rest of the videos here:  Crymes on Vimeo


the United States of STUPID could you see the likes of the's founders, Edward McSweegan and Durland Fish
not knowing spirochetes were their own phylum (are not regular "bacteria"), are relapsing fever organisms, that the nature of the relapse was antigenic variation (vaccines would do no good), and that the McSweegan-Fish gang never even asked what OspA was (fungal, TLR2/1 agonist, causes permanent immunosuppression, never could have been a vaccine).  The CDC deployed the stupid and cowardly Allen Steere who went into Rheumatology to avoid VietNam (there are no old ladies who need aspirin among deployed US combat forces), probably because he was known to be too stupid to ask any questions about ticks or spirochetes.

What is the simplest way to explain that the ALDF dot com / Yale / CDC Lyme cabal threw out of the Dearborn, "2-tiered" "case definition" the very same disabling, chronic neurologic, "New Great Imitator" disease that was caused by the fungal toxin, Pam3Cys or OspA?  They knew OspA vaccination causes neurologic, immunosuppression disease before the Oct 1994 Dearborn, Michigan fake consensus conference ever took place (chronology here).
In order for it to be prosecuted as a criminal case you have to show they knew ahead of time that there was this problem with their "vaccine" choice and that they CHANGED the testing definition after that, such as to leave out the chronic neurologic Lyme definition - the very disease caused by the fungal OspA vaccines

This is from June, 1993, by Alan Barbour and Durland Fish:

They said this ˄ BEFORE the CDC's Dearborn-"Let's-Leave-Out-the-Chronic-Neurologic-Form-of-the-Disease-Which-Is-Also-Caused-by-OspA-Injection-Alone" fake consensus conference took place (Oct, 1994). 
So, these criminals would have known there was a problem with OspA injections making people sick.  After all, OspA is a fungal endotoxin, TLR2/1-agonist.

The current, 1994, "Dearborn" case definition only detects the 15% of cases, only the late-, HLA-linked- arthritis cases, no early cases, and then these CDC (vaccine patent holding) criminals say this late manifestation needs no treatment.  The truth is that chronic neurologic disease is mainly caused by immunosuppression, reactivated Epstein-Barr and cross tolerance to other infections, or, technically, is post-sepsis syndrome or a kind of a B cell AIDS (see the Occam's Razor).

In this criminal case, you have to show to the DOJ that they CHANGED the diagnostic criteria from an older one that allowed more people to be diagnosed (the 1990 CDC version).  If the DOJ does not respond, clearly this was a bioweapon accident of some sort.  The circumstantial scientific, epidemiological evidence from Plum Island as the original outbreak area says so.    (Do the Russians know by now?  They knew in 2006.  The Chinese, a year later.  Site stats.)

The US press cant handle it, and no one has ever written about it in any book.


Whoever does not know what LYMErix-disease is, does not know what Lyme disease is.  This includes and all of the Lyme non-profits.  One has to know what the antigen is, in order to know what it does.  This is basic science. 

And it turns out that Lyme and Syphilis as "Great Imitators" are really Great Detonators of latent viruses and general immunosuppression - also known as post-sepsis syndrome.  It turns out that Pam3Cys (OspA), a triacyl lipopeptide, is a functional mimic of E. coli's Lipid A, a serious fungal (TLR2/1-agonist) ENDOTOXIN.  Think about that.  The CDC wanted to inject everyone with several blobs of bathroom scum.  And that's what happens with a tick bite, too. 

Baumgarth (UCDavis):

"For months after infection, those germinal centers fail to produce the specific cells — memory B cells and antibody-producing plasma cells — that are crucial for producing lasting immunity. In effect, the bacteria prevent the animal’s immune system from forming a “memory” of the invading bacteria and launching a protective immune response against future infections.

"The researchers found that following Borrelia burgdorferi infection, this process even prevented induction of strong immune responses to an influenza infection."



Cadavid on Septic Spirochetes:

"Finally, spirochetal lipoproteins have more prominent pro-inflammatory effects compared to other bacterial lipoproteins and synthetic lipopeptides ()."




1.)  Norman Latov on how OspA vaccination caused the same disease as chronic Lyme:

Neuropathy and cognitive impairment following vaccination with the OspA protein of Borrelia burgdorferi.

“Neurological syndromes that follow vaccination or infection are often attributed to autoimmune mechanisms. We report six patients who developed neuropathy or cognitive impairment, within several days to 2 months, following vaccination with the OspA antigen of Borrelia burgdorferi. Two of the patients developed cognitive impairment, one chronic inflammatory demyelinating polyneuropathy (CIDP), one multifocal motor neuropathy, one both cognitive impairment and CIDP, and one cognitive impairment and sensory axonal neuropathy. The patients with cognitive impairment had T2 hyperintense white matter lesions on magnetic resonance imaging. The similarity between the neurological sequelae observed in the OspA-vaccinated patients and those with chronic Lyme disease suggests a possible role for immune mechanisms in some of the manifestations of chronic Lyme disease that are resistant to antibiotic treatment.”



2.) Gary Wormser on OspA causing immunosuppression (which means it was the opposite of a "vaccine"):

Modulation of lymphocyte proliferative responses by a canine Lyme disease vaccine of recombinant outer surface protein A (OspA).

”… After exposure to either the unaltered vaccine preparation or OspA prepared in saline, normal lymphocyte responses to the mitogens concanavalin A, phytohemagglutinin-M or pokeweed mitogen, or the antigen BCG were consistently reduced. Whole cell extracts of B. burgdorferi also modulated immune responses but required a much greater quantity of protein than needed for theOspA preparation. The magnitude of modulation was directly dependent on the quantity of OspA. OspA interferes with the response of lymphocytes to proliferative stimuli including a blocking of cell cycle phase progression. Future studies designed to delete the particular region or component of theOspA molecule responsible for this effect may lead to improved vaccine preparations.”





3.) Donald H. Marks - an OspA vaccine trial administrator - on how LYMErix caused the same disease as chronic Lyme:

Neurological complications of vaccination with outer surface protein A (OspA).

”A wide range of neurological complications have been reported via the medical literature and the VAERS system after vaccination with recombinant outer surface protein A (OspA) of Borrelia. To explore this issue, 24 patients reporting neurological adverse events (AE) after vaccination with Lymerix, out of a group of 94 patients reporting adverse events after Lymerix vaccination, were examined for causation. Five reports of cerebral ischemia, two transient Ischemic attacks, five demyelinating events, two optic neuritis, two reports of transverse myelitis, and one non-specific demyelinating condition are evaluated in this paper. Caution is raised on not actively looking for neurologic AE, and for not considering causation when the incidence rate is too low to raise a calculable difference to natural occurrence.”

It’s not “Autoimmune.”  It’s Subimmune.  This Subimmunity represents the entire class of the DSM's VooDoo Somatoformia – as well as cancer.  Cancer is in the Subimmune class, at the other end of the immunity spectrum from Autoimmunity.   This fact or condition completely flips the entire medical paradigm where you have to have a biomarker that is above-, or more-than- the normal range.  Lyme is not an inflammatory disease.  There are always negative correlations to biomarkers of autoimmunity or illness or infection except when using sophisticated DNA techniques using spinal fluid, in particular in these chronic, waste-basket diseases.  Henceforth, Autoimmunity will be an obsolete word that connotes the previous Medical Establishment where BigPharma is going to “block” something with their drugs.  They are dinosaurs.  You can’t block a mechanism that is already permanently blocked and you can’t unblock it.

It could be that a person has an HLA-linked outcome to one of the secondary infections like Epstein-Barr or HHV-6, reactivated by the AIDS-like Lyme and LYMErix.  Those people would for instance have the official, hypersensitivity outcomes of MS or Lupus or whatever.  But they are not also called Incompetent Incantation-ators and they are not mistreated by the entire universe (family, friends, Social Security, “doctors,” everyone, including ILADS and the non profits).


4.) Ben Luft at the 1998 FDA Vaccine Meeting on LYMErix:

 "The point that I wanted to make in regard to the study is that there is very heavy dependence on serologic confirmation. And when we start thinking about the adverse events, *** it was stated originally when we got the overview of the disease that the disease is really quite protean. And actually the adverse events are very similar to what the disease manifestations are.**** And if you start to, as I think Dr. Hall was eluding to -- if you start to kind of say well how often do you actually become seropositive, you can start to have a different take on when someone has an adverse event or whether it is disease specific or infection specific versus vaccine specific. And I think that that is an important issue that we have to deal with. ..."



5.) Dave Persing who together with Yale’s Robert Schoen developed this test in 1994 or 1995 says this about the similarities between Lyme and LYMErix disease:

"Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure....”


Who else says OspA alone is responsible for the immunosuppression with brain inflammation that is characteristic of "Chronic Lyme?"

6.) THE NIH (Martin and Marques):

Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression (2006).

The spirochete Borrelia burgdorferi is the agent of Lyme disease, which causes central nervous system manifestations in up to 20% of patients. We investigated the response of human brain microglial cells, glial progenitors, neurons, astrocytes, as well as peripheral blood monocytes to stimulation with B. burgdorferi. We used oligoarrays to detect changes in the expression of genes important for shaping adaptive and innate immune responses. We found that stimulation with B. burgdorferi lysate increased the expression of Toll-like receptors (TLRs) 1 and 2 in all cell types except neurons. However, despite similarities in global gene profiles of monocytes and microglia, only microglial cells responded to the stimulation with a robust increase in HLA-DR, HLA-DQ, and also coexpressed CD11-c, a dendritic cell marker. In contrast, a large number of HLA-related molecules were repressed at both the RNA and the protein levels in stimulated monocytes, whereas secretion of IL-10 and TNF-alpha was strongly induced. These results show that signaling through TLR1/2 in response to B. burgdorferi can elicit opposite immunoregulatory effects in blood and in brain immune cells, which could play a role in the different susceptibility of these compartments to infection. 


The NIH patent explaining how Lyme causes LYMErix-disease (“stealth bomber”):

"The invention relates to novel antigens associated with Borrelia burgdorferi which are exported (or shed) in vivo and whose detection is a means of diagnosing Lyme disease. The antigens are extracellular membranevesicles and other bioproducts including the major extracellular protein antigen. Another object of the invention is to provide antibodies, monoclonal and/or polyclonal, labeled and/or unlabeled, that are raised against the antigens. A further object of the invention is to provide a method of diagnosing Lyme disease by detecting the antigens in a biological sample taken from a host using the antibodies in conventional immunoassay formats. Another object of the invention is to provide kits, for the diagnosis of Lyme disease, comprising the antibodies and ancillary reagents. The advantage of the antibodies used in the invention is that they react with the antigens from geographically diverse strains of Borrelia burgdorferi, but do not react with antigens from related Borrelia spirochetes.",217,872.PN.&OS=PN/5,217,872&RS=PN/5,217,872

The shed blebs (or exosomes or vesicles) have LYMErix on them (delayed fuse or “time bomb”):

Characterization of multiprotein complexes of the Borrelia burgdorferi outer membrane vesicles.

"Although we uncovered the existence of at least 10 distinct OM complexes harboring several unique subunits, the complexome is dominated by the frequent occurrence of a limited diversity of membrane proteins, most notably P13, outer surface protein (Osp) A, -B, -C, and -D and Lp6.6."



7.)  Dattwyler and Luft in 1989, in IDSA's own "journal," saying "treatment fails in half the cases." And the OspA vaccine caused the same systemic disease, say the crooks, themselves.  See the Occam's Razor report for who says so. 

Steere was at this 1994 FDA meeting: 

DATTWYLER:  "-- the ones that (sic) failed to mount a vigrous immune response tended to do worse.  So, there is an inverse correlation between the degree of serologic response and the outcome. 
"So, individuals with a poor immune response tend to have worse disease."

1994 FDA Meeting Transcripts where Ray Dattwyler tells the FDA Vaccine Committee that the
patients with low or no antibodies are the sickest:


Ray Dattwyler (SUNY-SB) says in 1988 that exposure to Lyme or even just its supernatants (OspA-like molecules) turn off the NK cell activity (or is immunosuppressive):

"Effect of B burgdorferi Culture on Normal PBL

"..when lymphocytes are cultured in the presence of growing Bb there is a marked inhibition ( p < .0005 ) of NK activity on days 3, 5, and 7 when compared to lymphocytes cultured in BSKII media in the absence of spirochetes.  This effect is not due to a selective depletion or or toxicity to endogenous NK since viability studies and monoclonal antibodies demonstrate no significant changes after culture with the organism.


"The inhibition is directly attributable to the organism or its supernatants (data not shown)."

From: Modulation of natural killer cell activity by Borrelia burgdorferi.

Golightly M, Thomas J, Volkman D, Dattwyler R.
Department of Pathology, State University of New York, Stony Brook 11794.
PMID: 3056196 [PubMed - indexed for MEDLINE]
Ann N Y Acad Sci. 1988;539:103-11.


Dattwyler on Seronegative Lyme and how that is like fungal diseases:

DOWNLOAD THE FULL TEXT of Dattwyler talking about seronegative, neuroLyme, HERE:
dattwyler1988 (1).pdf

Lyme is like Jeopardy!  Ask the right question:  "Something that causes a 'multi-system disease' and is IDSA's own 'New Great Imitator.'" 

The answer is, "What is OspA?"


UCSF says Lyme and LYMErix-Disease are a diseases of immunosuppression, and they say that half the Lyme/tick bite victims remain ill and are not cured with antibiotics
Feb 12, 2016:

“Early Lyme disease prior to antibiotic therapy was characterized by marked upregulation of Toll-like receptor signaling but lack of activation of the inflammatory T-cell apoptotic and B-cell developmental pathways seen in other acute infectious syndromes,” wrote the study’s authors. “Six months after completion of therapy, Lyme disease patients were found to have 31 to 60% of their pathways in common with three different immune-mediated chronic diseases. No differential gene expression signature was observed between Lyme disease patients with resolved illness to those with persistent symptoms at six months post-treatment.”

See more below from UC Davis,
and in the Occam's Razor report. The Great Imitator is EBV, the Great Initiator is the fungal antigen OspA, shed by spirochetes - their own phylum, and not regular "bacteria" - on blebs, says the NIH.  America is a really stupid country, when you consider that nearly every disease is an outcome of the first immune disordered step - inhibition of apoptosis of infected B cells.

Look at the experience of Lewis Bull, East Lyme, CT, back in the OspA "vaccines" trials days (late 1990s) to see exactly what these evil bastards of the CDC (Barbour, Johnson, Steere) had planned from the beginning to do with LYMErix - "multisystem (read: "sepsis")," Chronic Fatigue Syndrome-like - outcomes.   'Says it all.


University of California, Davis, says the CDC, Allen Steere, Gary Wormser, Edward McSweegan, et al must be lying their faces off:  Lyme and LYMErix cause permanent immunosuppression or is like a B cell AIDS. 


Lyme is not an "inflammatory," "autoimmune disease," it is the Great Initiator of latent viruses, as a fungal antigen shedder, SPIROCHETE (its own phylum).  Fungal antigens like triacyl lipopeptides (OspA) have always been known to cause immunosuppression. The complete opposite of an "autoimmune" or "inflammatory" disease.  The only biomarkers will be signs of nerve and brain degradation in the cerebral spinal fluid; no antibodies in the blood.




 So what exactly is OspA? 

“I did not get the vaccine so this does not concern me.”   Oh, yes, you got the vaccine.  Everyone with Lyme got LYMErix.  Here is what LYMErix is, and how this "vaccine-was-the-disease" works:

This is image is of Pam3Cys or OspA.  What is variable is the protein end "("peptide"), but what is immunogenic (seen as an invader to the immune system) are the fatty acids or acyl groups and the very electronegative cysteine core:


Pam3Cys is a triacylated lipoprotein, the degree of acylation is equated with its toxicity.   So what is acylation?  It’s the zig-zaggy lines that mean Carbon-Carbon-Carbon, yes, hydrocarbons, like margarine or octane.  Exactly, the name just refers to the number of carbons in each carboxyl or acyl group.  Palmitic (the Pam in Pam3Cys) has X number of carbons, gasoline, 8, linoleic acids, like 14. Look up what are alkanes then add a COOH group and you have one of these fatty acids. 

Something highly acylated like this (3 or more fatty acids hanging off) are managed by Toll-like Receptor (TLR) 2 and TLR1, together.  Therefore a “TLR2/1-agonist” is another term that generally refers to lipoproteins like those from Borrelia, mycoplasma, mycobacteria, and others like Brucella.  (But they can manage other compounds.)

This thing, Pam3Cys and fungal lipid molecules like it, is shed with the blebs.  In other words, like this:

The likes of OspA is on these blebs.  They go to the brain, inflame it, get eaten up by immune cells - which renders them incompetent-, they go to the kidneys (LUAT), etc. You will find this to be so in an NIH-owned patent (5,217,872) and elsewhere.

So, the fungal antigens are on the shed blebs and they go everywhere and they render the immune cells incompetent, resulting in an AIDS-like disease.  Everyone who has Lyme disease also has LYMErix disease. 


8.) Cadavid and the NIH say these shed vesicles or blebs go to the brain and inflame it:








In other words, the NIH is saying that thanks to exposure to shed blebs with TLR2/1 agonists like OspA on them, the HLA molecules will be downregulated and that you will make no antibodies.  This mechanism is mimicked elsewhere, in other fungal diseases models.  It is also CALLED endotoxin tolerance.


Endotoxin Tolerance Inhibits Lyn and c-Src Phosphorylation and Association with Toll-Like Receptor 4 but Increases Expression and Activity of Protein Phosphatases.

“Endotoxin tolerance protects the host by limiting excessive 'cytokine storm' during sepsis, but compromises the ability to counteract infections in septic shock survivors. It reprograms Toll-like receptor (TLR) 4 responses by attenuating the expression of proinflammatory cytokines without suppressing anti-inflammatory and antimicrobial mediators, but the mechanisms of reprogramming remain unclear. In this study, we demonstrate that the induction of endotoxin tolerance in human monocytes, THP-1 and MonoMac-6 cells inhibited lipopolysaccharide (LPS)-mediated phosphorylation of Lyn, c-Src and their recruitment to TLR4, but increased total protein phosphatase (PP) activity and the expression of protein tyrosine phosphatase (PTP) 1B, PP2A, PTP nonreceptor type (PTPN) 22 and mitogen-activated protein kinase phosphatase (MKP)-1. Chemical PP inhibitors, okadaic acid, dephostatin and cantharidic acid markedly decreased or completely abolished LPS tolerance, indicating the importance of phosphatases in endotoxin tolerization. Overexpression of PTPN22 decreased LPS-mediated nuclear factor (NF)-x03BA;B activation, p38 phosphorylation and CXCL8 gene expression, while PTPN22 ablation upregulated LPS-induced p65 NF-x03BA;B and p38 phosphorylation and the expression of TNF-α and pro-IL-1β mRNA, indicating PTPN22 as an inhibitor of TLR4 signaling. Thus, LPS tolerance interferes with TLR4 signaling by inhibiting Lyn and c-Src phosphorylation and their recruitment to TLR4, while increasing the phosphatase activity and expression of PP2A, PTPN22, PTP1B and MKP1.


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